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Cell reports (Cambridge), ISSN 2211-1247, 2016, Volume 16, Issue 10, pp. 2576 - 2592
The mechanisms underlying Zika virus (ZIKV)-related microcephaly and other neurodevelopment defects remain poorly understood. Here, we describe the derivation... 
NEURAL PROGENITORS | LONG-TERM | HUMAN BRAIN | ADAPTER | CENTRAL-NERVOUS-SYSTEM | INFECTION | MICE | BINDING KINASE 1 | ORGANOIDS | INNATE IMMUNITY | CELL BIOLOGY | Neocortex - pathology | Neurons - pathology | Transcription, Genetic - drug effects | Brain - embryology | Neuroglia - ultrastructure | Neuroglia - pathology | Humans | Brain - virology | Centrosome - drug effects | Gene Expression Profiling | Microcephaly - virology | Neural Stem Cells - ultrastructure | Zika Virus Infection - virology | Neural Stem Cells - immunology | Neuroepithelial Cells - immunology | Neuroprotective Agents - pharmacology | Spinal Cord - pathology | Microcephaly - pathology | Neuroepithelial Cells - virology | Nucleosides - pharmacology | Fetus - virology | Cell Death - drug effects | Phosphorylation - drug effects | Neurons - drug effects | Protein-Serine-Threonine Kinases - metabolism | Zika Virus - pathogenicity | Proto-Oncogene Proteins - metabolism | Zika Virus - ultrastructure | Neurons - virology | Virus Replication - drug effects | Neuroepithelial Cells - ultrastructure | Immunity, Innate - drug effects | Zika Virus Infection - pathology | Neural Stem Cells - virology | Zika Virus - physiology | Zika Virus - drug effects | Mitochondria - metabolism | Mitochondria - drug effects | Receptor Protein-Tyrosine Kinases - metabolism | Neural Stem Cells - enzymology | Centrosome - metabolism | Mitosis - drug effects | Brain - pathology | Protein Kinase Inhibitors - pharmacology | Neuroglia - virology | Neuroepithelial Cells - drug effects | Neurons/pathology | Zika Virus/pathogenicity | Mitochondria/metabolism | Virus Replication/drug effects | Microcephaly/pathology | Neurons/drug effects | Protein-Serine-Threonine Kinases/metabolism | Neural Stem Cells/immunology | Neuroglia/ultrastructure | Neuroepithelial Cells/drug effects | Neuroepithelial Cells/virology | Life Sciences | Brain/pathology | Zika Virus/drug effects | Brain/embryology | Mitochondria/drug effects | Fetus/virology | Neocortex/pathology | Neuroglia/pathology | Cell Death/drug effects | Mitosis/drug effects | Transcription, Genetic/drug effects | Nucleosides/pharmacology | Neural Stem Cells/enzymology | Neural Stem Cells/ultrastructure | Neuroepithelial Cells/ultrastructure | Receptor Protein-Tyrosine Kinases/metabolism | Microcephaly/virology | Proto-Oncogene Proteins/metabolism | Neuroprotective Agents/pharmacology | Zika Virus/ultrastructure | Neuroepithelial Cells/immunology | Brain/virology | Immunity, Innate/drug effects | Spinal Cord/pathology | Zika Virus/physiology | Neuroglia/virology | Microbiology and Parasitology | Zika Virus Infection/pathology | Neurons/virology | Zika Virus Infection/virology | Neural Stem Cells/virology | Centrosome/drug effects | Protein Kinase Inhibitors/pharmacology | Centrosome/metabolism | Phosphorylation/drug effects
Journal Article
Cancer Biology & Therapy, ISSN 1555-8576, 2014, Volume 11, Issue 5, pp. 464 - 473
....1 Current therapeutic strategies include surgery, radiation therapy and chemotherapy, but these are associated with significant side effects and only limited... 
Binding | Proteins | Landes | Calcium | Bioscience | Biology | Cell | Cycle | Cancer | Organogenesis | Hedgehog | Medulloblastoma | Nanocurcumin | Glioblastoma | IGF | Curcumin | NanoCurc | NEURAL PROGENITORS | SIGNALING PATHWAYS | ACTIVATION | nanocurcumin | medulloblastoma | PANCREATIC-CANCER | nanoCurc (TM) | MEDULLOBLASTOMA CELLS | INSULIN | glioblastoma | hedgehog | ONCOLOGY | FACTOR-I RECEPTOR | NF-KAPPA-B | EXPRESSION | GLIOBLASTOMA CELLS | curcumin | Apoptosis - drug effects | Neoplastic Stem Cells - drug effects | Curcumin - chemistry | Humans | Nanocapsules | Receptors, Notch - genetics | Glycoproteins - drug effects | Antineoplastic Agents - administration & dosage | Curcumin - administration & dosage | Hedgehog Proteins - genetics | Medulloblastoma - pathology | Glioblastoma - metabolism | Antineoplastic Agents - pharmacology | Polymers | Neoplastic Stem Cells - physiology | Tumor Stem Cell Assay | Curcumin - pharmacology | Hedgehog Proteins - analysis | STAT3 Transcription Factor - analysis | Medulloblastoma - metabolism | Antigens, CD - drug effects | Antineoplastic Agents - chemistry | Down-Regulation - drug effects | AC133 Antigen | Peptides - drug effects | Receptors, Notch - analysis | Mitosis - drug effects | Signal Transduction - drug effects | Glioblastoma - pathology | Somatomedins - genetics | Cell Line, Tumor | Cell Proliferation - drug effects | Glioblastoma - drug therapy | Medulloblastoma - drug therapy | nanoCurc | Research Paper
Journal Article
Plant physiology (Bethesda), ISSN 1532-2548, 2009, Volume 151, Issue 4, pp. 2162 - 2173
Replication protein A (RPA), a highly conserved single-stranded DNA-binding protein in eukaryotes, is a stable complex comprising three subunits termed RPA1,... 
Somatic cells | DNA | Irradiation | Systems Biology, Molecular Biology, and Gene Regulation | Meiosis | Megasporocytes | Plants | Embryo sac | DNA repair | Chromosomes | Rice | PATHWAYS | CELLS | STRAND BREAK REPAIR | GENE | HIGHER-PLANTS | MEIOSIS | ARABIDOPSIS | GENOME | DAMAGE | NUCLEOTIDE EXCISION-REPAIR | PLANT SCIENCES | RNA Interference - drug effects | Germ Cells, Plant - radiation effects | DNA Replication - drug effects | DNA Repair - radiation effects | Genetic Complementation Test | DNA Fragmentation - radiation effects | Meiosis - drug effects | Chromosomes, Plant - radiation effects | Ultraviolet Rays | Oryza - genetics | DNA Replication - radiation effects | Plants, Genetically Modified | Plant Proteins - metabolism | DNA Fragmentation - drug effects | Oryza - embryology | Pollen - cytology | Pollen - drug effects | Germ Cells, Plant - drug effects | Chromosomes, Plant - drug effects | DNA Repair - drug effects | Mitosis - radiation effects | Mutagens - pharmacology | Replication Protein A - metabolism | Oryza - drug effects | RNA Interference - radiation effects | Chromosomes, Plant - metabolism | Mutation - genetics | Germ Cells, Plant - growth & development | Pollen - radiation effects | Mitomycin - pharmacology | Phenotype | DNA, Bacterial - genetics | Meiosis - radiation effects | Mitosis - drug effects | Genes, Plant - genetics | Recombination, Genetic - radiation effects | Methyl Methanesulfonate - pharmacology | Oryza - cytology | Recombination, Genetic - drug effects | Arabidopsis thaliana | DNA replication | Physiological aspects | Environmental aspects | Genetic aspects | DNA binding proteins | Properties
Journal Article
The Journal of cell biology, ISSN 1540-8140, 2003, Volume 161, Issue 2, pp. 281 - 294
The proper segregation of sister chromatids in mitosis depends on bipolar attachment of all chromosomes to the mitotic spindle. We have identified the small... 
Mitosis | Kinetochores | Chromatids | Microtubules | HeLa cells | Cellular immunity | Chromosomes | Anaphase | Cells | Mitotic spindle apparatus | Chemical biology | Spindle assembly checkpoint | Chromosome segregation | KINASE-ACTIVITY | MAD2 | BUDDING YEAST | spindle assembly checkpoint | TENSION | G/M TRANSITION | mitosis | HISTONE H3 PHOSPHORYLATION | MAMMALIAN-CELLS | CELL BIOLOGY | kinetochores | chromosome segregation | chemical biology | VERTEBRATE SOMATIC-CELLS | Cell Cycle Proteins - drug effects | Protein Kinases - metabolism | RNA, Small Interfering - genetics | Paclitaxel - pharmacology | Humans | Nocodazole - pharmacology | Chromosome Segregation - drug effects | Mitosis - genetics | Aurora Kinase B | Microtubules - drug effects | Spindle Apparatus - genetics | Cell Cycle Proteins - genetics | Genes, cdc - physiology | Indoles - pharmacology | Kinetochores - enzymology | Protein-Serine-Threonine Kinases - metabolism | Aneugens - pharmacology | Eukaryotic Cells - enzymology | Polyploidy | Protein Kinases - drug effects | Separase | Protein-Serine-Threonine Kinases - genetics | Genes, cdc - drug effects | Aurora Kinases | Pyrimidines - pharmacology | Sulfonamides - pharmacology | Anaphase - drug effects | Phenotype | Anaphase - genetics | Animals | Eukaryotic Cells - drug effects | Mitosis - drug effects | Microtubules - genetics | Microtubules - enzymology | Protein-Serine-Threonine Kinases - drug effects | Eukaryotic Cells - cytology | Kinetochores - drug effects | Spindle Apparatus - drug effects | HeLa Cells | Thiones - pharmacology | Endopeptidases | Chromosome Segregation - genetics | Spindle Apparatus - enzymology | Research | mitosis; chromosome segregation; kinetochores; spindle assembly checkpoint; chemical biology
Journal Article
Journal Article
PloS one, ISSN 1932-6203, 04/2013, Volume 8, Issue 4, p. e62082
Some potent chemotherapy drugs including tubulin-binding agents had been developed from nature plants, such as podophyllotoxin and paclitaxel... 
MITOSIS | ANTIMITOTIC ACTIVITY | MICROTUBULES | NATURAL-PRODUCTS | MULTIDISCIPLINARY SCIENCES | DRUG DISCOVERY | RESISTANCE | TUBULIN | BINDING AGENTS | EXPRESSION | KINASES | Lung Neoplasms - drug therapy | Podophyllotoxin - pharmacology | Apoptosis - drug effects | Humans | Lung Neoplasms - metabolism | Apoptosis - genetics | Endoplasmic Reticulum Stress - genetics | Microtubules - metabolism | Podophyllotoxin - toxicity | Drug Evaluation, Preclinical | Disease Models, Animal | DNA Damage - drug effects | Lung Neoplasms - genetics | M Phase Cell Cycle Checkpoints - drug effects | Endoplasmic Reticulum Stress - drug effects | Antineoplastic Agents, Phytogenic - toxicity | Xenograft Model Antitumor Assays | Animals | Mitosis - drug effects | Signal Transduction - drug effects | Tumor Burden - drug effects | Cell Cycle Checkpoints - drug effects | Models, Biological | Cell Line, Tumor | Cell Proliferation - drug effects | Mice | Antineoplastic Agents, Phytogenic - pharmacology | Protein Multimerization - drug effects | Podophyllotoxin - analogs & derivatives | Chemotherapy | Podophyllotoxin | Analysis | Lung cancer | Polymerization | Stress (Physiology) | Tubulins | Health aspects | Apoptosis | Cancer | Drugs | Flow cytometry | Toxicity | Mitosis | Leukemia | DNA damage | Cytotoxicity | Selectivity | Biochemistry | Drug development | Kinases | Cancer therapies | Anticancer properties | Metastases | Proteins | Signal transduction | Tubulin | Paclitaxel | Xenografts | Cell cycle | Inhibition | Deoxyribonucleic acid--DNA | Stresses | Plants (botany) | Hematology | Injection | Survivin | Tumor cell lines | Gene expression | Stress | Aurora B protein | Signaling | Side effects | Colonization | Deoxyribonucleic acid | Stains | DNA
Journal Article
Molecular psychiatry, ISSN 1476-5578, 2008, Volume 14, Issue 8, pp. 764 - 773
The mechanisms underlying the initiation/onset of, and the recovery from, depression are still largely unknown; views that neurogenesis in the hippocampus may... 
Antidepressant | Neuroplasticity | Synapsin 1 | Depression | NCAM | Neurogenesis | TREATMENT INCREASES | ADULT NEUROGENESIS | PSYCHIATRY | BIOCHEMISTRY & MOLECULAR BIOLOGY | antidepressant | STRESS-INDUCED CHANGES | synapsin 1 | CELL-PROLIFERATION | NEUROSCIENCES | CHRONIC FLUOXETINE | neurogenesis | MEDIAL PREFRONTAL CORTEX | RAT HIPPOCAMPUS | depression | DENTATE GYRUS | PSA-NCAM | HIPPOCAMPAL NEUROGENESIS | neuroplasticity | Rats, Wistar | Neuronal Plasticity - drug effects | Male | Hippocampus - drug effects | Methylazoxymethanol Acetate - analogs & derivatives | Pyrrolidines - pharmacology | Depression - etiology | Depression - drug therapy | Neuronal Plasticity - physiology | Prefrontal Cortex - drug effects | Behavior, Animal - drug effects | Indoles - pharmacology | Antidepressive Agents - pharmacology | Neurogenesis - drug effects | Cytostatic Agents - pharmacology | Fluoxetine - pharmacology | Affect - drug effects | Affect - physiology | Rats | Behavior, Animal - physiology | Hippocampus - cytology | Prefrontal Cortex - cytology | Methylazoxymethanol Acetate - pharmacology | Animals | Mitosis - drug effects | Receptors, Corticotropin-Releasing Hormone - antagonists & inhibitors | Analysis of Variance | Neurogenesis - physiology | Antidiuretic Hormone Receptor Antagonists | Hippocampus - physiology | Imipramine - pharmacology | Prefrontal Cortex - physiology | Drug Combinations | Stress, Psychological - complications | Stress, Psychological - physiopathology | Antidepressants | Depression, Mental | Physiological aspects | Dosage and administration | Genetic aspects | Research | Hippocampus (Brain) | Drug therapy | Health aspects | Index Medicus
Journal Article
Nature genetics, ISSN 1546-1718, 2001, Volume 27, Issue 1, pp. 48 - 54
Journal Article
The New phytologist, ISSN 0028-646X, 2014, Volume 203, Issue 4, pp. 1175 - 1193
The role of YODA MITOGEN ACTIVATED PROTEIN KINASE KINASE KINASE 4 (MAPKKK4) upstream of MITOGEN ACTIVATED PROTEIN KINASE 6 (MPK6) was studied during... 
Phosphates | Full papers | Dehydrogenases | Ribosomal proteins | Carrier proteins | Adenosine triphosphatases | Heat shock proteins | Trip chaining | Peptide elongation factors | Families | Chaperonins | MAPKKK | MAP65‐1 | Arabidopsis | root | MPK6 | YODA | microtubules | cell division plane | MAP65-1 | Root | Microtubules | Cell division plane | MICROTUBULE ARRAYS | SITE | STOMATAL DEVELOPMENT | MAP KINASE | FATE | PHRAGMOPLAST | PLANT SCIENCES | CYTOKINESIS | THALIANA | GROWTH | GENE FAMILY | Meristem - cytology | Arabidopsis - enzymology | Arabidopsis - embryology | Protein Transport - drug effects | Arabidopsis Proteins - metabolism | Microtubules - metabolism | Microtubules - drug effects | Protein Binding - drug effects | Plant Epidermis - cytology | Meristem - drug effects | Phosphorylation - drug effects | Cytokinesis - drug effects | Arabidopsis - drug effects | Indoleacetic Acids - metabolism | Interphase | Arabidopsis - cytology | MAP Kinase Kinase Kinases - metabolism | Plant Roots - cytology | Mutation - genetics | Cell Division - drug effects | Up-Regulation - drug effects | Phenotype | Mitosis - drug effects | Indoleacetic Acids - pharmacology | Plant Roots - embryology | Fluorescent Antibody Technique | Proteomics | Plant Roots - anatomy & histology | Mitogen-Activated Protein Kinases - metabolism | Arabidopsis thaliana | Cell division | Embryonic development | Mitogens | Protein kinases | Analysis
Journal Article
Biomedicine & pharmacotherapy, ISSN 0753-3322, 2010, Volume 64, Issue 10, pp. 733 - 740
... in use of drug-coated stents for arresting VSMC cell proliferation following clinical procedures [1,3–6]. HDAC inhibitors (HDACIs) are a new class of anticancer... 
Internal Medicine | Medical Education | Histone modifications | Vascular smooth muscle cells | Butyrate | MEDICINE, RESEARCH & EXPERIMENTAL | MECHANISM | PROLIFERATION | COLON-CANCER | EPITHELIAL-CELLS | SODIUM-BUTYRATE | DEACETYLASE INHIBITORS | BIOLOGY | GENE-EXPRESSION | CARDIOVASCULAR-DISEASE | PHARMACOLOGY & PHARMACY | HUNTINGTONS-DISEASE | Cell Cycle Proteins - drug effects | Chromatin - metabolism | Cyclin-Dependent Kinases - metabolism | Muscle, Smooth, Vascular - metabolism | Atherosclerosis - genetics | Butyrates - pharmacology | Transcriptional Activation - drug effects | Cyclins - genetics | G1 Phase - drug effects | Cyclin-Dependent Kinase Inhibitor p21 - genetics | Cyclins - metabolism | Protein Processing, Post-Translational - drug effects | Cyclin-Dependent Kinase Inhibitor p15 - genetics | Cell Cycle Proteins - genetics | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Cyclin-Dependent Kinases - antagonists & inhibitors | Phosphorylation - drug effects | Cyclin-Dependent Kinases - genetics | Muscle, Smooth, Vascular - drug effects | Retinoblastoma Protein - metabolism | Cell Cycle Proteins - metabolism | Cells, Cultured | Rats | Down-Regulation - drug effects | Gene Expression Regulation - drug effects | Up-Regulation - drug effects | Acetylation - drug effects | Animals | Histones - genetics | Mitosis - drug effects | Methylation - drug effects | Retinoblastoma Protein - antagonists & inhibitors | Histone Deacetylase Inhibitors - pharmacology | Cell Proliferation - drug effects | Cyclin-Dependent Kinase Inhibitor p15 - metabolism | Histones - metabolism | Cell Cycle - drug effects | Esters | Chromatin | Gene expression | Analysis | Cell cycle
Journal Article