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Circulation Research, ISSN 0009-7330, 12/2009, Volume 105, Issue 12, pp. 1204 - 1212
RATIONALE:Angiotensin (Ang) II–induced apoptosis was reported to be mediated by different signaling molecules. Whether these molecules are either... 
Reactive oxygen species | CaMKII | Angiotensin II | Apoptosis | CARDIAC & CARDIOVASCULAR SYSTEMS | P38 MAPK | INHIBITION PROTECTS | apoptosis | VENTRICULAR MYOCYTES | CELL-DEATH | PHOSPHOLAMBAN PHOSPHORYLATION | HEART-DISEASE | METHIONINE OXIDATION | angiotensin II | reactive oxygen species | PERIPHERAL VASCULAR DISEASE | INDEPENDENT ACTIVATION | HEMATOLOGY | Free Radical Scavengers - pharmacology | Tiopronin - pharmacology | Reactive Oxygen Species - metabolism | Rats, Wistar | Species Specificity | Apoptosis - drug effects | Calcium - metabolism | NADPH Oxidases - metabolism | Peptides - genetics | Myocytes, Cardiac - enzymology | Time Factors | Egtazic Acid - analogs & derivatives | p38 Mitogen-Activated Protein Kinases - metabolism | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - metabolism | Cats | Calmodulin - metabolism | Angiotensin II - metabolism | Cell Survival | NADPH Oxidases - antagonists & inhibitors | Cells, Cultured | Enzyme Inhibitors - pharmacology | Rats | Mice, Transgenic | Imidazoles - pharmacology | Chelating Agents - pharmacology | Onium Compounds - pharmacology | Sulfonamides - pharmacology | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - antagonists & inhibitors | Peptides - pharmacology | Myocytes, Cardiac - pathology | Animals | Egtazic Acid - pharmacology | Myocytes, Cardiac - drug effects | Signal Transduction - drug effects | Mice | Protein Kinase Inhibitors - pharmacology | Pyridines - pharmacology | Enzyme Activation | Oxidative Stress - drug effects | Benzylamines - pharmacology | Calmodulin - antagonists & inhibitors
Journal Article
Apoptosis, ISSN 1360-8185, 4/2013, Volume 18, Issue 4, pp. 393 - 408
Journal Article
Journal Article
BRITISH JOURNAL OF PHARMACOLOGY, ISSN 0007-1188, 01/2015, Volume 172, Issue 2, pp. 375 - 387
Several studies suggest that heteromerization between (MOP) and (DOP) opioid receptors modulates the signalling properties of the individual receptors. For... 
METHIONINE ENKEPHALIN | antinociception | heteromer | opioid | high-throughput screening | PROTEIN-COUPLED RECEPTORS | SPINAL-CORD | NEUROBLASTOMA-CELL-LINE | MICE LACKING | CONCISE GUIDE | BIVALENT LIGANDS | dependence | PHARMACOLOGY & PHARMACY | trafficking | signalling | tolerance | MORPHINE-INDUCED ANALGESIA | IRRITABLE-BOWEL-SYNDROME | OPIATE RECEPTORS
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 01/2006, Volume 281, Issue 4, pp. 2012 - 2023
Journal Article
FASEB Journal, ISSN 0892-6638, 12/2012, Volume 26, Issue 12, pp. 5161 - 5171
Inflammatory pain can be controlled by endogenous opioid peptides. Here we blocked the degradation of opioids in peripheral injured tissue to locally augment... 
Immune cells | Analgesia | Ory nerves | DEGRADING ENZYMES | NEUROGENIC INFLAMMATION | SYNOVIAL-FLUID | CATABOLIZING ENZYMES | BIOCHEMISTRY & MOLECULAR BIOLOGY | analgesia | immune cells | SPINAL-CORD | BETA-ENDORPHIN | sensory nerves | CELL BIOLOGY | RECEPTORS MEDIATING ANTINOCICEPTION | BIOLOGY | INFLAMMATORY PAIN | RAT-BRAIN | NEUTRAL ENDOPEPTIDASE ENKEPHALINASE | Leucine - pharmacology | Opioid Peptides - pharmacology | Rats, Wistar | Thiorphan - pharmacology | Opioid Peptides - immunology | Male | Enkephalin, Methionine - pharmacology | Leukocytes - enzymology | Enkephalin, Methionine - metabolism | Dose-Response Relationship, Drug | Narcotic Antagonists | Alanine - analogs & derivatives | Enkephalin, Methionine - immunology | Hindlimb - physiopathology | Inflammation - complications | Flow Cytometry | Pain - complications | Pain - enzymology | Enkephalin, Leucine - pharmacology | Antibodies - immunology | Pain Threshold - drug effects | Dynorphins - metabolism | Neurons - drug effects | Leucine - analogs & derivatives | Alanine - pharmacology | Receptors, Opioid - metabolism | Amino Acid Sequence | Enkephalin, Leucine - metabolism | Pain - prevention & control | CD13 Antigens - antagonists & inhibitors | Enzyme Inhibitors - pharmacology | Rats | Hindlimb - innervation | Enkephalin, Leucine - immunology | Neprilysin - metabolism | CD13 Antigens - metabolism | Opioid Peptides - metabolism | Antibodies - pharmacology | Dynorphins - immunology | Animals | Neurons - enzymology | Phosphinic Acids - pharmacology | Neprilysin - antagonists & inhibitors | Hindlimb - drug effects | Leukocytes - drug effects | Dynorphins - pharmacology | Inflammation - prevention & control | Inflammation - enzymology
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 2006, Volume 40, Issue 6, pp. 1045 - 1055
Spermatozoa must undergo capacitation to acquire fertilizing ability. Reactive oxygen species (ROS), such as superoxide anion, hydrogen peroxide (H O ), and... 
Superoxide anion | Signal transduction | Spermatozoa | Hydrogen peroxide | Free radicals | Fertilization | Phosphoproteins | Nitric oxide | Protein kinases | phosphoproteins | KINASE SIGNAL-TRANSDUCTION | HUMAN SPERMATOZOA | nitric oxide | free radicals | fertilization | TYROSINE PHOSPHORYLATION | signal transduction | BIOCHEMISTRY & MOLECULAR BIOLOGY | hydrogen peroxide | protein kinases | PHOSPHOINOSITIDE 3-KINASE | spermatozoa | REDOX REGULATION | ENDOTHELIAL-CELLS | ENDOCRINOLOGY & METABOLISM | NITRIC-OXIDE | HYDROGEN-PEROXIDE | superoxide anion | ACROSOME REACTION | Okadaic Acid - pharmacology | Serum Albumin, Bovine - pharmacology | Butadienes - pharmacology | Phosphorylation | Nitriles - pharmacology | Nitric Oxide - pharmacology | Fetal Blood - physiology | Humans | Reactive Oxygen Species - pharmacology | Hydrogen Peroxide - pharmacology | MAP Kinase Kinase 1 - metabolism | Male | Methionine - pharmacology | Proto-Oncogene Proteins c-akt - physiology | Extracellular Signal-Regulated MAP Kinases - metabolism | Peptides - pharmacology | Tyrosine - metabolism | Androstadienes - pharmacology | Methionine - analogs & derivatives | Sperm Capacitation - drug effects | Phosphatidylinositol 3-Kinases - physiology | Benzamides - pharmacology | Tyrosine | Albumin | Oxides | Phosphotransferases | Free radicals (Chemistry)
Journal Article
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 10/2011, Volume 45, Issue 4, pp. 704 - 710
Rho/Ras signaling pathways may play an important role in the mechanism of LPS-induced inflammation and bronchoconstriction. In this study, we investigated the... 
EFS | Rho/Ras signaling pathways | Human bronchi | LPS | Statins | human bronchi | BIOCHEMISTRY & MOLECULAR BIOLOGY | RHO-KINASE INHIBITOR | LUNG INJURY | CONTRACTILE ACTIVITY | CELL BIOLOGY | LIPOPOLYSACCHARIDE EXPOSURE | IN-VITRO | RESPIRATORY SYSTEM | statins | NITRIC-OXIDE | INDUCED AIRWAY HYPERRESPONSIVENESS | INHALED ENDOTOXIN | TRACHEAL SMOOTH-MUSCLE | GERANYLGERANYLTRANSFERASE-I | Leucine - pharmacology | Electric Stimulation | Sesquiterpenes - metabolism | Humans | Middle Aged | Alkyl and Aryl Transferases - metabolism | Alkyl and Aryl Transferases - antagonists & inhibitors | ras Proteins - metabolism | Male | Methionine - pharmacology | Simvastatin - pharmacology | rho-Associated Kinases - antagonists & inhibitors | Bronchi - drug effects | Methionine - analogs & derivatives | Time Factors | rho-Associated Kinases - metabolism | Bronchoconstriction - drug effects | Bronchi - metabolism | Female | Leucine - analogs & derivatives | Amides - pharmacology | Protein Prenylation - drug effects | Enzyme Inhibitors - pharmacology | Imidazoles - pharmacology | Polyisoprenyl Phosphates - metabolism | Naphthalenes - pharmacology | Hydroxymethylglutaryl-CoA Reductase Inhibitors - pharmacology | Signal Transduction - drug effects | rho GTP-Binding Proteins - metabolism | Lipopolysaccharides - pharmacology | Protein Kinase Inhibitors - pharmacology | Pyridines - pharmacology | In Vitro Techniques
Journal Article
Journal Article