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NeuroReport, ISSN 0959-4965, 05/2019, Volume 30, Issue 7, pp. 479 - 484
Cerebral infarction is a common disease that threatens the health of humankind worldwide. Diabetes is one of the important causes of cerebral ischemic (CI)... 
liraglutide | mitochondrial ATP-sensitive potassium channel | STROKE | SUBTYPES | cerebral ischemia | inflammatory response | diabetes mellitus | NEUROSCIENCES | oxidative stress
Journal Article
Antioxidants & Redox Signaling, ISSN 1523-0864, 09/2012, Volume 17, Issue 6, pp. 849 - 859
Aims: Hydrogen sulfide (H 2 S), a novel gaseous mediator, has been recognized to protect neurons from overexcitation by enhancing the activity of the adenosine... 
Original Research Communications | CELLS | OVEREXPRESSION | OXIDATIVE STRESS | HOMEOSTASIS | SULFHYDRATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | ENDOCRINOLOGY & METABOLISM | ENDOPLASMIC-RETICULUM STRESS | K+ CHANNELS | BRAIN | PARKINSONS-DISEASE | PROTECTS | 1-Methyl-4-phenyl-1,2,3,6-tetrahydropyridine - pharmacology | Immunohistochemistry | Hydrogen Sulfide - pharmacology | KATP Channels - genetics | Reactive Oxygen Species - metabolism | Mice, Inbred C57BL | Ion Channels - genetics | Male | Potassium Channels, Inwardly Rectifying - genetics | Mitochondrial Proteins - genetics | Chromatography, High Pressure Liquid | Blotting, Western | Mice, Knockout | KATP Channels - metabolism | Animals | Ion Channels - metabolism | Mitochondrial Proteins - metabolism | Dopaminergic Neurons - metabolism | Dopaminergic Neurons - drug effects | Hydro-Lyases - metabolism | Mice | Uncoupling Protein 2 | Hydro-Lyases - genetics | Potassium Channels, Inwardly Rectifying - metabolism | Hydrogen sulfide | Physiological aspects | Research | Neurons | Analysis | Mitochondrial uncoupling protein 2 | Neuroprotection | Animal models | Reactive oxygen species | Dopamine | Parkinson's disease | Neurodegenerative diseases | Potassium channels (inwardly-rectifying) | Stress | Antioxidants | Sodium | Degeneration | Endoplasmic reticulum | Genotypes | Injuries | Movement disorders | Apoptosis
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 6/2001, Volume 98, Issue 13, pp. 7623 - 7628
Journal Article
Journal of Molecular and Cellular Cardiology, ISSN 0022-2828, 2014, Volume 79, pp. 42 - 53
Abstract ATP-sensitive potassium (KATP ) channels are abundantly expressed in the myocardium. Although a definitive role for the channel remains elusive they... 
Cardiovascular | ATP-sensitive potassium (KATP) channels | Ischemic preconditioning | Cardioprotection | Protein kinase C (PKC) | ATP-sensitive potassium (K | channels | CARDIAC & CARDIOVASCULAR SYSTEMS | METABOLIC INHIBITION | DIAZOXIDE | INFARCT-SIZE | CELL BIOLOGY | ISOLATED VENTRICULAR MYOCYTES | ATP-sensitive potassium (K-ATP) channels | ACTION-POTENTIAL DURATION | IN-VIVO | CARDIAC PROTECTION | MITOCHONDRIAL INNER MEMBRANE | STRESS | K+ CHANNELS | Diazoxide - pharmacology | Tetradecanoylphorbol Acetate - pharmacology | Rats, Wistar | Cardiotonic Agents - metabolism | Heart Failure - enzymology | Heart Failure - physiopathology | Myocardial Contraction - drug effects | Male | Time Factors | Isoenzymes - metabolism | Protein Kinase C-epsilon - antagonists & inhibitors | Sarcolemma - metabolism | Action Potentials - drug effects | Pinacidil - pharmacology | Cell Separation | Adenosine - pharmacology | Heart Failure - pathology | Enzyme Activation - drug effects | KATP Channels - metabolism | Myocytes, Cardiac - pathology | Phenotype | Animals | Myocytes, Cardiac - drug effects | Myocardial Reperfusion | Ischemic Preconditioning, Myocardial | Protein Kinase C-epsilon - metabolism | Myocytes, Cardiac - metabolism | Protein Kinase Inhibitors - pharmacology | Isoenzymes - antagonists & inhibitors | Ion Channel Gating - drug effects | Potassium channels | Index Medicus
Journal Article
Circulation Research: Journal of the American Heart Association, ISSN 0009-7330, 11/2001, Volume 89, Issue 10, pp. 891 - 898
Journal Article
Circulation Research: Journal of the American Heart Association, ISSN 0009-7330, 03/2001, Volume 88, Issue 6, pp. 570 - 577
Journal Article