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Journal Article
Journal Article
Cancer letters, ISSN 0304-3835, 2009, Volume 284, Issue 2, pp. 208 - 215
Abstract Arsenic trioxide (ATO) strongly induces apoptosis in acute promyelocytic leukemia (APL), but it induces cell cycle arrest in most solid tumors. In... 
Hematology, Oncology and Palliative Medicine | The Cbl proteins | ATO | G2/M arrest | Apoptosis | p53 | PI3K/Akt | Life Sciences & Biomedicine | Oncology | Science & Technology | Adenocarcinoma - pathology | Leukemia, Promyelocytic, Acute - pathology | Apoptosis - drug effects | Humans | Neoplasm Proteins - physiology | Stomach Neoplasms - metabolism | Stomach Neoplasms - pathology | Cell Line, Tumor - drug effects | Metaphase - drug effects | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | G2 Phase - drug effects | Neoplasm Proteins - drug effects | Tumor Suppressor Protein p53 - physiology | Leukemia, Promyelocytic, Acute - metabolism | Adenocarcinoma - metabolism | Ubiquitination - drug effects | Protein Processing, Post-Translational - drug effects | Proto-Oncogene Proteins c-cbl - drug effects | Antineoplastic Agents - pharmacology | Gene Expression Regulation, Neoplastic - drug effects | Cell Line, Tumor - metabolism | G2 Phase - physiology | Arsenicals - pharmacology | Gene Expression Regulation, Leukemic - drug effects | Proto-Oncogene Proteins c-akt - physiology | Proto-Oncogene Proteins c-cbl - physiology | Up-Regulation - drug effects | Oxides - pharmacology | Metaphase - physiology | Signal Transduction - drug effects | Phosphatidylinositol 3-Kinases - physiology | Signal Transduction - physiology | Apoptosis - physiology | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Research | Arsenic | Tumor proteins | Oncology, Experimental | Cancer | Index Medicus
Journal Article
PloS one, ISSN 1932-6203, 03/2015, Volume 10, Issue 3, pp. e0122370 - e0122370
Journal Article
PloS one, ISSN 1932-6203, 07/2016, Volume 11, Issue 7, pp. e0159341 - e0159341
.... However, the genotoxic effects on normal human cells when agents including PARPi disturb proficient cellular repair processes have not been... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | DNA Damage - drug effects | DNA Repair - drug effects | Gene Expression | Humans | Homologous Recombination | Poly(ADP-ribose) Polymerase Inhibitors - adverse effects | Cisplatin - pharmacology | Piperazines - adverse effects | Dose-Response Relationship, Drug | Drug Synergism | Poly(ADP-ribose) Polymerases - metabolism | Sister Chromatid Exchange - drug effects | Poly(ADP-ribose) Polymerases - genetics | Chromosome Aberrations - drug effects | Karyotyping | Genomic Instability - drug effects | Female | Benzimidazoles - adverse effects | Phthalazines - adverse effects | Care and treatment | DNA damage | Analysis | Risk factors | Ovarian cancer | Cancer | Toxicity | Epithelial cells | Sister chromatid exchange | Homologous recombination | Genotoxicity | Poly(ADP-ribose) | Clinical trials | Cytotoxicity | Homology | Kinases | DNA repair | Genomic instability | Proteins | Ribose | Rodents | Ribosylation | Repair | Deoxyribonucleic acid--DNA | ADP-ribosylation | BRCA2 protein | Medical research | Lymphoid cells | Stability | BRCA1 protein | Hypersensitivity | Poly(ADP-ribose) polymerase | Adenosine diphosphate | Breast cancer | Double-strand break repair | Cisplatin | Inhibitors | Mutation | Aberration | Prostate cancer | Prostate | Tumors | Index Medicus | Deoxyribonucleic acid | DNA
Journal Article
Molecular Cancer Therapeutics, ISSN 1535-7163, 04/2007, Volume 6, Issue 4, pp. 1317 - 1328
Journal Article