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PLoS Computational Biology, ISSN 1553-734X, 2014, Volume 10, Issue 2, p. e1003470
Interleukin-1 (IL-1) is a large cytokine family closely related to innate immunity and inflammation. IL-1 proteins are key players in signaling pathways such... 
DATA-BANK | KINASE | BIOCHEMICAL RESEARCH METHODS | MATHEMATICAL & COMPUTATIONAL BIOLOGY | INTERFACES | PROTEIN-PROTEIN INTERACTIONS | RECEPTOR | SCALE | MOLECULAR DOCKING | EXPRESSION | SOMATIC MUTATIONS | BRAIN-TUMORS | Interleukin-1 Receptor Accessory Protein - genetics | MAP Kinase Kinase 7 - genetics | Neoplasms - metabolism | Interleukin-1 - genetics | Interleukin-1 Receptor Accessory Protein - chemistry | Humans | MAP Kinase Kinase 7 - chemistry | Mitogen-Activated Protein Kinase 9 - genetics | Mitogen-Activated Protein Kinase 10 - genetics | MAP Kinase Kinase 7 - metabolism | Interleukin-1 Receptor Accessory Protein - metabolism | Receptors, Interleukin-1 Type I - metabolism | Inflammation - metabolism | MAP Kinase Kinase 4 - metabolism | Neoplasms - genetics | Computer Simulation | Receptors, Interleukin-1 Type I - chemistry | Interleukin-1 - chemistry | Oncogenes | Mitogen-Activated Protein Kinase 9 - chemistry | Interleukin-1 - metabolism | Protein Interaction Maps - immunology | Signal Transduction | Mitogen-Activated Protein Kinase 9 - metabolism | Computational Biology | Models, Molecular | Inflammation - immunology | Mitogen-Activated Protein Kinase 10 - metabolism | Mitogen-Activated Protein Kinase 10 - chemistry | Mutagenesis | Protein Interaction Maps - genetics | Models, Biological | Neoplasms - immunology | Receptors, Interleukin-1 Type I - genetics | Inflammation - genetics | MAP Kinase Kinase 4 - chemistry | MAP Kinase Kinase 4 - genetics | Polymorphism, Single Nucleotide | Mutation | Complications and side effects | Interleukin-1 | Patient outcomes | Physiological aspects | Development and progression | Inflammation | Genetic aspects | Research | Risk factors | Genetic polymorphisms | Apoptosis | Proteins | Cytokines | Disease | Kinases | Cancer
Journal Article
Journal of Medicinal Chemistry, ISSN 0022-2623, 01/2015, Volume 58, Issue 1, pp. 443 - 456
Journal Article
Journal Article
Nature Neuroscience, ISSN 1097-6256, 07/2009, Volume 12, Issue 7, pp. 864 - 871
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2014, Volume 9, Issue 1, p. e87067
Pancreatic beta-cell dysfunction is central to type 2 diabetes pathogenesis. Prolonged elevated levels of circulating free-fatty acids and hyperglycemia, also... 
OXIDATIVE STRESS | GENE | MULTIDISCIPLINARY SCIENCES | N-TERMINAL KINASE | FATTY-ACID | DEATH | ENDOPLASMIC-RETICULUM STRESS | DYSFUNCTION | ACTIVATING TRANSCRIPTION FACTOR-3 | PALMITATE | TYPE-2 | RNA, Small Interfering - genetics | Transcription Factor CHOP - genetics | Palmitic Acid - pharmacology | Caspase 9 - genetics | Caspase 9 - metabolism | Mitogen-Activated Protein Kinase 8 - antagonists & inhibitors | Apoptosis - drug effects | Diabetes Mellitus, Type 2 - genetics | Humans | Caspase 3 - metabolism | Mitogen-Activated Protein Kinase 9 - genetics | RNA, Messenger - metabolism | Mitogen-Activated Protein Kinase 10 - genetics | RNA, Messenger - antagonists & inhibitors | Caspase 3 - genetics | Apoptosis Regulatory Proteins - genetics | Mitogen-Activated Protein Kinase 8 - genetics | Cell Line | Signal Transduction | Mitogen-Activated Protein Kinase 9 - metabolism | Mitogen-Activated Protein Kinase 8 - metabolism | RNA, Messenger - genetics | Diabetes Mellitus, Type 2 - enzymology | Gene Expression Regulation | Rats | Glucose - pharmacology | Apoptosis Regulatory Proteins - metabolism | Mitogen-Activated Protein Kinase 10 - metabolism | Animals | Insulin-Secreting Cells - drug effects | Mitogen-Activated Protein Kinase 9 - antagonists & inhibitors | Diabetes Mellitus, Type 2 - pathology | Insulin-Secreting Cells - pathology | Mitogen-Activated Protein Kinase 10 - antagonists & inhibitors | Transcription Factor CHOP - metabolism | Insulin-Secreting Cells - enzymology | RNA, Small Interfering - metabolism | Type 2 diabetes | Pancreatic beta cells | RNA | Glucose | Fatty acids | Dextrose | Apoptosis | Oxidative stress | Transcription factors | Pathogenesis | JNK3 protein | Kinases | Palmitic acid | Proteins | Mitochondria | Hyperglycemia | Pancreas | Obesity | Immunoglobulins | Cytokines | Diabetes mellitus | Markers | c-Jun protein | Caspase | Exposure | Gene expression | Insulin | Beta cells | Cell lines | Isoforms | Caspase-9 | Insulin resistance | Diabetes | Endoplasmic reticulum | Endocrinology
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 06/2010, Volume 285, Issue 26, pp. 20358 - 20368
Journal Article
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 09/2010, Volume 114, Issue 5, pp. 1315 - 1322
J. Neurochem. (2010) 114, 1315–1322. The MAPK family is formed by extracellular signal‐regulated kinases p38 kinase and stress‐activated protein kinases... 
epilepsy | stress‐activated protein kinases | neuronal death | neuroprotection | stress-activated protein kinases | PROTEIN-KINASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | N-TERMINAL KINASE | SYMPATHETIC NEURONS | PEPTIDE INHIBITOR | NEUROSCIENCES | CELL-DEATH | GLUTAMATE-MEDIATED EXCITOTOXICITY | POLYGLUTAMINE-EXPANDED HUNTINGTIN | C-JUN ACTIVATION | SCAFFOLD PROTEINS | APOPTOTIC NEURONAL DEATH | Kainic Acid - pharmacology | Mice, Inbred C57BL | Mitogen-Activated Protein Kinase 3 - antagonists & inhibitors | Mitogen-Activated Protein Kinase 6 - metabolism | Excitatory Amino Acid Agonists - pharmacology | Enzyme Activation - drug effects | Mitogen-Activated Protein Kinase 10 - genetics | Mice, Knockout | Mitogen-Activated Protein Kinase 10 - metabolism | Phosphorylation - genetics | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Mice | Mitogen-Activated Protein Kinase 10 - deficiency | p38 Mitogen-Activated Protein Kinases - metabolism | Mitogen-Activated Protein Kinase 10 - antagonists & inhibitors | Phosphorylation - drug effects | Enzyme Activation - genetics | Mitogen-Activated Protein Kinase 1 - metabolism | Proteins | Nervous system diseases | Parkinson's disease | Huntington's chorea | Neurons | Analysis | Epilepsy | Protein kinases | Alzheimer's disease | Neurology | Genetics | Biochemistry | Kinases | Rodents
Journal Article