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Nature (London), ISSN 1476-4687, 12/2015, Volume 529, Issue 7584, pp. 110 - 114
Gain-of-function IDH mutations are initiating events that define major clinical and prognostic classes of gliomas(1,2). Mutant IDH protein produces a new... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Chromatin - metabolism | Up-Regulation | Humans | Glioma - genetics | Base Sequence | Glioma - pathology | Epigenesis, Genetic - drug effects | Gene Expression Regulation, Neoplastic - drug effects | Binding Sites | Chromatin - drug effects | Repressor Proteins - metabolism | Oncogenes - genetics | Insulator Elements - genetics | Glioma - enzymology | Chromosomal Proteins, Non-Histone - metabolism | Cell Cycle Proteins - metabolism | Cells, Cultured | Isocitrate Dehydrogenase - genetics | DNA Methylation - genetics | Down-Regulation - drug effects | Mutation - genetics | CRISPR-Cas Systems - genetics | Insulator Elements - drug effects | Phenotype | Isocitrate Dehydrogenase - chemistry | Receptor, Platelet-Derived Growth Factor alpha - genetics | CCCTC-Binding Factor | CpG Islands - genetics | Protein Binding | Isocitrate Dehydrogenase - metabolism | Cell Proliferation - drug effects | Enhancer Elements, Genetic - genetics | Glutarates - metabolism | Cell Transformation, Neoplastic - drug effects | Chromatin - genetics | DNA Methylation - drug effects | Glioma - drug therapy | Complications and side effects | Care and treatment | Platelet-derived growth factor | Gliomas | Analysis | Influence | Genetic aspects | Research | Methylation | Oncogenes | DNA methylation | Epigenetics | Genomes | Mutation | Gene expression | Binding sites | Deoxyribonucleic acid--DNA | Tumors | Index Medicus
Journal Article
Angewandte Chemie (International ed.), ISSN 1433-7851, 11/2013, Volume 52, Issue 47, pp. 12256 - 12267
Journal Article
International journal of cancer, ISSN 0020-7136, 08/2008, Volume 123, Issue 3, pp. 552 - 560
Genistein is a phytoestrogen that has been reported to suppress the AKT signaling pathway in several malignancies. However, the molecular mechanism of... 
Prostate cancer | Genistein | Tumor suppressor gene | Life Sciences & Biomedicine | Oncology | Science & Technology | PTEN Phosphohydrolase - drug effects | Prostatic Neoplasms - metabolism | Deubiquitinating Enzyme CYLD | CpG Islands - drug effects | Humans | Male | NF-kappa B - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Genes, Tumor Suppressor - drug effects | Genes, p53 - drug effects | Prostatic Neoplasms - genetics | Chromatin Immunoprecipitation | Forkhead Transcription Factors - metabolism | Sirtuin 1 | Antimetabolites, Antineoplastic - pharmacology | Electrophoretic Mobility Shift Assay | Gene Expression Regulation, Neoplastic - drug effects | Chromones - pharmacology | Anticarcinogenic Agents - pharmacology | Hydroxamic Acids - pharmacology | Phytoestrogens - pharmacology | Prostatic Neoplasms - drug therapy | PTEN Phosphohydrolase - genetics | Tumor Suppressor Proteins - metabolism | Enzyme Inhibitors - pharmacology | Morpholines - pharmacology | PTEN Phosphohydrolase - metabolism | Azacitidine - analogs & derivatives | Down-Regulation - drug effects | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Histones - drug effects | Gene Expression Regulation - drug effects | Up-Regulation - drug effects | Azacitidine - pharmacology | Acetylation - drug effects | Methylation - drug effects | Genistein - pharmacology | Cell Line, Tumor | Protein Kinase Inhibitors - pharmacology | Histones - metabolism | Forkhead Box Protein O3 | Sirtuins - metabolism | NF-kappa B - drug effects | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 12/2011, Volume 108, Issue 50, pp. 20119 - 20124
Natural killer cell lymphoma (NKCL) constitutes a rare and aggressive form of non-Hodgkin lymphoma, and there is little insight into its pathogenesis. Here we... 
B lymphocytes | T cell lymphoma | B cell lymphoma | Cell lines | Cell cycle | Tumor suppressor genes | Natural killer cells | Methylation | Tumors | Apoptosis | Neoplastic transformation | NK-cell activation and homeostasis | Biotage pyrosequencing | CCNG2 | CCNG1 | Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | G2 Phase - genetics | Gene Silencing - drug effects | Apoptosis - drug effects | Humans | Killer Cells, Natural - pathology | Promoter Regions, Genetic - genetics | G2 Phase - drug effects | Lymphoma, Non-Hodgkin - pathology | Gene Knockdown Techniques | Time Factors | DNA Mutational Analysis | Tumor Suppressor Proteins - genetics | DNA Copy Number Variations - drug effects | Gene Expression Regulation, Neoplastic - drug effects | Repressor Proteins - metabolism | Cell Division - genetics | Interleukin-2 - metabolism | Tumor Suppressor Proteins - metabolism | Transduction, Genetic | Repressor Proteins - genetics | DNA Copy Number Variations - genetics | DNA Methylation - genetics | Lymphoma, Non-Hodgkin - genetics | Cell Division - drug effects | Biopsy | Culture Media - pharmacology | Interleukin-2 - pharmacology | Killer Cells, Natural - drug effects | Killer Cells, Natural - metabolism | DNA Methylation - drug effects | Positive Regulatory Domain I-Binding Factor 1 | RNA, Small Interfering - metabolism | Development and progression | Killer cells | Genetic aspects | Non-Hodgkin's lymphomas | Health aspects | Index Medicus | Life Sciences | Biochemistry, Molecular Biology | Biological Sciences | biotage pyrosequencing | neoplastic transformation
Journal Article
The Journal of biological chemistry, ISSN 0021-9258, 05/2009, Volume 284, Issue 22, pp. 14838 - 14848
Journal Article
Lancet, The, ISSN 0140-6736, 2013, Volume 381, Issue 9883, pp. 2109 - 2117
Journal Article
PloS one, ISSN 1932-6203, 04/2013, Volume 8, Issue 4, pp. e61807 - e61807
The Notch pathway can have both oncogenic and tumor suppressor roles, depending on cell context. For example, Notch signaling promotes T cell differentiation... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Transcription, Genetic - drug effects | Apoptosis - drug effects | Receptors, Notch - metabolism | Humans | Apoptosis - genetics | Receptors, Notch - genetics | Gene Expression Profiling | Hematopoiesis - drug effects | Basic Helix-Loop-Helix Transcription Factors - metabolism | T-Lymphocytes - metabolism | Serrate-Jagged Proteins | T-Lymphocytes - drug effects | B-Lymphocytes - pathology | Precursor Cell Lymphoblastic Leukemia-Lymphoma - blood | B-Lymphocytes - metabolism | Repressor Proteins - metabolism | Bone Marrow - drug effects | Jagged-1 Protein | Basic Helix-Loop-Helix Transcription Factors - genetics | Membrane Proteins - genetics | Gene Expression Regulation, Leukemic - drug effects | Leukemia, B-Cell - pathology | Repressor Proteins - genetics | Hematopoiesis - genetics | Signal Transduction - genetics | Azacitidine - pharmacology | Leukemia, B-Cell - blood | Leukemia, B-Cell - genetics | Signal Transduction - drug effects | Precursor T-Cell Lymphoblastic Leukemia-Lymphoma - genetics | Cell Line, Tumor | Histones - metabolism | DNA Methylation - drug effects | Calcium-Binding Proteins - genetics | Gene Silencing - drug effects | Blood Cells - metabolism | Cell Lineage - drug effects | Intercellular Signaling Peptides and Proteins - metabolism | Epigenesis, Genetic - drug effects | Membrane Proteins - metabolism | T-Lymphocytes - pathology | Precursor Cell Lymphoblastic Leukemia-Lymphoma - pathology | Blood Cells - drug effects | Hydroxamic Acids - pharmacology | Cell Lineage - genetics | Calcium-Binding Proteins - metabolism | Intercellular Signaling Peptides and Proteins - genetics | Azacitidine - analogs & derivatives | DNA Methylation - genetics | Precursor Cell Lymphoblastic Leukemia-Lymphoma - genetics | B-Lymphocytes - drug effects | Bone Marrow - pathology | Receptor, Notch3 | Cell Proliferation - drug effects | Sulfites | Epigenetic inheritance | DNA microarrays | Genes | Genetic aspects | Genetic engineering | Acute lymphocytic leukemia | B cells | T cells | Methylation | Cell differentiation | Biotechnology | Transcription factors | Bisulfite | Pathogenesis | Leukemia | Differentiation (biology) | Lymphocytes T | Kinases | Inactivation | Signal transduction | Restoration | Lymphocytes | DNA methylation | Bone marrow | Tumorigenesis | Acetylation | Deoxyribonucleic acid--DNA | CpG islands | Acute lymphatic leukemia | Deactivation | Lymphatic leukemia | Tumor cell lines | Gene silencing | Signaling | Lymphocytes B | Regulatory mechanisms (biology) | Stem cells | Epigenetics | Ligands | Tumor suppressor genes | Leukemogenesis | Lymphomas | Notch protein | Aberration | Apoptosis | Cancer | Index Medicus | Deoxyribonucleic acid | DNA
Journal Article