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Nature Neuroscience, ISSN 1097-6256, 02/2009, Volume 12, Issue 2, pp. 125 - 131
Journal Article
Molecular Pharmacology, ISSN 0026-895X, 10/2018, Volume 94, Issue 4, pp. 1220 - 1231
Journal Article
Cell and Tissue Research, ISSN 0302-766X, 2/2007, Volume 327, Issue 2, pp. 313 - 322
The ontogeny of two gonadotropin-releasing-hormone (GnRH) systems, salmon GnRH (sGnRH) and chicken GnRH-II (cGnRH-II), was investigated in zebrafish (Danio... 
Human Genetics | Neuronal migration | Biomedicine | Proteomics | Reporter protein | Danio rerio ( Teleostei ) | Molecular Medicine | Gonadotropin-releasing hormone | In situ hybridization | Danio rerio (Teleostei) | DANIO-RERIO | CELLS | OLFACTORY PLACODE | reporter protein | GONADOTROPIN-RELEASING-HORMONE | CELL BIOLOGY | gonadotropin-releasing hormone | MESSENGER-RNA | neuronal migration | GOLDFISH | FISH | CHICK-EMBRYO | NEUROBLASTS | in situ hybridization | Mesencephalon - cytology | Mesencephalon - metabolism | Neurons - cytology | Green Fluorescent Proteins - genetics | Brain - growth & development | Preoptic Area - metabolism | RNA, Messenger - metabolism | Gonadotropin-Releasing Hormone - analogs & derivatives | Preoptic Area - cytology | Recombinant Fusion Proteins - metabolism | Brain - metabolism | In Situ Hybridization | Transfection | Larva - growth & development | Neurons - metabolism | Green Fluorescent Proteins - metabolism | Brain - cytology | Gene Expression | Olfactory Pathways - metabolism | Telencephalon - metabolism | Telencephalon - cytology | Gonadotropin-Releasing Hormone - genetics | Larva - metabolism | Neural Pathways - growth & development | RNA, Messenger - genetics | Zebrafish - growth & development | Zebrafish - genetics | Animals | Hypothalamus - metabolism | Hypothalamus - cytology | Olfactory Pathways - cytology | Zebrafish - metabolism | Gonadotropin-Releasing Hormone - metabolism | Neural Pathways - metabolism | Recombinant Fusion Proteins - genetics | Proteins | Brain | Neurons | Zebrafish | Hormones | Gene expression | Cell adhesion & migration
Journal Article
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2015, Volume 10, Issue 9, p. e0137136
The expression of soluble growth and survival promoting factors by neural precursor cells (NPCs) is suggested to be a prominent mechanism underlying the... 
STEM-CELLS | ADULT RATS | NEUROTROPHIC FACTOR | MULTIDISCIPLINARY SCIENCES | CENTRAL-NERVOUS-SYSTEM | SPINAL-CORD-INJURY | SUBVENTRICULAR ZONE | RAT MODEL | NEUROPROTECTION | PARKINSONS-DISEASE | TRANSPLANTATION | Substantia Nigra - growth & development | RNA, Small Interfering - genetics | Substantia Nigra - pathology | Dopaminergic Neurons - pathology | Mesencephalon - metabolism | Humans | Rats, Inbred F344 | Alkaline Phosphatase - metabolism | Hedgehog Proteins - metabolism | Glial Cell Line-Derived Neurotrophic Factor - antagonists & inhibitors | Substantia Nigra - metabolism | Neural Stem Cells - cytology | Oxidopamine | Neurogenesis - genetics | Glial Cell Line-Derived Neurotrophic Factor - metabolism | Hedgehog Proteins - genetics | Gene Expression Regulation, Developmental | Isoenzymes - metabolism | Dopaminergic Neurons - metabolism | Neural Stem Cells - transplantation | Neostriatum - metabolism | Transgenes | Disease Models, Animal | Mesencephalon - pathology | Animals, Newborn | Neostriatum - growth & development | Neostriatum - pathology | Alkaline Phosphatase - genetics | Rats, Transgenic | Signal Transduction | Isoenzymes - genetics | Glial Cell Line-Derived Neurotrophic Factor - genetics | Rats | Neurodegenerative Diseases - genetics | Neurodegenerative Diseases - therapy | Hedgehog Proteins - antagonists & inhibitors | Neurodegenerative Diseases - metabolism | GPI-Linked Proteins - metabolism | Stereotaxic Techniques | Phenotype | Animals | Neurodegenerative Diseases - chemically induced | Graft Survival - genetics | Mesencephalon - growth & development | Primary Cell Culture | Neural Stem Cells - metabolism | GPI-Linked Proteins - genetics | RNA, Small Interfering - metabolism | Stem cells | Physiological aspects | Nervous system | Development and progression | Genetic aspects | Degeneration | Models | Research | Neuroprotection | Brain | Biotechnology | Neurosciences | Nestin | Spinal cord | Mesencephalon | Substantia nigra | Parkinsons disease | Science | Transplantation | Neurogenesis | Grafting | Neuronal-glial interactions | Neurodegeneration | Efficiency | Precursors | Rodents | Neostriatum | Doublecortin protein | Growth factors | Phenotypes | Glial cell line-derived neurotrophic factor | Cell survival | Dopamine | RNA-mediated interference | 6-Hydroxydopamine | Ribonucleic acid--RNA | Molecular chains | Medicine | Hedgehog protein | Neural stem cells | Cell migration | RNA | Ribonucleic acid
Journal Article
European Journal of Neuroscience, ISSN 0953-816X, 03/2014, Volume 39, Issue 6, pp. 946 - 956
Variation within mesolimbic dopamine ( DA ) pathways has significant implications for behavioral responses to rewards, and previous studies have indicated... 
mother–infant | ventral tegmental area | conditioned place preference | mesolimbic | ong | vans rats | tyrosine hydroxylase | Long evans rats | Tyrosine hydroxylase | Conditioned place preference | Ventral tegmental area | Mesolimbic | Mother-infant | mother-infant | POSTTRANSCRIPTIONAL REGULATION | RECEPTOR-ALPHA EXPRESSION | HIGH-FAT DIET | Long Evans rats | JUVENILE RATS | SUBSTANTIA-NIGRA | NEUROSCIENCES | NUCLEUS-ACCUMBENS | MEDIAL PREOPTIC AREA | INDIVIDUAL-DIFFERENCES | TYROSINE-HYDROXYLASE GENE | Brain-Derived Neurotrophic Factor - genetics | Tyrosine 3-Monooxygenase - metabolism | LIM-Homeodomain Proteins - metabolism | Mesencephalon - metabolism | Receptors, Dopamine - metabolism | RNA, Messenger - genetics | Rats, Long-Evans | Rats | Transcription Factors - genetics | RNA, Messenger - metabolism | Transcription Factors - metabolism | Tyrosine 3-Monooxygenase - genetics | LIM-Homeodomain Proteins - genetics | Animals | Cyclin-Dependent Kinase Inhibitor p21 - genetics | Dopaminergic Neurons - metabolism | Maternal Behavior | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Female | Mesencephalon - growth & development | Brain-Derived Neurotrophic Factor - metabolism | Mesencephalon - physiology | Reward | Receptors, Dopamine - genetics | Phenols | Hydroxylases | RNA | Social aspects | Gene expression | Analysis
Journal Article
Molecular Neurobiology, ISSN 0893-7648, 07/2017, Volume 54, Issue 5, pp. 3825 - 3842
While environmental exposures are not the single cause of Parkinson's disease (PD), their interaction with genetic alterations is thought to contribute to... 
Metabolomics | α-Synuclein | Glucose transporters | Pesticides | Glycolysis | Adenosine monophosphate-activated kinase | Autophagy | Parkinson’s disease | OXIDATIVE STRESS | ENERGY STRESS | Parkinson's disease | ACTIVATED PROTEIN-KINASE | NEUROSCIENCES | COMPLEX-I | MOUSE MODEL | MITOCHONDRIAL DYSFUNCTION | MICE | alpha-Synuclein | PARKINSONS-DISEASE | Glucose Transport Proteins, Facilitative - metabolism | Cell Line | Pentose Phosphate Pathway - drug effects | Dopaminergic Neurons - pathology | alpha-Synuclein - toxicity | Mice, Inbred C57BL | Rats | Enzyme Activation - drug effects | Gene-Environment Interaction | Autophagy - drug effects | Mice, Knockout | Animals | Metabolome - drug effects | Signal Transduction - drug effects | Adenylate Kinase - metabolism | Dopaminergic Neurons - metabolism | Brain - pathology | Glucose - metabolism | Biological Transport - drug effects | Cell Death - drug effects | Paraquat - toxicity | Nitric Oxide Synthase Type II - metabolism | Proteins | Herbicides | Cell death | Nitric oxide | Genes | Physiological aspects | Biochemistry | Glucose | Adenylic acid | Protein kinases | Dextrose | Ascorbic acid | Alterations | Neurodegeneration | Pentose | Neostriatum | Inhibition | Movement disorders | Glucose transporter | AMP | Neurodegenerative diseases | Mortality | Rapamycin | Metabolism | Glucose transport | Nitric-oxide synthase | Sensitivity enhancement | Transport | Transporter | Phagocytosis | Phosphates | Mesencephalon | Toxicity | Activation | Kinases | Synuclein | Synergism | Glucose metabolism | Neurotoxicity | Rodents | Degeneration | Dopamine receptors | Deprivation | Dopamine | Adenosine monophosphate | Hexokinase | Cells | Protein kinase | Adenosine kinase | Pentose phosphate pathway | Paraquat | Galactose | Apoptosis | Index Medicus | autophagy | pesticides | metabolomics | glycolysis | adenosine monophosphate-activated kinase | glucose transporters | α-synuclein
Journal Article
Scientific Reports, ISSN 2045-2322, 02/2016, Volume 6, Issue 1, p. 20270
Generation of induced dopaminergic (iDA) neurons may provide a significant step forward towards cell replacement therapy for Parkinson's disease (PD). To study... 
CELLS | HUMAN ES | FATE | PATHWAY | MULTIDISCIPLINARY SCIENCES | DISEASE | Embryonic Stem Cells - metabolism | Embryonic Stem Cells - cytology | Mesencephalon - cytology | Hepatocyte Nuclear Factor 3-beta - genetics | LIM-Homeodomain Proteins - metabolism | Oligonucleotide Array Sequence Analysis | RNA, Messenger - isolation & purification | Humans | Transcriptome | Otx Transcription Factors - metabolism | Gene Expression Profiling | RNA, Messenger - metabolism | Neurogenesis | Dopaminergic Neurons - cytology | Otx Transcription Factors - genetics | Dopaminergic Neurons - metabolism | Nestin - genetics | Cell Differentiation | Parkinson Disease - metabolism | Induced Pluripotent Stem Cells - cytology | Induced Pluripotent Stem Cells - metabolism | Hepatocyte Nuclear Factor 3-beta - metabolism | Parkinson Disease - pathology | Pluripotent Stem Cells - cytology | Cells, Cultured | Metabolome | Transcription Factors - genetics | Nestin - metabolism | Pluripotent Stem Cells - metabolism | Transcription Factors - metabolism | LIM-Homeodomain Proteins - genetics | Sequence Analysis, RNA | RNA, Messenger - chemistry | Dopamine | Parkinson's disease | Transcription | Mesencephalon | Neurodegenerative diseases | Neurons | Parkinsons disease | Gene expression | Stem cells | Movement disorders | Pluripotency | Dopamine receptors | Inhibitory postsynaptic potentials
Journal Article
Molecular and Cellular Neuroscience, ISSN 1044-7431, 2005, Volume 28, Issue 3, pp. 547 - 555
Journal Article