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Journal of allergy and clinical immunology, ISSN 0091-6749, 09/2015, Volume 136, Issue 3, pp. 769 - 780
...). Mitochondrial oxidative stress might be involved in driving the oxidative stress–induced pathology. Objective We sought to determine the effects of oxidative stress... 
Allergy and Immunology | airway hyperresponsiveness | antioxidant | proliferation | MitoQ | inflammation | mitochondria | airway smooth muscle | Ozone | oxidative stress | chronic obstructive pulmonary disease | Allergy | Immunology | Life Sciences & Biomedicine | Science & Technology | Respiratory System - pathology | Reactive Oxygen Species - metabolism | Humans | Middle Aged | Male | Pulmonary Disease, Chronic Obstructive - pathology | Bronchial Hyperreactivity - genetics | Ubiquinone - pharmacology | Muscle, Smooth - drug effects | Smoking - physiopathology | Organophosphorus Compounds - pharmacology | Myocytes, Smooth Muscle - drug effects | Pneumonia - genetics | Pulmonary Disease, Chronic Obstructive - genetics | Bronchial Hyperreactivity - chemically induced | Signal Transduction | Mitochondria - pathology | Smoking - metabolism | Bronchial Hyperreactivity - pathology | Reactive Oxygen Species - antagonists & inhibitors | Electron Transport Chain Complex Proteins - metabolism | Pulmonary Disease, Chronic Obstructive - chemically induced | Respiratory System - drug effects | Airway Remodeling - genetics | Mice | Oxidative Stress - drug effects | Bronchial Hyperreactivity - drug therapy | Myocytes, Smooth Muscle - pathology | Pneumonia - pathology | Membrane Potential, Mitochondrial - drug effects | Pneumonia - chemically induced | Adult | Female | Pulmonary Disease, Chronic Obstructive - metabolism | Myocytes, Smooth Muscle - metabolism | Ubiquinone - analogs & derivatives | Gene Expression Regulation | Hydrogen Peroxide - pharmacology | Electron Transport Chain Complex Proteins - genetics | Mitochondria - metabolism | Antioxidants - pharmacology | Mitochondria - drug effects | Muscle, Smooth - metabolism | Animals | Pneumonia - drug therapy | Respiratory System - metabolism | Aged | Muscle, Smooth - pathology | Oxidative stress | Care and treatment | Lung diseases, Obstructive | Inflammation | Pharmaceutical industry | Biomedical research | Disease | Laboratories | Mortality | Colleges & universities | Smooth muscle | Mitochondrial DNA | Metabolism | Defects | Antioxidants | Mitochondria | Hospitals | Biopsy | Chronic obstructive pulmonary disease | Apoptosis | Smoking | Index Medicus | Abridged Index Medicus | NO, Nitric oxide | ΔΨm, Mitochondrial membrane potential | ASM, Airway smooth muscle | NAC, N-acetylcysteine | RL, Lung resistance | BAL, Bronchoalveolar lavage | OCR, Oxygen consumption rate | dTPP, Decyltriphenylphosphonium bromide | AHR, Airway hyperresponsiveness | logPC100, Concentration of acetylcholine that increased lung resistance by 100 | GOLD, Global Initiative for Chronic Obstructive Lung Disease | ATP, Adenosine triphosphate | KC, Keratinocyte-derived cytokine | JC-1, 5,5′,6,6′-Tetrachloro-1,1′,3,3′-tetraethylbenzimidazolylcarbocyanine iodide | COPD, Chronic obstructive pulmonary disease | ROS, Reactive oxygen species | Mechanisms of Allergy and Clinical Immunology
Journal Article
PloS one, ISSN 1932-6203, 10/2016, Volume 11, Issue 10, pp. e0165638 - e0165638
.... To explore whether AMPK activation could potentially prevent or reverse the effects of drug-induced mitochondrial and hepatocellular damage, we added an AMPK activator to collagen sandwich cultures... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Chemical and Drug Induced Liver Injury - prevention & control | Diclofenac - pharmacology | AMP-Activated Protein Kinases - metabolism | Rats | Hepatocytes - pathology | Male | Mitochondria - metabolism | Hepatocytes - metabolism | Mitochondria - drug effects | Enzyme Activation - drug effects | Rats, Sprague-Dawley | Autophagy - drug effects | Aminoimidazole Carboxamide - pharmacology | Ribonucleotides - pharmacology | Animals | Mitochondrial Dynamics - drug effects | Aminoimidazole Carboxamide - analogs & derivatives | Acetaminophen - pharmacology | Chemical and Drug Induced Liver Injury - drug therapy | Chemical and Drug Induced Liver Injury - pathology | Chemical and Drug Induced Liver Injury - enzymology | Hepatocytes - drug effects | Prevention | Liver diseases | Acetaminophen | Collagen | Quality control | Quality management | Drugs | Reactive oxygen species | Liver | Activation | Biosynthesis | Kinases | Autophagy | Damage prevention | Diclofenac | Proteins | Mitochondria | Analgesics | Rodents | Hepatology | Membrane potential | Nonsteroidal anti-inflammatory drugs | Elongation | Enzymes | Immunoglobulins | Oxygen | AMP | Injury prevention | Hepatocytes | Morphology | Pharmacy | ATP | Viability | Phagocytosis | Apoptosis | Index Medicus
Journal Article
PloS one, ISSN 1932-6203, 08/2015, Volume 10, Issue 8, pp. e0135083 - e0135083
The chemotherapeutic use of cisplatin is limited by its severe side effects. In this study, by conducting different omics data analyses, we demonstrated... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | L-Lactate Dehydrogenase - metabolism | Epithelial Cells - metabolism | Reactive Oxygen Species - metabolism | Apoptosis - drug effects | Epithelial Cells - drug effects | Humans | Male | Membrane Potential, Mitochondrial - drug effects | Glycolysis - drug effects | Reactive Oxygen Species - agonists | Tumor Suppressor Protein p53 - genetics | Antineoplastic Agents - toxicity | Dose-Response Relationship, Drug | Cisplatin - toxicity | Tumor Suppressor Protein p53 - agonists | Epithelial Cells - cytology | Kidney Tubules - metabolism | Cell Line | Kidney Tubules - drug effects | Citric Acid Cycle - drug effects | Gene Expression Regulation | Injections, Intraperitoneal | Tumor Suppressor Protein p53 - metabolism | Rats | Rats, Sprague-Dawley | Kidney Tubules - cytology | Animals | L-Lactate Dehydrogenase - genetics | Acetylcysteine - pharmacology | Cell Proliferation - drug effects | Physiological aspects | Reactive oxygen species | Genetic aspects | Research | Cisplatin | Bioengineering | Tricarboxylic acid cycle | Oxidative stress | Correlation | Toxicity | p53 Protein | Genes | Science | Cytotoxicity | Kinases | Ovarian cancer | Mitochondria | Cell growth | Pathways | Metabolites | Rodents | Cell cycle | Inhibition | Adenosine triphosphate | Urine | Oxygen | Abnormalities | Mortality | Data processing | Interdisciplinary aspects | Metabolism | Gene expression | Side effects | Cell death | Glycolysis | Apoptosis | Index Medicus
Journal Article
PloS one, ISSN 1932-6203, 03/2014, Volume 9, Issue 3, pp. e89351 - e89351
Although the anti-cancer effects of curcumin has been shown in various cancer cell types, in vitro, pre-clinical and clinical studies showed only a limited efficacy, even at high doses... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Reactive Oxygen Species - metabolism | Apoptosis - drug effects | Curcumin - chemistry | Humans | Cytosol - drug effects | Biological Availability | Curcumin - metabolism | Membrane Potential, Mitochondrial - drug effects | Antineoplastic Agents - metabolism | Biological Transport | Antineoplastic Agents - pharmacokinetics | Antineoplastic Agents - pharmacology | Epigenesis, Genetic - drug effects | Gene Expression Regulation, Neoplastic - drug effects | Proto-Oncogene Proteins c-akt - metabolism | STAT3 Transcription Factor - metabolism | Membrane Proteins - genetics | Curcumin - pharmacokinetics | Curcumin - pharmacology | Proto-Oncogene Proteins - genetics | Mitochondria - metabolism | Mitochondria - drug effects | Antineoplastic Agents - chemistry | MAP Kinase Signaling System - drug effects | Cell Cycle Checkpoints - drug effects | Cell Line, Tumor | Cytosol - metabolism | Care and treatment | Research | Oncology, Experimental | Analysis | Cancer | Drugs | Phosphorylation | Toxicity | Epithelial cells | Cytotoxicity | AKT protein | Drug delivery | Kinases | Tissues | Anticancer properties | Lipophilic | Mitochondria | Rodents | DNA methylation | Curcumin | Membrane potential | Mammary gland | Stat3 protein | Extracellular signal-regulated kinase | Breast cancer | Bioavailability | BNIP3 protein | Tagging | Epigenetics | Conjugation | Apoptosis | Index Medicus
Journal Article
BMC cancer, ISSN 1471-2407, 2010, Volume 10, Issue 1, pp. 210 - 210
Journal Article
Journal Article
Cardiovascular research, ISSN 0008-6363, 10/2016, Volume 112, Issue 1, pp. 491 - 501
Journal Article