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Molecular Neurobiology, ISSN 0893-7648, 5/2016, Volume 53, Issue 4, pp. 2451 - 2467
...) disruption and synaptic dysfunction, leading to AD pathology is not clear. Therefore, we hypothesized that the inhibition of neuronal NMDA-R by H2S and MK801 mitigate... 
Cerebrovascular pathology | Neurology | Neurosciences | Biomedicine | Blood–brain barrier dysfunction | Alzheimer’s disease | Neurobiology | Homocysteine | Cell Biology | Dementia | COGNITIVE PERFORMANCE | NEUROSCIENCES | MATRIX METALLOPROTEINASES | INDUCED MEMORY IMPAIRMENT | RISK-FACTOR | Blood-brain barrier dysfunction | RECEPTOR TRAFFICKING | MATRIX-METALLOPROTEINASE-9 | RAT-BRAIN | Alzheimer's disease | PLASMA HOMOCYSTEINE | EXPRESSION | Memory - drug effects | Glial Fibrillary Acidic Protein - genetics | Cadherins - metabolism | Microtubule-Associated Proteins - genetics | Microtubule-Associated Proteins - metabolism | Blood-Brain Barrier - physiopathology | Claudin-5 - metabolism | Microvessels - pathology | Male | Synapses - pathology | Glial Fibrillary Acidic Protein - metabolism | RNA, Messenger - metabolism | Antigens, CD - genetics | Alzheimer Disease - pathology | Antigens, CD - metabolism | Matrix Metalloproteinase 9 - metabolism | Cadherins - genetics | Synapses - drug effects | Alzheimer Disease - physiopathology | Hydrogen Sulfide - pharmacology | Cerebrovascular Circulation - drug effects | Matrix Metalloproteinase 2 - metabolism | Mice, Inbred C57BL | RNA, Messenger - genetics | Alzheimer Disease - drug therapy | Microvessels - drug effects | Claudin-5 - genetics | Cystathionine beta-Synthase - metabolism | Permeability | Blood-Brain Barrier - drug effects | Nerve Tissue Proteins - genetics | Blood-Brain Barrier - metabolism | Gene Expression Regulation - drug effects | Nerve Tissue Proteins - metabolism | Blood-Brain Barrier - pathology | Hydrogen Sulfide - therapeutic use | Intercellular Adhesion Molecule-1 - metabolism | Animals | Intercellular Adhesion Molecule-1 - genetics | Avoidance Learning - drug effects | Alzheimer Disease - genetics | Dizocilpine Maleate - pharmacology | Hydrogen sulfide | Methyl aspartate | Brain | RNA | Neurons | Intermediate filament proteins | cerebrovascular pathology | dementia | blood brain barrier dysfunction
Journal Article
Circulation (New York, N.Y.), ISSN 1524-4539, 2015, Volume 131, Issue 6, pp. 550 - 559
Journal Article
Diabetes (New York, N.Y.), ISSN 1939-327X, 2018, Volume 67, Issue 9, pp. 1867 - 1879
Intermittent fasting (IF) protects against the development of metabolic diseases and cancer, but whether it can prevent diabetic microvascular complications is... 
DIABETIC-RETINOPATHY | BILE-ACID | CELLS | ACTIVATION | BACTERIA | OBESITY | METABOLISM | FARNESOID X RECEPTOR | C57BL/6 MICE | ENDOCRINOLOGY & METABOLISM | TAUROURSODEOXYCHOLIC ACID | Retina - drug effects | Leukocytes - pathology | Receptors, G-Protein-Coupled - metabolism | Colon - drug effects | Microvessels - metabolism | Microvessels - pathology | Colon - immunology | Male | Ganglia, Sensory - metabolism | Retina - immunology | Intestinal Mucosa - drug effects | Diabetic Retinopathy - complications | Retinal Vessels - pathology | Feces - microbiology | Intestinal Mucosa - immunology | Bacteroidetes - isolation & purification | Firmicutes - growth & development | Diabetic Retinopathy - immunology | Firmicutes - isolation & purification | Retinal Vessels - metabolism | Colon - pathology | Retinal Vessels - drug effects | Dysbiosis - therapy | Goblet Cells - metabolism | Colon - metabolism | Ganglia, Sensory - pathology | Diabetic Retinopathy - prevention & control | Gastrointestinal Microbiome - drug effects | Bile Acids and Salts - therapeutic use | Survival Analysis | Leukocytes - drug effects | Diabetes Mellitus, Type 2 - pathology | Dysbiosis - pathology | Retina - pathology | Intestinal Mucosa - pathology | Intestinal Mucosa - metabolism | Retina - metabolism | Diabetes Mellitus, Type 2 - microbiology | Ganglia, Sensory - drug effects | Goblet Cells - drug effects | Diabetic Retinopathy - pathology | Receptors, G-Protein-Coupled - agonists | Dysbiosis - microbiology | Microvessels - immunology | Ganglia, Sensory - immunology | Goblet Cells - immunology | Leukocytes - immunology | Retinal Vessels - immunology | Mice, Mutant Strains | Diabetes Mellitus, Type 2 - therapy | Bacteroidetes - immunology | Mice, Inbred DBA | Verrucomicrobia - growth & development | Verrucomicrobia - immunology | Diabetes Mellitus, Type 2 - complications | Fasting | Microvessels - drug effects | Firmicutes - immunology | Verrucomicrobia - isolation & purification | Dysbiosis - complications | Animals | Bacteroidetes - growth & development | Gastrointestinal Microbiome - immunology | Goblet Cells - pathology | Prevention | Diabetic retinopathy | Usage | Diet therapy | Low-calorie diet | Rattus | Rats | Health aspects | Complications | 0107
Journal Article
PloS one, ISSN 1932-6203, 2012, Volume 7, Issue 5, p. e36893
...) and neurofibrillary tangles do not completely account for cognitive impairment, therefore other factors such as cardiovascular and cerebrovascular pathologies, may contribute to AD... 
TYPE-4 ALLELE | DIAGNOSIS | MICROVASCULAR PATHOLOGY | RISK-FACTORS | VASCULAR DEMENTIA | MULTIDISCIPLINARY SCIENCES | CEREBRAL-BLOOD-FLOW | APOLIPOPROTEIN-E | SENILE PLAQUES | DEGENERATION | AGE | Tyrosine 3-Monooxygenase - metabolism | Capillaries - pathology | Humans | Cerebral Cortex - pathology | Microvessels - metabolism | Microvessels - pathology | Male | Cerebral Cortex - metabolism | Alzheimer Disease - pathology | Brain - blood supply | Microcirculation | Vasoconstriction - physiology | Vesicular Acetylcholine Transport Proteins - metabolism | Dementia - metabolism | Aged, 80 and over | Neurons, Afferent - pathology | Female | Vasodilation - physiology | Capillaries - metabolism | Apolipoprotein E4 - metabolism | Dementia - pathology | Plaque, Amyloid - pathology | Cholinergic Neurons - metabolism | Aging - pathology | Immunohistochemistry - methods | Alzheimer Disease - metabolism | Plaque, Amyloid - metabolism | Aged | Neurons, Afferent - metabolism | Cholinergic Neurons - pathology | Aging - metabolism | Cohort Studies | Immunohistochemistry | Brain | Advertising executives | Dilatation | Blood vessels | Apolipoproteins | Alzheimer's disease | Pathogenesis | Innervation | Cognitive ability | Cardiovascular disease | Biochemistry | Vasodilation | Consortia | Cerebrovascular system | Angiogenesis | Immunotherapy | Aging | Tyrosine | Medical research | Neurodegenerative diseases | Hydroxylase | Cortex | Carriers | Neurofibrillary tangles | Morphology | Biomarkers | Transporter | Dementia | Structural integrity | Neuropathology | Staining | Tyrosine 3-monooxygenase | Substantia grisea | Vesicular acetylcholine transporter | Macular degeneration | Cerebral amyloid angiopathy | Rodents | Dementia disorders | Evolution | Amyloid | Capillaries | Plaques | Age | Medical imaging | Review boards | Vasoconstriction | Ethnicity | Brain research | Acetylcholine | Norepinephrine | Microvasculature
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 12/2013, Volume 127, Issue 5, pp. 691 - 700
Journal Article
Stroke (1970), ISSN 0039-2499, 01/2015, Volume 46, Issue 1, pp. 182 - 189
Journal Article
Journal Article
Journal of Endodontics, ISSN 0099-2399, 2012, Volume 38, Issue 3, pp. 367 - 371
Abstract Introduction The purpose of this study was to evaluate the biocompatibility of calcium aluminate cement (EndoBinder) in subcutaneous tissue of rats.... 
Endocrinology & Metabolism | Dentistry | tissue reaction | Biocompatibility | mineral trioxide aggregate | calcium aluminate cement | RESORPTION | SUBCUTANEOUS CONNECTIVE-TISSUE | PULP | PROROOT MTA | SOLUBILITY | DENTIN SIALOPROTEIN | ROOT-CANAL SEALER | DENTISTRY, ORAL SURGERY & MEDICINE | RADIOPACITY | HYDROXIDE | Inflammation - pathology | Neovascularization, Physiologic - drug effects | Rats, Wistar | Giant Cells - drug effects | Connective Tissue - drug effects | Microvessels - pathology | Male | Phagocytes - pathology | Necrosis | Plasma Cells - pathology | Plasma Cells - drug effects | Collagen - drug effects | Time Factors | Intubation - instrumentation | Calcium Compounds - pharmacology | Neutrophils - pathology | Silicates - pharmacology | Subcutaneous Tissue - pathology | Leukocytes, Mononuclear - drug effects | Connective Tissue - pathology | Macrophages - pathology | Phagocytes - drug effects | Neutrophils - drug effects | Microvessels - drug effects | Rats | Materials Testing | Biocompatible Materials - pharmacology | Fibroblasts - pathology | Polyethylene - chemistry | Leukocytes, Mononuclear - pathology | Oxides - pharmacology | Lymphocytes - pathology | Animals | Fibroblasts - drug effects | Lymphocytes - drug effects | Macrophages - drug effects | Root Canal Filling Materials - pharmacology | Aluminum Compounds - pharmacology | Edema - pathology | Drug Combinations | Giant Cells - pathology | Subcutaneous Tissue - drug effects
Journal Article
Cancer cell, ISSN 1535-6108, 2011, Volume 19, Issue 1, pp. 31 - 44
Polarization of tumor-associated macrophages (TAMs) to a proangiogenic/immune-suppressive (M2-like) phenotype and abnormal, hypoperfused vessels are hallmarks... 
Clodronic Acid - pharmacology | Gene Expression - genetics | Dendritic Cells - immunology | Humans | Pregnancy Proteins - genetics | Microvessels - pathology | Pregnancy Proteins - metabolism | Chemotactic Factors - metabolism | Neoplasm Metastasis - immunology | Antibodies - immunology | Neovascularization, Pathologic - immunology | Macrophages, Peritoneal - drug effects | Macrophages - pathology | Neoplasms - blood supply | Down-Regulation - genetics | Mice, Knockout | Macrophages - metabolism | Neoplasms - immunology | Cell Line, Tumor | Placenta Growth Factor | Mice | Mice, Inbred BALB C | CD8-Positive T-Lymphocytes - immunology | Macrophages, Peritoneal - metabolism | Neoplasms - metabolism | CD8-Positive T-Lymphocytes - pathology | Gene Expression - drug effects | Regional Blood Flow - genetics | Dendritic Cells - pathology | Culture Media, Conditioned - pharmacology | Lung Neoplasms - pathology | Hypoxia - metabolism | Neovascularization, Pathologic - pathology | Regional Blood Flow - drug effects | Transfection | Lung Neoplasms - secondary | Neoplasms - genetics | Dendritic Cells - metabolism | Macrophages - immunology | Lung Neoplasms - genetics | Microvessels - ultrastructure | Cytokines - metabolism | Mice, Inbred C57BL | Microvessels - drug effects | Pregnancy Proteins - immunology | Antibodies - pharmacology | Proteins - genetics | Neoplasm Metastasis - genetics | Hypoxia - genetics | Lung Neoplasms - immunology | Animals | Proteins - metabolism | Endothelial Cells - cytology | Neoplasm Metastasis - pathology | Macrophages - drug effects | Neovascularization, Pathologic - genetics | Cell Proliferation - drug effects | Proteins - pharmacology | Neoplasms - pathology | Endothelial Cells - drug effects
Journal Article
Journal Article
PloS one, ISSN 1932-6203, 2016, Volume 11, Issue 1, p. e0146829
.... This is partly due to bone marrow (BM) pathology leading to increased activity of BM-derived pro-inflammatory monocytes and impaired function of BM-derived reparative circulating angiogenic cells (CACs... 
PATHOGENESIS | ENDOTHELIAL PROGENITOR CELLS | MICROGLIAL RESPONSE | DENDRITIC CELLS | MULTIDISCIPLINARY SCIENCES | HEMATOPOIETIC STEM-CELLS | MONOCYTES | BLOOD-VESSEL FORMATION | MOUSE RETINA | DIFFERENTIATION | SDF-1/CXCR4 AXIS | Green Fluorescent Proteins - metabolism | Inflammation - pathology | Diabetic Retinopathy - immunology | Bone Marrow Cells - cytology | Cell Count | Mice, Inbred C57BL | Dendritic Cells - pathology | Microvessels - drug effects | Microvessels - pathology | Male | Diabetic Retinopathy - pathology | Animals | Bone Marrow Transplantation | Lipopolysaccharides - pharmacology | Monocytes - pathology | Microglia - pathology | Endothelial Cells - pathology | Spleen - pathology | Retina - pathology | Chimera | Bone marrow | Diabetic retinopathy | Complications and side effects | Development and progression | Diabetes | Health aspects | Homing behavior | Neurosciences | Retinopathy | Leukocyte migration | Laboratories | Streptozocin | Retina | Transplantation | Biology | Activation | Lipopolysaccharides | Homing | Angiogenesis | Cell activation | Splenocytes | Rodents | Physiology | Repair | Drug dosages | Spleen | Dendritic cells | Diabetes mellitus | Markers | Inflammation | Progenitor cells | Endothelial cells | Microglia | Pathology | Monocytes | Stem cells | Cells (biology) | Infiltration | Cell migration | Integrins
Journal Article