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Neuropharmacology, ISSN 0028-3908, 05/2014, Volume 80, pp. 18 - 27
.... Human exome sequencing and gene wide association studies have linked mutations in CRC subunits to intellectual disability disorders, autism spectrum disorder and schizophrenia... 
Intellectual disability disorder | Long-term potentiation | Epigenetics | Long-term memory | Nucleosome remodeling | Autism spectrum disorder | Chromatin remodeling | TRANSCRIPTION FACTORS | COMPLEX | LONG-TERM-MEMORY | DNA METHYLATION | MENTAL-RETARDATION | CORTICAL PYRAMIDAL NEURONS | SELF-RENEWAL | HISTONE ACETYLATION | NEUROSCIENCES | DE-NOVO MUTATIONS | DENDRITIC SPINE DENSITY | PHARMACOLOGY & PHARMACY | Memory Disorders - physiopathology | Autistic Disorder - physiopathology | Autistic Disorder - metabolism | Memory Disorders - genetics | Epigenesis, Genetic | Humans | Memory | Actins - metabolism | Brain - enzymology | Memory Disorders - metabolism | Cognition Disorders - metabolism | Actins - genetics | Brain - metabolism | DNA-Binding Proteins - metabolism | Nucleosomes - enzymology | Neurons - metabolism | Nuclear Proteins - genetics | DNA Helicases - genetics | Autistic Disorder - genetics | Cognition Disorders - physiopathology | Chromosomal Proteins, Non-Histone - metabolism | Gene Expression Regulation | Chromatin Assembly and Disassembly | Nucleosomes - metabolism | Nuclear Proteins - metabolism | Cognition Disorders - genetics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Nerve Tissue Proteins - genetics | Chromosomal Proteins, Non-Histone - genetics | Nerve Tissue Proteins - metabolism | Transcription Factors - metabolism | DNA Helicases - metabolism | Animals | Models, Biological | Neuronal Plasticity | Neurons - enzymology | Intellectual Disability - etiology | Mutation | Neurosciences | Chromatin | Neurons | Genes | Histones | Schizophrenia | Gene expression | chromatin remodeling | intellectual disability disorder | autism spectrum disorder | nucleosome remodeling | long-term memory | epigenetics | long-term potentiation
Journal Article
Molecular Neurobiology, ISSN 0893-7648, 5/2016, Volume 53, Issue 4, pp. 2451 - 2467
...Hydrogen Sulfide Ameliorates Homocysteine-Induced Alzheimer’s Disease-Like Pathology, Blood–Brain Barrier Disruption, and Synaptic Disorder Pradip K. Kamat 1... 
Cerebrovascular pathology | Neurology | Neurosciences | Biomedicine | Blood–brain barrier dysfunction | Alzheimer’s disease | Neurobiology | Homocysteine | Cell Biology | Dementia | COGNITIVE PERFORMANCE | NEUROSCIENCES | MATRIX METALLOPROTEINASES | INDUCED MEMORY IMPAIRMENT | RISK-FACTOR | Blood-brain barrier dysfunction | RECEPTOR TRAFFICKING | MATRIX-METALLOPROTEINASE-9 | RAT-BRAIN | Alzheimer's disease | PLASMA HOMOCYSTEINE | EXPRESSION | Memory - drug effects | Glial Fibrillary Acidic Protein - genetics | Cadherins - metabolism | Microtubule-Associated Proteins - genetics | Microtubule-Associated Proteins - metabolism | Blood-Brain Barrier - physiopathology | Claudin-5 - metabolism | Microvessels - pathology | Male | Synapses - pathology | Glial Fibrillary Acidic Protein - metabolism | RNA, Messenger - metabolism | Antigens, CD - genetics | Alzheimer Disease - pathology | Antigens, CD - metabolism | Matrix Metalloproteinase 9 - metabolism | Cadherins - genetics | Synapses - drug effects | Alzheimer Disease - physiopathology | Hydrogen Sulfide - pharmacology | Cerebrovascular Circulation - drug effects | Matrix Metalloproteinase 2 - metabolism | Mice, Inbred C57BL | RNA, Messenger - genetics | Alzheimer Disease - drug therapy | Microvessels - drug effects | Claudin-5 - genetics | Cystathionine beta-Synthase - metabolism | Permeability | Blood-Brain Barrier - drug effects | Nerve Tissue Proteins - genetics | Blood-Brain Barrier - metabolism | Gene Expression Regulation - drug effects | Nerve Tissue Proteins - metabolism | Blood-Brain Barrier - pathology | Hydrogen Sulfide - therapeutic use | Intercellular Adhesion Molecule-1 - metabolism | Animals | Intercellular Adhesion Molecule-1 - genetics | Avoidance Learning - drug effects | Alzheimer Disease - genetics | Dizocilpine Maleate - pharmacology | Hydrogen sulfide | Methyl aspartate | Brain | RNA | Neurons | Intermediate filament proteins | cerebrovascular pathology | dementia | blood brain barrier dysfunction
Journal Article
European Neuropsychopharmacology, ISSN 0924-977X, 2013, Volume 23, Issue 10, pp. 1165 - 1181
Journal Article
Journal Article
British journal of pharmacology, ISSN 0007-1188, 2015, Volume 172, Issue 9, pp. 2354 - 2368
Background and Purpose Mild cognitive deficit in early Parkinson's disease (PD) has been widely studied. Here we have examined the effects of memantine in... 
DEPRESSION | ACTIVATION | CONCISE GUIDE | NEUROTROPHIC FACTOR | PHARMACOLOGY | LONG-TERM-POTENTIATION | PHARMACOLOGY & PHARMACY | DOPAMINERGIC-NEURONS | MODEL | COGNITIVE FUNCTION | PARKINSONS-DISEASE | Memory Disorders - physiopathology | Antiparkinson Agents - pharmacology | Membrane Glycoproteins - metabolism | Protein-Tyrosine Kinases - metabolism | Memory Disorders - metabolism | Neuronal Plasticity - drug effects | Male | Extracellular Signal-Regulated MAP Kinases - metabolism | Hippocampus - drug effects | Parkinsonian Disorders - metabolism | Synapses - metabolism | Time Factors | Behavior, Animal - drug effects | Brain-Derived Neurotrophic Factor - metabolism | Disease Models, Animal | Parkinsonian Disorders - physiopathology | Synapses - drug effects | Memantine - pharmacology | Memory Disorders - psychology | 1-Methyl-4-phenylpyridinium - pharmacology | Mice, Inbred C57BL | Parkinsonian Disorders - psychology | Memory, Long-Term - drug effects | Parkinsonian Disorders - chemically induced | Hippocampus - metabolism | Memory Disorders - prevention & control | Animals | Signal Transduction - drug effects | Calcium Signaling - drug effects | 1-Methyl-4-phenyl-1,2,3,6-tetrahydropyridine | Parkinsonian Disorders - prevention & control | In Vitro Techniques | Hippocampus - physiopathology | Memory Disorders - chemically induced | Calpain | Mice | Pyridine | Analysis | Research Papers
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2010, Volume 107, Issue 52, pp. 22687 - 22692
Cognitive dysfunction and memory loss are common features of Alzheimer's disease (AD). Abnormalities in the expression profile of immediate early genes that... 
Memory disorders | Training | Pathology | Phosphorylation | Animal models | Neurons | N methyl D aspartate receptors | Memory | Amyloids | Alzheimers disease | Tangles | Presenilin | tangles | NEUROTROPHIC-FACTOR | MULTIDISCIPLINARY SCIENCES | AMYLOID-BETA-PROTEIN | MECHANISMS | TRANSGENIC MODEL | PATHOLOGY | CREB-BINDING PROTEIN | MESSENGER-RNA | presenilin | A-BETA | EXPRESSION | PRESENILIN-1 | Memory Disorders - physiopathology | Cricetulus | Humans | Memory Disorders - metabolism | tau Proteins - metabolism | Learning Disorders - physiopathology | Brain - metabolism | CREB-Binding Protein - genetics | CREB-Binding Protein - metabolism | Learning Disorders - therapy | Amyloid beta-Peptides - metabolism | Amyloid beta-Protein Precursor - metabolism | Lentivirus - genetics | Brain-Derived Neurotrophic Factor - metabolism | Neurons - metabolism | CHO Cells | Disease Models, Animal | Gene Transfer Techniques | Alzheimer Disease - physiopathology | Cricetinae | Mice, Inbred C57BL | Alzheimer Disease - therapy | Mice, Transgenic | Blotting, Western | Amyloid beta-Protein Precursor - genetics | Animals | Alzheimer Disease - metabolism | Cyclic AMP Response Element-Binding Protein - metabolism | Memory Disorders - therapy | Brain - pathology | Learning Disorders - metabolism | Mice | Genetic Therapy - methods | Genetic transformation | Amyloid beta-protein | Models | Genetic aspects | Research | Alzheimer's disease | Protein binding | Biological Sciences
Journal Article
British journal of psychiatry, ISSN 0007-1250, 03/2013, Volume 202, Issue 3, pp. 172 - 176
Background Bipolar disorder has a high co-occurrence with substance use disorders, but the pathophysiological mechanisms have not been adequately explored... 
Special Article | DRUG | DEPRESSION | PSYCHIATRY | EPIGENETIC REGULATION | EARLY-ONSET | STRESS | EXTINCTION | PREFRONTAL CORTEX | Behavior, Addictive - drug therapy | Free Radical Scavengers - pharmacology | Bipolar Disorder - metabolism | Recurrence | Dopamine Uptake Inhibitors - adverse effects | Humans | Cocaine-Related Disorders - epidemiology | Stress, Psychological - psychology | Cocaine-Related Disorders - metabolism | Stress, Psychological - metabolism | Stress, Psychological - epidemiology | Brain-Derived Neurotrophic Factor - metabolism | Free Radical Scavengers - therapeutic use | Acetylcysteine - therapeutic use | Cocaine - adverse effects | Disease Models, Animal | Epigenomics | Cocaine-Related Disorders - psychology | Comorbidity | Disease Progression | Bipolar Disorder - psychology | Animals | Acetylcysteine - pharmacology | Age of Onset | Central Nervous System Sensitization - physiology | Bipolar Disorder - epidemiology | Basal Ganglia - drug effects | Diagnosis, Dual (Psychiatry) | Brain | Early intervention | Emotional disorders | Dopamine | Hyperactivity | Acetylcysteine | Bipolar disorder | Alcohol | Addictions | Brain-derived neurotrophic factor | Mood | Stimulants | Intermittent | Epigenetics | Amphetamines | Cocaine | Psychiatry | Drug use | Memories | Substance abuse disorders | Depressive Disorders | Drug Abuse | Bipolar Disorders | Treatment | Neuroendocrinology | Etiology | Cocaine Abuse | Survivors | Stressors
Journal Article
BMC infectious diseases, ISSN 1471-2334, 2013, Volume 13, Issue 1, pp. 261 - 261
.... We evaluated the influence of rifampicin cotreatment, efavirenz pharmacokinetics and pharmacogenetics on neuropsychiatric disorders in Ugandan HIV patients with or without tuberculosis coinfection. Methods... 
CYP2B6 | Rifampicin | Tuberculosis | HIV | CNS | Ugandans | Efavirenz | Neuropsychiatric toxicity | Antiretroviral therapy | INFECTIOUS DISEASES | ANTITUBERCULAR THERAPY | SECONDARY METABOLISM | DRUG CONCENTRATIONS | HIV-1-INFECTED PATIENTS | BLOOD-BRAIN-BARRIER | NEUROPSYCHIATRIC ADVERSE EVENTS | PLASMA EXPOSURE | NEUROCOGNITIVE DISORDERS | INFECTED PATIENTS | Memory Disorders - virology | Prospective Studies | Humans | Mental Disorders - metabolism | Aryl Hydrocarbon Hydroxylases - genetics | Hallucinations - virology | Memory Disorders - metabolism | Uganda - epidemiology | HIV Infections - psychology | Male | Sleep Arousal Disorders - virology | HIV Infections - microbiology | Cytochrome P-450 CYP3A - genetics | Benzoxazines - pharmacokinetics | Sleep Arousal Disorders - metabolism | Tuberculosis - drug therapy | Adult | Benzoxazines - blood | Female | Rifampin - therapeutic use | Anti-HIV Agents - blood | Mental Disorders - virology | Tuberculosis - metabolism | Cognitive Dysfunction - drug therapy | Hallucinations - drug therapy | Cognitive Dysfunction - virology | Anti-HIV Agents - adverse effects | Cognitive Dysfunction - metabolism | Gene Frequency | Kaplan-Meier Estimate | Benzoxazines - adverse effects | Hallucinations - metabolism | Mental Disorders - microbiology | Antiretroviral Therapy, Highly Active | Tuberculosis - virology | Memory Disorders - drug therapy | Sleep Arousal Disorders - drug therapy | Mental Disorders - drug therapy | Anti-HIV Agents - pharmacokinetics | Antibiotics, Antitubercular - therapeutic use | HIV Infections - drug therapy | Cytochrome P-450 CYP2B6 | HIV Infections - metabolism | Sleep disorders | Hallucinations and illusions | Insomnia | Plasma | Genotype & phenotype | Health sciences | Brain research | Blood-brain barrier | Human immunodeficiency virus--HIV | Drug therapy | Patients | Psykiatri | Farmakologi och toxikologi | Klinisk medicin | Medicin och hälsovetenskap | Infektionsmedicin | Medicinska och farmaceutiska grundvetenskaper
Journal Article