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1. Full Text Mechanistic Studies of an Amine Oxidase Derived from d‑Amino Acid Oxidase
by Trimmer, Elizabeth E and Wanninayake, Udayanga S and Fitzpatrick, Paul F
Biochemistry (Easton), ISSN 0006-2960, 04/2017, Volume 56, Issue 14, pp. 2024 - 2030
Life Sciences & Biomedicine | Biochemistry & Molecular Biology | Science & Technology | Index Medicus
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2. Full Text Monoamine oxidase A mediates prostate tumorigenesis and cancer metastasis
by Wu, Jason Boyang and Shao, Chen and Li, Xiangyan and Li, Qinlong and Hu, Peizhen and Shi, Changhong and Li, Yang and Chen, Yi-Ting and Yin, Fei and Liao, Chun-Peng and Stiles, Bangyan L and Zhau, Haiyen E and Shih, Jean C and Chung, Leland W.K
The Journal of clinical investigation, ISSN 0021-9738, 07/2014, Volume 124, Issue 7, pp. 2891 - 2908
Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Up-Regulation | Humans | Prostatic Neoplasms - etiology | Male | Vascular Endothelial Growth Factor A - metabolism | Monoamine Oxidase - genetics | Vascular Endothelial Growth Factor A - genetics | Carcinogenesis | Gene Knockdown Techniques | Neoplasm Metastasis | Heterografts | Forkhead Transcription Factors - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Epithelial-Mesenchymal Transition | Nuclear Proteins - genetics | Promoter Regions, Genetic | Prostatic Neoplasms - pathology | Signal Transduction | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Mice, Inbred C57BL | Mice, SCID | Disease Progression | Animals | Mice, Nude | Models, Biological | Twist-Related Protein 1 - genetics | Cell Line, Tumor | Prostatic Neoplasms - enzymology | Forkhead Box Protein O1 | Mice | Monoamine Oxidase - metabolism | Development and progression | Genetic aspects | Monoamine oxidase | Metastasis | Properties | Prostate cancer | Medical research | Antigens | Oxidative stress | Enzymes | Gene expression | Proteins | Studies | Genotype & phenotype | Hypoxia | Tumorigenesis | Behavior | Tumors | Index Medicus | Abridged Index Medicus
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3. Full Text Monoamine oxidase A gene DNA hypomethylation – a risk factor for panic disorder?
by Domschke, Katharina and Tidow, Nicola and Kuithan, Henriette and Schwarte, Kathrin and Klauke, Benedikt and Ambrée, Oliver and Reif, Andreas and Schmidt, Hartmut and Arolt, Volker and Kersting, Anette and Zwanzger, Peter and Deckert, Jürgen
The international journal of neuropsychopharmacology, ISSN 1461-1457, 10/2012, Volume 15, Issue 9, pp. 1217 - 1228
Anxiety | gender | MAOA | methylation | epigenetics | Neurosciences | Clinical Neurology | Life Sciences & Biomedicine | Pharmacology & Pharmacy | Neurosciences & Neurology | Psychiatry | Science & Technology | Sulfites | Diagnostic and Statistical Manual of Mental Disorders | Epigenesis, Genetic | Humans | Life Change Events | Risk Factors | Molecular Sequence Data | Exons - genetics | Male | Smoking - genetics | DNA Methylation - genetics | Monoamine Oxidase - genetics | Sex Characteristics | Promoter Regions, Genetic - genetics | Pilot Projects | Mutagens | Base Sequence | Panic Disorder - epidemiology | Polymerase Chain Reaction | Minisatellite Repeats - genetics | Adult | Female | Panic Disorder - psychology | Panic Disorder - genetics | Index Medicus
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4. Full Text Monoamine oxidase inactivation: From pathophysiology to therapeutics
by Bortolato, Marco and Chen, Kevin and Shih, Jean C
Advanced drug delivery reviews, ISSN 0169-409X, 2008, Volume 60, Issue 13, pp. 1527 - 1533
Oxidative stress | Depression | Monoamine oxidase | Parkinson's disease | Life Sciences & Biomedicine | Pharmacology & Pharmacy | Science & Technology | Monoamine Oxidase Inhibitors - therapeutic use | Monoamine Oxidase Inhibitors - pharmacology | Tourette Syndrome - drug therapy | Isoenzymes - genetics | Humans | Alzheimer Disease - drug therapy | Tourette Syndrome - metabolism | Mitochondria - metabolism | Parkinson Disease - drug therapy | Monoamine Oxidase - genetics | Attention Deficit Disorder with Hyperactivity - drug therapy | Mice, Knockout | Attention Deficit Disorder with Hyperactivity - metabolism | Animals | Mitochondria - genetics | Isoenzymes - metabolism | Alzheimer Disease - metabolism | Anxiety Disorders - metabolism | Mice | Parkinson Disease - metabolism | Anxiety Disorders - drug therapy | Isoenzymes - antagonists & inhibitors | Monoamine Oxidase - metabolism | Index Medicus | depression | Parkinson’s disease | oxidative stress
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5. Full Text Monoamine Oxidase A–Mediated Enhanced Catabolism of Norepinephrine Contributes to Adverse Remodeling and Pump Failure in Hearts With Pressure Overload
by Kaludercic, Nina and Takimoto, Eiki and Nagayama, Takahiro and Feng, Ning and Lai, Edwin W and Bedja, Djahida and Chen, Kevin and Gabrielson, Kathleen L and Blakely, Randy D and Shih, Jean C and Pacak, Karel and Kass, David A and Di Lisa, Fabio and Paolocci, Nazareno
Circulation research, ISSN 0009-7330, 01/2010, Volume 106, Issue 1, pp. 193 - 202
Oxidative stress | Catecholamines | Monoamine oxidase A | Norepinephrine transporter | Congestive heart failure | Cardiac & Cardiovascular Systems | Peripheral Vascular Disease | Life Sciences & Biomedicine | Hematology | Cardiovascular System & Cardiology | Science & Technology | Fundamental and applied biological sciences. Psychology | Cardiology. Vascular system | Heart | Biological and medical sciences | Medical sciences | Heart failure, cardiogenic pulmonary edema, cardiac enlargement | Vertebrates: cardiovascular system | Blood Pressure | Monoamine Oxidase Inhibitors - pharmacology | Clorgyline - pharmacology | Heart Failure - enzymology | Oxidative Stress - genetics | Heart Failure - genetics | Mice, Transgenic | Cardiomegaly - enzymology | Monoamine Oxidase - genetics | Hydrogen Peroxide - metabolism | Ventricular Remodeling | Animals | Norepinephrine - metabolism | Ventricular Dysfunction, Left - genetics | Heart Ventricles - enzymology | Ventricular Dysfunction, Left - enzymology | Mice | Oxidative Stress - drug effects | Cardiomegaly - genetics | Monoamine Oxidase - metabolism | Index Medicus | congestive heart failure | catecholamines | serotonin | MAO-A | NET | oxidative stress
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6. Full Text A functional polymorphism in the monoamine oxidase A gene promoter
by Sabol, S. Z and Hu, Stella and Hamer, D
Human genetics, ISSN 0340-6717, 9/1998, Volume 103, Issue 3, pp. 273 - 279
Human Genetics | Gene Function | Molecular Medicine | Biomedicine | Metabolic Diseases | Life Sciences & Biomedicine | Genetics & Heredity | Science & Technology | Fundamental and applied biological sciences. Psychology | Classical genetics, quantitative genetics, hybrids | Human | Biological and medical sciences | Genetics of eukaryotes. Biological and molecular evolution | Humans | Middle Aged | Male | Monoamine Oxidase - genetics | Promoter Regions, Genetic - genetics | Haplotypes - genetics | Genetic Markers - genetics | Alleles | Minisatellite Repeats - genetics | Adult | Female | Polymorphism, Genetic - genetics | Genetic Linkage - genetics | Index Medicus
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7. Full Text Regulation of Monoamine Oxidase A by Circadian-Clock Components Implies Clock Influence on Mood
by Hampp, Gabriele and Ripperger, Jürgen A and Houben, Thijs and Schmutz, Isabelle and Blex, Christian and Perreau-Lenz, Stéphanie and Brunk, Irene and Spanagel, Rainer and Ahnert-Hilger, Gudrun and Meijer, Johanna H and Albrecht, Urs
Current biology, ISSN 0960-9822, 2008, Volume 18, Issue 9, pp. 678 - 683
MOLNEURO | Biochemistry & Molecular Biology | Biology | Life Sciences & Biomedicine - Other Topics | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Monoamine Oxidase Inhibitors - pharmacology | Promoter Regions, Genetic | Affect - physiology | Humans | Cell Cycle Proteins - metabolism | Circadian Rhythm Signaling Peptides and Proteins - genetics | Gene Expression Regulation | Rats | Nuclear Proteins - metabolism | Circadian Rhythm - physiology | Monoamine Oxidase - genetics | Transcription Factors - genetics | Period Circadian Proteins | Transcription Factors - metabolism | Animals | Circadian Rhythm Signaling Peptides and Proteins - metabolism | Biological Clocks - physiology | Cell Cycle Proteins - genetics | Mice | Nuclear Proteins - genetics | Dopamine - metabolism | Basal Ganglia - drug effects | Monoamine Oxidase - metabolism | Enzymes | Monoamine oxidase | Laws, regulations and rules | Analysis | Depression, Mental | Physiological aspects | Behavior modification | Genetic transcription | Cells | Index Medicus
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8. Full Text Sexual dimorphic effect in the genetic association of monoamine oxidase A (MAOA) markers with autism spectrum disorder
by Verma, Deepak and Chakraborti, Barnali and Karmakar, Arijit and Bandyopadhyay, Tirthankar and Singh, Asem Surindro and Sinha, Swagata and Chatterjee, Anindita and Ghosh, Saurabh and Mohanakumar, Kochupurackal P and Mukhopadhyay, Kanchan and Rajamma, Usha
Progress in neuro-psychopharmacology & biological psychiatry, ISSN 0278-5846, 04/2014, Volume 50, pp. 11 - 20
Allele | Monoamine oxidase A | Sexual dimorphism | Genetic association | Haplotype | Autism spectrum disorder | Neurosciences | Clinical Neurology | Life Sciences & Biomedicine | Pharmacology & Pharmacy | Neurosciences & Neurology | Psychiatry | Science & Technology | Psychology. Psychoanalysis. Psychiatry | Infantile autism | Pharmacology. Drug treatments | Psychopathology. Psychiatry | Developmental disorders | Biological and medical sciences | Medical sciences | Neuropharmacology | Child clinical studies | Genetic Predisposition to Disease - genetics | Genetic Association Studies | Humans | Child, Preschool | Linkage Disequilibrium - genetics | Male | Monoamine Oxidase - genetics | Sex Characteristics | Case-Control Studies | Young Adult | Haplotypes - genetics | Child Development Disorders, Pervasive - genetics | Genetic Markers - genetics | Adolescent | Alleles | Polymorphism, Single Nucleotide - genetics | Adult | Female | Child | Genetic research | Autism | Sex discrimination | Monoamine oxidase | Genetic transcription | Analysis | Index Medicus
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9. Full Text Monoamine oxidase B is elevated in Alzheimer disease neurons, is associated with γ-secretase and regulates neuronal amyloid β-peptide levels
by Schedin-Weiss, Sophia and Inoue, Mitsuhiro and Hromadkova, Lenka and Teranishi, Yasuhiro and Yamamoto, Natsuko Goto and Wiehager, Birgitta and Bogdanovic, Nenad and Winblad, Bengt and Sandebring-Matton, Anna and Frykman, Susanne and Tjernberg, Lars O
Alzheimer's research & therapy, ISSN 1758-9193, 08/2017, Volume 9, Issue 1, pp. 57 - 57
Monoamine oxidase B | Intraneuronal amyloid β-peptide | Alzheimer disease | γ-Secretase | Alzheimer disease pathogenesis | Amyloid β-peptide | Neurosciences | Clinical Neurology | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | RNA, Small Interfering - genetics | Humans | Middle Aged | Receptors, N-Methyl-D-Aspartate - metabolism | Male | Monoamine Oxidase - genetics | Alzheimer Disease - pathology | Brain - metabolism | Neurons - ultrastructure | Synapses - metabolism | Transfection | Amyloid beta-Peptides - metabolism | Aged, 80 and over | Female | Neurons - metabolism | Dendrites - metabolism | Gene Expression Regulation - genetics | Axons - metabolism | Models, Molecular | Presenilin-1 - genetics | Rats | Amyloid Precursor Protein Secretases - metabolism | Animals | Alzheimer Disease - metabolism | Brain - pathology | Aged | Mice | Cell Line, Transformed | Monoamine Oxidase - metabolism | RNA, Small Interfering - metabolism | Brain | Enzymes | Medical imaging | Peptides | Neurons | Pathogenesis | Kinases | Proteins | Neurotoxicity | Ligands | Scientific imaging | Mass spectrometry | Dementia | Index Medicus | Medicin och hälsovetenskap
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