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PLoS ONE, ISSN 1932-6203, 08/2017, Volume 12, Issue 8, pp. e0182407 - e0182407
Retinal ischemia is common in eye disorders, like diabetic retinopathy or retinal vascular occlusion. The goal of this study was to evaluate the potential... 
OPTIC-NERVE | AUTOIMMUNE GLAUCOMA MODEL | PROLIFERATIVE DIABETIC-RETINOPATHY | PRESSURE-INDUCED ISCHEMIA | MULTIDISCIPLINARY SCIENCES | REPERFUSION INJURY | RAT RETINA | GANGLION-CELLS | MACULAR DEGENERATION | GLIAL-CELLS | ENDOTHELIAL GROWTH-FACTOR | Amacrine Cells - drug effects | Microglia - metabolism | Cell Count | Humans | Photoreceptor Cells, Vertebrate - drug effects | Amacrine Cells - metabolism | Retina | Ranibizumab - therapeutic use | Vascular Endothelial Growth Factor A - metabolism | Protective Agents - therapeutic use | Glial Fibrillary Acidic Protein - metabolism | RNA, Messenger - metabolism | Retinal Ganglion Cells - metabolism | Rhodopsin - metabolism | Aqueous Humor - metabolism | Synapses - metabolism | Protective Agents - pharmacology | Protein Binding - drug effects | Microfilament Proteins - metabolism | Ischemia - pathology | Disease Models, Animal | Calcium-Binding Proteins - metabolism | Synapses - drug effects | Microglia - drug effects | Reperfusion Injury - pathology | Cholinergic Neurons - metabolism | RNA, Messenger - genetics | Rats | Cholinergic Neurons - drug effects | Ranibizumab - pharmacology | Animals | Ischemia - drug therapy | Mice | Photoreceptor Cells, Vertebrate - metabolism | Retinal Ganglion Cells - drug effects | Retinal diseases | Care and treatment | Occlusion | Immunohistochemistry | Intervention | Diabetic retinopathy | Neurosciences | Retinopathy | Disorders | Neurobiology | Activation | Eye | Macular degeneration | Proteins | Cell growth | Ischemia | Rodents | Eye (anatomy) | Vascular endothelial growth factor | Retinal cells | Glial fibrillary acidic protein | Diabetes mellitus | Brn-3 protein | Microglia | Vascular endothelial growth factor receptors | Studies | Polymerase chain reaction | Retinal ganglion cells | Pathology | Hospitals | Cell number | Morphology | Eye disorders | Amacrine cells | Photoreceptors | Diabetes | Glia | Apoptosis | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2013, Volume 8, Issue 5, pp. e64233 - e64233
Journal Article
Journal Article
PLoS Medicine, ISSN 1549-1277, 04/2007, Volume 4, Issue 4, pp. 675 - 689
In multiple sclerosis, inflammation can successfully be prevented, while promoting repair is still a major challenge. Microglial cells, the resident phagocytes... 
MEDICINE, GENERAL & INTERNAL | Genetic Therapy | Neurons - pathology | Nitric Oxide Synthase Type II - biosynthesis | Receptors, Immunologic - physiology | Humans | Myeloid Cells - physiology | Cells, Cultured - transplantation | Genetic Vectors - therapeutic use | Multiple Sclerosis | Membrane Glycoproteins - physiology | Flow Cytometry | Microglia - physiology | Female | Lentivirus - genetics | Myeloid Cells - drug effects | Monocytes - physiology | Cytokines - genetics | Genes, Reporter | Disease Models, Animal | Encephalomyelitis, Autoimmune, Experimental - surgery | Promoter Regions, Genetic | Encephalomyelitis, Autoimmune, Experimental - pathology | Transduction, Genetic | Mice, Inbred C57BL | Gene Expression Regulation | Genes, Synthetic | Cells, Cultured - physiology | Granulocyte-Macrophage Colony-Stimulating Factor - pharmacology | Hematopoietic Stem Cell Transplantation | Genetic Vectors - genetics | Membrane Glycoproteins - genetics | Cell Lineage | Encephalomyelitis, Autoimmune, Experimental - therapy | Animals | Nitric Oxide Synthase Type II - genetics | Cell Differentiation - drug effects | Interleukin-10 - genetics | Interleukin-10 - biosynthesis | Myeloid Cells - transplantation | Mice | Receptors, Immunologic - genetics | Cytokines - biosynthesis | Phagocytosis | Apoptosis | Cell Movement | Multiple sclerosis | Biochemical genetics | Inflammation | Research | Neuroglia | Central nervous system | Index Medicus | Neurological Disorders | Neurology | Neuroscience | Immunology | Immunology and Allergy
Journal Article
Journal Article
Journal Article
Nature Neuroscience, ISSN 1097-6256, 04/2018, Volume 21, Issue 4, pp. 530 - 540
Journal Article
STEM CELLS, ISSN 1066-5099, 05/2017, Volume 35, Issue 5, pp. 1259 - 1272
Mesenchymal stromal cells (MSCs) are believed to mobilize from the bone marrow in response to inflammation and injury, yet the effects of egress into the... 
Mesenchymal stromal cells | Shear stress | Biomechanical force | Inflammation | Traumatic brain injury | Immunomodulation | ACTIVATED MICROGLIAL/MACROPHAGE RESPONSE | STEM-CELLS | FLUID SHEAR-STRESS | MACROPHAGES | BLOOD-FLOW | CELL & TISSUE ENGINEERING | CELL BIOLOGY | TRAUMATIC BRAIN-INJURY | ONCOLOGY | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | ENDOTHELIAL-CELLS | GENE-EXPRESSION | HEME OXYGENASE-1 | HEMATOLOGY | Brain Injuries, Traumatic - therapy | Inflammation - pathology | Dinoprostone - biosynthesis | Humans | Stress, Mechanical | NF-kappa B - metabolism | Anti-Inflammatory Agents - metabolism | Mesenchymal Stromal Cells - immunology | Mesenchymal Stromal Cells - cytology | Bone Marrow Cells - immunology | Signal Transduction | Bone Marrow Cells - cytology | Rheology | Mice, Inbred C57BL | Mesenchymal Stromal Cells - metabolism | Rats | Administration, Intravenous | Biomechanical Phenomena | Phenotype | Animals | Brain Injuries, Traumatic - pathology | Bioreactors | Cyclooxygenase 2 - metabolism | Mesenchymal Stem Cell Transplantation | Antioxidants | Brain | Immunotherapy | Analysis | Stem cells | Stress (Psychology) | Injuries | Bone marrow | Tumor necrosis factor-TNF | Rodents | Mesenchyme | Prostaglandin E2 | Stem cell transplantation | Paracrine signalling | Egress | Recruitment | Head injuries | Conditioning | Cues | Microglia | Immune systems | Biomechanics | Tumor necrosis factor | Stromal cells | Mechanical stimuli | Cyclooxygenase-2 | Hippocampus | Chemokines | Apoptosis | Index Medicus | traumatic brain injury | immunomodulation | shear stress | inflammation | mesenchymal stromal cells
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 9/2008, Volume 105, Issue 38, pp. 14638 - 14643
Human mesenchymal stromal cells (hMSCs) were injected into the hippocampus of adult mice 1 day after transient global ischemia. The hMSCs both improved... 
Transcriptomes | Ischemia | Cytokines | Genes | Stromal cells | Mice | Macrophages | Hippocampus | Microglia | Mesenchymal stem cells | Mesenchymal stromal cells | Inflammation | MULTIDISCIPLINARY SCIENCES | mesenchymal stromal cells | LUNG INJURY | MICROGLIAL CELLS | MESENCHYMAL STEM-CELLS | TRANSPLANTATION | REPAIR | inflammation | mesenchymal stem cells | IMMUNOSUPPRESSION | MICE | STROMAL CELLS | microglia | ALTERNATIVE ACTIVATION | Humans | Cell Death - immunology | Neurons - cytology | Galectin 3 - immunology | Gene Expression Profiling | Mesenchymal Stromal Cells - immunology | Microglia - immunology | Bone Marrow Cells - immunology | beta-N-Acetylhexosaminidases - genetics | Cytokines - genetics | Ischemia - pathology | Hippocampus - immunology | Hippocampus - blood supply | Gene Expression Regulation - immunology | Macrophage Activation - immunology | Mice, Inbred C57BL | Neurons - immunology | Ischemia - therapy | Inflammation - immunology | Cerebrovascular Disorders - therapy | Hippocampus - cytology | Antigen-Presenting Cells - immunology | Animals | Lectins - genetics | Cerebrovascular Disorders - pathology | Mesenchymal Stem Cell Transplantation | Evaluation | Cerebral ischemia | Immune response | Stem cells | Bone marrow | Development and progression | Hippocampus (Brain) | Properties | Proteins | Brain | Neurons | Apoptosis | Index Medicus | Biological Sciences
Journal Article