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Nature immunology, ISSN 1529-2916, 2011, Volume 12, Issue 6, pp. 509 - 517
... have been made empirically, with little or no immunological insight. Recent advances in innate immunity have offered new insights about the mechanisms of vaccine-induced immunity and have facilitated a more rational approach to vaccine design... 
CXC CHEMOKINE RECEPTOR-5 | NATURAL-KILLER-CELLS | ANTIGEN-SPECIFIC MEMORY | DENDRITIC CELL SUBSETS | YELLOW-FEVER VACCINE | VARICELLA-ZOSTER-VIRUS | IN-VIVO | TOLL-LIKE RECEPTORS | INNATE IMMUNE-SYSTEM | IMMUNOLOGY | CD8 T-CELLS | Toll-like receptors | Research | HIV infection | Health aspects | Vaccination | Risk factors
Journal Article
Immunity (Cambridge, Mass.), ISSN 1074-7613, 2012, Volume 36, Issue 6, pp. 907 - 919
Celiac disease (CD) is a chronic enteropathy induced by dietary gluten in genetically predisposed people. The keystone of CD pathogenesis is an adaptive immune... 
NATURAL-KILLER | MULTIPLE COMMON | INTESTINAL INTRAEPITHELIAL LYMPHOCYTES | TRANSFERRIN RECEPTOR | DENDRITIC CELLS | REGULATORY T-CELLS | DERMATITIS-HERPETIFORMIS | DEAMIDATED GLIADIN | IMMUNOLOGY | REFRACTORY SPRUE | TISSUE TRANSGLUTAMINASE | T-Lymphocyte Subsets - immunology | Genes, MHC Class II | Lymphoma, T-Cell - immunology | Humans | Autoantibodies - biosynthesis | Glutens - chemistry | HLA-D Antigens - immunology | Virus Diseases - complications | Interleukin-15 - immunology | Transglutaminases - immunology | Lymphocyte Activation - genetics | Transglutaminases - analysis | Food Hypersensitivity - immunology | Celiac Disease - immunology | Intestinal Mucosa - immunology | Lymphoma, T-Cell - etiology | Disease Models, Animal | Adaptive Immunity | Autoimmune Diseases - epidemiology | Genetic Predisposition to Disease | Virus Diseases - immunology | Celiac Disease - genetics | Comorbidity | Autoimmune Diseases - immunology | Disease Progression | Celiac Disease - diagnosis | Food Hypersensitivity - complications | Celiac Disease - pathology | Autoantibodies - immunology | Models, Immunological | Animals | Immunoglobulin A - immunology | Age of Onset | Transglutaminases - physiology | Glutens - adverse effects | Celiac Disease - epidemiology | Mice | GTP-Binding Proteins | Celiac Disease - diet therapy | Intestinal Mucosa - pathology | Proteins | Autoimmunity | Celiac disease | Histocompatibility antigens | Peptides | Analysis | HLA histocompatibility antigens | Gluten | Disease | Pathogenesis | Histology | Lymphomas | Autoimmune diseases | Viral infections | Immune system
Journal Article
Cell (Cambridge), ISSN 0092-8674, 2016, Volume 167, Issue 5, pp. 1354 - 1368.e14
Journal Article
Science translational medicine, ISSN 1946-6242, 2015, Volume 7, Issue 277, pp. 277ra30 - 277ra30
Journal Article
The Journal of pathology, ISSN 1096-9896, 2019, Volume 247, Issue 5, pp. 606 - 614
...‐autonomous, immunological aspects of radiation‐induced cell death have been seen as increasingly important, alongside a greater understanding of the profound effects... 
macrophages | radiation‐induced DNA damage response | abscopal response | interferon response | stromal fibroblasts | CELLS | SINGLE | TUMOR | PATHOLOGY | BREAST-CANCER | THERAPY | ONCOLOGY | ATR INHIBITOR AZD6738 | RESISTANCE | RADIOTHERAPY | EXPRESSION | radiation-induced DNA damage response | Antigens, Neoplasm - genetics | Chromosomal Instability - immunology | Antigens, Neoplasm - immunology | Humans | Cancer-Associated Fibroblasts - immunology | Combined Modality Therapy | DNA Damage - immunology | Radiation Dosage | CD8-Positive T-Lymphocytes - radiation effects | Chromosomal Instability - genetics | Interferon Type I - biosynthesis | Signal Transduction - immunology | Animals | Immune Tolerance - immunology | Myeloid Cells - immunology | Neoplasms - immunology | Myeloid Cells - radiation effects | Interferon Type I - radiation effects | Neoplasms - radiotherapy | Mice | CD8-Positive T-Lymphocytes - immunology | DNA Damage - radiation effects | Disease Models, Animal | Immunologic Factors - therapeutic use | Interferon | Biological response modifiers | Cell death | DNA damage | DNA | Radiation | Animal models | Toxicity | Guanosine | Cytotoxicity | Macrophages | Cytosol | Genomic instability | Stimulators | Heterogeneity | Signal transduction | Immunology | Historical account | Cyclic GMP | Fibroblasts | Deoxyribonucleic acid--DNA | Immune system | Phenotypes | Immune response | Stability | Adenosine monophosphate | AMP | Mortality | Inflammation | Radiation therapy | Nucleic acids | Signaling | Immunosuppression | Radiation damage | Tumors | Apoptosis
Journal Article
PLoS pathogens, ISSN 1553-7374, 2017, Volume 13, Issue 12, p. e1006665
.... Here, we discuss the immunological mechanisms that control nasopharyngeal colonization, with an emphasis on findings from human studies. Increased understanding of these immunological mechanisms is required to identify correlates of protection against colonization that will facilitate the early testing and design of novel vaccines. 
COMMUNITY-ACQUIRED PNEUMONIA | CONJUGATE VACCINE | MICROBIOLOGY | HAEMOPHILUS-INFLUENZAE | NASOPHARYNGEAL CARRIAGE | STREPTOCOCCUS-PNEUMONIAE | VIROLOGY | BACTERIAL-COLONIZATION | EPIDEMIOLOGIC EVIDENCE | INFLUENZA-VIRUS | ANTIBODY-RESPONSES | PARASITOLOGY | TH17 CELLS | Pneumococcal Vaccines - immunology | Carrier State - immunology | Vaccines, Conjugate - immunology | Pneumococcal Infections - immunology | Nasopharynx - microbiology | Humans | Control | Disease transmission | Research | Pneumococcal vaccine | Microbial colonies | Health aspects | Pneumonia | Immunoglobulins | Disease | Communities | Mortality | Neutrophils | Nasopharynx | Vaccines | Whooping cough | Streptococcus infections | Studies | Immunology | Influenza | Meningitis | Adults | Colonization
Journal Article