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Cancer Research, ISSN 0008-5472, 10/2004, Volume 64, Issue 19, pp. 7099 - 7109
Journal Article
Biochemical Journal, ISSN 0264-6021, 12/2007, Volume 408, Issue 3, pp. 297 - 315
The specificities of 65 compounds reported to be relatively specific inhibitors of protein kinases have been profiled against a panel of 70-80 protein kinases.... 
Drug discovery | Kinase profiling | Protein kinase | Anti-cancer drugs | Inhibitor specificity | RHO-ASSOCIATED KINASE | TUMOR PROGRESSION | FAMILY-MEMBERS | BIOCHEMISTRY & MOLECULAR BIOLOGY | CELL-PROLIFERATION | protein kinase | P38 MAP KINASE | CYCLIN-DEPENDENT KINASES | RECEPTOR TYROSINE KINASES | drug discovery | kinase profiling | SB 203580 | anti-cancer drugs | ISOFORMS IN-VITRO | P90 RSK | inhibitor specificity | Amino Acid Sequence | Cell Line | Phosphorylation | Recombinant Proteins - antagonists & inhibitors | Animals | Mitogen-Activated Protein Kinases - antagonists & inhibitors | Humans | Drug Design | Protein Kinase Inhibitors - pharmacology | Enzyme Activation | Mitogen-Activated Protein Kinases - metabolism | Spodoptera | Index Medicus | Yes1, Yamaguchi sarcoma viral oncogene homologue 1 | CSK, C-terminal Src kinase | Lck, lymphocyte cell-specific protein-tyrosine kinase | EGF, epidermal growth factor | FGF-R, fibroblast-growth-factor receptor | PAK, p21-activated protein kinase | PDK, 3-phosphoinositide-dependent protein kinase | PI3K, phosphatidylinositol (phosphoinositide) 3-kinase | NEK, NIMA (never in mitosis in Aspergillus nidulans)-related kinase | RSK, p90 ribosomal S6 kinase | HEK-293 cells, human embryonic kidney-293 cells | VEGF, vascular endothelial growth factor (vasoendothelial growth factor) | EF2K, elongation-factor-2 kinase | CK, casein kinase | PTEN, phosphatase and tensin homologue deleted on chromosome 10 | ERK, extracellular-signal-regulated kinase | ATM, ataxia telangiectasia mutated | SRPK, serine-arginine protein kinase | IL-1, interleukin 1 | MNK, MAPK-integrating protein kinase | ROCK, Rho-dependent protein kinase | CaMKK, CaMK kinase | GST, glutathione transferase | MKK1, MAPK kinase-1 (also called MEK1, MAPK or ERK kinase 1) | GAK, cyclin G-associated kinase | FMK, fluoromethylketone | MST, mammalian homologue Ste20-like kinase | PKA, cAMP-dependent protein kinase | FKBP, FK506-binding protein | PPAR, peroxisome-proliferator-activated receptor | IKK, inhibitory κB kinase | PH, pleckstrin homology | MBP, myelin basic protein | AICAR, aminoimidazole-4-carboxamide-1-β-D-ribofuranoside | MAPKAP-K, MAPK-activated protein kinase | Sf21, Spodoptera frugiperda (fall armyworm) 21 | MARK, microtubule-affinity-regulating kinase | PIM, provirus integration site for Moloney murine leukaemia virus | LPS, lipopolysaccharide | MSK, mitogen- and stress-activated protein kinase | MAPK, mitogen-activated protein kinase | MELK, maternal embryonic leucine-zipper kinase | His6, hexahistidine | CAK, cyclin-dependent kinase-activating kinase | Eph-A2, Ephrin A2 receptor | PLK, polo-like kinase | ATF2, activating transcription factor 2 | PKD, protein kinase D | Src, sarcoma kinase | AMPK, AMP-activated protein kinase | MMS, methyl methanesulfonate | CHK, checkpoint kinase | JNK, c-Jun N-terminal kinase | TORC1, mTOR (mammalian target of rapamycin)–raptor (regulatory associated protein of mTOR) complex | BRSK, brain-specific kinase | RIP2, receptor-interacting protein 2 | IGF-1, insulin-like growth factor-1 | S6K1, S6 kinase 1 | DYRK, dual-specificity tyrosine-phosphorylated and -regulated kinase | HIPK, homeodomain-interacting protein kinase | ZMP, aminoimidazole-4-carboxamide-1-β-D-ribofuranoside monophosphate | PRAK, p38-regulated activated kinase | PKC, protein kinase C | Src-I1, Src inhibitor 1 | TANK, TRAF (tumour-necrosis-factor-receptor-associated factor)-family-member-associated nuclear factor κB activator | NFAT, nuclear factor for activated T-cells | PHK, phosphorylase kinase | GSK3, glycogen synthase kinase 3 | PKB, protein kinase B (also called Akt) | CaMK, calmodulin-dependent kinase | CDK, cyclin-dependent protein kinase | NDRG, N-myc downstream-regulated gene | SmMLCK, smooth-muscle myosin light-chain kinase | TBK1, TANK-binding kinase 1 | PRK, protein kinase C-related kinase | SGK, serum- and glucocorticoid-induced kinase
Journal Article
Biochemical Journal, ISSN 0264-6021, 04/2013, Volume 451, Issue 2, pp. 313 - 328
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 03/2012, Volume 287, Issue 13, pp. 9742 - 9752
An intensive recent effort to develop ATP-competitive mTOR inhibitors has resulted in several potent and selective molecules such as Torin1, PP242, KU63794,... 
ACTIVATION | COMPLEX | POTENT | TOR | BIOCHEMISTRY & MOLECULAR BIOLOGY | MODEL | PROTEINS | DISCOVERY | TOR Serine-Threonine Kinases - metabolism | Tumor Suppressor Proteins - antagonists & inhibitors | Humans | Multiprotein Complexes | Phosphatidylinositol 3-Kinases - metabolism | Adenosine Triphosphate | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Mechanistic Target of Rapamycin Complex 1 | Cell Cycle Proteins - antagonists & inhibitors | DNA-Binding Proteins - metabolism | Dose-Response Relationship, Drug | TOR Serine-Threonine Kinases - antagonists & inhibitors | Enzyme Inhibitors - pharmacokinetics | Protein-Serine-Threonine Kinases - antagonists & inhibitors | p38 Mitogen-Activated Protein Kinases - metabolism | Phosphorylation - drug effects | Proteomics - methods | Protein-Serine-Threonine Kinases - metabolism | Tumor Suppressor Proteins - metabolism | DNA-Binding Proteins - antagonists & inhibitors | Cell Cycle Proteins - metabolism | Enzyme Inhibitors - pharmacology | Transcription Factors - antagonists & inhibitors | Ataxia Telangiectasia Mutated Proteins | Transcription Factors - metabolism | Proteins - metabolism | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Cell Line, Tumor | Kinetics | Proteins - antagonists & inhibitors | Index Medicus | Slow Off-rate | PI3K | Serine | Threonine-protein Kinase | WYE-354 | mTOR | Tyrosine-protein Kinase (Tyrosine Kinase) | Enzyme Kinetics | KU63794 | Enzymology | PP242 | Torin1
Journal Article
Science, ISSN 0036-8075, 2/2007, Volume 315, Issue 5814, pp. 1000 - 1003
Pathogenic bacteria use the type III secretion system to deliver effector proteins into host cells to modulate the host signaling pathways. In this study, the... 
Phosphates | Phosphorylation | Phosphatases | HeLa cells | Mass spectroscopy | Immunoblotting | Bacteria | Reports | Infections | Chemical bonding | Family members | SHIGELLA-FLEXNERI | CELLS | ACTIVATION | VIRULENCE PLASMID | MULTIDISCIPLINARY SCIENCES | SECRETION SYSTEM | HOST | INFECTION | EXPRESSION | Shigella flexneri - metabolism | Humans | Mitogen-Activated Protein Kinase 3 - antagonists & inhibitors | Phosphoprotein Phosphatases - metabolism | Molecular Sequence Data | JNK Mitogen-Activated Protein Kinases - metabolism | NF-kappa B - metabolism | Salmonella typhimurium | MAP Kinase Signaling System | p38 Mitogen-Activated Protein Kinases - metabolism | Amino Acid Sequence | Cell Line | JNK Mitogen-Activated Protein Kinases - antagonists & inhibitors | Mitogen-Activated Protein Kinase 1 - antagonists & inhibitors | Bacterial Proteins - genetics | Tyrosine - metabolism | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | Mitogen-Activated Protein Kinases - antagonists & inhibitors | Mutagenesis | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Bacterial Proteins - metabolism | HeLa Cells | Shigella flexneri - physiology | Mitogen-Activated Protein Kinase 1 - metabolism | Mitogen-Activated Protein Kinases - metabolism | Physiological aspects | Immune response | Gram-negative bacteria | Research | Secretion | Enzymes | Pathogens | Biochemistry | Kinases | Spectrum analysis | Index Medicus
Journal Article
by Park, SE and Sapkota, K and Kim, S and Kim, H and Kim, SJ
British Journal of Pharmacology, ISSN 0007-1188, 10/2011, Volume 164, Issue 3, pp. 1008 - 1025
BACKGROUND AND PURPOSE Kaempferol, a dietary flavonoid and phyto‐oestrogen, is known to have anti‐inflammatory properties. Microglial activation has been... 
mitogen‐activated protein kinases | NF‐κB | kaempferol | neuroinflammation | AKT | lipopolysaccharide | microglia | NF-κB | mitogen-activated protein kinases | NITRIC-OXIDE SYNTHASE | ALZHEIMERS-DISEASE | NEURODEGENERATIVE DISEASES | MATRIX METALLOPROTEINASES | NF-kappa B | TOLL-LIKE RECEPTORS | GENE-EXPRESSION | PHARMACOLOGY & PHARMACY | RAT-BRAIN | NF-KAPPA-B | INNATE IMMUNITY | PARKINSONS-DISEASE | Microglia - metabolism | Reactive Oxygen Species - metabolism | Dinoprostone - biosynthesis | NF-kappa B - metabolism | Mitogen-Activated Protein Kinase Kinases - metabolism | Cyclooxygenase 2 - genetics | Nitric Oxide Synthase Type II - antagonists & inhibitors | Protein Binding - drug effects | p38 Mitogen-Activated Protein Kinases - metabolism | Phosphorylation - drug effects | Toll-Like Receptor 4 - antagonists & inhibitors | Proto-Oncogene Proteins c-akt - metabolism | Interleukin-1beta - antagonists & inhibitors | Cell Survival - drug effects | Nitric Oxide - biosynthesis | NF-kappa B - antagonists & inhibitors | Phagocytosis - drug effects | Microglia - drug effects | Signal Transduction | Anti-Inflammatory Agents - pharmacology | Nitric Oxide - antagonists & inhibitors | Cells, Cultured | Microglia - enzymology | Matrix Metalloproteinase 3 - metabolism | Toll-Like Receptor 4 - metabolism | Reactive Oxygen Species - antagonists & inhibitors | Animals | Nitric Oxide Synthase Type II - genetics | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Cyclooxygenase 2 - metabolism | Lipopolysaccharides - pharmacology | Matrix Metalloproteinase Inhibitors | Kaempferols - pharmacology | Mice | Dinoprostone - antagonists & inhibitors | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Kinases | Index Medicus | Research Papers
Journal Article
Nature, ISSN 0028-0836, 10/2017, Volume 550, Issue 7674, pp. 133 - 136
Targeted BRAF inhibition (BRAFi) and combined BRAF and MEK inhibition (BRAFi and MEKi) therapies have markedly improved the clinical outcomes of patients with... 
METASTATIC MELANOMA | MEK | EPITHELIAL-CELLS | PHOSPHORYLATION | PATHWAY | RAF | P21-ACTIVATED-KINASE-1 | MULTIDISCIPLINARY SCIENCES | KINASE | MECHANISMS | THERAPIES | TOR Serine-Threonine Kinases - metabolism | Apoptosis - drug effects | Humans | p21-Activated Kinases - antagonists & inhibitors | Melanoma - enzymology | JNK Mitogen-Activated Protein Kinases - metabolism | Mitogen-Activated Protein Kinase Kinases - metabolism | Melanoma - genetics | Proto-Oncogene Proteins c-raf - chemistry | Female | beta Catenin - chemistry | Phosphorylation - drug effects | p21-Activated Kinases - genetics | JNK Mitogen-Activated Protein Kinases - chemistry | Enzyme Activation - drug effects | p21-Activated Kinases - metabolism | beta Catenin - metabolism | Proto-Oncogene Proteins c-raf - metabolism | Mitogen-Activated Protein Kinase Kinases - chemistry | Drug Resistance, Neoplasm - genetics | Animals | MAP Kinase Signaling System - drug effects | Mitogen-Activated Protein Kinases - antagonists & inhibitors | Signal Transduction - drug effects | Proto-Oncogene Proteins B-raf - genetics | Melanoma - drug therapy | Cell Line, Tumor | Mice | Protein Kinase Inhibitors - pharmacology | Mutation | Drug Resistance, Neoplasm - drug effects | Melanoma | Physiological aspects | Cellular signal transduction | Drug resistance | Observations | Health aspects | Mitogen-activated protein kinases | TOR protein | Therapy | Phosphorylation | Activation | Metastasis | Drug development | Kinases | Cancer therapies | Metastases | Proteins | β-catenin | Signal transduction | Inhibition | Extracellular signal-regulated kinase | MAP kinase | JNK protein | Patients | Signaling | Protein arrays | Molecular modelling | Biopsy | Cell lines | Apoptosis | Index Medicus
Journal Article
Nature, ISSN 0028-0836, 05/2008, Volume 453, Issue 7194, pp. 519 - 523
In the three decades since pluripotent mouse embryonic stem (ES) cells were first described they have been derived and maintained by using various empirical... 
MAINTENANCE | ACTIVATION | COMMITMENT | MECHANISM | TERATOCARCINOMA | MULTIDISCIPLINARY SCIENCES | PLURIPOTENCY | MOUSE ES CELLS | DIFFERENTIATION | EXPRESSION | PROTEIN-KINASE INHIBITORS | Embryonic Stem Cells - metabolism | Diphenylamine - pharmacology | Embryonic Stem Cells - cytology | Diphenylamine - analogs & derivatives | STAT3 Transcription Factor - deficiency | Benzamides - pharmacology | STAT3 Transcription Factor - genetics | STAT3 Transcription Factor - metabolism | Cell Survival - drug effects | Pluripotent Stem Cells - cytology | Glycogen Synthase Kinase 3 - antagonists & inhibitors | Cells, Cultured | Pyrimidines - pharmacology | Glycogen Synthase Kinase 3 - metabolism | Regeneration - physiology | Pluripotent Stem Cells - metabolism | Regeneration - drug effects | Animals | MAP Kinase Signaling System - drug effects | Embryonic Stem Cells - drug effects | Mitogen-Activated Protein Kinases - antagonists & inhibitors | Cell Differentiation - drug effects | Pluripotent Stem Cells - drug effects | Cell Proliferation - drug effects | Mice | Pyridines - pharmacology | Mitogen-Activated Protein Kinases - metabolism | Morphogenesis | Cytokines | Influence | Regeneration (Biology) | Research | Genetic transcription | Embryonic stem cells | Properties | Protein kinases | Proteins | Signal transduction | Cyclin-dependent kinases | Stem cells | Cell cycle | Kinases | Apoptosis | Index Medicus
Journal Article
Journal of Immunology, ISSN 0022-1767, 08/2014, Volume 193, Issue 4, pp. 1954 - 1965
Canonical neutrophil antimicrobial effector mechanisms, such as degranulation, production of reactive oxygen species, and release of neutrophil extracellular... 
MAMMALIAN TARGET | NADPH OXIDASE | NETTING NEUTROPHILS | SIGNALING PATHWAYS | RESPIRATORY BURST | SYNOVIAL-FLUID | SUPEROXIDE-PRODUCTION | IN-VIVO | IMMUNOLOGY | RECEPTOR-III | CELL-DEATH | Reactive Oxygen Species - metabolism | Nitriles - pharmacology | Humans | Extracellular Signal-Regulated MAP Kinases - antagonists & inhibitors | Intracellular Signaling Peptides and Proteins - immunology | Macrophage-1 Antigen - immunology | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Receptors, IgG - antagonists & inhibitors | Protein-Tyrosine Kinases - immunology | Lymphocyte Function-Associated Antigen-1 - immunology | Macrophage-1 Antigen - metabolism | Syk Kinase | CD11a Antigen - metabolism | Butadienes - pharmacology | Neutrophil Activation - immunology | GPI-Linked Proteins - immunology | Cell Degranulation | Intracellular Signaling Peptides and Proteins - antagonists & inhibitors | src-Family Kinases - antagonists & inhibitors | Autoimmune Diseases - immunology | Cells, Cultured | Neutrophils - immunology | Imidazoles - pharmacology | Inflammation - immunology | Antioxidants - pharmacology | Onium Compounds - pharmacology | Pyrimidines - pharmacology | Receptors, IgG - immunology | src-Family Kinases - immunology | CD18 Antigens - metabolism | Mesalamine - pharmacology | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Aminopyrine - pharmacology | Ascorbic Acid - pharmacology | Pyridines - pharmacology | Antigen-Antibody Complex - immunology | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Protein-Tyrosine Kinases - antagonists & inhibitors | Index Medicus | Abridged Index Medicus
Journal Article
Journal of Cell Science, ISSN 0021-9533, 2013, Volume 126, Issue 17, pp. 3990 - 3999
The crucial role the Crumbs and Par polarity complexes play in tight junction integrity has long been established, however very few studies have investigated... 
Scribble | Polarity | Tight junctions | Mammary epithelium | EMT | ERK | CELL-CELL JUNCTIONS | PROTEIN-KINASE-C | BARRIER FUNCTION | E-CADHERIN | MAMMALIAN EPITHELIAL-CELLS | BETA-CATENIN | MESENCHYMAL TRANSITION | TIGHT JUNCTION | CELL BIOLOGY | MALIGNANT PROGRESSION | TUMOR-SUPPRESSOR SCRIBBLE | MAP Kinase Signaling System - physiology | Phosphorylation | Epithelial-Mesenchymal Transition - physiology | Homeodomain Proteins - metabolism | Humans | Membrane Glycoproteins - biosynthesis | Mitogen-Activated Protein Kinase 3 - antagonists & inhibitors | Transcriptional Activation | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | RNA Interference | Mitogen-Activated Protein Kinase 1 - genetics | HEK293 Cells | Transcription, Genetic | Flavonoids - pharmacology | Membrane Proteins - metabolism | Chromones - pharmacology | Tight Junctions - metabolism | MAP Kinase Kinase 1 - antagonists & inhibitors | Tumor Suppressor Proteins - metabolism | Homeodomain Proteins - biosynthesis | JNK Mitogen-Activated Protein Kinases - antagonists & inhibitors | Mitogen-Activated Protein Kinase 3 - genetics | Down-Regulation | Mitogen-Activated Protein Kinase 1 - antagonists & inhibitors | Morpholines - pharmacology | Transcription Factors - antagonists & inhibitors | Transcription Factors - biosynthesis | Anthracenes - pharmacology | Transcription Factors - metabolism | Membrane Proteins - biosynthesis | MAP Kinase Signaling System - drug effects | Mitogen-Activated Protein Kinase 3 - metabolism | Homeodomain Proteins - antagonists & inhibitors | Protein Kinase Inhibitors - pharmacology | RNA, Small Interfering | Tumor Suppressor Proteins - biosynthesis | Mitogen-Activated Protein Kinase 1 - metabolism | Zinc Finger E-box-Binding Homeobox 1 | Index Medicus
Journal Article