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1. Full Text Critical roles for CCR2 and MCP-3 in monocyte mobilization from bone marrow and recruitment to inflammatory sites
by Tsou, Chia-Lin and Peters, Wendy and Si, Yue and Slaymaker, Sarah and Aslanian, Ara M and Weisberg, Stuart P and Mack, Matthias and Charo, Israel F
Journal of Clinical Investigation, ISSN 0021-9738, 04/2007, Volume 117, Issue 4, pp. 902 - 909
Monocyte recruitment to sites of inflammation is regulated by members of the chemokine family of chemotactic cytokines. However, the mechanisms that govern the... 
HETEROGENEITY | MEDICINE, RESEARCH & EXPERIMENTAL | CELLS | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | RECOGNITION | DISEASE | RESISTANCE | CHEMOATTRACTANT PROTEIN-1 | MICE | CHEMOKINE RECEPTORS | EXPRESSION | Monocyte Chemoattractant Proteins - physiology | Chemokines - blood | Humans | Chemokine CCL7 | Bone Marrow Cells - physiology | Bone Marrow Transplantation - physiology | Monocytes - immunology | Adoptive Transfer | Blood Cell Count | Mice, Knockout | Polymorphism, Genetic | Receptors, Chemokine - deficiency | Monocyte Chemoattractant Proteins - genetics | Animals | Receptors, Chemokine - physiology | Mice | Monocytes - physiology | Receptors, Chemokine - genetics | Receptors, CCR2 | Inflammation - physiopathology | Physiological aspects | Monocytes | Inflammation | Research
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2. Full Text Monocyte-mediated defense against microbial pathogens
by Serbina, Natalya V and Jia, Ting and Hohl, Tobias M and Pamer, Eric G
Annual Review of Immunology, ISSN 0732-0582, 2008, Volume 26, Issue 1, pp. 421 - 452
Circulating blood monocytes supply peripheral tissues with macrophage and dendritic cell (DC) precursors and, in the setting of infection, also contribute... 
Inflammation | Microbial pathogens | Chemokines | Monocyte differentiation | microbial pathogens | HUMAN MONONUCLEAR PHAGOCYTES | TOXOPLASMA-GONDII INFECTION | CHEMOTACTIC PROTEIN-1 SECRETION | ORAL SALMONELLA INFECTION | IMMUNOLOGY | chemokines | NITRIC-OXIDE-SYNTHASE | INTESTINAL DENDRITIC CELLS | TUMOR-NECROSIS-FACTOR | CELLS IN-VIVO | inflammation | TOLL-LIKE RECEPTORS | CENTRAL-NERVOUS-SYSTEM | monocyte differentiation | Monocyte Chemoattractant Proteins - physiology | Receptors, CCR2 - physiology | Immunity, Mucosal - immunology | Infection - immunology | Monocytes - cytology | Humans | Infection - microbiology | Monocytes - immunology | Infection - physiopathology | Cell Differentiation - immunology | Immunity, Cellular - immunology | Animals | Chemotaxis - immunology
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3. Full Text Monocyte chemoattractant proteins in the pathogenesis of systemic sclerosis
by Distler, J. H. W and Akhmetshina, A and Schett, G and Distler, O
Rheumatology, ISSN 1462-0324, 2/2009, Volume 48, Issue 2, pp. 98 - 103
Activation of the immune system and increased synthesis of extracellular matrix proteins by fibroblasts are hallmarks in the pathogenesis of SSc. The molecular... 
MCP-1 | Systemic sclerosis | Chemokine receptors | Chemokines | RHEUMATOID-ARTHRITIS | INDUCED PULMONARY-FIBROSIS | SERUM-LEVELS | CHEMOKINE RECEPTOR | GENE-EXPRESSION | MONONUCLEAR LEUKOCYTES | GROWTH-FACTOR | RHEUMATOLOGY | TIGHT-SKIN MOUSE | FIBROBLASTS | Chemotaxis, Leukocyte | Monocyte Chemoattractant Proteins - physiology | Extracellular Matrix Proteins - biosynthesis | Skin - metabolism | Humans | Receptors, Chemokine - metabolism | Scleroderma, Systemic - metabolism | Scleroderma, Systemic - immunology | Animals | Scleroderma, Systemic - etiology | Models, Animal | Skin - pathology | Chemokines - immunology | Fibroblasts - metabolism | Skin - immunology
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4. Full Text Absence of CC Chemokine Ligand 2 Does Not Limit Obesity-Associated Infiltration of Macrophages Into Adipose Tissue
by Karen E. Inouye and Hang Shi and Jane K. Howard and Christine H. Daly and Graham M. Lord and Barrett J. Rollins and Jeffrey S. Flier
Diabetes, ISSN 0012-1797, 09/2007, Volume 56, Issue 9, pp. 2242 - 2250
Absence of CC Chemokine Ligand 2 Does Not Limit Obesity-Associated Infiltration of Macrophages Into Adipose Tissue Karen E. Inouye 1 , Hang Shi 1 , Jane K.... 
RECRUITMENT | MCP-1 | LINKED INSULIN-RESISTANCE | CC-CHEMOKINE | INFLAMMATION | ENDOCRINOLOGY & METABOLISM | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | WEIGHT-LOSS | NECROSIS-FACTOR-ALPHA | MICE | EXPRESSION | Immunohistochemistry | Macrophages - physiology | Monocyte Chemoattractant Proteins - physiology | Adipose Tissue - physiology | Mice, Inbred C57BL | Adipose Tissue - pathology | Adipose Tissue - physiopathology | Chemokine CCL7 | Chemokine CCL2 - genetics | Male | Obesity - physiopathology | Insulin - blood | Reverse Transcriptase Polymerase Chain Reaction | Chemokine CCL2 - deficiency | Energy Intake | Obesity - genetics | Mice, Knockout | Animals | Mice | Weight Gain | Adipose tissues | Obesity | Care and treatment | Macrophages | Health aspects | Risk factors
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5. Full Text Monocyte Chemotactic Protein‐3 Is a Myocardial Mesenchymal Stem Cell Homing Factor
by Schenk, Soren and Mal, Niladri and Finan, Amanda and Zhang, Ming and Kiedrowski, Matt and Popovic, Zoran and McCarthy, Patrick M and Penn, Marc S
STEM CELLS, ISSN 1066-5099, 01/2007, Volume 25, Issue 1, pp. 245 - 251
MSCs have received attention for their therapeutic potential in a number of disease states, including bone formation, diabetes, stem cell engraftment after... 
Homing | Chemokine | Bone marrow stromal cells | Chemokine receptors | bone marrow stromal cells | chemokine receptors | chemokine | BONE-MARROW | INFARCTED MYOCARDIUM | IMPROVES CARDIAC-FUNCTION | PROMOTING MIGRATION | DELIVERY | CELL BIOLOGY | ISCHEMIC CARDIOMYOPATHY | TRAUMATIC BRAIN-INJURY | ONCOLOGY | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | GROWTH-FACTORS | homing | NEOVASCULARIZATION | HEMATOLOGY | EXPRESSION | Monocyte Chemoattractant Proteins - physiology | Coronary Vessels - pathology | Echocardiography | Oligonucleotide Array Sequence Analysis | Heart - physiology | Chemokine CCL7 | Rats, Inbred Lew | Chemokines - physiology | Rats | Chemokines - genetics | Collagen - metabolism | Microscopy, Confocal | Animals | Polymerase Chain Reaction | Models, Animal | Receptors, Chemokine - genetics | Mesenchymal Stem Cell Transplantation | Cell Movement
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6. Full Text The Role of CCL12 in the Recruitment of Fibrocytes and Lung Fibrosis
by Moore, Bethany B and Murray, Lynne and Das, Anuk and Wilke, Carol A and Herrygers, Amy B and Toews, Galen B
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 08/2006, Volume 35, Issue 2, pp. 175 - 181
We have previously shown that mice that are genetically deficient in the CCR2 gene (CCR2-/- mice) are protected from fluorescein isothiocyanate (FITC)-induced... 
Fibrocytes | Lung | Chemokines | Fibrosis | CELLS | CC-CHEMOKINE | PERIPHERAL-BLOOD FIBROCYTES | BIOCHEMISTRY & MOLECULAR BIOLOGY | INJURY | FIBROBLAST PROLIFERATION | chemokines | CELL BIOLOGY | lung | fibrocytes | RESPIRATORY SYSTEM | fibrosis | ABSENCE | INFLAMMATORY CYTOKINES | PULMONARY-FIBROSIS | Monocyte Chemoattractant Proteins - physiology | Lung Diseases - chemically induced | Fibrosis - chemically induced | Fibroblasts - physiology | Mice, Inbred C57BL | Chemotaxis | Cell Movement - genetics | Mice, Knockout | Cell Movement - drug effects | Monocyte Chemoattractant Proteins - genetics | Animals | Adoptive Transfer - methods | Fluorescein-5-isothiocyanate | Fibroblasts - cytology | Mice | Mice, Inbred BALB C | Fibrosis - pathology | Kinetics | Lung Diseases - pathology | Fluorescent Dyes
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7. Full Text Recruited monocytic myeloid‐derived suppressor cells promote the arrest of tumor cells in the premetastatic niche through an IL‐1β‐mediated increase in E‐selectin expression
by Shi, Huifang and Zhang, Juechao and Han, Xiaoqing and Li, Huihan and Xie, Mingshu and Sun, Yingying and Liu, Wenguang and Ba, Xueqing and Zeng, Xianlu
International Journal of Cancer, ISSN 0020-7136, 03/2017, Volume 140, Issue 6, pp. 1370 - 1383
The tumor premetastatic niche initiated by primary tumors is constructed by multiple molecular factors and cellular components and provides permissive... 
premetastatic niche | IL‐1β | tumor cell arrest | E‐selectin | mo‐MDSCs | E-selectin | mo-MDSCs | IL-1β | PROGENITOR CELLS | CONTRIBUTE | METASTASIS | ANGIOGENESIS | SUBSETS | IL-1 beta | CANCER | RESPONSES | ONCOLOGY | INFLAMMATION | ENDOTHELIAL-CELLS | TNF-ALPHA | Monocyte Chemoattractant Proteins - physiology | Coculture Techniques | Tumor Microenvironment | Lung Neoplasms - pathology | Gene Knockdown Techniques | Melanoma, Experimental - immunology | Lung Neoplasms - secondary | E-Selectin - genetics | Melanoma, Experimental - secondary | E-Selectin - biosynthesis | Tumor Cells, Cultured | Monocytes - physiology | Neoplasm Proteins - genetics | Interleukin-1beta - physiology | Stem Cell Niche | Neoplasm Proteins - biosynthesis | Mice, Inbred C57BL | Melanoma, Experimental - pathology | Myeloid-Derived Suppressor Cells - physiology | Gene Expression Regulation, Neoplastic - immunology | Organ Specificity | Cell Adhesion | Myeloid-Derived Suppressor Cells - classification | Monocyte Chemoattractant Proteins - genetics | Lung Neoplasms - immunology | Macrophages - metabolism | Animals | Neoplastic Cells, Circulating | Endothelium, Vascular - pathology | Mice | Cell Movement
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8. Full Text Inhibition of glutaminyl cyclase attenuates cell migration modulated by monocyte chemoattractant proteins
by Chen, Yi-Ling and Huang, Kai-Fa and Kuo, Wen-Chih and Lo, Yan-Chung and Lee, Yu-May and Wang, Andrew H.-J
Biochemical Journal, ISSN 0264-6021, 03/2012, Volume 442, Issue 2, pp. 403 - 412
QC (glutaminyl cyclase) catalyses the formation of N-terminal pGlu (pyroglutamate) in peptides and proteins. pGlu formation in chemoattractants may participate... 
Chemokine | Monocyte chemoattractant protein (MCP) | Monocyte | Pyroglutamate (pGlu) | Glutaminyl cyclase (QC) | Lipopolysaccharide (LPS) | lipopolysaccharide (LPS) | chemokine | MCP-1 | monocyte chemoattractant protein (MCP) | CC-CHEMOKINE | pyroglutamate (pGlu) | BIOCHEMISTRY & MOLECULAR BIOLOGY | POSTTRANSLATIONAL MODIFICATIONS | CHEMOTACTIC PROTEIN-2 | IDENTIFICATION | THYROTROPIN-RELEASING HORMONE | PEPTIDES | PYROGLUTAMIC ACID | A-BETA | monocyte | glutaminyl cyclase (QC) | RECEPTORS | Aminoacyltransferases - antagonists & inhibitors | Monocyte Chemoattractant Proteins - physiology | Aminoacyltransferases - chemistry | RNA, Small Interfering - genetics | Humans | Aminoacyltransferases - genetics | Enzyme Inhibitors - pharmacology | Models, Molecular | Cell Movement - physiology | Gene Knockdown Techniques | Monocytes - drug effects | Cell Movement - drug effects | U937 Cells | Base Sequence | Lipopolysaccharides - pharmacology | Monocyte Chemoattractant Proteins - chemistry | Protein Interaction Domains and Motifs | Aminoacyltransferases - physiology | Monocytes - physiology | Inflammation - physiopathology
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9. Full Text Gene profiling in atherosclerosis reveals a key role for small inducible cytokines: validation using a novel monocyte chemoattractant protein monoclonal antibody
by Lutgens, Esther and Faber, Birgit and Schapira, Kitty and Evelo, Chris T. A and van Haaften, Rachel and Heeneman, Sylvia and Cleutjens, Kitty B. J. M and Bijnens, Ann Pascale and Beckers, Linda and Porter, J. Gordon and Mackay, Charles R and Rennert, Paul and Bailly, Veronique and Jarpe, Matthew and Dolinski, Brian and Koteliansky, Victor and de Fougerolles, Tony and Daemen, Mat J. A. P
Circulation, ISSN 0009-7322, 2005, Volume 111, Issue 25, pp. 3443 - 3452
Pathological aspects of atherosclerosis are well described, but gene profiles during atherosclerotic plaque progression are largely unidentified. Microarray... 
Genes | Inflammation | Atherosclerosis | atherosclerosis | ATHEROGENESIS | CARDIAC & CARDIOVASCULAR SYSTEMS | HUMAN ATHEROMA | genes | CC-CHEMOKINE | LESION FORMATION | RECEPTOR-DEFICIENT MICE | CDNA ARRAY ANALYSIS | E-KNOCKOUT MICE | inflammation | DIFFERENTIALLY EXPRESSED GENES | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | MICROARRAY DATA | Monocyte Chemoattractant Proteins - physiology | Apolipoproteins E - deficiency | Chemokine CCL2 - immunology | Peptide Hydrolases - genetics | Atherosclerosis - genetics | Extracellular Matrix - metabolism | RNA, Messenger - analysis | Male | Gene Expression Profiling | Monocyte Chemoattractant Proteins - immunology | Time Factors | Atherosclerosis - pathology | Atherosclerosis - drug therapy | Antibodies, Monoclonal - pharmacology | Chemokine CCL8 | Chemokines - physiology | Chemokines - genetics | Aorta, Thoracic | Disease Progression | Mice, Knockout | Animals | Antibodies, Monoclonal - administration & dosage | Inflammation - genetics | Mice | Cluster Analysis
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10. Full Text A Non-Glycosaminoglycan-Binding Variant of CC Chemokine Ligand 7 (Monocyte Chemoattractant Protein-3) Antagonizes Chemokine-Mediated Inflammation
by Ali, Simi and Robertson, Helen and Wain, Julie H and Isaacs, John D and Malik, Ghada and Kirby, John A
The Journal of Immunology, ISSN 0022-1767, 07/2005, Volume 175, Issue 2, pp. 1257 - 1266
A non-glycosaminoglycan (GAG)-binding variant of the pleiotropic chemokine CCL7 was generated by mutating to alanine the basic (B) amino acids within an... 
SITE | CELLS | MUTAGENESIS | RANTES | KINASE | HEPARAN-SULFATE | CHEMOTACTIC PROTEIN-3 | TNF-ALPHA | IMMUNOLOGY | HETEROLOGOUS DESENSITIZATION | IDENTIFICATION | Monocyte Chemoattractant Proteins - physiology | Heparin - metabolism | Calcium - metabolism | Humans | Synovial Fluid - metabolism | Monocytes - immunology | Cytokines - physiology | Genetic Variation | Arthritis, Rheumatoid - metabolism | Chemotaxis, Leukocyte - genetics | Chemokines - antagonists & inhibitors | Monocytes - pathology | Female | Inflammation Mediators - antagonists & inhibitors | Synovial Fluid - cytology | Cytokines - genetics | Inflammation Mediators - physiology | Endothelium, Vascular - immunology | Cell Line | Cytokines - metabolism | Glycosaminoglycans - metabolism | Chemokine CCL7 | Chemokines - physiology | Binding Sites - genetics | Monocyte Chemoattractant Proteins - metabolism | Arthritis, Rheumatoid - pathology | Synovial Fluid - immunology | Monocyte Chemoattractant Proteins - genetics | Point Mutation | Animals | Endothelium, Vascular - metabolism | Endothelium, Vascular - pathology | Ligands | Mice | Mice, Inbred BALB C | Amino Acid Motifs - genetics | Arthritis, Rheumatoid - immunology
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11. Full Text Neutrophil-Derived Heparin-Binding Protein (HBP/CAP37) Deposited on Endothelium Enhances Monocyte Arrest under Flow Conditions
by Soehnlein, Oliver and Xie, Xun and Ulbrich, Holger and Kenne, Ellinor and Rotzius, Pierre and Flodgaard, Hans and Eriksson, Einar E and Lindbom, Lennart
The Journal of Immunology, ISSN 0022-1767, 05/2005, Volume 174, Issue 10, pp. 6399 - 6405
In acute inflammation, infiltration of neutrophils often precedes a second phase of monocyte invasion, and data in the literature suggest that neutrophils may.... 
CELLS | LOCALIZATION | RECRUITMENT | HUMAN HBP/CAP37/AZUROCIDIN | INFLAMMATORY MEDIATOR | CHEMOATTRACTANT | CAP37 | IMMUNOLOGY | IDENTIFICATION | LEUKOCYTE | INFILTRATION | Monocyte Chemoattractant Proteins - physiology | Calcium - metabolism | Humans | Monocytes - metabolism | Monocytes - immunology | Blood Proteins - metabolism | Cell Migration Inhibition | Cattle | Monocytes - pathology | Calcium Signaling - immunology | Neutrophils - metabolism | Intracellular Fluid - metabolism | Endothelium, Vascular - immunology | Neutrophil Infiltration - immunology | Carrier Proteins - physiology | Cell Line | Adjuvants, Immunologic - physiology | Antimicrobial Cationic Peptides | Monocyte Chemoattractant Proteins - metabolism | Macrophage Activation | Animals | Carrier Proteins - metabolism | Endothelium, Vascular - metabolism | Blood Proteins - physiology | Protein Binding | Cytosol - metabolism | Endothelium, Vascular - pathology
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12. Full Text Human monocyte chemoattractant protein (MCP)-4 is a novel CC chemokine with activities on monocytes, eosinophils, and basophils induced in allergic and nonallergic inflammation that signals through the CC chemokine receptors (CCR)-2 and -3
by Garcia-Zepeda, EA and Combadiere, C and Rothenberg, ME and Sarafi, MN and Lavigne, F and Hamid, Q and Murphy, PM and Luster, AD
The Journal of Immunology, ISSN 0022-1767, 12/1996, Volume 157, Issue 12, pp. 5613 - 5626
The chemokines are a large family of cytokines that regulate the complex and precise recruitment of immune cells into inflammatory foci. To fully appreciate... 
MCP-2 | MESSENGER-RNA | RANTES | FUNCTIONAL EXPRESSION | ENDOTHELIAL-CELLS | GENE-EXPRESSION | CHEMOTACTIC PROTEIN-1 | MOLECULAR-CLONING | CYTOKINE | IMMUNOLOGY | T-CELLS | Chemotaxis, Leukocyte | Monocyte Chemoattractant Proteins - physiology | Gene Expression | Signal Transduction | Humans | DNA, Complementary - genetics | Molecular Sequence Data | Receptors, Cytokine - physiology | Endothelium, Vascular - physiology | Sequence Homology, Amino Acid | Tissue Distribution | Eosinophils - physiology | Sequence Alignment | Calcium - physiology | Base Sequence | Basophils - physiology | Cloning, Molecular | Histamine Release | Sinusitis - physiopathology | Nasal Mucosa - physiology | Epithelium - physiology | Monocytes - physiology
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13. Full Text Role of the monocyte chemoattractant protein and eotaxin subfamily of chemokines in allergic inflammation
by A D Luster and M E Rothenberg
Journal of Leukocyte Biology, ISSN 0741-5400, 11/1997, Volume 62, Issue 5, pp. 620 - 633
Allergic inflammation is characterized by the tissue accumulation and activation of leukocytes rich in eosinophils. During these responses, there is marked... 
gene structure | gene targeting | cytokines | asthma | receptor usage | Receptor usage | Gene targeting | Cytokines | Gene structure | Asthma | CC-CHEMOKINE | POTENT ACTIVATOR | CHEMOTACTIC PROTEIN-3 | IMMUNOLOGY | PULMONARY EOSINOPHILIA | CELL BIOLOGY | CD4+ LYMPHOCYTES-T | MESSENGER-RNA | FUNCTIONAL EXPRESSION | MOLECULAR-CLONING | EOSINOPHIL ACCUMULATION | GROWTH-FACTOR | HEMATOLOGY | Monocyte Chemoattractant Proteins - physiology | Amino Acid Sequence | Inflammation - pathology | Monocyte Chemoattractant Proteins - biosynthesis | Humans | Molecular Sequence Data | Chemokine CCL11 | Chemokines, CC | Cytokines - physiology | Sequence Homology, Amino Acid | Inflammation - metabolism | Animals | Hypersensitivity - metabolism | Hypersensitivity - pathology | Mice | Cytokines - biosynthesis
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14. Full Text CCL7 and CXCL10 Orchestrate Oxidative Stress-Induced Neutrophilic Lung Inflammation
by Michalec, Lidia and Choudhury, Barun K and Postlethwait, Edward and Wild, James S and Alam, Rafeul and Lett-Brown, Michael and Sur, Sanjiv
The Journal of Immunology, ISSN 0022-1767, 01/2002, Volume 168, Issue 2, pp. 846 - 852
Oxidative stress from ozone (03) exposure augments airway neutrophil recruitment and chemokine production. We and others have shown that severe and sudden... 
OBSTRUCTIVE PULMONARY-DISEASE | AIR-POLLUTION | INDUCED SPUTUM | IFN-GAMMA | CHEMOKINES | GENE-EXPRESSION | OZONE-INDUCED INFLAMMATION | ASTHMATIC SUBJECTS | IMMUNOLOGY | CHEMOATTRACTANT CINC | HYPERRESPONSIVENESS | Monocyte Chemoattractant Proteins - physiology | Antibody Specificity | Inflammation - pathology | Chemokines, CXC - immunology | Dose-Response Relationship, Immunologic | Macrophages, Alveolar - pathology | Oxidative Stress - immunology | Monocyte Chemoattractant Proteins - immunology | Ozone - administration & dosage | Inflammation - metabolism | Respiratory Mucosa - immunology | Receptors, Chemokine - biosynthesis | Lung - metabolism | Chemokines, CXC - biosynthesis | Chemokines - immunology | Neutrophils - pathology | Adjuvants, Immunologic - physiology | Chemokines - biosynthesis | Lung - pathology | Monocyte Chemoattractant Proteins - biosynthesis | Cytokines | Neutrophils - drug effects | Chemokine CCL7 | Neutrophils - immunology | Administration, Inhalation | Inflammation - immunology | Adjuvants, Immunologic - biosynthesis | Chemokine CXCL10 | Up-Regulation - drug effects | Animals | Up-Regulation - immunology | Macrophages, Alveolar - drug effects | Lung - drug effects | Chemokines, CXC - physiology | Mice | Mice, Inbred BALB C | Receptors, CCR3 | Oxidative Stress - drug effects | Respiratory Mucosa - metabolism | Lung - immunology
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