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Journal of Biological Chemistry, ISSN 0021-9258, 07/2008, Volume 283, Issue 28, pp. 19704 - 19713
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2012, Volume 7, Issue 5, p. e35997
We have recently shown that silencing of the brain/islet specific c-Jun N-terminal Kinase3 (JNK3) isoform enhances both basal and cytokine-induced beta-cell... 
FORKHEAD TRANSCRIPTION FACTOR | APOPTOSIS | HUMAN ISLETS | PROTEIN-KINASE-B | TUMOR SUPPRESSION | GLYCOGEN-SYNTHASE KINASE-3 | BIOLOGY | PANCREATIC BETA-CELLS | JUN NH2-TERMINAL KINASE | PHYSIOLOGICAL FUNCTIONS | MICE LACKING | Phosphorylation | Glycogen Synthase Kinase 3 beta | Insulinoma - genetics | Insulin Receptor Substrate Proteins - metabolism | Mitogen-Activated Protein Kinase 9 - genetics | Mitogen-Activated Protein Kinase 10 - genetics | Insulin-Secreting Cells - metabolism | RNA Interference | Forkhead Transcription Factors - metabolism | Mitogen-Activated Protein Kinase 8 - genetics | Proto-Oncogene Proteins c-akt - metabolism | Insulin Secretion | Signal Transduction | Mitogen-Activated Protein Kinase 9 - metabolism | Mitogen-Activated Protein Kinase 8 - metabolism | Rats | Glycogen Synthase Kinase 3 - metabolism | Blotting, Western | Mitogen-Activated Protein Kinase 10 - metabolism | Insulin - metabolism | Animals | Insulin-Secreting Cells - drug effects | Insulinoma - metabolism | Cell Line, Tumor | Insulinoma - pathology | Insulin-Secreting Cells - pathology | Forkhead Box Protein O3 | Cytokines - pharmacology | Pancreatic beta cells | Synthesis | Cytokines | Glycogen | Gene expression | Insulin | Apoptosis | Brain | Transcription factors | AKT1 protein | Glycogen synthase | JNK3 protein | AKT2 protein | c-Jun protein | AKT protein | Glucose | Kinases | Beta cells | Signal transduction | Signaling | Rodents | Insulin resistance | Forkhead protein | Cytoplasm | FOXO3 protein
Journal Article
Neurochemical Research, ISSN 0364-3190, 9/2012, Volume 37, Issue 9, pp. 1868 - 1878
The epileptogenesis may involve a variety of signaling events that culminate with synaptic reorganization. Mitogen-activated protein kinases (MAPKs) and AKT... 
Biochemistry, general | Neurochemistry | Neurology | Neurosciences | Biomedicine | Epilepsy | Cortex | Pilocarpine | AKT | Hippocampus | Cell Biology | MAPKs | NERVOUS-SYSTEM | LIMBIC STRUCTURES | TEMPORAL-LOBE EPILEPSY | TYROSINE PHOSPHORYLATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | P38 MAPK | INDUCED STATUS-EPILEPTICUS | SYNAPTIC PLASTICITY | NEUROSCIENCES | MAPK SIGNALING PATHWAYS | REGULATED KINASE | SPONTANEOUS RECURRENT SEIZURES | Epilepsy - enzymology | Rats, Wistar | Status Epilepticus - enzymology | Mitogen-Activated Protein Kinase 9 - metabolism | Mitogen-Activated Protein Kinase 8 - metabolism | Status Epilepticus - chemically induced | Rats | Epilepsy - metabolism | Male | Blotting, Western | Cerebral Cortex - metabolism | Mitogen-Activated Protein Kinase 10 - metabolism | Muscarinic Agonists | Hippocampus - metabolism | Animals | MAP Kinase Signaling System - drug effects | Epilepsy - chemically induced | p38 Mitogen-Activated Protein Kinases - metabolism | Oncogene Protein v-akt - metabolism | Status Epilepticus - metabolism | Mitogen-Activated Protein Kinases - metabolism | Enzymes | Oxidative stress | Temporal lobe | Phosphorylation | Cytokines | Extracellular signal-regulated kinase | AKT protein | MAP kinase | Sclerosis | Western blotting | Neurotransmitters | Plasticity (synaptic) | Protein kinase | Mitogens | Latent period
Journal Article
Molecular Neurobiology, ISSN 0893-7648, 5/2018, Volume 55, Issue 5, pp. 4437 - 4452
Journal Article
EMBO reports, ISSN 1469-221X, 05/2012, Volume 13, Issue 5, pp. 455 - 461
Journal Article
Journal of Medicinal Chemistry, ISSN 0022-2623, 06/2006, Volume 49, Issue 12, pp. 3563 - 3580
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2015, Volume 10, Issue 4, pp. e0119204 - e0119204
Introduction and Aims The ASK1-JNK3 signaling pathway plays a pivotal role in the pathogenesis of Parkinson's disease (PD). The specific binding of... 
APOPTOSIS | ACTIVATION | MULTIDISCIPLINARY SCIENCES | N-TERMINAL KINASE | MOUSE MODEL | NEURODEGENERATION | INDUCTION | AP-1 | EXPRESSION | BRAIN | PARKINSONS-DISEASE | Arrestins - chemistry | Apoptosis - drug effects | Dopaminergic Neurons - pathology | Humans | Molecular Sequence Data | Male | Arrestins - metabolism | Dopaminergic Neurons - cytology | Protein Kinase Inhibitors - chemistry | Dopaminergic Neurons - metabolism | Cytoprotection - drug effects | Dopaminergic Neurons - drug effects | MAP Kinase Kinase Kinase 5 - metabolism | Protein Structure, Tertiary | Amino Acid Sequence | Parkinson Disease - pathology | Peptides - chemistry | 1-Methyl-4-phenyl-1,2,3,6-tetrahydropyridine - adverse effects | Mice, Inbred C57BL | Mitochondria - drug effects | Mitochondria - pathology | Peptides - pharmacology | Mitogen-Activated Protein Kinase 10 - metabolism | Animals | Mitogen-Activated Protein Kinase 10 - chemistry | Signal Transduction - drug effects | beta-Arrestins | Cell Line, Tumor | Mice | Protein Kinase Inhibitors - pharmacology | Physiological aspects | Enzyme inhibitors | Research | Neurons | Cell culture | Neurosciences | Parkinson's disease | Peptides | Toxicity | Pathogenesis | Downstream effects | JNK3 protein | Parkinsons disease | Amino acids | Activation | MPP | Kinases | Proteins | Signal transduction | Mitochondria | Neurotoxicity | Neurodegeneration | Biocompatibility | Inhibition | Movement disorders | Dopamine receptors | Binding | Dopamine | Neurodegenerative diseases | MAP kinase | JNK protein | Medicine | Neurology | Signaling | Hospitals | MPTP | Laboratory animals | Apoptosis | Index Medicus
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