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2013, 1
Reference
2013, Advances in molecular and cellular microbiology, ISBN 1780641621, Volume 26
Web Resource
2013, Advances in molecular and cellular microbiology, ISBN 1780641621, Volume 26
Web Resource
2013, Advances in molecular and cellular microbiology, ISBN 1780641621, Volume 26
Web Resource
Nature, ISSN 0028-0836, 01/2009, Volume 457, Issue 7226, pp. 191 - 195
Journal Article
Journal Article
Nature Neuroscience, ISSN 1097-6256, 06/2017, Volume 20, Issue 6, pp. 774 - 783
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2018, Volume 13, Issue 5, pp. e0196893 - e0196893
Borrelia burgdorferi, the causative agent of Lyme disease, is a vector-borne bacterial infection that is transmitted through the bite of an infected tick. If... 
NERVOUS-SYSTEM | INTRADERMAL INOCULATION | NONHUMAN PRIMATE MODEL | LYME NEUROBORRELIOSIS | ANTIBODY-PRODUCTION | MULTIDISCIPLINARY SCIENCES | MOUSE MODEL | DISEASE | INFECTION | CEREBROSPINAL-FLUID | BINDING PROTEIN | Meninges - immunology | Capillaries - pathology | Meninges - microbiology | Humans | Dura Mater - microbiology | Capillaries - microbiology | Meninges - blood supply | Borrelia burgdorferi - physiology | Male | Lyme Disease - pathology | CD4-Positive T-Lymphocytes - immunology | Lyme Disease - microbiology | Dura Mater - blood supply | Disease Models, Animal | Injections, Intradermal | CD4-Positive T-Lymphocytes - microbiology | CD8-Positive T-Lymphocytes - microbiology | Meninges - pathology | Dura Mater - pathology | Dura Mater - immunology | Mice, Inbred C3H | Bacterial Adhesion | Host-Pathogen Interactions | Lyme Disease - immunology | Animals | Borrelia burgdorferi - pathogenicity | Capillaries - immunology | Mice | CD8-Positive T-Lymphocytes - immunology | Cell Movement | Dura mater | Models | Medical examination | Immune response | Spirochetes | Lyme disease | Cell culture | Health sciences | Flow cytometry | Pathogenesis | CD8 antigen | Trafficking | Central nervous system | Disseminated infection | Borreliosis | Lymphocytes T | Infections | Leukocytes | Tissues | Lymphocytes | Primates | Physiology | Meninges | Vector-borne diseases | Arachnids | Blood vessels | CD3 antigen | Investigations | CD4 antigen | Immune systems | Medicine | White blood cells | Antibiotics | Plasmids | Index Medicus
Journal Article
2009, Volume 3, 1
Reference
Nature, ISSN 0028-0836, 2014, Volume 505, Issue 7482, pp. 223 - 228
Traumatic brain injury (TBI) is increasingly appreciated to be highly prevalent and deleterious to neurological function(1,2). At present, no effective... 
BIOMARKERS | GREEN FLUORESCENT PROTEIN | HEAD-INJURY | INSERTION | STERILE INFLAMMATION | FLUID | CORTEX | MULTIDISCIPLINARY SCIENCES | IN-VIVO | MICE | RELEASE | Encephalitis - prevention & control | Neuroprotective Agents - therapeutic use | Reactive Oxygen Species - metabolism | Brain Injuries - drug therapy | Humans | Astrocytes - pathology | Brain Injuries - complications | Male | Intracranial Hemorrhages - diagnosis | Microglia - physiology | Encephalitis - drug therapy | Glasgow Coma Scale | Purinergic P2 Receptor Antagonists - pharmacology | Encephalitis - complications | Cell Death - drug effects | Neuroprotective Agents - administration & dosage | Disease Models, Animal | Microglia - cytology | Glutathione - therapeutic use | Skull - metabolism | Meninges - pathology | Microglia - drug effects | Neutrophils - drug effects | Neutrophils - physiology | Encephalitis - pathology | Rats | Purinergic P2 Receptor Antagonists - therapeutic use | Meninges - drug effects | Rats, Sprague-Dawley | Brain Injuries - diagnosis | Brain - drug effects | Antioxidants - therapeutic use | Animals | Glutathione - administration & dosage | Receptors, Purinergic P2 - metabolism | Antioxidants - administration & dosage | Brain - pathology | Receptors, Purinergic P2X7 - metabolism | Purinergic P2 Receptor Antagonists - administration & dosage | Mice | Administration, Topical | Brain Injuries - pathology | Intracranial Hemorrhages - complications | Brain | Medical examination | Cell death | Physiological aspects | Skull | Models | Meninges | Observations | Injuries | Brain damage | Emergency medical care | Head injuries | Rodents | Index Medicus
Journal Article
NATURE, ISSN 0028-0836, 08/2018, Volume 560, Issue 7717, pp. 185 - 185
Ageing is a major risk factor for many neurological pathologies, but its mechanisms remain unclear. Unlike other tissues, the parenchyma of the central nervous... 
AMYLOID PROTEIN | BRAIN INTERSTITIAL FLUID | MULTIDISCIPLINARY SCIENCES | PLAQUE-FORMATION | CENTRAL-NERVOUS-SYSTEM | GROWTH FACTOR-C | GENE-THERAPY | EXPRESSION | MOUSE MODELS | TRANSGENIC MICE | BETA | Humans | Homeostasis | Male | Cognition | Aging - cerebrospinal fluid | Alzheimer Disease - pathology | Brain - metabolism | Meninges - physiopathology | Amyloid - metabolism | Amyloid beta-Peptides - metabolism | Female | Disease Models, Animal | Alzheimer Disease - physiopathology | Cognition Disorders - physiopathology | Meninges - pathology | Lymph Nodes - metabolism | Mice, Transgenic | Alzheimer Disease - cerebrospinal fluid | Aging - pathology | Extracellular Fluid - metabolism | Animals | Perfusion | Lymphatic Vessels - pathology | Mice | Cognition Disorders - therapy | Lymphatic Vessels - physiopathology | Alzheimer's disease | Genetic aspects | Brain | Animal models | Nuclear magnetic resonance--NMR | Memory | Central nervous system | Impairment | Cognitive ability | Parenchyma | Genomes | Cerebrospinal fluid | Lymph nodes | Risk factors | Learning | Rodents | Aging | Amyloid | Meninges | Vascular endothelial growth factor | Growth factors | Bioinformatics | Age | Efflux | Lymphatic system | Neurodegenerative diseases | Computational fluid dynamics | Therapeutic applications | Macromolecules | Health risks | Fluid | Transgenic mice | Blood vessels | Risk analysis | Gene expression | Neurological diseases | Pathology | Vascular endothelial growth factor C | Disruption | Alzheimers disease | Index Medicus
Journal Article
STEM CELLS, ISSN 1066-5099, 12/2011, Volume 29, Issue 12, pp. 2062 - 2076
Adult spinal cord has little regenerative potential, thus limiting patient recovery following injury. In this study, we describe a new population of cells... 
Stem cell niche | Neural stem cells | Meninges | Spinal cord injury | NEURONAL MIGRATION | SUBVENTRICULAR ZONE | NG2 PROTEOGLYCAN | CEREBRAL-CORTEX | CELL & TISSUE ENGINEERING | CELL BIOLOGY | MAMMALIAN BRAIN | ONCOLOGY | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | EMBRYONIC STEM-CELLS | STEM/PROGENITOR CELLS | CENTRAL-NERVOUS-SYSTEM | NEURAL PROGENITOR CELLS | GROWTH-FACTOR | HEMATOLOGY | Adult Stem Cells - physiology | Oligodendroglia - metabolism | Nestin | Cell Proliferation | Microtubule-Associated Proteins - genetics | Microtubule-Associated Proteins - metabolism | Gene Expression Profiling | Laminectomy | Regenerative Medicine | Neural Stem Cells - cytology | Neurogenesis | Meninges - metabolism | Lentivirus - metabolism | Oligodendroglia - physiology | Intermediate Filament Proteins - genetics | Lentivirus - genetics | Cell Differentiation | Neuropeptides - genetics | Oligodendroglia - cytology | Spinal Cord Injuries - therapy | Adult Stem Cells - cytology | Stem Cell Niche | Neural Stem Cells - physiology | Rats | Meninges - cytology | Neuropeptides - metabolism | Nerve Tissue Proteins - genetics | Rats, Sprague-Dawley | Nerve Tissue Proteins - metabolism | Adult Stem Cells - metabolism | Patch-Clamp Techniques | Animals | Meninges - physiology | Electrophysiologic Techniques, Cardiac | Intermediate Filament Proteins - metabolism | Cell Movement | Index Medicus | Tissue-Specific Stem Cells
Journal Article