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Immunity, ISSN 1074-7613, 06/2015, Volume 42, Issue 6, pp. 1033 - 1047
Expansion and acquisition of Th1 cell effector function requires metabolic reprogramming; however, the signals instructing these adaptations remain poorly... 
COFACTOR PROTEIN CD46 | MAMMALIAN TARGET | ACTIVATION | HEMOLYTIC-UREMIC SYNDROME | AMINO-ACIDS | EFFECTOR FUNCTION | IMMUNOLOGY | GLUCOSE-UPTAKE | EXPRESSION | T-CELLS | GLYCOLYTIC SWITCH | RNA, Small Interfering - genetics | Up-Regulation | Th1 Cells - physiology | TOR Serine-Threonine Kinases - metabolism | Large Neutral Amino Acid-Transporter 1 - metabolism | Homeodomain Proteins - metabolism | Humans | Complement System Proteins - immunology | Glucose Transporter Type 1 - metabolism | Ras Homolog Enriched in Brain Protein | Interferon-gamma - metabolism | Mechanistic Target of Rapamycin Complex 1 | Multiprotein Complexes - metabolism | Membrane Cofactor Protein - metabolism | Immunity, Cellular - genetics | Cells, Cultured | Oxidative Phosphorylation | Neuropeptides - metabolism | Hemolytic-Uremic Syndrome - immunology | Cellular Reprogramming - immunology | Cell Differentiation - immunology | Glucose Transporter Type 1 - genetics | Monomeric GTP-Binding Proteins - metabolism | Glycolysis | Membrane Cofactor Protein - genetics | Adaptor Proteins, Signal Transducing - metabolism | Glucose metabolism | Glucose | T cells | Dextrose | Biomedical research | Cytokines | Colleges & universities | T cell receptors | Kinases | Metabolism | Gene expression | Experiments | Proteins | Scholarships & fellowships | Bioenergetics | Lymphocytes | Arrays
Journal Article
Kidney International, ISSN 0085-2538, 08/2012, Volume 82, Issue 4, pp. 454 - 464
Dense deposit disease and glomerulonephritis with isolated C3 deposits are glomerulopathies characterized by deposits of C3 within or along the glomerular... 
complement | glomerulonephritis | clinical immunology | membranoproliferative glomerulonephritis (MPGN) | LONG-TERM | HEMOLYTIC-UREMIC SYNDROME | MESANGIOCAPILLARY GLOMERULONEPHRITIS | GLOMERULONEPHRITIS TYPE-II | MACULAR DEGENERATION | FACTOR-H DEFICIENCY | GLOMERULAR-FILTRATION-RATE | UROLOGY & NEPHROLOGY | MUTATIONS | MEMBRANOPROLIFERATIVE GLOMERULONEPHRITIS | MEMBRANE COFACTOR PROTEIN | Humans | Child, Preschool | Male | Complement System Proteins - metabolism | Young Adult | Time Factors | DNA Mutational Analysis | Renal Insufficiency - immunology | Child | Glomerulonephritis, Membranoproliferative - pathology | Genetic Predisposition to Disease | Risk Assessment | Complement C3 - metabolism | Gene Frequency | Risk Factors | Glomerulonephritis - genetics | Glomerulonephritis - immunology | Glomerulonephritis - mortality | Biomarkers - blood | Kidney Glomerulus - pathology | Disease Progression | Glomerulonephritis, Membranoproliferative - genetics | Glomerulonephritis, Membranoproliferative - immunology | Phenotype | Complement Factor H - metabolism | Glomerulonephritis, Membranoproliferative - mortality | Glomerulonephritis - pathology | Adolescent | Age of Onset | Membrane Cofactor Protein - genetics | Kidney Glomerulus - immunology | Mutation | Complement Factor I - metabolism | Haplotypes | Complement C3 Nephritic Factor - genetics | Glomerulonephritis - therapy | Infant | Case-Control Studies | Adult | Complement System Proteins - genetics | Female | Membrane Cofactor Protein - metabolism | Complement Pathway, Alternative - genetics | France | Renal Insufficiency - genetics | Kaplan-Meier Estimate | Complement Factor I - genetics | Treatment Outcome | Chi-Square Distribution | Glomerulonephritis, Membranoproliferative - therapy | Biopsy | Complement C3 Nephritic Factor - metabolism | Complement Factor H - genetics
Journal Article
Blood, ISSN 0006-4971, 01/2010, Volume 115, Issue 2, pp. 379 - 387
Journal Article
Journal Article
Neuron, ISSN 0896-6273, 2006, Volume 49, Issue 4, pp. 489 - 502
Microglia are the immune cells of the brain. Here we show a massive infiltration of highly ramified and elongated microglia within the core of amyloid plaques... 
PROTEINS | STEMCELL | HUMDISEASE | CELLS | PROGENITORS | ORIGIN | PHAGOCYTOSIS | GLUCOCORTICOIDS | AMYLOID-BETA-PEPTIDE | ALPHA | APPSW TRANSGENIC MICE | NEUROSCIENCES | BRAIN | NEUROPROTECTION | Tumor Necrosis Factor-alpha - metabolism | Presenilin-1 | Age Factors | Gene Expression - drug effects | Amyloid beta-Peptides - pharmacology | Humans | Imaging, Three-Dimensional - methods | Bone Marrow Cells - physiology | Peptide Fragments - pharmacology | Green Fluorescent Proteins - genetics | RNA, Messenger - metabolism | Bone Marrow Transplantation - methods | Alzheimer Disease - pathology | Microglia - physiology | Time Factors | Amyloid beta-Protein Precursor - metabolism | Membrane Cofactor Protein - metabolism | Membrane Proteins - metabolism | Indoles | In Situ Hybridization - methods | Disease Models, Animal | Calcium-Binding Proteins - metabolism | Green Fluorescent Proteins - metabolism | Interleukin-1 - metabolism | Microglia - drug effects | Plaque, Amyloid - pathology | Membrane Proteins - genetics | Mice, Inbred C57BL | Cells, Cultured | Phagocytosis - physiology | Mice, Transgenic | Toll-Like Receptor 2 - metabolism | Microscopy, Confocal - methods | Immunohistochemistry - methods | Whole-Body Irradiation - methods | Amyloid beta-Protein Precursor - genetics | Animals | Microfilament Proteins | Alzheimer Disease - metabolism | Plaque, Amyloid - metabolism | Mice | Alzheimer Disease - genetics | Injections, Intraventricular - methods | Lysosomal-Associated Membrane Protein 2 - metabolism | Proteins | Physiological aspects | Neurons | Alzheimer's disease | Analysis | Studies | Medical research | Brain | Neurotoxicity | Brain research | Microscopy | Transgenic animals | Software | Nonsteroidal anti-inflammatory drugs | Alzheimers disease | Exports | Immune system | Basic Medicine | Neurosciences | Medical and Health Sciences | Medicin och hälsovetenskap | Medicinska och farmaceutiska grundvetenskaper | Neurovetenskaper
Journal Article
Journal Article
Journal of Thoracic and Cardiovascular Surgery, The, ISSN 0022-5223, 2014, Volume 148, Issue 3, pp. 1106 - 1114
Journal Article
The Journal of Pathology, ISSN 0022-3417, 04/2013, Volume 229, Issue 5, pp. 729 - 742
Dysregulated complement is thought to play a central role in age‐related macular degeneration ( AMD ) pathogenesis, but the specific mechanisms have yet to be... 
apoptosis | ageing | age‐related macular degeneration | complement | exosome | oxidative stress | age-related macular degeneration | POLY(ADP-RIBOSE) POLYMERASE | OXIDIZED LDL | BRUCHS MEMBRANE | PATHOLOGY | BASAL DEPOSITS | LOW-DENSITY LIPOPROTEINS | ONCOLOGY | HUMAN EYES | FACTOR-H POLYMORPHISM | HEPARAN-SULFATE | MEDIATED CELL-DEATH | GEOGRAPHIC ATROPHY | Exosomes - metabolism | Endoribonucleases - genetics | Age Factors | Complement Activation | Macular Degeneration - immunology | Humans | Middle Aged | Male | RNA, Messenger - metabolism | Complement System Proteins - metabolism | Young Adult | Retinal Pigment Epithelium - pathology | Transfection | RNA Interference | Cell Membrane - pathology | Aged, 80 and over | Complement System Proteins - genetics | Female | Membrane Cofactor Protein - metabolism | Lipoproteins, LDL - metabolism | Membrane Proteins - metabolism | Retinal Drusen - immunology | Protein-Serine-Threonine Kinases - metabolism | Cell Line | Endoribonucleases - metabolism | Membrane Proteins - genetics | Retinal Drusen - pathology | Down-Regulation | Protein-Serine-Threonine Kinases - genetics | CD59 Antigens - genetics | Epithelial Cells - pathology | Disease Progression | Poly(ADP-ribose) Polymerases - metabolism | Macular Degeneration - genetics | Epithelial Cells - immunology | Retinal Pigment Epithelium - immunology | Membrane Cofactor Protein - genetics | Aged | Cell Membrane - immunology | CD59 Antigens - metabolism | Apoptosis | Macular Degeneration - pathology
Journal Article