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PLoS ONE, ISSN 1932-6203, 03/2013, Volume 8, Issue 3, pp. e57917 - e57917
Journal Article
Brain Pathology, ISSN 1015-6305, 09/2016, Volume 26, Issue 5, pp. 569 - 580
Somatic mutations of the H3F3A and HIST1H3B genes encoding the histone H3 variants, H3.3 and H3.1, were recently identified in high‐grade gliomas arising in... 
glioblastoma | astrocytoma | diffuse midline glioma | H3F3A | K27M mutation | histone H3.3 | DIPG | histone H3.1 | HIST1H3B | diffuse intrinsic pontine glioma | Diffuse intrinsic pontine glioma | Glioblastoma | Histone H3.1 | Diffuse midline glioma | Histone H3.3 | Astrocytoma | histone H3 | INTRINSIC PONTINE GLIOMA | ACTIVATING ACVR1 MUTATIONS | MOLECULAR CHARACTERISTICS | SUBGROUPS | PATHOLOGY | BRAIN-STEM GLIOMAS | PEDIATRIC GLIOBLASTOMA | NEUROSCIENCES | CLINICAL NEUROLOGY | GLIONEURONAL TUMORS | H3F3A K27M MUTATIONS | HIGH-FREQUENCY | HIGH-GRADE ASTROCYTOMAS | Receptor, Epidermal Growth Factor - genetics | Humans | Middle Aged | Brain Neoplasms - pathology | Child, Preschool | Male | X-linked Nuclear Protein - metabolism | Brain Neoplasms - metabolism | Tumor Suppressor Protein p53 - genetics | Glioma - metabolism | Glioma - genetics | Young Adult | Receptor, Epidermal Growth Factor - metabolism | Glioma - pathology | Adult | Female | Child | Gene Expression Regulation, Neoplastic - genetics | Glycine - genetics | PTEN Phosphohydrolase - genetics | Genetic Association Studies | Tumor Suppressor Protein p53 - metabolism | Brain Neoplasms - genetics | PTEN Phosphohydrolase - metabolism | Isocitrate Dehydrogenase - genetics | Mutation - genetics | Histones - genetics | Adolescent | Isocitrate Dehydrogenase - metabolism | Aged | Methionine - genetics | Tumor proteins | Gliomas | Genetic aspects | Spinal cord | Mutation | Rodents | Tumors | Index Medicus
Journal Article
BMC Genomics, ISSN 1471-2164, 03/2012, Volume 13, Issue 1, pp. 101 - 101
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 6, pp. e21102 - e21102
Background: One-carbon metabolism appears to play an important role in DNA methylation reaction. Evidence suggests that a low intake of B vitamins or high... 
POPULATION-BASED SAMPLE | DNA METHYLATION | METHYLENETETRAHYDROFOLATE REDUCTASE POLYMORPHISM | MICROSATELLITE INSTABILITY | MULTIDISCIPLINARY SCIENCES | SPORADIC COLORECTAL-CANCER | MGMT PROMOTER METHYLATION | MULTIVITAMIN USE | COMPETING RISKS | DIETARY-FOLATE | ONE-CARBON METABOLISM | MutL Protein Homolog 1 | Colonic Neoplasms - genetics | Folic Acid - pharmacology | Humans | Middle Aged | Suppressor of Cytokine Signaling 1 Protein | Methionine - pharmacology | Folic Acid - administration & dosage | Receptors, Retinoic Acid - genetics | Poly-ADP-Ribose Binding Proteins | Insulin-Like Growth Factor II - genetics | Polymerase Chain Reaction | Cell Cycle Proteins - genetics | Adult | Female | Surveys and Questionnaires | Calcium Channels, T-Type - genetics | Neoplasm Proteins - genetics | Nuclear Proteins - genetics | Basic Helix-Loop-Helix Transcription Factors - genetics | Risk Factors | Colonic Neoplasms - epidemiology | Cyclin-Dependent Kinase Inhibitor p16 - genetics | Suppressor of Cytokine Signaling Proteins - genetics | DNA Methylation - genetics | Methionine - administration & dosage | Nerve Tissue Proteins - genetics | Ubiquitin-Protein Ligases | Proto-Oncogene Proteins B-raf - genetics | Adaptor Proteins, Signal Transducing - genetics | Core Binding Factor Alpha 3 Subunit - genetics | CpG Islands - genetics | Mutation | DNA Methylation - drug effects | Kruppel-Like Transcription Factors - genetics | Oncology, Experimental | Amino acids | Drinking of alcoholic beverages | Research | Epidemiology | Risk factors | Folic acid | Colon cancer | Gene mutations | Gastrointestinal diseases | Nurses | Physiological aspects | Genetic aspects | Methylation | Cancer | Colorectal carcinoma | Colorectal cancer | Alcohol | DNA methylation | Insulin-like growth factor-binding protein 3 | Nutrients | Colon | Insulin-like growth factor II | Deoxyribonucleic acid--DNA | CpG islands | Medical personnel | Statistical analysis | MLH1 protein | Health risks | Methionine | Metabolism | Carbon | Low level | Vitamins | Alcoholic beverages | Health risk assessment | Vitamin B6 | Runx3 protein | Tumors | Index Medicus | Risk | Deoxyribonucleic acid | DNA
Journal Article
PLoS Biology, ISSN 1544-9173, 03/2012, Volume 10, Issue 3, pp. e1001288 - e1001288
An increasing number of genes required for mitochondrial biogenesis, dynamics, or function have been found to be mutated in metabolic disorders and... 
RETINAL DEGENERATION | DNA DEPLETION | DROSOPHILA-PARKIN MUTANTS | UNFOLDED PROTEIN RESPONSE | PINK1/PARKIN PATHWAY | GENE | FRIEDREICHS-ATAXIA | COPY-NUMBER | GENOMIC HYBRIDIZATION-ARRAY | BIOCHEMISTRY & MOLECULAR BIOLOGY | DISEASE | BIOLOGY | Mitochondria - enzymology | Electron Transport | Electroretinography - methods | Leukoencephalopathies - genetics | Retina - metabolism | Cell Proliferation | Reactive Oxygen Species - metabolism | Humans | Middle Aged | Muscles - physiopathology | Child, Preschool | Male | Mitochondrial Proteins - genetics | Drosophila Proteins - metabolism | Drosophila - physiology | Drosophila - enzymology | Young Adult | Mitochondria - genetics | Mitochondrial Proteins - metabolism | HEK293 Cells | Adult | Female | Methionine-tRNA Ligase - metabolism | Photoreceptor Cells - pathology | Ataxia - genetics | Muscles - metabolism | Ataxia - metabolism | Child | Drosophila - genetics | Oxidative Phosphorylation | Neurodegenerative Diseases - genetics | Leukoencephalopathies - metabolism | Longevity | Neurodegenerative Diseases - metabolism | Unfolded Protein Response | Gene Expression Regulation, Enzymologic | Phenotype | Animals | Pedigree | Photoreceptor Cells - metabolism | Adolescent | Drosophila Proteins - genetics | Mutation | Methionine-tRNA Ligase - genetics | Retina - pathology | Proteins | Medical research | Genetics | Neurodegeneration | Genes | Index Medicus
Journal Article
Science, ISSN 0036-8075, 8/2012, Volume 337, Issue 6098, pp. 1081 - 1084
Identification of the causal genes that control complex trait variation remains challenging, limiting our appreciation of the evolutionary processes that... 
Enzymes | Genetic variation | Ecological competition | REPORTS | Alleles | Glucosinolates | Genetic loci | Herbivores | Infestation | Genotypes | Phenotypic traits | CYTOCHROME-P450 | CYP79F1 | BIOSYNTHESIS | CYP83B1 | MULTIDISCIPLINARY SCIENCES | BOECHERA-STRICTA | PLANTS | ARABIDOPSIS | ECOLOGICAL GENOMICS | Herbivory - physiology | Molecular Sequence Data | Gene-Environment Interaction | Glucosinolates - genetics | Amino Acid Sequence | Plant Leaves - parasitology | Plants, Genetically Modified - genetics | Quantitative Trait, Heritable | Methionine - metabolism | Selection, Genetic | Gene Dosage | Brassicaceae - metabolism | Arabidopsis - metabolism | Polymorphism, Genetic | Arabidopsis - genetics | Brassicaceae - genetics | Glucosinolates - biosynthesis | Plants, Genetically Modified - metabolism | Plant Leaves - genetics | Plant Leaves - metabolism | Plants, Genetically Modified - parasitology | Cytochrome P-450 Enzyme System - genetics | Arabidopsis - parasitology | Brassicaceae - parasitology | Quantitative Trait Loci | Methionine - genetics | Amino Acid Substitution | Quantitative genetics | Research | Phytochemistry | Genetic polymorphisms | Plant genetics | Plant biology | Biochemistry | Defense mechanisms | Evolutionary biology | Molecular biology | Polymorphism | Index Medicus | cyp79f1 | cyp83b1 | ecological genomics | cytochrome-p450 | plants | biosynthesis | boechera-stricta | arabidopsis
Journal Article
The American Journal of Human Genetics, ISSN 0002-9297, 01/2015, Volume 96, Issue 1, pp. 170 - 177
Journal Article
Journal of Oral Pathology & Medicine, ISSN 0904-2512, 02/2013, Volume 42, Issue 2, pp. 180 - 185
J Oral Pathol Med (2013) 42 : 180–185 Background:  The capacity for DNA repair is essential in maintaining cellular functions and homeostasis; however, this... 
XRCC1 | histopathological parameters | polymorphisms | oral cancer | XRCC3 | Oral cancer | Polymorphisms | Histopathological parameters | HEAD | STRAND BREAK REPAIR | RISK | PATHOLOGY | XPD | NECK-CANCER | ACID SUBSTITUTION VARIANTS | DENTISTRY, ORAL SURGERY & MEDICINE | SQUAMOUS-CELL CARCINOMA | DNA-REPAIR | ASSOCIATION | SINGLE-NUCLEOTIDE POLYMORPHISMS | Gene Frequency - genetics | Carcinoma, Squamous Cell - genetics | Carcinoma, Squamous Cell - pathology | Humans | Middle Aged | Male | DNA Repair - genetics | Case-Control Studies | Young Adult | Arginine - genetics | Threonine - genetics | Aged, 80 and over | Adult | Female | Codon - genetics | Mouth Neoplasms - genetics | Genetic Predisposition to Disease - genetics | Risk Factors | Tryptophan - genetics | Genotype | DNA-Binding Proteins - genetics | Alleles | Glutamine - genetics | Lymphatic Metastasis - genetics | Mouth Neoplasms - pathology | Polymorphism, Single Nucleotide - genetics | Polymorphism, Genetic - genetics | Aged | Genetic Variation - genetics | X-ray Repair Cross Complementing Protein 1 | Methionine - genetics | Squamous cell carcinoma | Oncology, Experimental | Genes | Disease susceptibility | Research | Cancer patients | Anopheles | Codon | Analysis | DNA | Genetic research | Genetic aspects | Mouth cancer | Cancer | Index Medicus | Dentistry | Codons
Journal Article
Cancer Epidemiology Biomarkers & Prevention, ISSN 1055-9965, 11/2009, Volume 18, Issue 11, pp. 3086 - 3096
Journal Article
Oncogene, ISSN 0950-9232, 07/2007, Volume 26, Issue 32, pp. 4635 - 4647
Telomerase-negative cancer cells can maintain their telomeres by a recombination-mediated alternative lengthening of telomeres (ALT) process. We reported... 
Telomeres | siRNA | Alternative lengthening of telomeres | Methionine restriction | PML | ALT-associated PML bodies | CANCER-CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | telomeres | DNA-DAMAGE | MAMMALIAN-CELLS | PROTEIN COMPLEX | CELL BIOLOGY | HUMAN RAP1 | METABOLIC DEFECT | LARGE-BODY FORMATION | NUCLEAR-BODIES | ONCOLOGY | alternative lengthening of telomeres | methionine restriction | GENETICS & HEREDITY | PROMYELOCYTIC LEUKEMIA BODIES | TUMOR-CELL-LINES | Humans | Neoplasm Proteins - antagonists & inhibitors | Resting Phase, Cell Cycle | Autoantigens - genetics | RNA Interference | Neoplasm Proteins - genetics | Intracellular Signaling Peptides and Proteins - genetics | Telomeric Repeat Binding Protein 2 - antagonists & inhibitors | DNA Repair Enzymes - physiology | DNA-Binding Proteins - physiology | DNA-Binding Proteins - antagonists & inhibitors | Telomeric Repeat Binding Protein 2 - physiology | Genetic Techniques | Autoantigens - physiology | Tumor Suppressor Proteins - physiology | G1 Phase | Cell Line, Tumor | Intracellular Signaling Peptides and Proteins - physiology | Cell Cycle Proteins - physiology | Telomeric Repeat Binding Protein 1 - physiology | Tumor Suppressor Proteins - antagonists & inhibitors | Neoplasm Proteins - physiology | DNA Repair Enzymes - genetics | Telomeric Repeat Binding Protein 1 - genetics | Cell Cycle Proteins - antagonists & inhibitors | MRE11 Homologue Protein | Telomere-Binding Proteins - genetics | Tumor Suppressor Proteins - genetics | Telomere-Binding Proteins - physiology | Cell Cycle Proteins - genetics | Telomere - metabolism | DNA Repair Enzymes - antagonists & inhibitors | Nuclear Proteins - genetics | Methionine - deficiency | Telomere - genetics | Transcription Factors - physiology | Intracellular Signaling Peptides and Proteins - antagonists & inhibitors | RNA, Small Interfering - pharmacology | Antigens, Nuclear - physiology | Transcription Factors - antagonists & inhibitors | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Telomere-Binding Proteins - antagonists & inhibitors | Antigens, Nuclear - genetics | Nuclear Proteins - antagonists & inhibitors | Telomeric Repeat Binding Protein 1 - antagonists & inhibitors | Cell Proliferation - drug effects | Nuclear Proteins - physiology | Telomeric Repeat Binding Protein 2 - genetics | Tumor Suppressor p53-Binding Protein 1 | Promyelocytic Leukemia Protein | Physiological aspects | Genetic aspects | Research | Cancer cells | Proteins | Ribonucleic acid | Cell cycle | Genetics | Oncology | Ribonucleic acid--RNA | DNA repair | Cancer | Index Medicus
Journal Article