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Circulation Research, ISSN 0009-7330, 02/2009, Volume 104, Issue 4, pp. 476 - 487
MicroRNAs (miRNAs) comprise a novel class of endogenous, small, noncoding RNAs that negatively regulate gene expression. Functionally, an individual miRNA is... 
Vascular disease | Proliferation | Gene regulation | MicroRNAs | Vascular smooth muscle cells | microRNAs | CARDIAC & CARDIOVASCULAR SYSTEMS | proliferation | DOWN-REGULATION | CARDIOVASCULAR BIOLOGY | P27(KIP1) | BREAST-CANCER | INHIBITION | vascular disease | MESSENGER-RNA | gene regulation | RAT CAROTID-ARTERY | GROWTH | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | vascular smooth muscle cells | MICRORNA EXPRESSION | PROGRESSION | Carotid Artery Diseases - prevention & control | Cyclin-Dependent Kinase Inhibitor p57 - metabolism | Cell Proliferation | Muscle, Smooth, Vascular - metabolism | Hyperplasia | Myocytes, Smooth Muscle - pathology | Oligonucleotides, Antisense - metabolism | Male | MicroRNAs - metabolism | Angioplasty, Balloon - adverse effects | Gene Knockdown Techniques | Cyclin-Dependent Kinase Inhibitor p27 - metabolism | RNA Interference | Time Factors | Carotid Artery Diseases - pathology | Carotid Artery Diseases - metabolism | Myocytes, Smooth Muscle - metabolism | Disease Models, Animal | Carotid Artery, Common - metabolism | Proto-Oncogene Proteins c-sis | Cells, Cultured | Rats | Rats, Sprague-Dawley | Tunica Intima - metabolism | Muscle, Smooth, Vascular - pathology | Platelet-Derived Growth Factor - metabolism | Animals | Tunica Intima - pathology | Carotid Artery Diseases - genetics | Carotid Artery, Common - pathology | RNA, Small Interfering - metabolism | Index Medicus
Journal Article
Cancer Cell, ISSN 1535-6108, 2008, Volume 14, Issue 5, pp. 382 - 393
A key step in angiogenesis is the upregulation of growth factor receptors on endothelial cells. Here, we demonstrate that a small regulatory microRNA, miR-296,... 
CELLCYCLE | THERAPY | ONCOLOGY | VASCULAR INTEGRITY | IN-VIVO | GENE-EXPRESSION | DEGRADATION | TARGETS | OCULAR NEOVASCULARIZATION | TUMOR ANGIOGENESIS | MICRORNAS | CANCER | CELL BIOLOGY | Endothelium, Vascular - cytology | Luciferases - metabolism | MicroRNAs - antagonists & inhibitors | Humans | Neovascularization, Pathologic | Molecular Sequence Data | MicroRNAs - metabolism | Brain Neoplasms - blood supply | Phosphoproteins - antagonists & inhibitors | Phosphoproteins - metabolism | RNA, Messenger - metabolism | Receptor, Platelet-Derived Growth Factor beta - genetics | Brain Neoplasms - metabolism | Glioma - metabolism | Vascular Endothelial Growth Factor Receptor-2 - genetics | Vascular Endothelial Growth Factor Receptor-2 - antagonists & inhibitors | Kidney - metabolism | Receptor, Platelet-Derived Growth Factor beta - antagonists & inhibitors | Oligonucleotides - pharmacology | Base Sequence | RNA, Messenger - antagonists & inhibitors | Glioma - therapy | Umbilical Veins - cytology | Receptor, Platelet-Derived Growth Factor beta - metabolism | Signal Transduction | Endosomal Sorting Complexes Required for Transport | RNA, Messenger - genetics | RNA, Small Interfering - pharmacology | Cells, Cultured | Angiogenesis Inhibitors - pharmacology | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Kidney - cytology | Phosphoproteins - genetics | Reverse Transcriptase Polymerase Chain Reaction | Sequence Homology, Nucleic Acid | Blotting, Western | Hepatocyte Growth Factor - metabolism | Glioma - blood supply | Xenograft Model Antitumor Assays | Magnetic Resonance Imaging | Animals | Endothelium, Vascular - metabolism | Brain Neoplasms - therapy | Fluorescent Antibody Technique | Mice | MicroRNAs - genetics | Cell Movement | Umbilical Veins - metabolism | Tyrosine | Medical colleges | Neurosciences | Messenger RNA | Gliomas | Oncology, Experimental | Brain tumors | Research | Growth factors | Cancer | Endothelium | Index Medicus | PDGFR | VEGFR | miRNA | cancer | HGS | angiogenesis
Journal Article
Oncotarget, ISSN 1949-2553, 2015, Volume 6, Issue 27, pp. 24017 - 24031
Up to now, the molecular mechanisms underlying the stemness of prostate cancer stem cells (PCSCs) are still poorly understood. In this study, we demonstrated... 
miR-7 | Tumorigenesis | PI3K/Akt pathway | Cancer stem cells | KLF4 | Prostate cancer | PHOSPHORYLATION | SELF-RENEWAL | PROLIFERATION | ACUTE MYELOID-LEUKEMIA | MESENCHYMAL TRANSITION | TUMOR-INITIATING CELLS | CELL BIOLOGY | prostate cancer | cancer stem cells | tumorigenesis | CD44 | Index Medicus
Journal Article
Nature Communications, ISSN 2041-1723, 12/2016, Volume 7, Issue 1, pp. 13616 - 13616
Journal Article
PLoS Pathogens, ISSN 1553-7366, 04/2013, Volume 9, Issue 4, pp. e1003248 - e1003248
Upon recognition of viral components by pattern recognition receptors, such as the toll-like receptors (TLRs) and retinoic acid-inducible gene I (RIG-I)-like... 
RNA REPLICATION | HEPATITIS-C-VIRUS | MICROBIOLOGY | PROTEIN-SYNTHESIS | ALPHA-INTERFERON | I INTERFERON | VIROLOGY | SIGNALING PATHWAY | GENE-EXPRESSION | CYCLOOXYGENASE-2 EXPRESSION | NF-KAPPA-B | DEPENDENT ACTIVATION | PARASITOLOGY | Interleukin-1 Receptor-Associated Kinases - metabolism | RNA Helicases - metabolism | Up-Regulation | Phosphorylation | Interleukin-1 Receptor-Associated Kinases - genetics | Hepacivirus - immunology | Humans | Adaptor Proteins, Vesicular Transport - genetics | Hepacivirus - genetics | Hepacivirus - pathogenicity | JNK Mitogen-Activated Protein Kinases - metabolism | MicroRNAs - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Interferon Regulatory Factor-3 - genetics | MAP Kinase Signaling System | Hepatitis C - immunology | RNA Interference | Protein Kinase C - metabolism | HEK293 Cells | Interferon-alpha - metabolism | Toll-Like Receptor 7 - metabolism | Interferon-alpha - biosynthesis | Myeloid Differentiation Factor 88 - genetics | Proto-Oncogene Proteins c-fos - metabolism | MicroRNAs - immunology | Immune Evasion | Cell Line, Tumor | Viral Nonstructural Proteins - metabolism | MicroRNAs - genetics | RNA, Small Interfering | Serine Endopeptidases - metabolism | Myeloid Differentiation Factor 88 - metabolism | Virus diseases | Immune response | MicroRNA | Cellular control mechanisms | Research | Observations | Hepatitis C virus | Properties | Testing | Index Medicus | Proteins | Hepatitis | Kinases | Viral infections | Immune system
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 08/2010, Volume 207, Issue 8, pp. 1589 - 1597
Uncontrolled extracellular matrix production by fibroblasts in response to tissue injury contributes to fibrotic diseases, such as idiopathic pulmonary... 
MEDICINE, RESEARCH & EXPERIMENTAL | RESPONSES | PHOSPHATASE | INJURY | TENSIN HOMOLOG | MECHANISMS | GROWTH-FACTOR | IMMUNOLOGY | MYOFIBROBLAST DIFFERENTIATION | EXPRESSION | CANCER | MICRORNA | Oligonucleotides - genetics | Pulmonary Fibrosis - therapy | Idiopathic Pulmonary Fibrosis - genetics | Gene Expression - drug effects | Gene Expression - genetics | Humans | Actins - metabolism | MicroRNAs - metabolism | Pulmonary Fibrosis - genetics | Idiopathic Pulmonary Fibrosis - metabolism | Actins - genetics | Antisense Elements (Genetics) - therapeutic use | Collagen - genetics | Lung - metabolism | Phosphorylation - drug effects | Smad7 Protein - genetics | Extracellular Matrix Proteins - metabolism | Transforming Growth Factor beta1 - pharmacology | Fibroblasts - metabolism | Smad7 Protein - metabolism | Cell Line | Lung - pathology | Extracellular Matrix Proteins - genetics | Mice, Inbred C57BL | Smad2 Protein - metabolism | Mice, Transgenic | Pulmonary Fibrosis - pathology | Transforming Growth Factor beta1 - genetics | Fibroblasts - pathology | Fibronectins - metabolism | Collagen - metabolism | Animals | Fibroblasts - drug effects | Antisense Elements (Genetics) - genetics | Idiopathic Pulmonary Fibrosis - pathology | Pulmonary Fibrosis - chemically induced | Bleomycin - pharmacology | Fibronectins - genetics | Mice | MicroRNAs - genetics | Index Medicus | Brief Definitive Report
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 9/2008, Volume 105, Issue 35, pp. 12885 - 12890
Progress in understanding the biology of multiple myeloma (MM), a plasma cell malignancy, has been slow. The discovery of microRNAs (miRNAs), a class of small... 
Tumor cell line | Cell growth | MicroRNA | Transfection | Cell lines | Multiple myeloma | Oligonucleotides | Plasma cells | Signatures | Tumors | SOCS-1 | MGUS | PCAF | Tumor suppressor gene | CELLS | MULTIDISCIPLINARY SCIENCES | BONE-MARROW | plasma cells | CANCER | SIGNATURE | TUMOR-SUPPRESSOR | TARGETS | LEUKEMIA | DIFFERENTIATION | EXPRESSION | tumor suppressor gene | IN-SITU | Up-Regulation | Genes, Neoplasm | Humans | Gene Expression Regulation, Neoplastic | Molecular Sequence Data | Suppressor of Cytokine Signaling 1 Protein | MicroRNAs - metabolism | Gene Expression Profiling | Monoclonal Gammopathy of Undetermined Significance | Plasma Cells - pathology | Bcl-2-Like Protein 11 | Base Sequence | Plasma Cells - metabolism | Membrane Proteins - metabolism | Repressor Proteins - metabolism | STAT3 Transcription Factor - metabolism | Proto-Oncogene Proteins - metabolism | Reproducibility of Results | Health | Tumor Suppressor Protein p53 - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Apoptosis Regulatory Proteins - metabolism | p300-CBP Transcription Factors - metabolism | Multiple Myeloma - pathology | Animals | Mice, Nude | Cell Line, Tumor | Mice | MicroRNAs - genetics | Suppressor of Cytokine Signaling Proteins - metabolism | Multiple Myeloma - genetics | Receptors, Interleukin-6 - metabolism | Genetic aspects | Gene expression | Health aspects | Studies | Pathology | Rodents | Ribonucleic acid--RNA | Cells | Index Medicus | Biological Sciences
Journal Article
Proceedings of the National Academy of Sciences, ISSN 0027-8424, 02/2014, Volume 111, Issue 5, pp. E572 - E581
The roles of microRNAs (miRNAs) and the miRNA processing machinery in the regulation of stem cell biology are not well understood. Here, we show that the p53... 
LIMB | STEM-CELLS | METASTASIS | MULTIDISCIPLINARY SCIENCES | PLURIPOTENCY | EPITHELIAL DEVELOPMENT | MICRORNAS | P63 | EPIDERMAL MORPHOGENESIS | TAP63 | P53 | RNA-Binding Proteins - genetics | Cell Proliferation | Homeodomain Proteins - metabolism | Humans | Multipotent Stem Cells - metabolism | Transcription Factors - deficiency | MicroRNAs - metabolism | Gene Expression Profiling | Phosphoproteins - metabolism | RNA, Messenger - metabolism | SOXB1 Transcription Factors - metabolism | Tumor Suppressor Proteins - deficiency | Tumor Suppressor Proteins - genetics | Trans-Activators - genetics | Adult | Transcription, Genetic | Cell Differentiation | Induced Pluripotent Stem Cells - cytology | Induced Pluripotent Stem Cells - metabolism | Cell Line | Tumor Suppressor Proteins - metabolism | Nanog Homeobox Protein | RNA, Messenger - genetics | Keratinocytes - cytology | Phosphoproteins - genetics | Transcription Factors - genetics | Down-Regulation - genetics | Proteins - genetics | Transcription Factors - metabolism | Cell Lineage | Animals | Proteins - metabolism | Trans-Activators - deficiency | Embryo, Mammalian - cytology | Keratinocytes - metabolism | Multipotent Stem Cells - cytology | Octamer Transcription Factor-3 - metabolism | Phosphoproteins - deficiency | Trans-Activators - metabolism | Mice | MicroRNAs - genetics | RNA-Binding Proteins - metabolism | Chimera | Epidermis - cytology | Index Medicus | PNAS Plus | Biological Sciences
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 07/2014, Volume 124, Issue 7, pp. 3093 - 3106
Dysregulation of epigenetic controls is associated with tumorigenesis in response to microenvironmental stimuli; however, the regulatory pathways involved in... 
EPITHELIAL-MESENCHYMAL TRANSITION | BREAST-CANCER | MEDICINE, RESEARCH & EXPERIMENTAL | DIVISIONS | NOTCH | MIR-205 | SLUG | ESTROGEN-RECEPTOR | GLAND | Cell Polarity | Transcription Factor HES-1 | Cell Proliferation | MicroRNAs - antagonists & inhibitors | Epigenesis, Genetic | Homeodomain Proteins - metabolism | Humans | Gene Expression Regulation, Neoplastic | Tumor Microenvironment | MicroRNAs - metabolism | Mammary Neoplasms, Experimental - metabolism | RNA, Neoplasm - metabolism | Mammary Neoplasms, Experimental - genetics | Breast Neoplasms - metabolism | Gene Knockdown Techniques | Intercellular Signaling Peptides and Proteins - metabolism | Basic Helix-Loop-Helix Transcription Factors - metabolism | Neoplastic Stem Cells - metabolism | Kruppel-Like Transcription Factors - metabolism | Serrate-Jagged Proteins | Epithelial-Mesenchymal Transition | Mammary Neoplasms, Experimental - pathology | Neoplastic Stem Cells - pathology | Female | Membrane Proteins - metabolism | Jagged-1 Protein | Calcium-Binding Proteins - metabolism | Basic Helix-Loop-Helix Transcription Factors - genetics | Signal Transduction | Membrane Proteins - genetics | Carcinogenesis - genetics | Intercellular Signaling Peptides and Proteins - genetics | Receptor, Notch2 - metabolism | Homeodomain Proteins - genetics | Transcription Factors - metabolism | Animals | Breast Neoplasms - genetics | Breast Neoplasms - pathology | Cell Line, Tumor | RNA, Neoplasm - genetics | Mice | MicroRNAs - genetics | Calcium-Binding Proteins - genetics | Zinc Finger E-box-Binding Homeobox 1 | MicroRNA | Oncology, Experimental | Stem cells | Genetic research | Genetic aspects | Research | Genetic regulation | Properties | Carcinogenesis | Cancer | Medical research | Breast cancer | Rodents | Index Medicus | Abridged Index Medicus
Journal Article
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 09/2017, Volume 491, Issue 2, pp. 329 - 336
Hyperglycemia plays a crucial role in the pathogenesis of diabetic complications; however, the mechanisms underlying diabetic cardiac fibrosis remain unclear.... 
MicroRNA | EndMT | Notch2 | Fibrosis | High glucose | TARGET | CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | HEART-FAILURE | MICRORNAS | DIABETIC CARDIOMYOPATHY | BIOPHYSICS | ALAGILLE-SYNDROME | EXPRESSION | Dependovirus - genetics | Cadherins - metabolism | Genetic Vectors - administration & dosage | Humans | Aortic Valve - drug effects | Diabetes Mellitus, Experimental - genetics | Male | MicroRNAs - metabolism | Diabetes Mellitus, Experimental - chemically induced | Cadherins - genetics | Endomyocardial Fibrosis - prevention & control | Dependovirus - metabolism | Genetic Vectors - chemistry | Signal Transduction | Endothelial Cells - metabolism | Genetic Vectors - metabolism | Glucose - pharmacology | Heart Ventricles - metabolism | MicroRNAs - genetics | Calcium-Binding Proteins - genetics | Platelet Endothelial Cell Adhesion Molecule-1 - genetics | Streptozocin | Vimentin - metabolism | Actins - metabolism | Receptor, Notch2 - genetics | Actins - genetics | Platelet Endothelial Cell Adhesion Molecule-1 - metabolism | Vimentin - genetics | Epithelial-Mesenchymal Transition | Diabetic Cardiomyopathies - prevention & control | Endomyocardial Fibrosis - genetics | Endomyocardial Fibrosis - chemically induced | Endomyocardial Fibrosis - pathology | Diabetes Mellitus, Experimental - metabolism | Heart Ventricles - pathology | Calcium-Binding Proteins - metabolism | Cell Line | Mice, Inbred C57BL | Gene Expression Regulation | Receptor, Notch2 - metabolism | Aortic Valve - cytology | Aortic Valve - metabolism | Fibronectins - metabolism | Diabetic Cardiomyopathies - genetics | Animals | Endothelial Cells - cytology | Diabetes Mellitus, Experimental - pathology | Diabetic Cardiomyopathies - pathology | Fibronectins - genetics | Diabetic Cardiomyopathies - chemically induced | Endothelial Cells - drug effects | Fibronectins | Hyperglycemia | Stem cells | Glucose | Muscle proteins | Dextrose | Endothelium | Index Medicus
Journal Article
by Zhang, J and Hu, M and Teng, ZQ and Tang, YP and Chen, C
JOURNAL OF NEUROSCIENCE, ISSN 0270-6474, 11/2014, Volume 34, Issue 45, pp. 14919 - 14933
Journal Article
Nature Cell Biology, ISSN 1465-7392, 07/2017, Volume 19, Issue 7, pp. 820 - 832
Understanding the roles of splicing factors and splicing events during tumorigenesis would open new avenues for targeted therapies. Here we identify an... 
NONCODING RNA | PAXILLIN | COLORECTAL-CANCER | GENE-EXPRESSION | SELF-RENEWAL | PROLIFERATION | LIVER DEVELOPMENT | PROGRESSION | TUMOR-INITIATING CELLS | CANCER STEM-CELLS | CELL BIOLOGY | Paxillin - metabolism | RNA-Binding Proteins - genetics | Up-Regulation | Paxillin - genetics | Cell Proliferation | Alternative Splicing | Exons | Humans | Gene Expression Regulation, Neoplastic | Male | MicroRNAs - metabolism | SOXB1 Transcription Factors - metabolism | Octamer Transcription Factor-3 - genetics | Transfection | RNA Interference | SOXB1 Transcription Factors - genetics | Time Factors | Carcinoma, Hepatocellular - genetics | 3' Untranslated Regions | Binding Sites | Nanog Homeobox Protein - genetics | Argonaute Proteins - metabolism | Argonaute Proteins - genetics | Liver Neoplasms - genetics | Mice, Inbred C57BL | Gene Expression Profiling - methods | RNA, Long Noncoding - genetics | Tumor Burden | Hep G2 Cells | Animals | Carrier Proteins - metabolism | Mice, Nude | Octamer Transcription Factor-3 - metabolism | Liver Neoplasms - metabolism | Protein Binding | High-Throughput Nucleotide Sequencing | Mice | MicroRNAs - genetics | RNA, Long Noncoding - metabolism | Nanog Homeobox Protein - metabolism | RNA-Binding Proteins - metabolism | Carcinoma, Hepatocellular - metabolism | Alternative splicing | Translation | Transcription | Splicing | MiRNA | Hepatocellular carcinoma | Gene expression | Argonaute 2 protein | Gene sequencing | Degradation | Liver cancer | 3' Untranslated regions | Ribonucleic acids | Translation elongation | Tumorigenesis | Splicing factors | Elongation | Index Medicus
Journal Article