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Cell reports (Cambridge), ISSN 2211-1247, 2016, Volume 16, Issue 10, pp. 2576 - 2592
The mechanisms underlying Zika virus (ZIKV)-related microcephaly and other neurodevelopment defects remain poorly understood. Here, we describe the derivation... 
NEURAL PROGENITORS | LONG-TERM | HUMAN BRAIN | ADAPTER | CENTRAL-NERVOUS-SYSTEM | INFECTION | MICE | BINDING KINASE 1 | ORGANOIDS | INNATE IMMUNITY | CELL BIOLOGY | Neocortex - pathology | Neurons - pathology | Transcription, Genetic - drug effects | Brain - embryology | Neuroglia - ultrastructure | Neuroglia - pathology | Humans | Brain - virology | Centrosome - drug effects | Gene Expression Profiling | Microcephaly - virology | Neural Stem Cells - ultrastructure | Zika Virus Infection - virology | Neural Stem Cells - immunology | Neuroepithelial Cells - immunology | Neuroprotective Agents - pharmacology | Spinal Cord - pathology | Microcephaly - pathology | Neuroepithelial Cells - virology | Nucleosides - pharmacology | Fetus - virology | Cell Death - drug effects | Phosphorylation - drug effects | Neurons - drug effects | Protein-Serine-Threonine Kinases - metabolism | Zika Virus - pathogenicity | Proto-Oncogene Proteins - metabolism | Zika Virus - ultrastructure | Neurons - virology | Virus Replication - drug effects | Neuroepithelial Cells - ultrastructure | Immunity, Innate - drug effects | Zika Virus Infection - pathology | Neural Stem Cells - virology | Zika Virus - physiology | Zika Virus - drug effects | Mitochondria - metabolism | Mitochondria - drug effects | Receptor Protein-Tyrosine Kinases - metabolism | Neural Stem Cells - enzymology | Centrosome - metabolism | Mitosis - drug effects | Brain - pathology | Protein Kinase Inhibitors - pharmacology | Neuroglia - virology | Neuroepithelial Cells - drug effects | Neurons/pathology | Zika Virus/pathogenicity | Mitochondria/metabolism | Virus Replication/drug effects | Microcephaly/pathology | Neurons/drug effects | Protein-Serine-Threonine Kinases/metabolism | Neural Stem Cells/immunology | Neuroglia/ultrastructure | Neuroepithelial Cells/drug effects | Neuroepithelial Cells/virology | Life Sciences | Brain/pathology | Zika Virus/drug effects | Brain/embryology | Mitochondria/drug effects | Fetus/virology | Neocortex/pathology | Neuroglia/pathology | Cell Death/drug effects | Mitosis/drug effects | Transcription, Genetic/drug effects | Nucleosides/pharmacology | Neural Stem Cells/enzymology | Neural Stem Cells/ultrastructure | Neuroepithelial Cells/ultrastructure | Receptor Protein-Tyrosine Kinases/metabolism | Microcephaly/virology | Proto-Oncogene Proteins/metabolism | Neuroprotective Agents/pharmacology | Zika Virus/ultrastructure | Neuroepithelial Cells/immunology | Brain/virology | Immunity, Innate/drug effects | Spinal Cord/pathology | Zika Virus/physiology | Neuroglia/virology | Microbiology and Parasitology | Zika Virus Infection/pathology | Neurons/virology | Zika Virus Infection/virology | Neural Stem Cells/virology | Centrosome/drug effects | Protein Kinase Inhibitors/pharmacology | Centrosome/metabolism | Phosphorylation/drug effects
Journal Article
Molecular and cellular biology, ISSN 0270-7306, 2007, Volume 27, Issue 13, pp. 4953 - 4967
Article Usage Stats Services MCB Citing Articles Google Scholar PubMed Related Content Social Bookmarking CiteULike Delicious Digg Facebook Google+ Mendeley... 
CELLS | OXIDATIVE STRESS | ACTIVATED PROTEIN-KINASE | PGC-1-ALPHA | BIOCHEMISTRY & MOLECULAR BIOLOGY | GROWTH | GENE-EXPRESSION | TRANSCRIPTIONAL COACTIVATOR | DIFFERENTIATION | MICROARRAY DATA | TRANSGENIC MICE | CELL BIOLOGY | Gene Expression Regulation, Enzymologic - drug effects | Acetyltransferases - metabolism | Multienzyme Complexes - metabolism | Adipocytes - drug effects | Glucose Intolerance | AMP-Activated Protein Kinases | Oxidative Phosphorylation - drug effects | Adipose Tissue, White - cytology | Body Composition - drug effects | Isoenzymes - metabolism | Adenosine Triphosphate - metabolism | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Trans-Activators - genetics | Food Deprivation | p38 Mitogen-Activated Protein Kinases - metabolism | Homeostasis - drug effects | Phosphorylation - drug effects | Protein-Serine-Threonine Kinases - metabolism | Fibroblasts - metabolism | Isoenzymes - genetics | Hydrogen Peroxide - pharmacology | Protein-Serine-Threonine Kinases - genetics | Mitochondria - metabolism | Multienzyme Complexes - genetics | Macrophages - cytology | Mitochondria - drug effects | Feeding Behavior - drug effects | Polyamines - metabolism | Macrophages - metabolism | Organ Size - drug effects | Animals | Adipocytes - metabolism | Fibroblasts - drug effects | Adipose Tissue, White - enzymology | Adipose Tissue, White - growth & development | Glucose - metabolism | Macrophages - drug effects | Trans-Activators - metabolism | Mice | Transcription Factors | Adipose Tissue, White - drug effects | Energy Metabolism - drug effects | White/cytology/drug effects/enzymology/growth | Hydrogen Peroxide/pharmacology | Macrophages/cytology/drug effects/metabolism | Mitochondria/drug effects/metabolism | Fibroblasts/drug effects/metabolism | p38 Mitogen-Activated Protein Kinases/metabolism | MEDICIN OCH HÄLSOVETENSKAP | Homeostasis/drug effects | Multienzyme Complexes/genetics/metabolism | Protein-Serine-Threonine Kinases/genetics/metabolism | Trans-Activators/genetics/metabolism | Enzymologic/drug effects | Glucose/metabolism | Adipose Tissue | Feeding Behavior/drug effects | Organ Size/drug effects | Oxidative Phosphorylation/drug effects | MEDICAL AND HEALTH SCIENCES | development | Acetyltransferases/metabolism | Adipocytes/drug effects/metabolism | Gene Expression Regulation | Adenosine Triphosphate/metabolism | Body Composition/drug effects | Polyamines/metabolism | Energy Metabolism/drug effects | Isoenzymes/genetics/metabolism | Phosphorylation/drug effects
Journal Article
EMBO Molecular Medicine, ISSN 1757-4676, 03/2018, Volume 10, Issue 3, p. n/a
Journal Article
Brain Research, ISSN 0006-8993, 2009, Volume 1311, pp. 189 - 196
Journal Article
Cell metabolism, ISSN 1550-4131, 2016, Volume 24, Issue 6, pp. 795 - 806
.... Without any obvious toxicity or deleterious effects, NMN suppressed age-associated body weight gain, enhanced energy metabolism, promoted physical activity, improved insulin sensitivity and plasma... 
NAD | NAD+ precursor | insulin sensitivity | eye function | anti-aging | mitochondria | aging | nicotinamide mononucleotide | glucose metabolism | NMN | energy metabolism | precursor | LIFE-SPAN | STEM-CELLS | ACTIVATION | MITOCHONDRIAL | NAD BIOSYNTHESIS | INSULIN-SECRETION | ENDOCRINOLOGY & METABOLISM | GENE-EXPRESSION | RIBOSIDE | SIRT1 | ADIPOSE-TISSUE | CELL BIOLOGY | Darkness | Aging - drug effects | Male | Muscle, Skeletal - metabolism | Nicotinamide Mononucleotide - administration & dosage | Aging - genetics | Time Factors | Lipids - blood | Muscle, Skeletal - drug effects | Cell Respiration - drug effects | Myeloid Cells - drug effects | Weight Gain - drug effects | NAD - metabolism | Physical Conditioning, Animal | Food | Lymphocytes - metabolism | Insulin - pharmacology | Administration, Oral | Mice, Inbred C57BL | Mitochondria - metabolism | Mitochondria - drug effects | Gene Expression Regulation - drug effects | Eating - drug effects | Bone Density - drug effects | Animals | Aging - physiology | Lymphocytes - drug effects | Myeloid Cells - metabolism | Nicotinamide Mononucleotide - pharmacology | Drinking - drug effects | Nicotinamide Mononucleotide - blood | Energy Metabolism - drug effects | Niacinamide | Medical colleges | Exercise | Pharmacy | Genes | Body weight | Physiological aspects | Muscles | Mice | Ophthalmology | Gene expression
Journal Article
PloS one, ISSN 1932-6203, 11/2012, Volume 7, Issue 11, p. e50778
.... Moreover, the effect on mitochondrial function upon lycopene exposure was assessed. Methods and Findings... 
ISCHEMIA-REPERFUSION INJURY | INFARCT SIZE | OXYGEN | INDUCED ACTIVATION | PATHWAY | MULTIDISCIPLINARY SCIENCES | DISEASE | CAROTENOIDS | ANEMIC RATS | AMERICAN-HEART-ASSOCIATION | CELL-DEATH | Animals, Newborn | Cell Survival - drug effects | Cell Hypoxia - drug effects | Mitochondrial Membrane Transport Proteins - chemistry | Mitochondrial Membrane Transport Proteins - metabolism | Myocytes, Cardiac - cytology | Apoptosis - drug effects | Mice, Inbred C57BL | Protein Conformation - drug effects | Mitochondria - metabolism | Mitochondria - drug effects | Carotenoids - pharmacology | Lycopene | Oxygen - metabolism | Animals | Myocytes, Cardiac - drug effects | Signal Transduction - drug effects | Cytoprotection - drug effects | Myocytes, Cardiac - metabolism | Mice | Oxidative Stress - drug effects | Cytochrome c | Infants (Newborn) | Antioxidants | Analysis | Heart cells | Permeability | Apoptosis | Cytochrome | Neonates | Reactive oxygen species | Heart attacks | Mitochondrial permeability transition pore | Membrane permeability | Cardiovascular disease | Activation | Caspase-3 | Neurotoxicity | Mitochondria | Reperfusion | Carotenoids | Ischemia | Rodents | Penicillin | Occupational health | Membrane potential | Pretreatment | Cardiology | Cardiovascular system | Oxygen | Caspase | Cardiomyocytes | Pharmacology | Malondialdehyde | Hypotheses | Hospitals | Hypoxia | MPTP | Laboratory animals | Cytoplasm
Journal Article
Human molecular genetics, ISSN 0964-6906, 2014, Volume 23, Issue 17, pp. 4491 - 4509
A novel mutation in the alpha-Synuclein (alpha-Syn) gene "G51D" was recently identified in two familial cases exhibiting features of Parkinson's disease (PD)... 
WILD-TYPE | INCLUSION FORMATION | OXIDATIVE STRESS | HUMAN PLASMA | BIOCHEMISTRY & MOLECULAR BIOLOGY | GENETICS & HEREDITY | RAT-BRAIN NEURONS | MONOCLONAL-ANTIBODY | SOLUTION NMR-SPECTROSCOPY | PULSED ESR MEASUREMENTS | TRANSGENIC MICE | PHOSPHOLIPID-BINDING | Neurons - pathology | Humans | alpha-Synuclein - ultrastructure | Saccharomyces cerevisiae - drug effects | Protein Transport - drug effects | Brain - metabolism | Saccharomyces cerevisiae - metabolism | Cell Nucleus - metabolism | Protein Binding - drug effects | Sodium Dodecyl Sulfate - pharmacology | Cell Membrane - metabolism | Neurons - metabolism | alpha-Synuclein - genetics | Phosphorylation - drug effects | Buffers | Inclusion Bodies - metabolism | Neurons - drug effects | Protein Aggregation, Pathological - genetics | Cell Membrane - drug effects | Neuroblastoma - pathology | Protein Aggregates - drug effects | Cell Line | Parkinson Disease - pathology | Protein Structure, Secondary | Subcellular Fractions - drug effects | Cells, Cultured | Inclusion Bodies - drug effects | Mitochondria - metabolism | alpha-Synuclein - secretion | Mitochondria - drug effects | Parkinson Disease - genetics | Mutation - genetics | Subcellular Fractions - metabolism | alpha-Synuclein - chemistry | Brain - drug effects | Unilamellar Liposomes - metabolism | Nuclear Envelope - metabolism | Nuclear Envelope - drug effects | Cell Differentiation - drug effects | Brain - pathology | Cell Nucleus - drug effects | Index Medicus
Journal Article
Nucleic acids research, ISSN 1362-4962, 2019, Volume 47, Issue 8, pp. 4026 - 4038
Abstract Eukaryotic Primase-Polymerase (PrimPol) is an enzyme that maintains efficient DNA duplication by repriming replication restart downstream of replicase... 
SURVIVAL | REPAIR | PHOTOPRODUCTS | BYPASS | POLYMERASE-ETA | DEFICIENT XERODERMA-PIGMENTOSUM | TOLERANCE | BIOCHEMISTRY & MOLECULAR BIOLOGY | MECHANISMS | GENOME | Multifunctional Enzymes - genetics | DNA Primase - deficiency | DNA Replication - drug effects | Humans | Multifunctional Enzymes - deficiency | Quinolones - pharmacology | DNA-Directed DNA Polymerase - deficiency | DNA-Directed DNA Polymerase - genetics | Mitochondria - genetics | Cell Nucleus - radiation effects | 4-Nitroquinoline-1-oxide - pharmacology | DNA Replication - radiation effects | Gene Deletion | Micronuclei, Chromosome-Defective - drug effects | DNA Primase - genetics | Mitochondria - radiation effects | Micronuclei, Chromosome-Defective - radiation effects | DNA - drug effects | Fibroblasts - metabolism | Osteoblasts - radiation effects | Osteoblasts - drug effects | Mutagens - pharmacology | Cisplatin - pharmacology | DNA - metabolism | Mitochondria - drug effects | Ultraviolet Rays - adverse effects | DNA - genetics | Sister Chromatid Exchange - drug effects | Cell Nucleus - genetics | Fibroblasts - radiation effects | Fibroblasts - drug effects | Cell Line, Tumor | Sister Chromatid Exchange - radiation effects | Bleomycin - pharmacology | Cell Nucleus - drug effects | Osteoblasts - metabolism | Cell Line, Transformed | Genome Integrity, Repair and
Journal Article
The Journal of clinical investigation, ISSN 1558-8238, 2015, Volume 125, Issue 11, pp. 4223 - 4238
.... We have recently shown that salt has a proinflammatory effect and boosts the activation of Th17 cells and the activation of classical, LPS-induced macrophages (M1... 
MEDICINE, RESEARCH & EXPERIMENTAL | M2 MACROPHAGES | NA+ STORAGE | TISSUE | GENE-EXPRESSION | URINARY SODIUM | TRANSCRIPTION FACTOR | POTASSIUM EXCRETION | ALTERNATIVE ACTIVATION | BLOOD-PRESSURE | T-CELLS | Sodium Chloride, Dietary - pharmacology | Sodium Chloride - pharmacology | Male | Glycolysis - drug effects | Proto-Oncogene Proteins c-akt - genetics | Oxidative Phosphorylation - drug effects | Interleukin-4 - pharmacology | Histone Code - drug effects | Bone Marrow Cells - drug effects | TOR Serine-Threonine Kinases - physiology | Wound Healing - drug effects | Macrophages - immunology | Macrophages - classification | Immunity, Innate - drug effects | Mice, Inbred C57BL | Cells, Cultured | Mice, Transgenic | Inflammation | Proto-Oncogene Proteins c-akt - physiology | Mitochondria - drug effects | Random Allocation | Interleukin-13 - pharmacology | Gene Expression Regulation - drug effects | Animals | Signal Transduction - drug effects | Sodium Chloride, Dietary - toxicity | Chitin - toxicity | Macrophages - drug effects | Mice | Macrophage Activation - drug effects | Salt | Medical research | Immune response | Medicine, Experimental | Research | Macrophages | Properties | Observations | Biological control systems | Health aspects | Hypertension | Wound healing | Sodium | Cytokines | Kinases | Metabolism | Gene expression | Experiments | Acquisitions & mergers
Journal Article
PloS one, ISSN 1932-6203, 2011, Volume 6, Issue 6, p. e20786
Photodynamic therapy, unlikely to elicit drug-resistance, deserves attention as a strategy to counter this outstanding problem common to the chemotherapy of... 
MHC CLASS-I | CUTANEOUS LEISHMANIASIS | IMMUNOGENICITY | THERAPY | UROPORPHYRIA | DENDRITIC CELLS | CLINICAL-TRIAL | EFFICACY | LEISH-F1+MPL-SE VACCINE | MULTIDISCIPLINARY SCIENCES | SINGLET OXYGEN | Endocytosis - radiation effects | Ovalbumin - immunology | Photochemotherapy - methods | Dendritic Cells - immunology | CD8-Positive T-Lymphocytes - parasitology | Phagosomes - radiation effects | Substrate Specificity | Dendritic Cells - radiation effects | Macrophages - parasitology | CD8-Positive T-Lymphocytes - radiation effects | Leishmania - parasitology | Indoles - metabolism | Light | Leishmania - drug effects | Mitochondria - radiation effects | Dendritic Cells - drug effects | Indoles - pharmacology | Host-Parasite Interactions | Macrophages - radiation effects | Intracellular Space - radiation effects | Macrophages - immunology | Phagosomes - drug effects | Cell Line | Dendritic Cells - parasitology | Intracellular Space - drug effects | Antigen Presentation - radiation effects | Endocytosis - drug effects | Phagosomes - metabolism | Solubility | HLA Antigens - immunology | Mitochondria - metabolism | Mitochondria - drug effects | Drug Discovery | Phagosomes - parasitology | Photolysis - radiation effects | Photolysis - drug effects | Animals | CD8-Positive T-Lymphocytes - drug effects | Intracellular Space - metabolism | Indoles - therapeutic use | Macrophages - drug effects | Antigen Presentation - drug effects | Leishmania - physiology | Mice | CD8-Positive T-Lymphocytes - immunology | Indoles - chemistry | Photolysis | Dendritic cells | Vaccination | Genetic engineering | Drug resistance | T cells | Photochemotherapy | Cancer | Porphyrins | Phagolysosomes | CD8 antigen | Transgenic | Biosynthesis | Lymphocytes T | Parasites | Macrophages | Inactivation | Immunity | Accumulation | Immunity (cell-mediated) | Homing | Mitochondria | Immunology | Lymphocytes | Parasitic diseases | Infectivity | Protozoa | Ovalbumin | Antigens | Photodynamic therapy | Deactivation | Host specificity | Mutants | Medicine | Chemistry | Chemotherapy | Major histocompatibility complex | Antigen-presenting cells | Lysis | Intracellular | Cell migration
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2016, Volume 113, Issue 6, pp. E782 - E790
Epstein–Barr virus (EBV) is an oncogenic herpesvirus that has been causally linked to the development of B-cell and epithelial malignancies. Early after... 
B cell | Epstein-Barr virus | Metabolism | Oncogene-induced senescence | Autophagy | TRANSFORMATION | SURVIVAL | autophagy | DNA-DAMAGE RESPONSE | MULTIDISCIPLINARY SCIENCES | CANCER | REPLICATION | SIGNALING PATHWAY | metabolism | TUMORIGENESIS | oncogene-induced senescence | LYMPHOCYTES | Metabolomics | Cell Transformation, Viral - drug effects | TOR Serine-Threonine Kinases - metabolism | DNA Replication - drug effects | Humans | Herpesvirus 4, Human - drug effects | Cellular Senescence - drug effects | Dimethylformamide - pharmacology | Autophagy - drug effects | Mechanistic Target of Rapamycin Complex 1 | Multiprotein Complexes - metabolism | Deoxyglucose - pharmacology | Cell Respiration - drug effects | B-Lymphocytes - virology | Stress, Physiological - drug effects | B-Lymphocytes - pathology | B-Lymphocytes - ultrastructure | Oncogenes | DNA Damage - drug effects | Herpesvirus 4, Human - physiology | Tumor Suppressor Protein p53 - metabolism | Mitochondria - metabolism | Transcriptome - genetics | Mitochondria - drug effects | Transcription Factors - metabolism | B-Lymphocytes - drug effects | Signal Transduction - drug effects | Cell Cycle Checkpoints - drug effects | Cell Proliferation - drug effects | Cell interaction | B cells | Host-virus relationships | Observations | Health aspects | Biological Sciences | PNAS Plus | Epstein–Barr virus
Journal Article