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Autophagy, ISSN 1554-8635, 10/2014, Volume 5, Issue 1, pp. 19 - 32
Journal Article
Nature (London), ISSN 1476-4687, 06/2015, Volume 522, Issue 7556, pp. 354 - 358
Journal Article
Neuron (Cambridge, Mass.), ISSN 0896-6273, 10/2003, Volume 40, Issue 3, pp. 501 - 514
Targeted deletion of focal adhesion kinase ( fak) in the developing dorsal forebrain resulted in local disruptions of the cortical basement membrane located... 
Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Immunohistochemistry | Membrane Glycoproteins - metabolism | Embryo, Mammalian | Protein-Tyrosine Kinases - metabolism | Homeodomain Proteins - metabolism | Astrocytes - pathology | Cerebral Cortex - pathology | Glial Fibrillary Acidic Protein - metabolism | Muscular Dystrophies - genetics | Protein-Tyrosine Kinases - deficiency | Focal Adhesion Kinase 1 | Calbindins | Neurons - metabolism | Serine Endopeptidases | Disease Models, Animal | Fibroblasts - metabolism | Muscular Dystrophies - metabolism | Muscular Dystrophies - congenital | Silver Staining | Nerve Tissue Proteins | Basement Membrane - metabolism | Blotting, Western | Precipitin Tests | Mice, Knockout | Infection | Staining and Labeling | Mice | Mutation | Dura Mater | Astrocytes - metabolism | Neurons - pathology | Microtubule-Associated Proteins - metabolism | Otx Transcription Factors | Phosphopyruvate Hydratase - metabolism | Basement Membrane - pathology | Dystroglycans | Cerebral Cortex - metabolism | Cerebral Cortex - ultrastructure | Phosphotyrosine - metabolism | DNA-Binding Proteins - metabolism | Protein-Tyrosine Kinases - genetics | Lamins - metabolism | src-Family Kinases - metabolism | Cytoskeletal Proteins - metabolism | Cell Adhesion Molecules, Neuronal - metabolism | Cerebral Cortex - abnormalities | Extracellular Matrix Proteins - metabolism | Fibroblasts - virology | Cells, Cultured | Focal Adhesion Protein-Tyrosine Kinases | Microscopy, Electron | Muscular Dystrophies - pathology | S100 Calcium Binding Protein G - metabolism | Transcription Factors - metabolism | Animals | Carrier Proteins - metabolism | Calbindin 2 | Bacterial Proteins - metabolism | Heterozygote | Proteins | Neurons | Rodents | Protein expression | Kinases | Embryos | Muscular dystrophy | Deoxyribonucleic acid--DNA | Cell adhesion & migration | Index Medicus
Journal Article
Autophagy, ISSN 1554-8627, 11/2012, Volume 8, Issue 11, pp. 1628 - 1642
.... We previously demonstrated that deficiency or blockade of the ubiquitously expressed receptor CD47 results in remarkable cell and tissue protection against ischemic and radiation stress... 
BECN1 | CD47 | autophagosome | ATG7 | ionizing radiation | MAP1A/1BLC3 | ATG5 | Binding | Proteins | Landes | Calcium | Bioscience | Biology | Cell | Cycle | Cancer | Organogenesis | Ionizing radiation | Autophagosome | p62 | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Whole-Body Irradiation | Human Umbilical Vein Endothelial Cells - metabolism | Microtubule-Associated Proteins - genetics | Apoptosis - radiation effects | Microtubule-Associated Proteins - metabolism | Sequestosome-1 Protein | Gene Silencing - radiation effects | Humans | Up-Regulation - radiation effects | Radiation Protection | Human Umbilical Vein Endothelial Cells - cytology | Apoptosis Regulatory Proteins - genetics | Phagosomes - ultrastructure | Membrane Proteins - metabolism | Autophagy - genetics | Radiation, Ionizing | Beclin-1 | CD47 Antigen - genetics | Ubiquitin-Activating Enzymes - genetics | Jurkat Cells | Membrane Proteins - genetics | Mice, Inbred C57BL | Phagosomes - metabolism | CD47 Antigen - metabolism | Ubiquitin-Activating Enzymes - metabolism | Apoptosis Regulatory Proteins - metabolism | Autophagy-Related Protein 7 | Organ Specificity - radiation effects | Animals | Autophagy-Related Protein 5 | Gene Expression Regulation - radiation effects | Mice | Adaptor Proteins, Signal Transducing - metabolism | Human Umbilical Vein Endothelial Cells - radiation effects | Index Medicus | MAP1A | Basic Research Paper | 1BLC3
Journal Article
Neurobiology of Disease, ISSN 0969-9961, 2014, Volume 71, Issue 1, pp. 234 - 244
Journal Article
Arthritis & rheumatology (Hoboken, N.J.), ISSN 2326-5191, 07/2017, Volume 69, Issue 7, pp. 1418 - 1428
Objective Regulated in development and DNA damage response 1 (REDD1) is an endogenous inhibitor of mechanistic target of rapamycin (mTOR) that regulates... 
Life Sciences & Biomedicine | Rheumatology | Science & Technology | Immunohistochemistry | AMP-Activated Protein Kinases - metabolism | Arthritis, Experimental - genetics | Oxidative Stress | Microtubule-Associated Proteins - metabolism | Humans | Apoptosis - genetics | Arthritis, Experimental - metabolism | DNA-Binding Proteins - metabolism | Organelle Biogenesis | Cartilage, Articular - metabolism | Menisci, Tibial - surgery | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha - metabolism | Autophagy - genetics | Chondrocytes - metabolism | Disease Models, Animal | High Mobility Group Proteins - metabolism | Mitochondria - metabolism | Transcription Factors - genetics | Synovial Membrane - metabolism | Mice, Knockout | Animals | Osteoarthritis, Knee - genetics | Autophagy-Related Protein 5 - metabolism | Mice | Osteoarthritis, Knee - metabolism | TOR protein | Oxidative stress | DNA damage | Homeostasis | Biosynthesis | Kinases | AMP-activated protein kinase | Autophagy | Synovium | Proteins | Cartilage | Mitochondria | Biomedical materials | Clonal deletion | Surgery | Microtubule-associated protein 1 | Biocompatibility | Damage | Menisci | Deoxyribonucleic acid--DNA | Meniscus | AMP | Markers | Rapamycin | Cartilage (articular) | Bone (subchondral) | Inhibitors | Protein kinase | Chondrocytes | Death | In vivo methods and tests | Osteoarthritis | ATP | Destabilization | In vitro methods and tests | Phagocytosis | Cellular stress response | Apoptosis | Cartilage diseases | Index Medicus | Abridged Index Medicus
Journal Article
Molecular cell, ISSN 1097-2765, 11/2011, Volume 44, Issue 4, pp. 660 - 666
How pseudouridylation (Ψ), the most common and evolutionarily conserved modification of rRNA, regulates ribosome activity is poorly understood. Medically, Ψ is... 
Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Protein Biosynthesis | Hydro-Lyases - deficiency | Microcephaly - genetics | Microtubule-Associated Proteins - genetics | Saccharomyces cerevisiae - genetics | Humans | Ribonucleoproteins, Small Nuclear - genetics | Ribosomes - metabolism | Fetal Growth Retardation - genetics | Microcephaly - enzymology | Dyskeratosis Congenita - genetics | RNA, Ribosomal - genetics | Intellectual Disability - genetics | Nuclear Proteins - deficiency | Cell Cycle Proteins - genetics | Intellectual Disability - enzymology | RNA, Transfer - genetics | Hydro-Lyases - metabolism | Hydro-Lyases - genetics | Microtubule-Associated Proteins - deficiency | Nuclear Proteins - genetics | RNA, Transfer - chemistry | Binding Sites | Fetal Growth Retardation - enzymology | Genes, Reporter | Transduction, Genetic | Luciferases - analysis | RNA, Transfer - metabolism | RNA, Ribosomal - chemistry | Ribosomes - chemistry | RNA, Ribosomal - metabolism | Ribonucleoproteins, Small Nuclear - deficiency | Cell Cycle Proteins - deficiency | Saccharomyces cerevisiae Proteins - genetics | Dyskeratosis Congenita - enzymology | Sequence Homology, Amino Acid | Animals | Plasmids | Saccharomyces cerevisiae - enzymology | Mice | Mutation | Cricket | Chemical properties | Paralysis | Molecular genetics | Cells | Transfer RNA | Index Medicus | Dyskeratosis | Fidelity | RNA modification | Internal ribosome entry site | Translation | tRNA | rRNA | Ribosomes | X chromosome | pseudouridylation | Evolution
Journal Article
EMBO molecular medicine, ISSN 1757-4684, 02/2013, Volume 5, Issue 3, pp. 397 - 412
Alpha‐1‐anti‐trypsin deficiency is the most common genetic cause of liver disease in children and liver transplantation is currently the only available treatment... 
TFEB | gene therapy | alpha‐1‐anti‐trypsin | autophagy | helper‐dependent adenoviral vector | Alpha-1-anti-trypsin | Helper-dependent adenoviral vector | Gene therapy | Autophagy | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Liver - pathology | Liver - enzymology | Microtubule-Associated Proteins - genetics | Humans | NF-kappa B - metabolism | Mutation, Missense | Lysosomes - metabolism | Transfection | Time Factors | Liver Cirrhosis - metabolism | Autophagy - genetics | Microtubule-Associated Proteins - deficiency | Interleukin-6 - metabolism | Liver Cirrhosis - genetics | Disease Models, Animal | alpha 1-Antitrypsin Deficiency - genetics | Gene Transfer Techniques | Genetic Predisposition to Disease | alpha 1-Antitrypsin - genetics | alpha 1-Antitrypsin Deficiency - therapy | Mice, Inbred C57BL | Mice, Transgenic | Liver Cirrhosis - therapy | alpha 1-Antitrypsin Deficiency - metabolism | Mice, Knockout | Papio | Autophagy-Related Protein 7 | Phenotype | Animals | alpha 1-Antitrypsin Deficiency - complications | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - genetics | Liver Cirrhosis - pathology | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - metabolism | Mice | alpha 1-Antitrypsin - metabolism | HeLa Cells | Apoptosis | Genetic Therapy - methods | Proteins | Trypsin | Liver diseases | Genes | Genetic engineering | Genetic transcription | Health aspects | Phenotypes | Gene transfer | Syngeneic grafts | Cloning | Transgenic mice | Gene expression | Medical screening | Studies | Allografts | Plasmids | Fibrosis | Polymers | Phagocytosis | Deoxyribonucleic acid--DNA | Liver transplantation | Index Medicus | helper-dependent adenoviral vector | alpha-1-anti-trypsin
Journal Article