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Genes and Development, ISSN 0890-9369, 06/2013, Volume 27, Issue 12, pp. 1365 - 1377
Journal Article
Cell, ISSN 0092-8674, 10/2016, Volume 167, Issue 3, pp. 722 - 738.e23
Journal Article
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 08/2012, Volume 287, Issue 35, pp. 30024 - 30034
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 06/2014, Volume 71, pp. 208 - 220
Cardiac aging is associated with compromised myocardial function and morphology although the underlying mechanism remains elusive. Aldehyde dehydrogenase 2... 
Free radicals | ALDH2 | Cardiac geometry | Contractile function | Aging | AMPK | Sirt1 | INSULIN-LIKE-GROWTH-FACTOR-1 | MYOCARDIAL-INFARCTION | BIOCHEMISTRY & MOLECULAR BIOLOGY | HEART-FAILURE | THERAPEUTIC OPPORTUNITIES | RISK | AUTOPHAGY | OVEREXPRESSION | ALCOHOL | ENDOCRINOLOGY & METABOLISM | ESSENTIAL-HYPERTENSION | PROTEIN DAMAGE | Sirtuin 1 - metabolism | AMP-Activated Protein Kinases - metabolism | Apoptosis - drug effects | Calcium - metabolism | Mitochondria, Heart - pathology | Aldehyde Dehydrogenase, Mitochondrial | Ion Channels - genetics | Cardiomegaly - pathology | Mitochondrial Proteins - genetics | Mitochondria, Heart - drug effects | Stilbenes - pharmacology | Sirtuin 1 - genetics | Heterocyclic Compounds, 4 or More Rings - pharmacology | Mitochondrial Proteins - metabolism | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Superoxides - metabolism | Benzamides - pharmacology | Mitochondria, Heart - genetics | Aldehyde Dehydrogenase - metabolism | Myocardial Contraction | Signal Transduction | Atrial Remodeling - genetics | Gene Expression Regulation | Mitochondria, Heart - enzymology | Aldehyde Dehydrogenase - genetics | Mice, Transgenic | Myocardium - pathology | Cardiomegaly - enzymology | Transcription Factors - genetics | Aging - pathology | Transcription Factors - metabolism | Myocardium - enzymology | Animals | Ion Channels - metabolism | Fibrosis | Mice | Uncoupling Protein 2 | Cardiomegaly - genetics | AMP-Activated Protein Kinases - genetics | Benzodioxoles - pharmacology | Aging - metabolism | Heart | Genetic engineering | Aldehydes | Alternative medicine | Resveratrol
Journal Article
Journal of the American Heart Association, ISSN 2047-9980, 03/2017, Volume 6, Issue 3, p. n/a
Background The cardiac microvascular system ischemia/reperfusion injury following percutaneous coronary intervention is a clinical thorny problem. This study... 
apoptosis | ischemia/reperfusion injury | endothelial cell | mitochondria | Endothelial cell | Mitochondria | Ischemia/reperfusion injury | Apoptosis | CARDIAC & CARDIOVASCULAR SYSTEMS | PERMEABILITY | HEXOKINASE | NO-REFLOW PHENOMENON | REPERFUSION | ISCHEMIA | ENDOTHELIAL-CELLS | ACUTE MYOCARDIAL-INFARCTION | CYTOCHROME-C RELEASE | CARDIOPROTECTION | Immunohistochemistry | Mitochondria, Heart - ultrastructure | Microvessels - physiopathology | Mitochondria, Heart - metabolism | Reactive Oxygen Species - metabolism | Mitochondrial Dynamics - genetics | Apoptosis - drug effects | Male | Mitochondrial Proteins - genetics | Chromatography, High Pressure Liquid | Microcirculation - drug effects | Voltage-Dependent Anion Channel 1 - biosynthesis | Mitochondrial Membrane Transport Proteins - biosynthesis | Mitochondrial Membrane Transport Proteins - genetics | DNA, Mitochondrial - genetics | Hexokinase - biosynthesis | Hexokinase - genetics | Mitochondria, Heart - genetics | Myocardial Reperfusion Injury - genetics | Cardiolipins - genetics | Disease Models, Animal | Oxidation-Reduction | Endothelial Cells - metabolism | Membrane Proteins - genetics | Cells, Cultured | Gene Expression Regulation | Mitochondrial Proteins - biosynthesis | Blotting, Western | Myocardial Reperfusion Injury - physiopathology | Myocardial Reperfusion Injury - metabolism | Membrane Proteins - biosynthesis | Animals | Mitochondrial Dynamics - drug effects | Myocytes, Cardiac - metabolism | Cardiolipins - biosynthesis | Mice | Myocytes, Cardiac - ultrastructure | Voltage-Dependent Anion Channel 1 - genetics | Endothelial Cells - drug effects
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 12/2007, Volume 117, Issue 12, pp. 3730 - 3741
The clinical utility of anthracycline anticancer agents, especially doxorubicin, is limited by a progressive toxic cardiomyopathy linked to mitochondrial... 
MEDICINE, RESEARCH & EXPERIMENTAL | APOPTOSIS-INDUCING FACTOR | INDUCED CARDIOTOXICITY | HEME OXYGENASE | CARBON-MONOXIDE | NITRIC-OXIDE SYNTHASE | CARDIAC MYOCYTES | HEART-FAILURE | CYTOCHROME-C RELEASE | TRANSCRIPTION FACTOR GATA-4 | SUPEROXIDE-DISMUTASE | Antibiotics, Antineoplastic - pharmacology | Apoptosis - drug effects | Mitochondria, Heart - pathology | Antibiotics, Antineoplastic - adverse effects | Apoptosis - genetics | Male | Cardiomyopathies - enzymology | Necrosis - pathology | Proto-Oncogene Proteins c-akt - genetics | Sarcomeres - pathology | Cardiomyopathies - genetics | DNA, Mitochondrial - genetics | Carbon Monoxide - pharmacology | Sarcomeres - genetics | Caspase 3 - genetics | Caspase 3 - biosynthesis | Heme Oxygenase (Decyclizing) - metabolism | Mitochondria, Heart - genetics | Proto-Oncogene Proteins c-akt - metabolism | Heme Oxygenase (Decyclizing) - genetics | 3-Phosphoinositide-Dependent Protein Kinases | Protein-Serine-Threonine Kinases - metabolism | Antimetabolites - pharmacology | GATA4 Transcription Factor - metabolism | DNA, Mitochondrial - metabolism | Cells, Cultured | Gene Silencing | Mitochondria, Heart - enzymology | Protein-Serine-Threonine Kinases - genetics | Rats | Cardiomyopathies - pathology | GATA4 Transcription Factor - genetics | Guanylate Cyclase - metabolism | Myocardium - pathology | Necrosis - chemically induced | Myocardium - enzymology | Animals | Fibrosis | Guanylate Cyclase - genetics | Mice | Necrosis - genetics | Sarcomeres - enzymology | Doxorubicin - adverse effects | Doxorubicin - pharmacology | Necrosis - enzymology | Cardiomyopathies - chemically induced | Cardiomyopathy | Dosage and administration | Research | Drug therapy | Doxorubicin | Heart diseases | Risk factors | Apoptosis
Journal Article
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 08/2011, Volume 286, Issue 34, pp. 29610 - 29620
Journal Article
Journal of Pineal Research, ISSN 0742-3098, 09/2017, Volume 63, Issue 2, pp. e12418 - n/a
Journal Article