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Cell Reports, ISSN 2211-1247, 09/2016, Volume 16, Issue 10, pp. 2576 - 2592
The mechanisms underlying Zika virus (ZIKV)-related microcephaly and other neurodevelopment defects remain poorly understood. Here, we describe the derivation... 
NEURAL PROGENITORS | LONG-TERM | HUMAN BRAIN | ADAPTER | CENTRAL-NERVOUS-SYSTEM | INFECTION | MICE | BINDING KINASE 1 | ORGANOIDS | INNATE IMMUNITY | CELL BIOLOGY | Neocortex - pathology | Neurons - pathology | Transcription, Genetic - drug effects | Brain - embryology | Neuroglia - ultrastructure | Neuroglia - pathology | Humans | Brain - virology | Centrosome - drug effects | Gene Expression Profiling | Microcephaly - virology | Neural Stem Cells - ultrastructure | Zika Virus Infection - virology | Neural Stem Cells - immunology | Neuroepithelial Cells - immunology | Neuroprotective Agents - pharmacology | Spinal Cord - pathology | Microcephaly - pathology | Neuroepithelial Cells - virology | Nucleosides - pharmacology | Fetus - virology | Cell Death - drug effects | Phosphorylation - drug effects | Neurons - drug effects | Protein-Serine-Threonine Kinases - metabolism | Zika Virus - pathogenicity | Proto-Oncogene Proteins - metabolism | Zika Virus - ultrastructure | Neurons - virology | Virus Replication - drug effects | Neuroepithelial Cells - ultrastructure | Immunity, Innate - drug effects | Zika Virus Infection - pathology | Neural Stem Cells - virology | Zika Virus - physiology | Zika Virus - drug effects | Mitochondria - metabolism | Mitochondria - drug effects | Receptor Protein-Tyrosine Kinases - metabolism | Neural Stem Cells - enzymology | Centrosome - metabolism | Mitosis - drug effects | Brain - pathology | Protein Kinase Inhibitors - pharmacology | Neuroglia - virology | Neuroepithelial Cells - drug effects | Index Medicus | Neurons/pathology | Zika Virus/pathogenicity | Mitochondria/metabolism | Virus Replication/drug effects | Microcephaly/pathology | Neurons/drug effects | Protein-Serine-Threonine Kinases/metabolism | Neural Stem Cells/immunology | Neuroglia/ultrastructure | Neuroepithelial Cells/drug effects | Neuroepithelial Cells/virology | Life Sciences | Brain/pathology | Zika Virus/drug effects | Brain/embryology | Mitochondria/drug effects | Fetus/virology | Neocortex/pathology | Neuroglia/pathology | Cell Death/drug effects | Mitosis/drug effects | Transcription, Genetic/drug effects | Nucleosides/pharmacology | Neural Stem Cells/enzymology | Neural Stem Cells/ultrastructure | Neuroepithelial Cells/ultrastructure | Receptor Protein-Tyrosine Kinases/metabolism | Microcephaly/virology | Proto-Oncogene Proteins/metabolism | Neuroprotective Agents/pharmacology | Zika Virus/ultrastructure | Neuroepithelial Cells/immunology | Brain/virology | Immunity, Innate/drug effects | Spinal Cord/pathology | Zika Virus/physiology | Neuroglia/virology | Microbiology and Parasitology | Zika Virus Infection/pathology | Neurons/virology | Zika Virus Infection/virology | Neural Stem Cells/virology | Centrosome/drug effects | Protein Kinase Inhibitors/pharmacology | Centrosome/metabolism | Phosphorylation/drug effects
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2013, Volume 8, Issue 4, pp. e62082 - e62082
Some potent chemotherapy drugs including tubulin-binding agents had been developed from nature plants, such as podophyllotoxin and paclitaxel. However, poor... 
MITOSIS | ANTIMITOTIC ACTIVITY | MICROTUBULES | NATURAL-PRODUCTS | MULTIDISCIPLINARY SCIENCES | DRUG DISCOVERY | RESISTANCE | TUBULIN | BINDING AGENTS | EXPRESSION | KINASES | Lung Neoplasms - drug therapy | Podophyllotoxin - pharmacology | Apoptosis - drug effects | Humans | Lung Neoplasms - metabolism | Apoptosis - genetics | Endoplasmic Reticulum Stress - genetics | Microtubules - metabolism | Podophyllotoxin - toxicity | Drug Evaluation, Preclinical | Disease Models, Animal | DNA Damage - drug effects | Lung Neoplasms - genetics | M Phase Cell Cycle Checkpoints - drug effects | Endoplasmic Reticulum Stress - drug effects | Antineoplastic Agents, Phytogenic - toxicity | Xenograft Model Antitumor Assays | Animals | Mitosis - drug effects | Signal Transduction - drug effects | Tumor Burden - drug effects | Cell Cycle Checkpoints - drug effects | Models, Biological | Cell Line, Tumor | Cell Proliferation - drug effects | Mice | Antineoplastic Agents, Phytogenic - pharmacology | Protein Multimerization - drug effects | Podophyllotoxin - analogs & derivatives | Chemotherapy | Podophyllotoxin | Analysis | Lung cancer | Polymerization | Stress (Physiology) | Tubulins | Health aspects | Apoptosis | Cancer | Drugs | Flow cytometry | Toxicity | Mitosis | Leukemia | Lung | DNA damage | Cytotoxicity | Selectivity | Drug development | Kinases | Cancer therapies | Anticancer properties | Metastases | Proteins | Signal transduction | Tubulin | Paclitaxel | Xenografts | Cell cycle | Inhibition | Stains | Deoxyribonucleic acid--DNA | Stresses | Plants (botany) | Hematology | Injection | Survivin | Tumor cell lines | Gene expression | Stress | Aurora B protein | Signaling | Side effects | Cell lines | Colonization | Index Medicus | Deoxyribonucleic acid | DNA
Journal Article
Journal of Cellular Physiology, ISSN 0021-9541, 05/2005, Volume 203, Issue 2, pp. 398 - 409
Human mesenchymal stem cells (hMSCs) expanded with and without fibroblast growth factor (FGF) supplementation were compared with respect to their proliferation... 
PROGENITOR CELLS | CROSS-TALK | PROTEIN-KINASE-C | IN-VITRO | PHYSIOLOGY | ATDC5 CELLS | DEFICIENT MDX MICE | STROMAL CELLS | GENE-EXPRESSION SIGNATURES | CHONDROCYTE DIFFERENTIATION | N-CADHERIN | CELL BIOLOGY | Chondrocytes - cytology | Chondrogenesis - drug effects | Age Factors | Oligonucleotide Array Sequence Analysis | Humans | Fibroblast Growth Factor 2 - pharmacology | Bone Marrow Cells - physiology | Gene Expression Profiling | Cell Culture Techniques - methods | Proteoglycans - drug effects | Cell Differentiation - genetics | Chondrocytes - drug effects | Mesenchymal Stromal Cells - cytology | Chondrocytes - physiology | Collagen - drug effects | Up-Regulation - physiology | Bone Marrow Cells - drug effects | Mesenchymal Stromal Cells - physiology | Mesenchymal Stromal Cells - drug effects | Tissue Engineering - methods | Bone Marrow Cells - cytology | Gene Expression Regulation - genetics | Extracellular Matrix Proteins - genetics | Cells, Cultured | Proteoglycans - metabolism | Down-Regulation - drug effects | Extracellular Matrix Proteins - drug effects | Down-Regulation - physiology | Gene Expression Regulation - drug effects | Up-Regulation - drug effects | Chondrogenesis - physiology | Collagen - metabolism | Mitosis - drug effects | Mitosis - physiology | Signal Transduction - drug effects | Cell Differentiation - drug effects | Signal Transduction - physiology | Cell Proliferation - drug effects | Index Medicus
Journal Article
Plant Physiology, ISSN 0032-0889, 4/2013, Volume 161, Issue 4, pp. 1930 - 1951
Phytohormones regulate plant growth from cell division to organ development. Jasmonates (JAs) are signaling molecules that have been implicated in... 
Cell growth | Plant growth regulators | Developmental biology | Genes | Epidermal cells | Cell cycle | Gene expression regulation | Plants | GENES, DEVELOPMENT, AND EVOLUTION | Plant cells | Seedlings | ARABIDOPSIS MUTANT CEV1 | TOBACCO BY-2 CELLS | METHYL JASMONATE | PROBE LEVEL DATA | GENOME-WIDE ANALYSIS | GENE-EXPRESSION | A-TYPE CYCLIN | PLANT DEVELOPMENT | TRANSCRIPTION FACTOR | DNA-REPLICATION | PLANT SCIENCES | Meristem - cytology | Arabidopsis - growth & development | Cell Nucleus Size - drug effects | DNA Replication - drug effects | Cell Count | Cotyledon - drug effects | Gene Expression Profiling | Mitosis - genetics | Ribosomal Proteins - metabolism | Arabidopsis Proteins - metabolism | Cell Nucleus - metabolism | Plant Leaves - drug effects | Meristem - drug effects | Arabidopsis Proteins - genetics | Arabidopsis - drug effects | Endoreduplication - genetics | Ribosomal Proteins - genetics | Arabidopsis - cytology | Cell Size - drug effects | Endoreduplication - drug effects | Cyclopentanes - pharmacology | DNA, Plant - metabolism | Down-Regulation - drug effects | Plant Leaves - cytology | Arabidopsis - genetics | Gene Expression Regulation, Plant - drug effects | Phenotype | Mitosis - drug effects | Models, Biological | Plant Leaves - growth & development | Oxylipins - pharmacology | Cotyledon - growth & development | Acetates - pharmacology | Cell Proliferation - drug effects | Cell Nucleus - drug effects | Cluster Analysis | Cell proliferation | Plant genetics | Research | Jasmonates | Properties | Observations | Testing | Index Medicus | Life Sciences | Molecular biology | Cellular Biology | Biochemistry, Molecular Biology
Journal Article
Developmental Cell, ISSN 1534-5807, 10/2010, Volume 19, Issue 4, pp. 612 - 624
Journal Article
Cell Metabolism, ISSN 1550-4131, 05/2016, Volume 23, Issue 5, pp. 797 - 810
Journal Article
Molecular Psychiatry, ISSN 1359-4184, 08/2009, Volume 14, Issue 8, pp. 764 - 773
The mechanisms underlying the initiation/onset of, and the recovery from, depression are still largely unknown; views that neurogenesis in the hippocampus may... 
Antidepressant | Neuroplasticity | Synapsin 1 | Depression | NCAM | Neurogenesis | TREATMENT INCREASES | ADULT NEUROGENESIS | PSYCHIATRY | BIOCHEMISTRY & MOLECULAR BIOLOGY | antidepressant | STRESS-INDUCED CHANGES | synapsin 1 | CELL-PROLIFERATION | NEUROSCIENCES | CHRONIC FLUOXETINE | neurogenesis | MEDIAL PREFRONTAL CORTEX | RAT HIPPOCAMPUS | depression | DENTATE GYRUS | PSA-NCAM | HIPPOCAMPAL NEUROGENESIS | neuroplasticity | Rats, Wistar | Neuronal Plasticity - drug effects | Male | Hippocampus - drug effects | Methylazoxymethanol Acetate - analogs & derivatives | Pyrrolidines - pharmacology | Depression - etiology | Depression - drug therapy | Neuronal Plasticity - physiology | Prefrontal Cortex - drug effects | Behavior, Animal - drug effects | Indoles - pharmacology | Antidepressive Agents - pharmacology | Neurogenesis - drug effects | Cytostatic Agents - pharmacology | Fluoxetine - pharmacology | Affect - drug effects | Affect - physiology | Rats | Behavior, Animal - physiology | Hippocampus - cytology | Prefrontal Cortex - cytology | Methylazoxymethanol Acetate - pharmacology | Animals | Mitosis - drug effects | Receptors, Corticotropin-Releasing Hormone - antagonists & inhibitors | Analysis of Variance | Neurogenesis - physiology | Antidiuretic Hormone Receptor Antagonists | Hippocampus - physiology | Imipramine - pharmacology | Prefrontal Cortex - physiology | Drug Combinations | Stress, Psychological - complications | Stress, Psychological - physiopathology | Antidepressants | Depression, Mental | Physiological aspects | Dosage and administration | Genetic aspects | Research | Hippocampus (Brain) | Drug therapy | Health aspects | Index Medicus
Journal Article
The Journal of Cell Biology, ISSN 0021-9525, 4/2003, Volume 161, Issue 2, pp. 267 - 280
The Aurora/Ipl1 family of protein kinases plays multiple roles in mitosis and cytokinesis. Here, we describe ZM447439, a novel selective Aurora kinase... 
Mitotic index | Mitosis | Kinetochores | Microtubules | DNA | Cell lines | HeLa cells | Cultured cells | Chromosomes | Cells | Chemical biology | Aneuploidy | Spindle checkpoint | ZM447439 | KINASE-ACTIVITY | LOCALIZATION | BUDDING YEAST | PHOSPHORYLATION | TENSION | mitosis | MAMMALIAN-CELLS | CELL BIOLOGY | chemical biology | CENTROSOME AMPLIFICATION | HISTONE H3 | aneuploidy | spindle checkpoint | Protein Kinases - metabolism | Protein Kinases - genetics | Paclitaxel - pharmacology | DNA Replication - drug effects | Humans | Chromosome Segregation - drug effects | Mitosis - genetics | Tumor Suppressor Protein p53 - genetics | Aurora Kinase B | Spindle Apparatus - genetics | Eukaryotic Cells - ultrastructure | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Benzamides - pharmacology | Mad2 Proteins | Phosphorylation - drug effects | Calcium-Binding Proteins - metabolism | Eukaryotic Cells - enzymology | Chromosomal Proteins, Non-Histone - metabolism | Kinetochores - metabolism | Enzyme Inhibitors - pharmacology | Protein-Serine-Threonine Kinases - genetics | Repressor Proteins - genetics | Genes, cdc - drug effects | Aurora Kinases | Tumor Suppressor Protein p53 - drug effects | Anaphase - drug effects | Chromosomal Proteins, Non-Histone - genetics | Anaphase - genetics | Eukaryotic Cells - drug effects | Mitosis - drug effects | DNA Replication - genetics | Repressor Proteins - drug effects | Kinetochores - drug effects | Spindle Apparatus - drug effects | HeLa Cells | Quinazolines - pharmacology | Cell Cycle Proteins | Calcium-Binding Proteins - genetics | Chromosome Segregation - genetics | Research | Cytokinesis | Protein kinases | Proteins | Genes | Index Medicus | mitosis; spindle checkpoint; chemical biology; aneuploidy; ZM447439
Journal Article