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Journal of Clinical Investigation, ISSN 0021-9738, 05/2009, Volume 119, Issue 5, pp. 1298 - 1311
Idiopathic pulmonary fibrosis (IPF) can lead to the development of secondary pulmonary hypertension (PH) and ultimately death. Despite this known association,... 
MEDICINE, RESEARCH & EXPERIMENTAL | EPITHELIUM-DERIVED FACTOR | GROWTH-FACTOR-BETA | ANGIOGENESIS | ARTERIAL-HYPERTENSION | DISEASE | SMOOTH-MUSCLE-CELLS | PROLIFERATION | GENE-TRANSFER | EXPRESSION | PIGMENT-EPITHELIUM | Genetic Therapy | Idiopathic Pulmonary Fibrosis - therapy | Apoptosis - drug effects | Gene Expression - genetics | Humans | Transforming Growth Factor beta1 - metabolism | Caspase 3 - metabolism | Hypertension, Pulmonary - physiopathology | Microvessels - pathology | Idiopathic Pulmonary Fibrosis - chemically induced | Nerve Growth Factors - metabolism | Vascular Endothelial Growth Factor A - metabolism | Hypertension, Pulmonary - therapy | Idiopathic Pulmonary Fibrosis - metabolism | Vascular Endothelial Growth Factor Receptor-2 - genetics | Pulmonary Artery - metabolism | Idiopathic Pulmonary Fibrosis - complications | Vascular Endothelial Growth Factor A - pharmacology | Transforming Growth Factor beta1 - pharmacology | Disease Models, Animal | Fibroblasts - metabolism | Serpins - genetics | Endothelial Cells - metabolism | Smad2 Protein - metabolism | Rats | Lung - physiopathology | Nitric Oxide Synthase Type III - genetics | Rats, Sprague-Dawley | Eye Proteins - metabolism | Models, Biological | Nerve Growth Factors - genetics | Fibroblasts - drug effects | Idiopathic Pulmonary Fibrosis - pathology | Bronchoalveolar Lavage Fluid - chemistry | Hypertension, Pulmonary - etiology | Serum Albumin - metabolism | Phosphorylation | Female | Lung - metabolism | Eye Proteins - genetics | Lung - pathology | Serpins - metabolism | Vascular Endothelial Growth Factor A - therapeutic use | Microvessels - drug effects | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Transforming Growth Factor beta1 - genetics | Animals | Endothelial Cells - cytology | Pulmonary Artery - pathology | Endothelial Cells - drug effects | Immunohistochemistry | Usage | Care and treatment | Genetic aspects | Research | Health aspects | Vascular endothelial growth factor | Pulmonary hypertension | Risk factors | Apoptosis | Index Medicus | Abridged Index Medicus
Journal Article
The American Journal of Pathology, ISSN 0002-9440, 10/2017, Volume 187, Issue 10, pp. 2337 - 2347
Journal Article
PLoS ONE, ISSN 1932-6203, 12/2012, Volume 7, Issue 12, pp. e52279 - e52279
Angiogenesis, the formation of new blood vessels from pre-existing vascular beds, is essential for tumor growth, invasion, and metastasis. Luteolin is a common... 
MAMMALIAN TARGET | MATRIX METALLOPROTEINASES | APOPTOSIS | NECROSIS-FACTOR | ACTIVATION | PATHWAY | VEGF | MULTIDISCIPLINARY SCIENCES | CELL INVASION | DOWN-REGULATION | CANCER | TOR Serine-Threonine Kinases - metabolism | Humans | Male | Extracellular Signal-Regulated MAP Kinases - metabolism | Human Umbilical Vein Endothelial Cells - cytology | Chorioallantoic Membrane - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Prostatic Neoplasms - blood supply | Cell Survival - drug effects | Prostatic Neoplasms - pathology | Human Umbilical Vein Endothelial Cells - drug effects | Ribosomal Protein S6 Kinases, 70-kDa - metabolism | Neoplasm Invasiveness | Aorta - drug effects | Microvessels - drug effects | Rats | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Luteolin - therapeutic use | Luteolin - pharmacology | Chick Embryo | Xenograft Model Antitumor Assays | Cell Movement - drug effects | Microvessels - physiology | Animals | Signal Transduction - drug effects | Neovascularization, Pathologic - drug therapy | Cell Line, Tumor | Cell Proliferation - drug effects | Mice | Matrix Metalloproteinases - metabolism | In Vitro Techniques | Aorta - physiology | Cytokines - biosynthesis | Immunohistochemistry | Flavonoids | Care and treatment | Flavones | Bioflavonoids | Analysis | Neovascularization | Vascular endothelial growth factor | Prostate cancer | Tumors | Cancer | Vegetables | Matrix metalloproteinases | AKT protein | Metastasis | Assaying | Metastases | Angiogenesis | Signal transduction | Toxicology | Cell growth | Gelatin | Biocompatibility | Aorta | Physiology | Inhibition | Medical research | Cytokines | Internal medicine | Blood vessels | Diet | Antiangiogenics | Weight reduction | Hypoxia | Viability | Cell migration | Cell proliferation | Animal models | Phosphorylation | Kinases | Interleukin 6 | Rodents | Xenografts | Extracellular matrix | Tumor necrosis factor-TNF | Vascular endothelial growth factor receptor 2 | Interleukin 8 | CD34 antigen | Extracellular signal-regulated kinase | Inflammation | Endothelial cells | Gelatinase B | Gelatinase A | Medicine | Chorioallantoic membrane | In vivo methods and tests | Prostate | Apoptosis | Index Medicus
Journal Article
BMC Cancer, ISSN 1471-2407, 11/2010, Volume 10, Issue 1, pp. 659 - 659
Journal Article
Journal Article
Cancer Cell, ISSN 1535-6108, 2011, Volume 19, Issue 1, pp. 31 - 44
Polarization of tumor-associated macrophages (TAMs) to a proangiogenic/immune-suppressive (M2-like) phenotype and abnormal, hypoperfused vessels are hallmarks... 
Clodronic Acid - pharmacology | Gene Expression - genetics | Dendritic Cells - immunology | Humans | Pregnancy Proteins - genetics | Microvessels - pathology | Pregnancy Proteins - metabolism | Chemotactic Factors - metabolism | Neoplasm Metastasis - immunology | Antibodies - immunology | Neovascularization, Pathologic - immunology | Macrophages, Peritoneal - drug effects | Macrophages - pathology | Neoplasms - blood supply | Down-Regulation - genetics | Mice, Knockout | Macrophages - metabolism | Neoplasms - immunology | Cell Line, Tumor | Placenta Growth Factor | Mice | Mice, Inbred BALB C | CD8-Positive T-Lymphocytes - immunology | Macrophages, Peritoneal - metabolism | Neoplasms - metabolism | CD8-Positive T-Lymphocytes - pathology | Gene Expression - drug effects | Regional Blood Flow - genetics | Dendritic Cells - pathology | Culture Media, Conditioned - pharmacology | Lung Neoplasms - pathology | Hypoxia - metabolism | Neovascularization, Pathologic - pathology | Regional Blood Flow - drug effects | Transfection | Lung Neoplasms - secondary | Neoplasms - genetics | Dendritic Cells - metabolism | Macrophages - immunology | Lung Neoplasms - genetics | Microvessels - ultrastructure | Cytokines - metabolism | Mice, Inbred C57BL | Microvessels - drug effects | Pregnancy Proteins - immunology | Antibodies - pharmacology | Proteins - genetics | Neoplasm Metastasis - genetics | Hypoxia - genetics | Lung Neoplasms - immunology | Animals | Proteins - metabolism | Endothelial Cells - cytology | Neoplasm Metastasis - pathology | Macrophages - drug effects | Neovascularization, Pathologic - genetics | Cell Proliferation - drug effects | Proteins - pharmacology | Neoplasms - pathology | Endothelial Cells - drug effects | Index Medicus
Journal Article
Biomaterials, ISSN 0142-9612, 2013, Volume 34, Issue 36, pp. 9201 - 9209
Journal Article
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 04/2009, Volume 15, Issue 8, pp. 2695 - 2702
Journal Article
International Journal of Cancer, ISSN 0020-7136, 02/2013, Volume 132, Issue 4, pp. 755 - 765
New approaches are needed to the therapy of advanced prostate cancer. This study determined the effect of growth hormone‐releasing hormone (GHRH) antagonists,... 
prostate cancer | GHRH antagonists | metastasis | GHRH | angiogenesis | MICROVESSEL DENSITY | PROLIFERATION | E-CADHERIN | BETA-CATENIN | CELL-LINES | BREAST-CANCER | INVASION | ONCOLOGY | GENE-EXPRESSION | Cell Proliferation | Cadherins - metabolism | Humans | Male | Receptors, Neuropeptide - metabolism | Growth Hormone-Releasing Hormone - pharmacology | Matrix Metalloproteinase 9 - metabolism | Neoplasm Metastasis - drug therapy | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Receptors, Pituitary Hormone-Regulating Hormone - metabolism | Sermorelin - pharmacology | Sermorelin - analogs & derivatives | Antineoplastic Agents - pharmacology | Prostatic Neoplasms - drug therapy | Prostatic Neoplasms - pathology | Matrix Metalloproteinase 2 - metabolism | Neoplasm Invasiveness | Random Allocation | Cell Adhesion | beta Catenin - metabolism | Xenograft Model Antitumor Assays | Animals | Mice, Nude | Neovascularization, Pathologic - drug therapy | Cell Line, Tumor | Vascular Endothelial Growth Factors - metabolism | Growth Hormone-Releasing Hormone - analogs & derivatives | Mice | Growth Hormone-Releasing Hormone - antagonists & inhibitors | Care and treatment | Pituitary hormones | Somatotropin releasing hormone | Growth | Oncology, Experimental | Development and progression | Metastasis | Research | Cells | Endothelium | Heterocyclic compounds | Analysis | Health aspects | Vascular endothelial growth factor | Prostate cancer | Enzyme-linked immunosorbent assay | Cancer | Growth hormones | Cancer therapies | Index Medicus
Journal Article