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Journal Article
Journal of Digestive Diseases, ISSN 1751-2972, 09/2017, Volume 18, Issue 9, pp. 504 - 510
OBJECTIVEEvaluations of upper gastrointestinal toxicity from ketamine abuse are uncommon. This study investigated the clinical pattern of upper... 
gastrointestinal diseases | stomach | substance‐related disorders | ketamine | upper gastrointestinal tract | substance-related disorders | COMMON | GASTROENTEROLOGY & HEPATOLOGY | HONG-KONG | ABUSERS | Prevalence | Intestine, Small - pathology | Anemia - epidemiology | Gastrointestinal Diseases - epidemiology | Melena - chemically induced | Humans | Middle Aged | Esophagitis - chemically induced | Metaplasia - chemically induced | Duodenal Ulcer - chemically induced | Male | Case-Control Studies | Ketamine - administration & dosage | Young Adult | Urologic Diseases - epidemiology | Ketamine - adverse effects | Metaplasia - epidemiology | Duodenal Ulcer - epidemiology | Adult | Female | Hematemesis - chemically induced | Retrospective Studies | Analgesics - adverse effects | Stomach Ulcer - chemically induced | Abdominal Pain - chemically induced | Cross-Sectional Studies | Administration, Inhalation | Analgesics - administration & dosage | Anemia - chemically induced | Endoscopy, Gastrointestinal | Melena - epidemiology | Hematemesis - epidemiology | Substance-Related Disorders - complications | Abdominal Pain - epidemiology | Gastritis - epidemiology | Vomiting - chemically induced | Gastritis - chemically induced | Stomach Ulcer - epidemiology | Esophagitis - epidemiology | Adolescent | Urologic Diseases - chemically induced | Vomiting - epidemiology | Chronic Disease | Gastrointestinal Diseases - chemically induced | Ketamine | Pain | Abuse | Anemia | Vomiting | Toxicity | Index Medicus
Journal Article
International Journal of Cancer, ISSN 0020-7136, 09/2016, Volume 139, Issue 5, pp. 1171 - 1181
Journal Article
American Journal of Hematology, ISSN 0361-8609, 05/2012, Volume 87, Issue 5, pp. 552 - 554
Journal Article
Journal Article
Journal of Clinical Oncology, ISSN 0732-183X, 10/2009, Volume 27, Issue 28, pp. 4760 - 4766
Journal Article
Journal Article
Gastroenterology, ISSN 0016-5085, 2015, Volume 148, Issue 5, pp. 1024 - 1034.e9
Background & Aims Oncogenic mutations in KRAS contribute to the development of pancreatic ductal adenocarcinoma, but are not sufficient to initiate... 
Gastroenterology and Hepatology | Gene Regulation | Signal Transduction | ChIP | Mouse Model | CANCER-CELLS | ONCOGENIC KRAS | C-MYC | FACTOR RECEPTOR | K-RAS | INFLAMMATION | EPIDERMAL-GROWTH-FACTOR | MICE | GASTROENTEROLOGY & HEPATOLOGY | EXPRESSION | PROGRESSION | Receptor, Epidermal Growth Factor - genetics | Carcinoma, Pancreatic Ductal - chemically induced | Pancreatic Neoplasms - metabolism | Proto-Oncogene Proteins p21(ras) - genetics | Humans | Transcriptional Activation | Carcinoma, Pancreatic Ductal - metabolism | Pancreas, Exocrine - pathology | Male | Precancerous Conditions - metabolism | Pancreatic Ducts - pathology | Cyclosporine | Pancreatic Ducts - metabolism | Carcinoma, Pancreatic Ductal - genetics | Pancreatitis - genetics | Receptor, Epidermal Growth Factor - metabolism | Cell Transformation, Neoplastic - genetics | Cell Transdifferentiation | Pancreatic Neoplasms - chemically induced | Precancerous Conditions - pathology | Disease Models, Animal | SOX9 Transcription Factor - metabolism | Cell Line | Tissue Culture Techniques | Mice, Inbred C57BL | NFATC Transcription Factors - metabolism | Pancreatic Neoplasms - pathology | Gene Expression Regulation | Pancreatic Neoplasms - genetics | Pancreatitis - chemically induced | Carcinoma, Pancreatic Ductal - pathology | Cell Transformation, Neoplastic - metabolism | Precancerous Conditions - genetics | Mice, Knockout | Metaplasia | Animals | Pancreatitis - pathology | Pancreas, Exocrine - metabolism | Mutation | Pancreatitis - metabolism | Precancerous Conditions - chemically induced | Cell Transformation, Neoplastic - pathology | Ceruletide | NFATC Transcription Factors - deficiency | SOX9 Transcription Factor - genetics | NFATC Transcription Factors - genetics | Genetic transcription | Index Medicus | Abridged Index Medicus | NFATc1 | mouse model | signal transduction | transcriptional regulation | EGFR
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2013, Volume 8, Issue 11, pp. e79182 - e79182
There are few mouse models that adequately mimic large bowel cancer in humans or the gastrointestinal inflammation which frequently precedes it. Dextran sodium... 
GALECTIN-3 EXPRESSION | MUTANT MICE | CARCINOGENESIS | EXPERIMENTAL COLITIS | NEOPLASTIC PROGRESSION | COLORECTAL-CANCER | MULTIDISCIPLINARY SCIENCES | INTESTINAL CANCER | MOUSE MODELS | BOWEL-DISEASE | LYMPHOCYTES | Inflammation - chemically induced | Inflammation - pathology | Colitis - genetics | Colorectal Neoplasms - genetics | Humans | Colitis - pathology | Smad3 Protein - metabolism | DNA-Binding Proteins - genetics | Colorectal Neoplasms - chemically induced | Dextran Sulfate - toxicity | Mice, Knockout | DNA-Binding Proteins - metabolism | Smad3 Protein - genetics | Inflammation - metabolism | Animals | Transforming Growth Factor beta - genetics | Colitis - chemically induced | Colitis - metabolism | Inflammation - genetics | Mice | Colorectal Neoplasms - pathology | Transforming Growth Factor beta - metabolism | Colorectal Neoplasms - metabolism | Adenocarcinoma | Animal models | Transforming growth factor-b | Colorectal carcinoma | Colorectal cancer | Viruses | Smad3 protein | Sodium sulfates | Signal transduction | Carcinogens | Cell growth | Colon cancer | Azoxymethane | Lymphocytes | Intestine | Rodents | Sodium sulfate | Tumorigenesis | Colon | Lesions | Medical research | Phenotypes | Dysplasia | Inflammation | Disease control | Heterozygosity | Medicine | Inflammatory bowel disease | Dextran | Pathology | Metaplasia | Signaling | RAG2 protein | Lymphocytes B | Sodium | Colitis | Mutation | Sulfate | Laboratory animals | Polyps | Peritoneum | Tumors | Cancer | Index Medicus
Journal Article
Journal Article
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 04/2014, Volume 50, Issue 4, pp. 690 - 698
Journal Article