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The Journal of clinical investigation, ISSN 0021-9738, 01/2005, Volume 115, Issue 1, pp. 118 - 127
The angiogenic mechanism and therapeutic potential of PDGF-CC, a recently discovered member of the VEGF/PDGF superfamily, remain incompletely characterized.... 
Neovascularization, Physiologic - drug effects | Humans | Stem Cells - cytology | Microcirculation - drug effects | Lymphokines | Phosphotyrosine - metabolism | Myocardium - metabolism | Ischemia - pathology | Coronary Vessels - drug effects | Signal Transduction | Cells, Cultured | Ischemia - metabolism | Myocardium - pathology | Platelet-Derived Growth Factor - pharmacology | Cell Movement - drug effects | Animals | Ischemia - drug therapy | Cell Differentiation - drug effects | Coronary Vessels - cytology | Endothelial Cells - cytology | Hindlimb - blood supply | Receptors, Platelet-Derived Growth Factor - metabolism | Stem Cells - drug effects | Hindlimb - drug effects | Mice | Ischemia - chemically induced | Endothelial Cells - drug effects | Index Medicus | Abridged Index Medicus | Hindlimb/blood supply/drug effects | Cell Differentiation/drug effects | Receptors; Platelet-Derived Growth Factor/metabolism | Medical and Health Sciences | Medicin och hälsovetenskap | Cell Movement/drug effects | Cells; Cultured | Stem Cells/cytology/drug effects | Myocardium/metabolism/pathology | Endothelial Cells/cytology/drug effects | MEDICINE | Platelet-Derived Growth Factor/pharmacology | Ischemia/chemically induced/drug therapy/metabolism/pathology | MEDICIN | Phosphotyrosine/metabolism | Research Support; Non-U.S. Gov't | Coronary Vessels/cytology/drug effects | Microcirculation/drug effects | Neovascularization; Physiologic/drug effects
Journal Article
Journal of Cerebral Blood Flow & Metabolism, ISSN 0271-678X, 11/2015, Volume 35, Issue 11, pp. 1871 - 1881
Journal Article
Scientific Reports, ISSN 2045-2322, 05/2014, Volume 4, Issue 1, pp. 4871 - 4871
Journal Article
Journal Article
Circulation, ISSN 0009-7322, 12/2002, Volume 106, Issue 23, pp. 2973 - 2979
Background-Erythropoietin (EPO) is a critical regulator for the proliferation of immature erythroid precursors, but its role as a potential cytoprotectant in... 
Cytochrome c | Proteins, mitochondrial | Proteins, proto-oncogene | Cysteine endopeptidases | Apoptosis | CARDIAC & CARDIOVASCULAR SYSTEMS | IN-VIVO EVIDENCE | INJURY | apoptosis | PROGRAMMED CELL-DEATH | proteins, proto-oncogene | cysteine endopeptidases | ENDOTHELIAL-CELLS | NEURONS | PERIPHERAL VASCULAR DISEASE | cytochrome c | HEMATOLOGY | proteins, mitochondrial | BRAIN | ERYTHROID PROGENITORS | Endothelium, Vascular - cytology | Erythropoietin - pharmacology | Mitochondria - enzymology | Cell Hypoxia - physiology | Microcirculation - metabolism | Intracellular Membranes - physiology | Apoptosis - drug effects | Endothelium, Vascular - drug effects | Cytoprotection - physiology | Cysteine Endopeptidases - drug effects | Microcirculation - drug effects | Brain - blood supply | Cysteine Endopeptidases - metabolism | Cytoprotection - drug effects | Cell Membrane - metabolism | Proto-Oncogene Proteins | DNA Fragmentation - drug effects | Mitochondria - chemistry | Cell Membrane - drug effects | Protein-Serine-Threonine Kinases - metabolism | Membrane Potentials - drug effects | Cell Survival - drug effects | Phosphatidylserines - metabolism | Cells, Cultured | Enzyme Inhibitors - pharmacology | Rats | Mitochondria - drug effects | Enzyme Activation - drug effects | Rats, Sprague-Dawley | Antibodies - pharmacology | Proto-Oncogene Proteins c-akt | Microcirculation - cytology | Animals | Signal Transduction - drug effects | Endothelium, Vascular - metabolism | Erythropoietin - antagonists & inhibitors | Intracellular Membranes - drug effects | Index Medicus | Abridged Index Medicus
Journal Article
Diabetes Care, ISSN 0149-5992, 2009, Volume 32, Issue 11, pp. 2068 - 2074
Journal Article
European Journal of Pharmacology, ISSN 0014-2999, 09/2013, Volume 715, Issue 1-3, pp. 381 - 394
Okadaic acid (OKA) has been observed to cause memory impairment in human subjects having seafood contaminated with dinoflagellate ( ). OKA induces tau... 
Cholinergic dysfunction | Okadaic acid | Neuroinflammation | Curcumin | Cerebral blood flow | Memory | COGNITIVE PERFORMANCE | PROTEIN | ACETYLCHOLINE | ALZHEIMERS-DISEASE | DAMAGE | REDUCTION | INJECTION | PHARMACOLOGY & PHARMACY | HYDROGEN-PEROXIDE | RAT-BRAIN | CEREBRAL-BLOOD-FLOW | Memory - drug effects | Transcription, Genetic - drug effects | Reactive Oxygen Species - metabolism | Apoptosis - drug effects | Calcium - metabolism | Glutathione - metabolism | Motor Activity - drug effects | Male | Neurons - cytology | Brain - physiology | Microcirculation - drug effects | Okadaic Acid - adverse effects | Brain - metabolism | Brain - blood supply | Neuroprotective Agents - pharmacology | Adenosine Triphosphate - metabolism | Phosphorylation - drug effects | Neurons - drug effects | Malondialdehyde - metabolism | Maze Learning - physiology | Curcumin - pharmacology | Mitochondria - metabolism | Avoidance Learning - physiology | Mitochondria - drug effects | Maze Learning - drug effects | Organ Size - drug effects | Animals | Atrophy - prevention & control | Brain - pathology | Acetylcholine | Avoidance Learning - drug effects | Mice | Acetylcholinesterase - metabolism | Energy Metabolism - drug effects | Memory - physiology | Antioxidants | Seafood | Intermediate filament proteins | Analysis | Contamination | Index Medicus
Journal Article
American Journal of Pathology, The, ISSN 0002-9440, 2010, Volume 176, Issue 3, pp. 1306 - 1315
Brain hemodynamics in cerebral malaria (CM) is poorly understood, with apparently conflicting data showing microcirculatory hypoperfusion and normal or even... 
Pathology | SEQUESTRATION | PATHOGENESIS | AFRICAN CHILDREN | PLASMODIUM-FALCIPARUM | IN-VIVO | VOLUME | SUBARACHNOID HEMORRHAGE | RED-BLOOD-CELLS | MICE | BERGHEI | PATHOLOGY | Body Temperature - drug effects | Plasmodium berghei - physiology | Parasitemia - drug therapy | Nimodipine - pharmacology | Vasospasm, Intracranial - complications | Malaria, Cerebral - physiopathology | Microcirculation - drug effects | Microcirculation - physiology | Artemisinins - pharmacology | Arterioles - physiopathology | Erythrocytes - pathology | Parasitemia - physiopathology | Vasospasm, Intracranial - parasitology | Malaria, Cerebral - parasitology | Cerebrovascular Circulation - drug effects | Mice, Inbred C57BL | Parasitemia - complications | Cell Adhesion - drug effects | Arterioles - drug effects | Vasoconstriction - drug effects | Erythrocytes - drug effects | Nimodipine - therapeutic use | Arterioles - pathology | Animals | Artemisinins - therapeutic use | Leukocytes - parasitology | Malaria, Cerebral - complications | Vasospasm, Intracranial - physiopathology | Malaria, Cerebral - drug therapy | Survival Analysis | Parasitemia - parasitology | Leukocytes - drug effects | Mice | Vasodilation - drug effects | Erythrocytes - parasitology | Plasmodium berghei - drug effects | Vasospasm, Intracranial - drug therapy | Index Medicus | Abridged Index Medicus | Regular
Journal Article
Journal Article
Annals of Oncology, ISSN 0923-7534, 5/2008, Volume 19, Issue 5, pp. 927 - 934
Arterial hypertension (HT) has been reported in all studies involving bevacizumab, an antiangiogenic agent designed to target vascular endothelial growth... 
Hypertension | Angiogenesis | Microcirculation | Endothelial function | Capillary rarefaction | endothelial function | LIES DOWNSTREAM | PERMEABILITY | angiogenesis | NITRIC-OXIDE SYNTHASE | KINASE | ANTIBODY | MECHANISMS | microcirculation | THERAPY | ONCOLOGY | capillary rarefaction | DRUGS | hypertension | ENDOTHELIAL GROWTH-FACTOR | Cholinergic Agents - administration & dosage | Cholinergic Agents - pharmacology | Humans | Middle Aged | Antibodies, Monoclonal - adverse effects | Antibodies, Monoclonal - therapeutic use | Capillaries - drug effects | Endothelium, Vascular - drug effects | Male | Microscopy, Video | Microcirculation - drug effects | Bevacizumab | Antibodies, Monoclonal, Humanized | Iontophoresis | Colorectal Neoplasms - drug therapy | Hypertension - chemically induced | Forearm - blood supply | Angiogenesis Inhibitors - therapeutic use | Female | Blood Pressure - drug effects | Pilocarpine - administration & dosage | Angiogenesis Inhibitors - adverse effects | Diabetes Mellitus, Type 2 - complications | Colorectal Neoplasms - blood supply | Antibodies, Monoclonal - pharmacology | Endothelium, Vascular - physiopathology | Angiogenesis Inhibitors - pharmacology | Laser-Doppler Flowmetry | Adenocarcinoma - drug therapy | Adenocarcinoma - secondary | Adenocarcinoma - blood supply | Diabetes Mellitus, Type 2 - physiopathology | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Neovascularization, Pathologic - drug therapy | Pilocarpine - pharmacology | Fingers - blood supply | Aged | Vasodilation - drug effects | Index Medicus
Journal Article
Journal Article
American Journal of Physiology - Renal Physiology, ISSN 0363-6127, 04/2014, Volume 306, Issue 7, pp. F734 - F743
Acute kidney injury (AKI) is a complication of sepsis and leads to a high mortality rate. Human and animal studies suggest that mitochondrial dysfunction plays... 
Mitochondrial antioxidant | Oxidative stress | Sepsis | Mitochondria | Kidney | ACUTE KIDNEY INJURY | GRAM-NEGATIVE SEPSIS | PHYSIOLOGY | STAPHYLOCOCCUS-AUREUS SEPSIS | PERITUBULAR CAPILLARY DYSFUNCTION | CRITICAL ILLNESS | mitochondria | kidney | BIOGENESIS | SEPTIC SHOCK | UROLOGY & NEPHROLOGY | mitochondrial antioxidant | MICE | GENERATION | sepsis | oxidative stress | Mitochondria - enzymology | Electron Transport Complex III - metabolism | Kidney - blood supply | Kidney - pathology | Kidney - enzymology | Male | Electron Transport Complex I - metabolism | Microcirculation - drug effects | Electron Transport Complex IV - metabolism | Sepsis - drug therapy | Sepsis - pathology | Piperidines - pharmacology | Time Factors | Adenosine Triphosphate - metabolism | Cell Respiration - drug effects | Organophosphorus Compounds - pharmacology | Body Temperature Regulation - drug effects | Renal Circulation - drug effects | Superoxide Dismutase - metabolism | Disease Models, Animal | Kidney - drug effects | Acute Kidney Injury - pathology | Mice, Inbred C57BL | Antioxidants - pharmacology | Mitochondria - drug effects | Mitochondria - pathology | Acute Kidney Injury - enzymology | Sepsis - physiopathology | Acute Kidney Injury - physiopathology | Acute Kidney Injury - prevention & control | Animals | Electron Transport Complex II - metabolism | Sepsis - enzymology | Electron Transport Chain Complex Proteins - metabolism | Mice | Oxidative Stress - drug effects | Antioxidants | Superoxide | Genetic aspects | Health aspects | Enzymes | Kidney diseases | Rodents | Index Medicus
Journal Article