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The Journal of Immunology, ISSN 0022-1767, 04/2003, Volume 170, Issue 7, pp. 3883 - 3889
Journal Article
FASEB Journal, ISSN 0892-6638, 03/2006, Volume 20, Issue 3, pp. 533 - 535
Although metallothionein (MT) can be induced by inflammatory mediators, its roles in coagulatory disturbance during inflammation are poorly defined. We... 
cytokine | OXIDATIVE STRESS | BIOCHEMISTRY & MOLECULAR BIOLOGY | INDUCTION | INTERLEUKIN-6 | CELL BIOLOGY | SHOCK | systemic inflammatory response syndrome | TUMOR-NECROSIS-FACTOR | ACUTE-PHASE | PROTECTIVE ROLE | BIOLOGY | NULL MICE | endothelial damage | EXPRESSION | BACTERIAL-ENDOTOXIN | Inflammation - chemically induced | Interleukin-1 - genetics | Liver - pathology | Kidney - pathology | Multiple Organ Failure - chemically induced | Systemic Inflammatory Response Syndrome - physiopathology | Interleukin-1 - biosynthesis | Chemotactic Factors - biosynthesis | Kidney - metabolism | Granulocyte-Macrophage Colony-Stimulating Factor - biosynthesis | Chemotactic Factors - genetics | Lung - metabolism | Systemic Inflammatory Response Syndrome - chemically induced | Cytokines - genetics | Neutrophils - pathology | Chemokine CCL4 | Lung - pathology | Chemokine CCL3 | Lipopolysaccharides - toxicity | Interleukin-6 - genetics | Thrombophilia - physiopathology | Liver - metabolism | Mice, Inbred C57BL | Chemokine CCL2 - genetics | Macrophage Inflammatory Proteins - genetics | Monokines - biosynthesis | Monokines - genetics | Granulocyte-Macrophage Colony-Stimulating Factor - genetics | Metallothionein - physiology | Mice, Knockout | Gene Expression Regulation - drug effects | Animals | Metallothionein - genetics | Homeostasis - physiology | Lipopolysaccharides - pharmacology | Chemokine CCL2 - biosynthesis | Chemokine CXCL2 | Interleukin-6 - biosynthesis | Thrombophilia - chemically induced | Mice | Metallothionein - deficiency | Multiple Organ Failure - physiopathology | Cytokines - biosynthesis | Inflammation - physiopathology | Macrophage Inflammatory Proteins - biosynthesis
Journal Article
Microbes and Infection, ISSN 1286-4579, 2006, Volume 8, Issue 4, pp. 974 - 983
Journal Article
Shock, ISSN 1073-2322, 01/2005, Volume 23, Issue 1, pp. 39 - 44
Journal Article
Journal Article
The Journal of Immunology, ISSN 0022-1767, 02/2002, Volume 168, Issue 4, pp. 1919 - 1925
Journal Article
Environmental Health Perspectives, ISSN 0091-6765, 3/2006, Volume 114, Issue 3, pp. 328 - 333
Journal Article
Critical Care Medicine, ISSN 0090-3493, 04/2007, Volume 35, Issue 4, pp. 1113 - 1122
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 06/2000, Volume 20, Issue 12, pp. 4398 - 4404
Broad spectrum caspase inhibitors have been found to reduce neurodegeneration caused by cerebral ischemia. We studied whether blockade of group I caspases,... 
Neuroprotection | Caspase inhibition | TNF-σ | Permanent focal cerebral ischemia | Ac- YVAD.cmk | IL-1β | Apoptosis | Ac-YVAD.cmk | TNF-alpha | NITRIC-OXIDE SYNTHASE | apoptosis | IL-1 beta | NEUROSCIENCES | caspase inhibition | DAMAGE | INTERLEUKIN-1-BETA CONVERTING-ENZYME | FACTOR-ALPHA | PROGRAMMED CELL-DEATH | STROKE | TUMOR-NECROSIS-FACTOR | permanent focal cerebral ischemia | neuroprotection | BRAIN INJURY | RAT-BRAIN | EXPRESSION | Apoptosis - drug effects | Cerebral Cortex - pathology | Injections, Intraventricular | Nucleosomes - drug effects | Male | Cerebral Infarction - prevention & control | Interleukin-1 - biosynthesis | Cerebral Infarction - physiopathology | Neuroprotective Agents - pharmacology | Time Factors | Amino Acid Chloromethyl Ketones - pharmacology | Cerebral Cortex - drug effects | Cerebral Infarction - pathology | Ischemic Attack, Transient - pathology | Ischemic Attack, Transient - physiopathology | In Situ Nick-End Labeling | Rats | Inflammation | Caspase Inhibitors | Monokines - biosynthesis | Rats, Sprague-Dawley | Amino Acid Chloromethyl Ketones - administration & dosage | Animals | Cerebral Cortex - immunology | Cysteine Proteinase Inhibitors - pharmacology | Chemokine CCL2 - biosynthesis | Chemokine CXCL2 | Interleukin-10 - biosynthesis | Nucleosomes - pathology | Tumor Necrosis Factor-alpha - biosynthesis | Cytokines - biosynthesis | ARTICLE | TNF-α
Journal Article
The Journal of Immunology, ISSN 0022-1767, 07/2002, Volume 169, Issue 2, pp. 974 - 982
We postulated that the seleno-organic compound ebselen would attenuate neutrophil recruitment and activation after aerosolized challenge with endotoxin (LPS)... 
ENDOTHELIAL ADHESION MOLECULES | SELENOORGANIC COMPOUND | IN-VIVO | ACUTE LUNG INJURY | ATTENUATED RESPONSE GENES | FUNCTIONAL-CHARACTERIZATION | IMMUNOLOGY | CHEMOATTRACTANT CINC | MONOCLONAL-ANTIBODIES | ISCHEMIA-REPERFUSION | NF-KAPPA-B | Interleukin-1 - genetics | Tumor Necrosis Factor-alpha - genetics | Organoselenium Compounds - administration & dosage | Bronchoalveolar Lavage Fluid - immunology | Male | Azoles - therapeutic use | Bronchi - immunology | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Interleukin-1 - biosynthesis | Bronchi - drug effects | RNA, Messenger - biosynthesis | Chemotactic Factors - biosynthesis | Intercellular Adhesion Molecule-1 - biosynthesis | RNA, Messenger - antagonists & inhibitors | Bronchi - metabolism | Bronchoalveolar Lavage Fluid - cytology | Interleukin-1 - antagonists & inhibitors | Chemokines, CXC - biosynthesis | Neutrophil Infiltration - drug effects | Organoselenium Compounds - therapeutic use | Disease Models, Animal | Neutrophil Infiltration - immunology | Azoles - administration & dosage | Lung - pathology | Lipopolysaccharides - toxicity | Intercellular Signaling Peptides and Proteins | Injections, Intraperitoneal | Rats | Inflammation - immunology | Monokines - biosynthesis | Up-Regulation - drug effects | Animals | Up-Regulation - immunology | Lung - drug effects | Aerosols | Chemokine CXCL2 | Chemokine CXCL1 | Chemokines, CXC - physiology | Growth Substances - biosynthesis | Tumor Necrosis Factor-alpha - biosynthesis | Lung - immunology | Tumor Necrosis Factor-alpha - antagonists & inhibitors
Journal Article
Clinical & Experimental Ophthalmology, ISSN 1442-6404, 06/2002, Volume 30, Issue 3, pp. 196 - 199
Ocular infection with Pseudomonas aeruginosa triggers extensive host inflammatory response and corneal damage. The purpose of present study was to investigate... 
cytokine | cornea | chemokine | Pseudomonas aeruginosa | Chemokine | Cornea | Cytokine | CELLS | PULMONARY INFLAMMATION | INJURY | MODEL | INTERLEUKIN-6 | MESSENGER-RNA | OPHTHALMOLOGY | MICE | TNF-ALPHA | PROTEIN-2 | Interleukin-1 - genetics | Interleukin-8 - genetics | Corneal Ulcer - genetics | Tumor Necrosis Factor-alpha - genetics | Pseudomonas aeruginosa - physiology | RNA, Messenger - metabolism | Interleukin-1 - biosynthesis | Pseudomonas Infections - metabolism | Eye Infections, Bacterial - microbiology | Gene Expression - physiology | Corneal Ulcer - microbiology | Female | Models, Animal | Eye Infections, Bacterial - metabolism | Cytokines - genetics | Cornea - microbiology | Chemokines - biosynthesis | Corneal Ulcer - metabolism | Interleukin-6 - genetics | Eye Infections, Bacterial - genetics | Interleukin-8 - biosynthesis | Reverse Transcriptase Polymerase Chain Reaction | Chemokines - genetics | Monokines - biosynthesis | Pseudomonas Infections - microbiology | Monokines - genetics | Pseudomonas Infections - genetics | Animals | Chemokine CXCL2 | Interleukin-6 - biosynthesis | Mice | Mice, Inbred BALB C | Tumor Necrosis Factor-alpha - biosynthesis | Cytokines - biosynthesis | Corneal Ulcer, metabolism | Interleukin-8, biosynthesis | Tumor Necrosis Factor, genetics | Eye Infections, Bacterial, metabolism | Interleukin-1, biosynthesis | Corneal Ulcer, microbiology | Gene Expression, physiology | Pseudomonas Infections, microbiology | Corneal Ulcer, genetics | Monokines, biosynthesis | Monokines, genetics | Chemokines, genetics | Cytokines, genetics | Pseudomonas aeruginosa, physiology | RNA, Messenger, metabolism | Eye Infections, Bacterial, genetics | Cytokines, biosynthesis | Cornea, microbiology | Interleukin-6, genetics | Tumor Necrosis Factor, biosynthesis | Interleukin-8, genetics | Pseudomonas Infections, metabolism | Interleukin-1, genetics | Eye Infections, Bacterial, microbiology | Pseudomonas Infections, genetics | Animal | Interleukin-6, biosynthesis | Chemokines, biosynthesis
Journal Article
Journal Article
Journal Article
Circulation, ISSN 0009-7322, 09/2001, Volume 104, Issue 12, pp. 1413 - 1418
Background-Complement consists of a complex cascade of proteins involved in innate and adaptive immunity. The cascade can be activated through 3 distinct... 
Antibodies | Infarction | Inflammation | Ischemia | Cell adhesion molecules | cell adhesion molecules | CARDIAC & CARDIOVASCULAR SYSTEMS | COMPLEMENT ACTIVATION | infarction | ischemia | NECROSIS | THERAPY | inflammation | PATHWAY | PURIFICATION | PERIPHERAL VASCULAR DISEASE | antibodies | SERINE-PROTEASE | PROTEINS | HEMATOLOGY | GLOMERULAR DEPOSITION | Myocardial Reperfusion Injury - etiology | Surface Plasmon Resonance | Antibody Specificity | Myocardial Ischemia - metabolism | Cell Adhesion Molecules - genetics | Male | Dose-Response Relationship, Drug | Interleukins - genetics | RNA, Messenger - biosynthesis | Myocardial Reperfusion Injury - pathology | Carrier Proteins - pharmacology | Myocardium - metabolism | Interleukins - biosynthesis | Complement Activation - drug effects | Disease Models, Animal | Collectins | Cell Adhesion Molecules - biosynthesis | Complement C3 - metabolism | Antibodies, Monoclonal - pharmacology | Carrier Proteins - antagonists & inhibitors | Rats | Myocardium - pathology | Monokines - biosynthesis | Rats, Sprague-Dawley | Monokines - genetics | Myocardial Ischemia - pathology | Animals | Signal Transduction - drug effects | Myocardial Ischemia - complications | Creatine Kinase - metabolism | Mannose - pharmacology | Antibodies, Monoclonal - metabolism | Myocardial Reperfusion Injury - prevention & control | Peroxidase - metabolism
Journal Article
Journal Article