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2013, Johns Hopkins biographies of disease, ISBN 9781421411323, xxiv, 289
Book
Journal of inherited metabolic disease, ISSN 0141-8955, 1978
Journal
Journal of Neurology, Neurosurgery and Psychiatry, ISSN 0022-3050, 02/2019, Volume 90, Issue 2, pp. 211 - 218
In adulthood, spinal cord MRI abnormalities such as T2-weighted hyperintensities and atrophy are commonly associated with a large variety of causes... 
hereditary spastic paraplegia | metabolic disease | MRI | neurogenetics | myelopathy | Urine | Plasma | Multiple sclerosis | Spinal cord | Genetic disorders | Nuclear magnetic resonance--NMR | Disease | Dietary minerals | Vitamin deficiency | Mitochondrial DNA | Atrophy | Neurology | Vitamin E | Vitamin B | Nitrous oxide | Adults | Paralysis | Mutation | Copper | Homocysteine | Metabolic disorders | Life Sciences
Journal Article
1993, ISBN 3805555814, Volume 17., xi, 189
Book
by Monies, Dorota and Abouelhoda, Mohamed and AlSayed, Moeenaldeen and Alhassnan, Zuhair and Alotaibi, Maha and Kayyali, Husam and Al-Owain, Mohammed and Shah, Ayaz and Rahbeeni, Zuhair and Al-Muhaizea, Mohammad A and Alzaidan, Hamad I and Cupler, Edward and Bohlega, Saeed and Faqeih, Eissa and Faden, Maha and Alyounes, Banan and Jaroudi, Dyala and Goljan, Ewa and Elbardisy, Hadeel and Akilan, Asma and Albar, Renad and Aldhalaan, Hesham and Gulab, Shamshad and Chedrawi, Aziza and Al Saud, Bandar K and Kurdi, Wesam and Makhseed, Nawal and Alqasim, Tahani and El Khashab, Heba Y and Al-Mousa, Hamoud and Alhashem, Amal and Kanaan, Imaduddin and Algoufi, Talal and Alsaleem, Khalid and Basha, Talal A and Al-Murshedi, Fathiya and Khan, Sameena and Al-Kindy, Adila and Alnemer, Maha and Al-Hajjar, Sami and Alyamani, Suad and Aldhekri, Hasan and Al-Mehaidib, Ali and Arnaout, Rand and Dabbagh, Omar and Shagrani, Mohammad and Broering, Dieter and Tulbah, Maha and Alqassmi, Amal and Almugbel, Maisoon and AlQuaiz, Mohammed and Alsaman, Abdulaziz and Al-Thihli, Khalid and Sulaiman, Raashda A and Al-Dekhail, Wajeeh and Alsaegh, Abeer and Bashiri, Fahad A and Qari, Alya and Alhomadi, Suzan and Alkuraya, Hisham and Alsebayel, Mohammed and Hamad, Muddathir H and Szonyi, Laszlo and Abaalkhail, Faisal and Al-Mayouf, Sulaiman M and Almojalli, Hamad and Alqadi, Khalid S and Elsiesy, Hussien and Shuaib, Taghreed M and Seidahmed, Mohammed Zain and Abosoudah, Ibraheem and Akleh, Hana and AlGhonaium, Abdulaziz and Alkharfy, Turki M and Al Mutairi, Fuad and Eyaid, Wafa and Alshanbary, Abdullah and Sheikh, Farrukh R and Alsohaibani, Fahad I and Alsonbul, Abdullah and Al Tala, Saeed and Balkhy, Soher and Bassiouni, Randa and Alenizi, Ahmed S and Hussein, Maged H and Hassan, Saeed and Khalil, Mohamed and Tabarki, Brahim and Alshahwan, Saad and Oshi, Amira and Sabr, Yasser and Alsaadoun, Saad and Salih, Mustafa A and Mohamed, Sarar and Sultana, Habiba and Tamim, Abdullah and El-Haj, Moayad and Alshahrani, Saif and Bubshait, Dalal K and Alfadhel, Majid and ...
Human Genetics, ISSN 0340-6717, 8/2017, Volume 136, Issue 8, pp. 921 - 939
Journal Article
Nature Biotechnology, ISSN 1087-0156, 01/2012, Volume 30, Issue 2, pp. 159 - 64
Journal Article
The New England Journal of Medicine, ISSN 0028-4793, 07/2014, Volume 371, Issue 1, pp. 42 - 49
In this cohort study involving 6403 children at increased risk for celiac disease on the basis of HLA haplotype, the risks of celiac disease autoimmunity and... 
ANTIBODIES | MEDICINE, GENERAL & INTERNAL | GLUTEN | AUTOIMMUNITY | GENE DOSAGE | SUSCEPTIBILITY | TEDDY | CHILDHOOD | CHILDREN | Autoimmune Diseases - epidemiology | United States - epidemiology | Genetic Predisposition to Disease | Prospective Studies | Celiac Disease - genetics | Humans | Kaplan-Meier Estimate | Child, Preschool | Europe - epidemiology | Infant | Male | Risk | Transglutaminases - immunology | Autoimmune Diseases - genetics | Homozygote | HLA-DR4 Antigen - genetics | HLA-DR3 Antigen - genetics | HLA-DQ Antigens - genetics | Antibodies - blood | Female | Celiac Disease - epidemiology | Infant, Newborn | Haplotypes | Viral antibodies | Celiac disease | Histocompatibility antigens | Analysis | HLA histocompatibility antigens | Antibodies | Children | Health aspects | Risk factors | Diabetes mellitus | Heterozygotes | Environmental factors | Small intestine | Transglutaminase 2 | Children & youth | Homozygotes | Genotype & phenotype | Biopsy | Histocompatibility antigen HLA | Autoimmune diseases | Age | Genotypes | Immune system | Index Medicus | Abridged Index Medicus | Autoimmune Diseases: epidemiology | Celiac Disease: genetics | Medical and Health Sciences | Medicin och hälsovetenskap | Europe: epidemiology | Antibodies: blood | HLA-DQ Antigens: genetics | HLA-DR3 Antigen: genetics | Klinisk medicin | Transglutaminases: immunology | United States: epidemiology | HLA-DR4 Antigen: genetics | Clinical Medicine | Endokrinologi och diabetes | Autoimmune Diseases: genetics | Celiac Disease: epidemiology | Endocrinology and Diabetes
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2018, Volume 13, Issue 1, pp. e0191319 - e0191319
The Pediatric Cardiac Genomics Consortium (PCGC) designed the Congenital Heart Disease Genetic Network Study to provide phenotype and genotype data for a large... 
UNITED-STATES | METROPOLITAN ATLANTA | DE-NOVO MUTATIONS | MULTIDISCIPLINARY SCIENCES | SEX | BIRTH-DEFECTS | PREVALENCE | OUTCOMES | INFANT | MATERNAL SMOKING | CHILDREN | Congenital heart disease | Research | Children | Health aspects | Index Medicus
Journal Article
Journal Article
Molecular Systems Biology, ISSN 1744-4292, 2011, Volume 7, Issue 1, pp. 494 - n/a
Proteins targeting the same subcellular localization tend to participate in mutual protein–protein interactions (PPIs) and are often functionally associated.... 
comorbidity | human disease | subcellular localization | cellular networks | BIOCHEMISTRY & MOLECULAR BIOLOGY | MOUSE | DISORDERS | IDENTIFICATION | INTERACTOME | INTERACTION NETWORK | PHENOTYPE NETWORK | CONSTRUCTION | MUTATIONS | EXPRESSION | MAP | Disease - genetics | Comorbidity | Humans | Molecular Epidemiology - methods | Data Collection - methods | Cell Compartmentation | Disease - etiology | Proteins - genetics | Systems Biology - methods | Disease - classification | Algorithms | Proteins - metabolism | Databases, Protein | Subcellular Fractions - chemistry | Cluster Analysis | Health care | Enrichment | Cluster analysis | Modules | Genes | Correlation coefficients | Spatial discrimination | Data bases | Risk factors | Proteins | Mitochondria | Databases | Pathways | Mathematical analysis | Etiology | Compartments | Risk assessment | Genetics | Localization | Protein transport | Alzheimer's disease | Correlation coefficient | Translocation | Progressions | Integration | Phenotypes | Neurodegenerative diseases | Angiotensin I | Diabetes mellitus | Health risks | Metabolism | Clustering | Organelles | Patients | Molecular chains | Golgi apparatus | Diseases | Government programs | Proteomics | Angiotensin | Metabolic pathways | Mutation | Position (location) | Alzheimers disease | Protein interaction | Endoplasmic reticulum | Index Medicus
Journal Article
Cell Metabolism, ISSN 1550-4131, 05/2012, Volume 15, Issue 5, pp. 665 - 674
Nonalcoholic fatty liver disease (NAFLD) is associated with increased cardiovascular and liver-related mortality. NAFLD is characterized by both triglyceride... 
RAT-LIVER | RISK-FACTORS | MACROPHAGE APOPTOSIS | PROTEIN-KINASE | ENDOCRINOLOGY & METABOLISM | HMG-COA REDUCTASE | STEATOHEPATITIS | 3-HYDROXY-3-METHYLGLUTARYL COENZYME | PREVALENCE | MICRORNA EXPRESSION | MODULATION | CELL BIOLOGY | Sirtuin 1 - metabolism | Up-Regulation | Cholesterol - blood | Humans | Middle Aged | Sterol Esterase - metabolism | Male | MicroRNAs - metabolism | Desmosterol - metabolism | Cardiovascular Diseases - genetics | Cholesterol - genetics | Sirtuin 1 - genetics | Case-Control Studies | Phosphorylation - genetics | Hydroxymethylglutaryl CoA Reductases - metabolism | Adenylate Kinase - metabolism | Non-alcoholic Fatty Liver Disease | Sterol O-Acyltransferase - metabolism | Adult | Female | Lipid Metabolism - genetics | Sterol Regulatory Element Binding Protein 2 - genetics | Sterol Regulatory Element Binding Protein 2 - metabolism | Fatty Liver - genetics | Receptors, LDL - genetics | Gene Expression | Fatty Liver - metabolism | Cardiovascular Diseases - metabolism | Fatty Liver - blood | Liver - metabolism | Receptors, LDL - metabolism | Cholesterol - metabolism | Cholesterol, LDL - genetics | Phenotype | Sterol Esterase - genetics | Desmosterol - blood | Cholesterol, LDL - metabolism | MicroRNAs - genetics | Sterol O-Acyltransferase - genetics | Adenylate Kinase - genetics | Hydroxymethylglutaryl CoA Reductases - genetics | Enzymes | Liver diseases | Low density lipoproteins | Genes | Esters | Triglycerides | Cholesterol | MicroRNA | Fatty liver | Blood cholesterol | Physiological aspects | Hydrolases | Blood lipids |