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Journal Article
Nature Genetics, ISSN 1061-4036, 02/2014, Volume 46, Issue 2, pp. 188 - 193
Mitochondrial Ca2+ uptake has key roles in cell life and death. Physiological Ca2+ signaling regulates aerobic metabolism, whereas pathological Ca2+ overload... 
UNIPORTER | DISEASE | GENETICS & HEREDITY | MUSCULAR-DYSTROPHY | CA2+ UPTAKE | CELL-SURVIVAL | PROTEINS | MCU | Immunohistochemistry | Calcium Channels - metabolism | Calcium Signaling - physiology | Humans | DNA, Complementary - genetics | Molecular Sequence Data | Mitochondrial Membrane Transport Proteins - genetics | Histological Techniques | Cation Transport Proteins - metabolism | Base Sequence | Membrane Potential, Mitochondrial - genetics | Cation Transport Proteins - genetics | Quadriceps Muscle - pathology | Real-Time Polymerase Chain Reaction | Calcium-Binding Proteins - metabolism | Mitochondrial Membrane Transport Proteins - metabolism | Mitochondria - metabolism | Sequence Analysis, DNA | Exome - genetics | Phenotype | Analysis of Variance | Pedigree | Extrapyramidal Tracts - pathology | Fluorescent Antibody Technique | Polymorphism, Single Nucleotide - genetics | Learning Disorders - genetics | Movement Disorders - genetics | Muscular Diseases - genetics | Calcium Signaling - genetics | Calcium-Binding Proteins - genetics | Gene mutations | Cellular signal transduction | Muscle diseases | Genetic aspects | Research | Health aspects | Risk factors | Proteins | Medical research | Cell culture | Mitochondria | Genealogy | Biomedical research | Nuclear magnetic resonance--NMR | Mutation | Kinases | Muscular dystrophy | Index Medicus
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2013, Volume 8, Issue 2, pp. e55785 - e55785
Mitochondrial calcium uptake is present in nearly all vertebrate tissues and is believed to be critical in shaping calcium signaling, regulating ATP synthesis... 
PROTEIN | ESSENTIAL COMPONENT | RNAI THERAPEUTICS | METABOLISM | RESPIRATION | MULTIDISCIPLINARY SCIENCES | RAT KIDNEY MITOCHONDRIA | CA2+ UPTAKE | OSCILLATIONS | MCU | DELIVERY | Multigene Family | Calcium Channels - metabolism | Calcium - metabolism | Humans | Mitochondrial Membrane Transport Proteins - genetics | Multiprotein Complexes - metabolism | Cell Respiration - genetics | Mitochondria - genetics | RNA Interference | Cation Transport Proteins - metabolism | HEK293 Cells | Membrane Potential, Mitochondrial - genetics | Cation Transport Proteins - genetics | Protein Stability | Calcium Channels - genetics | Calcium Signaling | Calcium-Binding Proteins - metabolism | Amino Acid Sequence | Mitochondrial Membrane Transport Proteins - metabolism | Liver - metabolism | Mitochondria - metabolism | Protein Transport | Sequence Alignment | Animals | Calcium Channels - chemistry | Protein Binding | Mice | HeLa Cells | Calcium-Binding Proteins - genetics | Proteins | Genomes | Cell death | Genes | Genomics | Calcium | Liver | Science | Tissues | Calcium influx | Medical schools | Calcium signalling | Mitochondria | Membrane potential | Bioinformatics | Medical research | Departments | RNA-mediated interference | Metabolism | Gene expression | Calcium (mitochondrial) | Medicine | Silence | Hospitals | Gene duplication | Molecular biology | Electron transport | Pore formation | Pharmaceuticals | Index Medicus
Journal Article
Molecular Cell, ISSN 1097-2765, 01/2015, Volume 57, Issue 1, pp. 69 - 82
Journal Article
Journal of Cell Science, ISSN 0021-9533, 02/2013, Volume 126, Issue 3, pp. 789 - 802
Journal Article
Journal Article
Cell Metabolism, ISSN 1550-4131, 2011, Volume 13, Issue 6, pp. 668 - 678
Here we show that yeast strains with reduced target of rapamycin (TOR) signaling have greater overall mitochondrial electron transport chain activity during... 
TRANSCRIPTION FACTORS | MOLECULAR-MECHANISM | STATIONARY-PHASE | OXIDATIVE STRESS | BUDDING YEAST | INCREASED RESPIRATION | CALORIC RESTRICTION | SCH9 | ENDOCRINOLOGY & METABOLISM | GENE-EXPRESSION | SACCHAROMYCES-CEREVISIAE | CELL BIOLOGY | Protein Kinases - metabolism | Protein Kinases - genetics | Adaptation, Physiological - drug effects | Multiprotein Complexes - genetics | Saccharomyces cerevisiae - drug effects | Phosphatidylinositol 3-Kinases - metabolism | Colony Count, Microbial | Membrane Potential, Mitochondrial - drug effects | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Multiprotein Complexes - metabolism | Microbiological Phenomena - genetics | Superoxides - metabolism | Membrane Potential, Mitochondrial - genetics | Protein-Serine-Threonine Kinases - metabolism | Microbiological Phenomena - drug effects | Saccharomyces cerevisiae - physiology | Saccharomyces cerevisiae Proteins - antagonists & inhibitors | Protein-Serine-Threonine Kinases - genetics | Mitochondria - metabolism | Mitochondria - drug effects | Saccharomyces cerevisiae Proteins - genetics | Gene Knockout Techniques | Sirolimus - pharmacology | Dinitrophenols - pharmacology | Phosphatidylinositol 3-Kinases - genetics | Vitamin K 3 - pharmacology | Oxygen Consumption - genetics | Saccharomyces cerevisiae Proteins - metabolism | Saccharomyces cerevisiae - growth & development | Medical colleges | Chronologies | Superoxide | TOR protein | Reactive oxygen species | Menadione | Conservation | Acidification | Superoxide dismutase | Rapamycin | Longevity | Media (transport) | Electron transport chain | Mitochondria | Life span | Membrane potential | Electron transport | ATP | Manganese | Index Medicus
Journal Article
Neuron, ISSN 0896-6273, 04/2013, Volume 78, Issue 1, pp. 57 - 64
Valosin-containing protein (VCP) is a highly expressed member of the type II AAA+ ATPase family. mutations are the cause of inclusion body myopathy, Paget’s... 
LIPID-PEROXIDATION | SPINAL-CORD PATHOLOGY | MOUSE MODEL | ALS | AMYOTROPHIC-LATERAL-SCLEROSIS | DYSFUNCTION | BONE | NEUROSCIENCES | PAGET-DISEASE | TRANSGENIC MICE | REVEALS | RNA, Small Interfering - genetics | Humans | Middle Aged | Male | Frontotemporal Dementia - metabolism | Neurons - ultrastructure | Muscular Dystrophies, Limb-Girdle - genetics | Adenosine Triphosphate - metabolism | Membrane Potential, Mitochondrial - genetics | Muscular Dystrophies, Limb-Girdle - pathology | NAD - metabolism | Fibroblasts - metabolism | Animals, Newborn | Frontotemporal Dementia - genetics | Magnesium - metabolism | Mitochondria - pathology | Fibroblasts - pathology | Mutation - genetics | Myositis, Inclusion Body - genetics | Osteitis Deformans - pathology | Muscular Dystrophies, Limb-Girdle - metabolism | Analysis of Variance | Luminescent Proteins - genetics | Adenosine Triphosphatases - genetics | Mice | Lipid Peroxidation - genetics | RNA, Small Interfering - metabolism | Valosin Containing Protein | Osteitis Deformans - metabolism | Family Health | Cerebral Cortex - cytology | Case-Control Studies | Osteitis Deformans - genetics | Transfection | Mitochondria - genetics | Cell Cycle Proteins - genetics | Myositis, Inclusion Body - pathology | Adult | Female | Neuroblastoma - pathology | Frontotemporal Dementia - pathology | Adenosine Triphosphatases - deficiency | Mice, Inbred C57BL | Cells, Cultured | Cell Cycle Proteins - deficiency | Mitochondria - metabolism | Animals | Oxygen Consumption - genetics | Myositis, Inclusion Body - metabolism | Aged | Nervous system diseases | Neurosciences | Genes | Amyotrophic lateral sclerosis | Genetic aspects | Adenosine triphosphatase | Dementia | Proteins | Medical research | Phosphorylation | Biomedical research | Disease | Rodents | Respiration | Experiments | Patients | Index Medicus | Report
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 09/2008, Volume 283, Issue 36, pp. 24770 - 24780