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Cell reports (Cambridge), ISSN 2211-1247, 2016, Volume 16, Issue 10, pp. 2576 - 2592
The mechanisms underlying Zika virus (ZIKV)-related microcephaly and other neurodevelopment defects remain poorly understood. Here, we describe the derivation... 
NEURAL PROGENITORS | LONG-TERM | HUMAN BRAIN | ADAPTER | CENTRAL-NERVOUS-SYSTEM | INFECTION | MICE | BINDING KINASE 1 | ORGANOIDS | INNATE IMMUNITY | CELL BIOLOGY | Neocortex - pathology | Neurons - pathology | Transcription, Genetic - drug effects | Brain - embryology | Neuroglia - ultrastructure | Neuroglia - pathology | Humans | Brain - virology | Centrosome - drug effects | Gene Expression Profiling | Microcephaly - virology | Neural Stem Cells - ultrastructure | Zika Virus Infection - virology | Neural Stem Cells - immunology | Neuroepithelial Cells - immunology | Neuroprotective Agents - pharmacology | Spinal Cord - pathology | Microcephaly - pathology | Neuroepithelial Cells - virology | Nucleosides - pharmacology | Fetus - virology | Cell Death - drug effects | Phosphorylation - drug effects | Neurons - drug effects | Protein-Serine-Threonine Kinases - metabolism | Zika Virus - pathogenicity | Proto-Oncogene Proteins - metabolism | Zika Virus - ultrastructure | Neurons - virology | Virus Replication - drug effects | Neuroepithelial Cells - ultrastructure | Immunity, Innate - drug effects | Zika Virus Infection - pathology | Neural Stem Cells - virology | Zika Virus - physiology | Zika Virus - drug effects | Mitochondria - metabolism | Mitochondria - drug effects | Receptor Protein-Tyrosine Kinases - metabolism | Neural Stem Cells - enzymology | Centrosome - metabolism | Mitosis - drug effects | Brain - pathology | Protein Kinase Inhibitors - pharmacology | Neuroglia - virology | Neuroepithelial Cells - drug effects | Neurons/pathology | Zika Virus/pathogenicity | Mitochondria/metabolism | Virus Replication/drug effects | Microcephaly/pathology | Neurons/drug effects | Protein-Serine-Threonine Kinases/metabolism | Neural Stem Cells/immunology | Neuroglia/ultrastructure | Neuroepithelial Cells/drug effects | Neuroepithelial Cells/virology | Life Sciences | Brain/pathology | Zika Virus/drug effects | Brain/embryology | Mitochondria/drug effects | Fetus/virology | Neocortex/pathology | Neuroglia/pathology | Cell Death/drug effects | Mitosis/drug effects | Transcription, Genetic/drug effects | Nucleosides/pharmacology | Neural Stem Cells/enzymology | Neural Stem Cells/ultrastructure | Neuroepithelial Cells/ultrastructure | Receptor Protein-Tyrosine Kinases/metabolism | Microcephaly/virology | Proto-Oncogene Proteins/metabolism | Neuroprotective Agents/pharmacology | Zika Virus/ultrastructure | Neuroepithelial Cells/immunology | Brain/virology | Immunity, Innate/drug effects | Spinal Cord/pathology | Zika Virus/physiology | Neuroglia/virology | Microbiology and Parasitology | Zika Virus Infection/pathology | Neurons/virology | Zika Virus Infection/virology | Neural Stem Cells/virology | Centrosome/drug effects | Protein Kinase Inhibitors/pharmacology | Centrosome/metabolism | Phosphorylation/drug effects
Journal Article
Plant physiology (Bethesda), ISSN 1532-2548, 2009, Volume 151, Issue 4, pp. 2162 - 2173
Replication protein A (RPA), a highly conserved single-stranded DNA-binding protein in eukaryotes, is a stable complex comprising three subunits termed RPA1,... 
Somatic cells | DNA | Irradiation | Systems Biology, Molecular Biology, and Gene Regulation | Meiosis | Megasporocytes | Plants | Embryo sac | DNA repair | Chromosomes | Rice | PATHWAYS | CELLS | STRAND BREAK REPAIR | GENE | HIGHER-PLANTS | MEIOSIS | ARABIDOPSIS | GENOME | DAMAGE | NUCLEOTIDE EXCISION-REPAIR | PLANT SCIENCES | RNA Interference - drug effects | Germ Cells, Plant - radiation effects | DNA Replication - drug effects | DNA Repair - radiation effects | Genetic Complementation Test | DNA Fragmentation - radiation effects | Meiosis - drug effects | Chromosomes, Plant - radiation effects | Ultraviolet Rays | Oryza - genetics | DNA Replication - radiation effects | Plants, Genetically Modified | Plant Proteins - metabolism | DNA Fragmentation - drug effects | Oryza - embryology | Pollen - cytology | Pollen - drug effects | Germ Cells, Plant - drug effects | Chromosomes, Plant - drug effects | DNA Repair - drug effects | Mitosis - radiation effects | Mutagens - pharmacology | Replication Protein A - metabolism | Oryza - drug effects | RNA Interference - radiation effects | Chromosomes, Plant - metabolism | Mutation - genetics | Germ Cells, Plant - growth & development | Pollen - radiation effects | Mitomycin - pharmacology | Phenotype | DNA, Bacterial - genetics | Meiosis - radiation effects | Mitosis - drug effects | Genes, Plant - genetics | Recombination, Genetic - radiation effects | Methyl Methanesulfonate - pharmacology | Oryza - cytology | Recombination, Genetic - drug effects | Arabidopsis thaliana | DNA replication | Physiological aspects | Environmental aspects | Genetic aspects | DNA binding proteins | Properties
Journal Article
PloS one, ISSN 1932-6203, 04/2013, Volume 8, Issue 4, p. e62082
.... However, poor cytotoxic selectivity, serious side-effects, and limited effectiveness are still the major concerns in their therapeutic application... 
MITOSIS | ANTIMITOTIC ACTIVITY | MICROTUBULES | NATURAL-PRODUCTS | MULTIDISCIPLINARY SCIENCES | DRUG DISCOVERY | RESISTANCE | TUBULIN | BINDING AGENTS | EXPRESSION | KINASES | Lung Neoplasms - drug therapy | Podophyllotoxin - pharmacology | Apoptosis - drug effects | Humans | Lung Neoplasms - metabolism | Apoptosis - genetics | Endoplasmic Reticulum Stress - genetics | Microtubules - metabolism | Podophyllotoxin - toxicity | Drug Evaluation, Preclinical | Disease Models, Animal | DNA Damage - drug effects | Lung Neoplasms - genetics | M Phase Cell Cycle Checkpoints - drug effects | Endoplasmic Reticulum Stress - drug effects | Antineoplastic Agents, Phytogenic - toxicity | Xenograft Model Antitumor Assays | Animals | Mitosis - drug effects | Signal Transduction - drug effects | Tumor Burden - drug effects | Cell Cycle Checkpoints - drug effects | Models, Biological | Cell Line, Tumor | Cell Proliferation - drug effects | Mice | Antineoplastic Agents, Phytogenic - pharmacology | Protein Multimerization - drug effects | Podophyllotoxin - analogs & derivatives | Chemotherapy | Podophyllotoxin | Analysis | Lung cancer | Polymerization | Stress (Physiology) | Tubulins | Health aspects | Apoptosis | Cancer | Drugs | Flow cytometry | Toxicity | Mitosis | Leukemia | DNA damage | Cytotoxicity | Selectivity | Biochemistry | Drug development | Kinases | Cancer therapies | Anticancer properties | Metastases | Proteins | Signal transduction | Tubulin | Paclitaxel | Xenografts | Cell cycle | Inhibition | Deoxyribonucleic acid--DNA | Stresses | Plants (botany) | Hematology | Injection | Survivin | Tumor cell lines | Gene expression | Stress | Aurora B protein | Signaling | Side effects | Colonization | Deoxyribonucleic acid | Stains | DNA
Journal Article
Nature genetics, ISSN 1546-1718, 2001, Volume 27, Issue 1, pp. 48 - 54
Journal Article
Cell Cycle, ISSN 1551-4005, 2014, Volume 7, Issue 18, pp. 2922 - 2928
Mouse embryonic stem cells (mESC) are characterized by high proliferation activity. mESC are highly sensitive to genotoxic stresses and do not undergo G1/S... 
Binding | Proteins | Landes | Calcium | Bioscience | Biology | Cell | Cycle | Cancer | Organogenesis | H2AX | Embryonic stem cells | DNA damage response | DNA breaks | Comet assay | Cell cycle | SOMATIC-CELLS | APOPTOSIS | DEFENSE | cell cycle | embryonic stem cells | PROLIFERATION | SEGREGATION | CELL BIOLOGY | IMMORTAL STRAND HYPOTHESIS | STRESS RESPONSES | comet assay | ATM | DIFFERENTIATION | CYCLE | Embryonic Stem Cells - metabolism | Gamma Rays | Embryonic Stem Cells - cytology | Nocodazole - pharmacology | DNA Repair - radiation effects | Phosphoproteins - metabolism | G2 Phase - drug effects | DNA-Binding Proteins - metabolism | DNA Breaks, Single-Stranded - drug effects | Flow Cytometry | Embryonic Stem Cells - radiation effects | Signal Transduction - radiation effects | Protein-Serine-Threonine Kinases - metabolism | Cell Differentiation - radiation effects | DNA Repair - drug effects | Tumor Suppressor Proteins - metabolism | Mitosis - radiation effects | G2 Phase - radiation effects | DNA Breaks, Single-Stranded - radiation effects | Cell Cycle Proteins - metabolism | Tumor Suppressor Protein p53 - metabolism | Ataxia Telangiectasia Mutated Proteins | Animals | Mitosis - drug effects | Embryonic Stem Cells - drug effects | Signal Transduction - drug effects | Cell Differentiation - drug effects | Mice | DNA Damage | Histones - metabolism | Kinetics
Journal Article
by Yu, Z and Chen, T and Hebert, J and Li, E and Richard, S
Molecular and cellular biology, ISSN 0270-7306, 2009, Volume 29, Issue 11, pp. 2982 - 2996
Journal Article
Developmental cell, ISSN 1534-5807, 2010, Volume 19, Issue 4, pp. 612 - 624
Journal Article
The EMBO Journal, ISSN 1460-2075, 2006, Volume 25, Issue 24, pp. 5775 - 5782
The phosphatidyl inositol 3‐kinase‐like kinases (PIKKs), ataxia‐telangiectasia mutated (ATM) and ATM‐ and Rad3‐related (ATR) regulate parallel damage response... 
ataxia telangiectasia‐mutated protein | PIKKs | phosphorylation | DNA damage responses | Phosphorylation | Ataxia telangiectasia-mutated protein | RECRUITMENT | BIOCHEMISTRY & MOLECULAR BIOLOGY | ataxia telangiectasia-mutated protein | DNA-DAMAGE | COMPLEXES | PROTEIN-KINASES | CELL BIOLOGY | CHK1 | SECKEL-SYNDROME | CHECKPOINT | IONIZING-RADIATION | ATAXIA-TELANGIECTASIA | BINDING | Protein Kinases - metabolism | Hydroxyurea - pharmacology | Protein-Serine-Threonine Kinases - deficiency | DNA Replication - drug effects | Humans | G2 Phase - drug effects | DNA-Binding Proteins - metabolism | Ultraviolet Rays | DNA Replication - radiation effects | Phosphorylation - drug effects | Protein-Serine-Threonine Kinases - metabolism | Protein Structure, Tertiary | Tumor Suppressor Proteins - metabolism | Mitosis - radiation effects | G2 Phase - radiation effects | Cell Cycle Proteins - metabolism | Nuclear Proteins - metabolism | Ataxia Telangiectasia Mutated Proteins | Enzyme Activation - drug effects | Fibroblasts - pathology | Phosphoserine - metabolism | Enzyme Activation - radiation effects | Histones - deficiency | Phosphorylation - radiation effects | Animals | Mitosis - drug effects | Fibroblasts - radiation effects | Models, Biological | Fibroblasts - drug effects | Phosphoproteins - deficiency | Checkpoint Kinase 2 | Checkpoint Kinase 1 | Fibroblasts - cytology | Mice | Ions | Ultraviolet radiation | Molecular biology | Kinases | DNA damage
Journal Article
Journal Article
The Plant cell, ISSN 1040-4651, 9/2007, Volume 19, Issue 9, pp. 2763 - 2775
Controlling microtubule dynamics and spatial organization is a fundamental requirement of eukaryotic cell function. Members of the ORBIT/MAST/CLASP family of... 
Proteins | Dendritic cells | Microtubules | Epidermal cells | Plant roots | Fluorescence | Plants | Microtubule associated proteins | Animal cells | Plant cells | PLUS-END | BAND ORGANIZATION | ROOT | BIOCHEMISTRY & MOLECULAR BIOLOGY | PLANT-CELLS | MICROFILAMENT CROSS-TALK | PLANT SCIENCES | CELL BIOLOGY | CORTICAL MICROTUBULES | CELLULOSE MICROFIBRIL ALIGNMENT | MITOTIC SPINDLE | DYNAMICS | HELICAL GROWTH | Microtubule-Associated Proteins - chemistry | Microtubule-Associated Proteins - genetics | Dinitrobenzenes - pharmacology | Phylogeny | Plant Epidermis - ultrastructure | Recombinant Fusion Proteins - metabolism | Plant Roots - drug effects | Microtubules - metabolism | Microtubules - drug effects | Plant Epidermis - cytology | Plant Leaves - drug effects | Plant Roots - growth & development | Cytokinesis - drug effects | Protein Structure, Tertiary | Green Fluorescent Proteins - metabolism | Arabidopsis Proteins - genetics | Arabidopsis - drug effects | Arabidopsis - cytology | Cell Size - drug effects | Plant Roots - cytology | Plant Epidermis - drug effects | Mutation - genetics | Cell Division - drug effects | Plant Leaves - cytology | Arabidopsis - metabolism | Arabidopsis - genetics | Gene Expression Regulation, Plant - drug effects | Mitosis - drug effects | Arabidopsis Proteins - chemistry | Cell Proliferation - drug effects | Interphase - drug effects | Plant Leaves - ultrastructure | Sulfanilamides - pharmacology | Arabidopsis thaliana | Growth | Mitosis | Kinetochores | Genetic aspects | Research | Properties | Observations | Gene expression
Journal Article
Cancer Biology & Therapy, ISSN 1555-8576, 2014, Volume 11, Issue 5, pp. 464 - 473
....1 Current therapeutic strategies include surgery, radiation therapy and chemotherapy, but these are associated with significant side effects and only limited... 
Binding | Proteins | Landes | Calcium | Bioscience | Biology | Cell | Cycle | Cancer | Organogenesis | Hedgehog | Medulloblastoma | Nanocurcumin | Glioblastoma | IGF | Curcumin | NanoCurc | NEURAL PROGENITORS | SIGNALING PATHWAYS | ACTIVATION | nanocurcumin | medulloblastoma | PANCREATIC-CANCER | nanoCurc (TM) | MEDULLOBLASTOMA CELLS | INSULIN | glioblastoma | hedgehog | ONCOLOGY | FACTOR-I RECEPTOR | NF-KAPPA-B | EXPRESSION | GLIOBLASTOMA CELLS | curcumin | Apoptosis - drug effects | Neoplastic Stem Cells - drug effects | Curcumin - chemistry | Humans | Nanocapsules | Receptors, Notch - genetics | Glycoproteins - drug effects | Antineoplastic Agents - administration & dosage | Curcumin - administration & dosage | Hedgehog Proteins - genetics | Medulloblastoma - pathology | Glioblastoma - metabolism | Antineoplastic Agents - pharmacology | Polymers | Neoplastic Stem Cells - physiology | Tumor Stem Cell Assay | Curcumin - pharmacology | Hedgehog Proteins - analysis | STAT3 Transcription Factor - analysis | Medulloblastoma - metabolism | Antigens, CD - drug effects | Antineoplastic Agents - chemistry | Down-Regulation - drug effects | AC133 Antigen | Peptides - drug effects | Receptors, Notch - analysis | Mitosis - drug effects | Signal Transduction - drug effects | Glioblastoma - pathology | Somatomedins - genetics | Cell Line, Tumor | Cell Proliferation - drug effects | Glioblastoma - drug therapy | Medulloblastoma - drug therapy | nanoCurc | Research Paper
Journal Article
Journal of experimental botany, ISSN 1460-2431, 2012, Volume 63, Issue 14, pp. 5323 - 5335
Journal Article