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Cancer Cell, ISSN 1535-6108, 2008, Volume 13, Issue 3, pp. 193 - 205
Tumor vasculature is derived from sprouting of local vessels (angiogenesis) and bone marrow (BM)-derived circulating cells (vasculogenesis). By using a model... 
CELLCYCLE | BREAST-CANCER | ENDOTHELIAL PROGENITOR CELLS | RECTAL-CANCER | MAMMARY-TUMORS | ONCOLOGY | HEMATOPOIETIC STEM-CELLS | IN-VIVO | GROWTH-FACTOR | PRECURSOR CELLS | CANCER-THERAPY | RADIATION-THERAPY | CELL BIOLOGY | Myeloid Cells - enzymology | Bone Marrow Cells - enzymology | Neoplasms, Experimental - enzymology | Antineoplastic Agents - therapeutic use | Monocytes - immunology | Subcutaneous Tissue - surgery | Neovascularization, Pathologic - pathology | Neoplasms, Experimental - pathology | Neovascularization, Pathologic - enzymology | Protease Inhibitors - pharmacology | Matrix Metalloproteinase 9 - metabolism | Monocytes - transplantation | Melanoma, Experimental | Stem Cells - enzymology | Time Factors | Matrix Metalloproteinase 9 - genetics | Myeloid Cells - immunology | Bone Marrow Cells - immunology | Bone Marrow Transplantation | Diphosphonates - therapeutic use | Bone Marrow Cells - drug effects | Myeloid Cells - drug effects | Neovascularization, Pathologic - prevention & control | Antineoplastic Agents - pharmacology | Imidazoles - therapeutic use | Protease Inhibitors - therapeutic use | Signal Transduction | Mice, Inbred C57BL | Imidazoles - pharmacology | Matrix Metalloproteinase 9 - deficiency | Neoplasms, Experimental - radiotherapy | Mice, Inbred C3H | Mice, Knockout | Monocytes - drug effects | Monocytes - enzymology | Subcutaneous Tissue - radiation effects | Animals | Subcutaneous Tissue - blood supply | Matrix Metalloproteinase Inhibitors | Myeloid Cells - transplantation | Mice | CD11b Antigen - metabolism | Endothelial Cells - enzymology | Neoplasms, Experimental - drug therapy | Diphosphonates - pharmacology | Neoplasms, Experimental - blood supply | Neovascularization | Nuclear radiation | Analysis | Tumors | Index Medicus
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 05/2012, Volume 287, Issue 19, pp. 15966 - 15980
Journal Article
Nature, ISSN 0028-0836, 01/2009, Volume 457, Issue 7229, pp. 599 - 602
BRAF and NRAS are common targets for somatic mutations in benign and malignant neoplasms that arise from melanocytes situated in epithelial structures, and... 
CELLS | ALPHA-SUBUNIT | PATHWAY | MULTIDISCIPLINARY SCIENCES | KINASE-C | BRAF | CANCER | TUMORS | MELANOCYTOSIS | Cell Proliferation | GTP-Binding Protein alpha Subunits - chemistry | Humans | Melanoma - enzymology | Nevus, Blue - pathology | Uveal Neoplasms - enzymology | MAP Kinase Signaling System | Skin Neoplasms - enzymology | Nevus, Blue - genetics | DNA Mutational Analysis | Melanoma - genetics | ras Proteins - chemistry | Melanocytes - pathology | Codon - genetics | Protein Structure, Tertiary | Skin Neoplasms - pathology | Oncogenes - genetics | Uveal Neoplasms - genetics | Melanocytes - enzymology | GTP-Binding Protein alpha Subunits - metabolism | Uveal Neoplasms - pathology | Cells, Cultured | Nevus, Blue - enzymology | GTP-Binding Protein alpha Subunits - genetics | Melanoma - pathology | Mutation - genetics | Mitogen-Activated Protein Kinases - antagonists & inhibitors | Biopsy | Cell Transformation, Neoplastic | Skin Neoplasms - genetics | GTP-Binding Protein alpha Subunits - deficiency | Enzyme Activation | Genes, Dominant - genetics | Apoptosis | Mitogen-Activated Protein Kinases - metabolism | Antibody diversity | Evaluation | Gene mutations | Uvea | Melanoma | Genetic aspects | Research | Health aspects | Cancer | Proteins | Studies | Enzymes | Cell cycle | Mutation | Kinases | Risk factors | Skin cancer | Index Medicus
Journal Article
Nature, ISSN 0028-0836, 07/2017, Volume 547, Issue 7664, pp. 453 - 457
Plasticity of the cell state has been proposed to drive resistance to multiple classes of cancer therapies, thereby limiting their effectiveness(1-4). A... 
EPITHELIAL-MESENCHYMAL TRANSITION | METABOLISM | MULTIDISCIPLINARY SCIENCES | PROSTATE-CANCER | HUMAN-MELANOMA CELLS | SENSITIVITY | DEATH | INHIBITOR | EXPRESSION | SIGNATURE | REVEALS | Prostatic Neoplasms - metabolism | Cadherins - metabolism | Humans | Mesoderm - drug effects | Melanoma - enzymology | Male | Lipid Peroxides - metabolism | Zinc Finger E-box-Binding Homeobox 1 - genetics | Neoplasms - genetics | Mesoderm - enzymology | Cell Death | Epithelial-Mesenchymal Transition | Lipid Peroxidation - drug effects | Cell Transdifferentiation | Prostatic Neoplasms - drug therapy | Melanoma - metabolism | Glutathione Peroxidase - metabolism | Prostatic Neoplasms - pathology | Reproducibility of Results | Neoplasms - enzymology | Melanoma - pathology | Iron - metabolism | Neoplasms - drug therapy | Cell Lineage | Drug Resistance, Neoplasm - genetics | Proto-Oncogene Proteins B-raf - genetics | Melanoma - drug therapy | Proteomics | Cell Line, Tumor | Prostatic Neoplasms - enzymology | Mesoderm - metabolism | Neoplasms - pathology | Mesoderm - pathology | Peroxidase | Research | Chemical properties | Chemical synthesis | Analysis | Cancer cells | Therapy | Biotechnology | Carcinoma | Mesenchyme | Genes | Lipid peroxidation | Lipids | Iron | Genomes | Phospholipids | Kinases | Cancer therapies | Proteins | Organoids | Dissipation | Peroxides | Lipid metabolism | Glutathione | Transfer RNA | Peroxidation | Glutathione peroxidase | Enzymes | Mortality | Melanoma | Selenocysteine | Tumor cell lines | Metabolism | Substrates | Cell death | Lipoxygenase | Prostate cancer | Prostate | Tumors | Apoptosis | Cancer | Index Medicus
Journal Article
Experimental Neurology, ISSN 0014-4886, 2009, Volume 219, Issue 1, pp. 146 - 155
Cancer pain significantly affects the quality of cancer patients, and current treatments for this pain are limited. C-Jun N-terminal kinase (JNK) has been... 
NEUROPATHIC PAIN | MURINE MODEL | MECHANICAL ALLODYNIA | ACTIVATED PROTEIN-KINASE | N-TERMINAL KINASE | SPINAL NERVE LIGATION | FUNCTIONAL INTERACTIONS | CELL-CYCLE ARREST | PRIMARY SENSORY NEURONS | C-JUN | NEUROSCIENCES | Melanoma - complications | Skin Neoplasms - drug therapy | Analgesics, Opioid - pharmacology | Melanoma - enzymology | JNK Mitogen-Activated Protein Kinases - metabolism | Male | Peripheral Nervous System Diseases - physiopathology | Skin Neoplasms - enzymology | Pain - enzymology | Pain - drug therapy | Pain - etiology | Sensory Receptor Cells - metabolism | Disease Models, Animal | Peripheral Nervous System Diseases - etiology | JNK Mitogen-Activated Protein Kinases - antagonists & inhibitors | Morphine - pharmacology | Mice, Inbred C57BL | Enzyme Inhibitors - pharmacology | Peripheral Nervous System Diseases - enzymology | Activating Transcription Factor 3 - metabolism | Enzyme Inhibitors - therapeutic use | Peptides - pharmacology | Skin Neoplasms - complications | Animals | Sensory Receptor Cells - cytology | Sensory Receptor Cells - drug effects | Hyperalgesia - drug therapy | Melanoma - drug therapy | Cell Line, Tumor | Hyperalgesia - etiology | Cell Proliferation - drug effects | Mice | Pain Measurement | Hyperalgesia - enzymology | Peptides - therapeutic use | Allergy | Care and treatment | Pain | Peptides | Growth | Analysis | Melanoma | Fluorescence | Cancer pain | Allergic reaction | Skin cancer | Cancer | Index Medicus
Journal Article
American Journal of Veterinary Research, ISSN 0002-9645, 01/2018, Volume 79, Issue 1, pp. 90 - 97
Journal Article
The Journal of Pathology, ISSN 0022-3417, 06/2006, Volume 209, Issue 2, pp. 213 - 219
Journal Article
Science, ISSN 0036-8075, 7/2011, Volume 333, Issue 6039, pp. 233 - 238
Journal Article
JOURNAL OF IMMUNOLOGY, ISSN 0022-1767, 01/2009, Volume 182, Issue 1, pp. 111 - 120
The underlying mechanisms of tumor-induced immune suppression need to be fully understood. Regulatory T (Treg) cells have been shown to play an important role... 
APOPTOSIS | GROWTH-FACTOR-BETA | ACTIVATION | DENDRITIC CELLS | CD4(+)CD25(+) | IMMUNOSUPPRESSION | CD69 | INDUCTION | IMMUNOLOGY | CANCER | TUMOR-IMMUNITY | T-Lymphocyte Subsets - immunology | Carcinoma, Lewis Lung - enzymology | Antigens, CD - biosynthesis | Coculture Techniques | Carcinoma, Lewis Lung - immunology | Lung Neoplasms - pathology | Extracellular Signal-Regulated MAP Kinases - metabolism | Enzyme Activation - immunology | T-Lymphocytes, Regulatory - pathology | T-Lymphocytes, Regulatory - immunology | Melanoma, Experimental - immunology | Membrane Proteins - physiology | Antigens, Differentiation, T-Lymphocyte - physiology | Membrane Proteins - metabolism | Melanoma, Experimental - enzymology | Lung Neoplasms - enzymology | Lectins, C-Type | Mice, Inbred C57BL | Melanoma, Experimental - pathology | Mice, Transgenic | Protein Binding - immunology | Transforming Growth Factor beta1 - physiology | Disease Progression | Lung Neoplasms - immunology | Animals | T-Lymphocyte Subsets - pathology | Interleukin-2 Receptor alpha Subunit - metabolism | T-Lymphocyte Subsets - enzymology | Carcinoma, Lewis Lung - pathology | Cell Line, Tumor | Growth Inhibitors - physiology | Antigens, CD - physiology | Mice | Mice, Inbred BALB C | Antigens, Differentiation, T-Lymphocyte - biosynthesis | CD4 Antigens - biosynthesis | T-Lymphocytes, Regulatory - enzymology | Index Medicus | Abridged Index Medicus
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 08/2013, Volume 123, Issue 8, pp. 3231 - 3242
Journal Article