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Nature Immunology, ISSN 1529-2908, 11/2001, Volume 2, Issue 11, pp. 1025 - 1031
The predisposition of nonobese diabetic (NOD) mice to develop autoimmune disease is usually attributed to defects in peripheral tolerance mechanisms. Here,... 
APOPTOSIS | THYMUS | ACTIVATION | CD4(+)CD8(+) THYMOCYTES | NONOBESE DIABETIC MICE | IN-VIVO | MOUSE | T-CELL REPERTOIRE | NEGATIVE SELECTION | DEATH | IMMUNOLOGY | T-Lymphocyte Subsets - immunology | Antigens, CD - biosynthesis | Interleukin-7 - pharmacology | Antigens, CD - genetics | Autoimmune Diseases - genetics | RNA, Messenger - biosynthesis | Interleukin-4 - pharmacology | Membrane Glycoproteins - physiology | Mice, Inbred MRL lpr | Immune Tolerance - immunology | Thymus Gland - pathology | fas Receptor - physiology | Diabetes Mellitus, Type 1 - immunology | CD28 Antigens - physiology | Genetic Predisposition to Disease | Carrier Proteins - biosynthesis | Lectins, C-Type | Mice, Inbred C57BL | Autoimmune Diseases - immunology | Gene Expression Regulation | Diabetes Mellitus, Type 1 - genetics | Intracellular Signaling Peptides and Proteins | Mice, Inbred NOD - immunology | Clonal Deletion - immunology | Mice, Inbred NZB | CD8 Antigens - analysis | Receptors, Antigen, T-Cell - physiology | Fas Ligand Protein | Carrier Proteins - genetics | Animals | T-Lymphocyte Subsets - pathology | CD4 Antigens - analysis | Antigens, Differentiation, T-Lymphocyte - genetics | CASP8 and FADD-Like Apoptosis Regulating Protein | Mice | Antigens, Differentiation, T-Lymphocyte - biosynthesis | Apoptosis | Mice, Inbred NOD - genetics
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