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1. Full Text Mitochondrial Diseases of the Brain
by Chaturvedi, Rajnish K and Flint Beal, M
Free Radical Biology and Medicine, ISSN 0891-5849, 10/2013, Volume 63, pp. 1 - 29
Neurodegenerative disorders are debilitating diseases of the brain, characterized by behavioral, motor and cognitive impairments. Ample evidence underpins... 
Sirtuins | Charcot-Marie-Tooth disease and Friedreich’s ataxia | Mitochondrial dysfunction | Free radicals | Alzheimer’s disease | Neurodegenerative diseases | Amyotrophic lateral sclerosis | Co-Q10 | Huntington’s disease | PGC-1α | Creatine | Parkinson’s disease | Charcot-Marie-Tooth disease and | Parkinson's disease | Huntington's disease | Friedreich's ataxia | Alzheimer's disease | KETOGLUTARATE DEHYDROGENASE COMPLEX | PGC-1 alpha | BIOCHEMISTRY & MOLECULAR BIOLOGY | EARLY PARKINSON-DISEASE | NEURONAL CELL-DEATH | AMYOTROPHIC-LATERAL-SCLEROSIS | AMYLOID PRECURSOR PROTEIN | Charcot-Marie-Tooth disease and Friedreich's ataxia | CYTOCHROME-C-OXIDASE | TARGETED ANTIOXIDANT MITOQ | ENDOCRINOLOGY & METABOLISM | PLACEBO-CONTROLLED TRIAL | TRANSGENIC MOUSE MODEL | COMPLEX-I DEFICIENCY | Mitochondrial Diseases - pathology | Friedreich Ataxia - metabolism | Parkinson Disease - pathology | Free Radicals - toxicity | Friedreich Ataxia - pathology | Humans | Huntington Disease - pathology | Nerve Degeneration - physiopathology | Mitochondria - metabolism | Mitochondria - pathology | Charcot-Marie-Tooth Disease - pathology | Huntington Disease - metabolism | Nerve Degeneration - metabolism | Alzheimer Disease - pathology | Brain - metabolism | Amyotrophic Lateral Sclerosis - pathology | Alzheimer Disease - metabolism | Amyotrophic Lateral Sclerosis - metabolism | Brain - pathology | Parkinson Disease - metabolism | Charcot-Marie-Tooth Disease - metabolism | Proteins | Nervous system diseases | Mitochondrial DNA | Gene mutations | Analysis | Index Medicus
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2. Full Text Mitochondria and quality control defects in a mouse model of gaucher disease - Links to parkinson's disease
by Osellame, Laura D and Rahim, Ahad A and Hargreaves, Iain P and Gegg, Matthew E and Richard-Londt, Angela and Brandner, Sebastian and Waddington, Simon N and Schapira, Anthony H.V and Duchen, Michael R
Cell Metabolism, ISSN 1550-4131, 06/2013, Volume 17, Issue 6, pp. 941 - 953
Mutations in the glucocerebrosidase (gba) gene cause Gaucher disease (GD), the most common lysosomal storage disorder, and increase susceptibility to... 
OXIDATIVE STRESS | PINK1 | INHIBITION | GENE | ENDOCRINOLOGY & METABOLISM | LYSOSOMAL STORAGE DISEASES | ALPHA-SYNUCLEIN | AUTOPHAGY | DYSFUNCTION | GLUCOCEREBROSIDASE MUTATIONS | COMPLEX-I DEFICIENCY | CELL BIOLOGY | Astrocytes - cytology | Mitochondrial Diseases - genetics | Electron Transport | Glucosylceramidase - genetics | Gaucher Disease - metabolism | Sequestosome-1 Protein | Heat-Shock Proteins - metabolism | Humans | Lysosomes - genetics | Cells, Cultured | Neurons - cytology | Mitochondria - metabolism | Parkinson Disease - genetics | Mice, Knockout | Animals | Gaucher Disease - genetics | Lysosomes - metabolism | Mitochondria - genetics | Mice | Parkinson Disease - metabolism | Autophagy - genetics | Adaptor Proteins, Signal Transducing - metabolism | alpha-Synuclein - metabolism | Disease Models, Animal | Proteins | High technology industry | Neurosciences | Developmental biology | Analysis | Quality control | Mitochondrial DNA
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3. Full Text Parkinson's disease
by Lees, Andrew J, Prof and Hardy, John, Prof and Revesz, Tamas, Prof
Lancet, The, ISSN 0140-6736, 2009, Volume 373, Issue 9680, pp. 2055 - 2066
Summary Parkinson's disease is a common progressive bradykinetic disorder that can be accurately diagnosed. It is characterised by the presence of severe... 
Internal Medicine | MULTIPLE SYSTEM ATROPHY | MEDICINE, GENERAL & INTERNAL | VASCULAR PARKINSONISM | PHASE-I TRIAL | ALPHA-SYNUCLEIN PATHOLOGY | PYRAMIDAL DEGENERATION | MITOCHONDRIAL DYSFUNCTION | SUBCUTANEOUS APOMORPHINE | LEWY BODY DISEASE | DEEP-BRAIN-STIMULATION | PROGRESSIVE SUPRANUCLEAR PALSY | Parkinson Disease - therapy | Genetic Therapy | Genetic Predisposition to Disease - genetics | Glucosylceramidase - genetics | Age Distribution | Humans | Middle Aged | Protein-Serine-Threonine Kinases | Risk Factors | Male | Dopamine Agents - therapeutic use | Mutation - genetics | Cause of Death | Leucine-Rich Repeat Serine-Threonine Protein Kinase-2 | Embryonic Stem Cells - transplantation | Age of Onset | Parkinson Disease - diagnosis | Parkinson Disease - epidemiology | Sex Distribution | Female | Parkinsonian Disorders - genetics | Aged | alpha-Synuclein - genetics | Parkinson Disease - etiology | Care and treatment | Parkinson's disease | Patient outcomes | Stem cells | Transplantation | Diagnosis | Gene therapy | Health aspects | Risk factors | Proteins | Tobacco smoke | Rodents | Mortality | Parkinsons disease | Mutation | Smoking | Caffeine
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4. Full Text Mitochondrial disease
by Schapira, Anthony HV
The Lancet, ISSN 0140-6736, 2006, Volume 368, Issue 9529, pp. 70 - 82
Defects of mitochondrial metabolism cause a wide range of human diseases that include examples from all medical subspecialties. This review updates the topic... 
PROGRESSIVE EXTERNAL OPHTHALMOPLEGIA | ELECTRON-TRANSPORT CHAIN | CYTOCHROME-C-OXIDASE | AMYLOID-BETA PEPTIDE | MEDICINE, GENERAL & INTERNAL | ALZHEIMERS-DISEASE | GAMMA-GENE POLG | HEREDITARY OPTIC NEUROPATHY | RESPIRATORY-CHAIN DISORDERS | INTRACYTOPLASMIC SPERM INJECTION | PARKINSONS-DISEASE | Mitochondrial Diseases - genetics | Mitochondrial Diseases - drug therapy | Humans | Ubiquinone - deficiency | Genotype | Male | Mitochondria - metabolism | Phenotype | Animals | Mitochondria - genetics | Ubiquinone - therapeutic use | Female | Ubiquinone - physiology | Mutation | Mitochondria - physiology | Mitochondrial Diseases - physiopathology | Analysis | Mitochondrial diseases | Research | Genetic aspects | Gene mutations | Proteins | Mitochondrial DNA | Inheritances | Minority & ethnic groups | Cells | Apoptosis
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5. Full Text Parkinson's disease
by Thomas, Bobby and Flint Beal, M
Human Molecular Genetics, ISSN 0964-6906, 10/2007, Volume 16, Issue 2, pp. R183 - R194
Parkinson's disease (PD) is a chronic progressive neurodegenerative movement disorder characterized by a profound and selective loss of nigrostriatal... 
SUBSTANTIA-NIGRA NEURONS | RICH REPEAT KINASE-2 | UBIQUITIN-PROTEIN LIGASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | GENETICS & HEREDITY | A30P TRANSGENIC MICE | C-TERMINAL TRUNCATION | LEWY BODY DISEASE | MITOCHONDRIAL-DNA DELETIONS | ALPHA-SYNUCLEIN NEUROTOXICITY | CYCLIN-DEPENDENT KINASE-5 | EARLY-ONSET PARKINSONISM | Oncogene Proteins - genetics | Genetic Predisposition to Disease | Protein Kinases - genetics | Oxidation-Reduction | Humans | Protein-Serine-Threonine Kinases - genetics | Mitochondria - metabolism | Parkinson Disease - genetics | Leucine-Rich Repeat Serine-Threonine Protein Kinase-2 | Protein Deglycase DJ-1 | Models, Biological | Parkinson Disease - metabolism | alpha-Synuclein - genetics | Mutation | Ubiquitin-Protein Ligases - genetics | Intracellular Signaling Peptides and Proteins - genetics | Parkinson Disease - etiology
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6. Full Text Mitochondrial diseases
by Schapira, Anthony HV, Prof
Lancet, The, ISSN 0140-6736, 2012, Volume 379, Issue 9828, pp. 1825 - 1834
Summary Mitochondria have a crucial role in cellular bioenergetics and apoptosis, and thus are important to support cell function and in determination of cell... 
Internal Medicine | ELECTRON-TRANSPORT CHAIN | MEDICINE, GENERAL & INTERNAL | SUBSTANTIA-NIGRA NEURONS | CHAPERONE-MEDIATED AUTOPHAGY | ALZHEIMERS-DISEASE | EARLY PARKINSON-DISEASE | COMPLEX I DEFICIENCY | PLACEBO-CONTROLLED TRIAL | HEREDITARY OPTIC NEUROPATHY | ALPHA-SYNUCLEIN | HUNTINGTONS-DISEASE | Mitochondrial Diseases - genetics | Humans | Mitochondrial Diseases - metabolism | Mitochondrial Proteins - physiology | Apoptosis - genetics | Mitochondria - metabolism | Mitochondrial Proteins - genetics | Disease Progression | Mitochondria - genetics | Mitochondrial Proteins - metabolism | Apoptosis - physiology | Mutation | Mitochondria - physiology | Mitochondrial Diseases - physiopathology | Care and treatment | Gene mutations | Physiological aspects | Mitochondrial DNA | Mitochondrial diseases | Genetic aspects | Research | Risk factors | Proteins | Kinases | Rodents | Parkinsons disease
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