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Cancer research (Chicago, Ill.), ISSN 0008-5472, 09/2017, Volume 77, Issue 18, pp. 5054 - 5067
Journal Article
Life sciences (1973), ISSN 0024-3205, 11/2018, Volume 212, pp. 150 - 158
Ultraviolet A radiation | Inflammation | Alpha Pinene | DNA damage | Apoptosis | Pharmacology & Pharmacy | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Lipid Peroxidation - radiation effects | Apoptosis - drug effects | Apoptosis - radiation effects | Keratinocytes - radiation effects | Membrane Potential, Mitochondrial - radiation effects | Humans | NF-kappa B - metabolism | Membrane Potential, Mitochondrial - drug effects | Monoterpenes - pharmacology | Ultraviolet Rays | Lipid Peroxidation - drug effects | Oxidative Stress - radiation effects | Skin - pathology | DNA Damage - drug effects | Epidermis - pathology | Epidermis - radiation effects | Epidermis - drug effects | Cells, Cultured | Keratinocytes - pathology | Keratinocytes - drug effects | NF-kappa B - genetics | Skin - radiation effects | Oxidative Stress - drug effects | DNA Damage - radiation effects | Skin - drug effects | Oxidative stress | Radiation | Fluorescence | Prevention | Antioxidants | Pyrimidines | Analysis | DNA | Genetic research | Skin | Tumor proteins | Fluorescence microscopy | Reactive oxygen species | Bax protein | Bcl-2 protein | Toxicity | Essential oils | p53 Protein | Lipid peroxidation | Cytotoxicity | Lipids | Mitochondrial DNA | Interleukin 6 | Proteins | Mitochondria | Membrane potential | Pretreatment | Deoxyribonucleic acid--DNA | Peroxidation | NF-κB protein | U.V. radiation | Markers | Keratinocytes | Nucleotide excision repair | Ultraviolet radiation | Cell death | Spectrofluorometry | Irradiation | Pinene | Dimers | Cyclobutane | Cyclobutane pyrimidine dimers | Index Medicus
Journal Article
Brain research, ISSN 0006-8993, 2009, Volume 1311, pp. 189 - 196
Journal Article
Journal of food science, ISSN 0022-1147, 11/2020, Volume 85, Issue 11, pp. 4070 - 4079
apoptosis | piperine | estrogen receptor | colon cancer | radiation sensitizer | mitochondrial membrane potential | Life Sciences & Biomedicine | Food Science & Technology | Science & Technology | Neoplasms - metabolism | Alkaloids - pharmacology | Apoptosis - drug effects | Apoptosis - radiation effects | Humans | Caspase 3 - metabolism | Radiation | Poly (ADP-Ribose) Polymerase-1 - metabolism | Mitochondria - metabolism | Mitochondria - drug effects | HT29 Cells | Neoplasms - genetics | Piperidines - pharmacology | Signal Transduction - drug effects | Caspase 3 - genetics | Polyunsaturated Alkamides - pharmacology | Cell Line, Tumor | Mitochondria - radiation effects | Cell Proliferation - drug effects | Neoplasms - physiopathology | Signal Transduction - radiation effects | Benzodioxoles - pharmacology | Poly (ADP-Ribose) Polymerase-1 - genetics | Cell Proliferation - radiation effects | Alkaloids | Care and treatment | Colon cancer | Analysis | Estrogen | Radiotherapy | Sugars | Apoptosis | Cancer | Monosaccharides | Cell proliferation | Adenocarcinoma | Biotechnology | Downstream effects | Estrogens | DNA damage | Colorectal cancer | Estrogen receptors | Assaying | Cancer therapies | Anticancer properties | Synergistic effects | Signal transduction | Mitochondria | Radiosensitization | Pathways | Ribose | Colon | Pretreatment | Deoxyribonucleic acid--DNA | Sensitizing | Adenosine diphosphate | Caspase | Radio | Radiation therapy | Mode of action | Signaling | Radiation tolerance | Immunofluorescence | Index Medicus
Journal Article
Journal of cellular physiology, ISSN 0021-9541, 06/2018, Volume 233, Issue 6, pp. 4666 - 4676
benzoxazole derivative | apoptosis | cell cycle arrest | mitochondria | p53 | Physiology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Benzoxazoles - pharmacology | Transcription, Genetic - drug effects | Apoptosis - drug effects | Apoptosis - radiation effects | Colorectal Neoplasms - genetics | Humans | Radiation Tolerance | Transcription, Genetic - radiation effects | Chemoradiotherapy | Tumor Suppressor Protein p53 - genetics | Dose-Response Relationship, Drug | G2 Phase Cell Cycle Checkpoints - radiation effects | Colorectal Neoplasms - therapy | G2 Phase Cell Cycle Checkpoints - drug effects | Mitochondria - radiation effects | Female | Antineoplastic Agents - pharmacology | Gene Expression Regulation, Neoplastic - drug effects | Signal Transduction - radiation effects | Colorectal Neoplasms - metabolism | HCT116 Cells | Radiation-Sensitizing Agents - pharmacology | Tumor Suppressor Protein p53 - metabolism | Mitochondria - metabolism | Mitochondria - drug effects | Mitochondria - pathology | HT29 Cells | Xenograft Model Antitumor Assays | Animals | Signal Transduction - drug effects | Mice, Nude | Cell Proliferation - drug effects | Gene Expression Regulation, Neoplastic - radiation effects | Mice, Inbred BALB C | HeLa Cells | Colorectal Neoplasms - pathology | Cell Proliferation - radiation effects | Cytochrome c | Ionizing radiation | Chemotherapy | Analysis | Colorectal cancer | Lymphomas | Biochemistry | Tumor proteins | Radiotherapy | Apoptosis | Cancer | Cytochrome | Phosphorylation | Radiosensitivity | Transcription | p53 Protein | Xenotransplantation | Colorectal carcinoma | Cancer therapies | Anticancer properties | Proteins | Mitochondria | Cell activation | Colon cancer | Cell cycle | Xenografts | Colon | Sensitizing | I.R. radiation | Radiation therapy | Gene expression | Lymphoma | Lymphocytes B | Cell death | Tumor suppressor genes | Combined treatment | Index Medicus
Journal Article