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Journal of Comparative Neurology, ISSN 0021-9967, 10/2001, Volume 439, Issue 2, pp. 127 - 139
The specific mechanisms by which nervous system injury becomes a chronic pain state remain undetermined. Historically, it has been believed that injuries... 
neuropathy | nociception | radiculopathy | behavioral sensitivity | TNF | IL‐6 | Nociception | Neuropathy | Radiculopathy | Behavioral sensitivity | IL-6 | MECHANICAL ALLODYNIA | RAT | ROOT | DISORDERS | MODEL | NEUROSCIENCES | NEUROPATHIC PAIN | ZOOLOGY | A77-1726 | LEFLUNOMIDE | METABOLITE | MONONEUROPATHY | Ganglia, Spinal - injuries | Immunohistochemistry | Membrane Glycoproteins - metabolism | Neuroglia - pathology | Tumor Necrosis Factor-alpha - genetics | Male | Ganglia, Spinal - physiopathology | Peripheral Nervous System Diseases - physiopathology | Glial Fibrillary Acidic Protein - metabolism | RNA, Messenger - metabolism | Cytokines - genetics | Interleukin-6 - metabolism | Disease Models, Animal | Interleukin-6 - genetics | Rats | Rats, Sprague-Dawley | Peripheral Nervous System Diseases - metabolism | Blood Proteins | Pain - physiopathology | Interleukin-10 - genetics | Ganglia, Spinal - pathology | Antigens, CD | Tumor Necrosis Factor-alpha - metabolism | Interleukin-1 - genetics | Spinal Cord - drug effects | Spinal Cord - metabolism | Gliosis - physiopathology | Neuroglia - drug effects | Receptors, Tumor Necrosis Factor - therapeutic use | Basigin | Dose-Response Relationship, Drug | Pain - drug therapy | Sialoglycoproteins - pharmacology | Spinal Cord - pathology | Interleukin-10 - metabolism | Peripheral Nervous System Diseases - pathology | Gliosis - etiology | Interleukin-1 - metabolism | Antigens, Neoplasm | Cytokines - metabolism | Hyperalgesia - metabolism | Gliosis - metabolism | Hyperalgesia - physiopathology | Animals | Hyperalgesia - etiology | Avian Proteins | Interleukin 1 Receptor Antagonist Protein | Neuroglia - metabolism | Antigens, Surface | Cytokines - antagonists & inhibitors | Lumbar Vertebrae
Journal Article
Journal of Neurosurgery: Spine, ISSN 1547-5654, 08/2016, Volume 25, Issue 2, pp. 154 - 164
OBJECTIVE It has been hypothesized that the recombinant human bone morphogenetic protein-2 (rhBMP-2) amplification of the host inflammatory response interacts... 
Bone morphogenetic protein-2 | Inflammation | Rat | Chronic constriction injury | SURGERY | ACTIVATION | rat | INJURY | chronic constriction injury | DISC MATERIAL | bone morphogenetic protein-2 | CLINICAL NEUROLOGY | inflammation | GLUTAMATE | ANIMAL-MODELS | HYPERALGESIA | PERIPHERAL MONONEUROPATHY | Constriction, Pathologic - pathology | Drug Implants | Motor Activity - drug effects | Male | Recombinant Proteins - adverse effects | Constriction, Pathologic - physiopathology | Hyperalgesia - pathology | Sciatic Neuropathy - surgery | Bone Morphogenetic Protein 2 - adverse effects | Bone Morphogenetic Protein 2 - administration & dosage | Transforming Growth Factor beta - adverse effects | Pain - etiology | Sciatic Nerve - drug effects | Sciatic Neuropathy - drug therapy | Disease Models, Animal | Sciatic Nerve - pathology | Osteogenesis - drug effects | Sciatic Neuropathy - pathology | Hot Temperature | Random Allocation | Pain - pathology | Rats, Sprague-Dawley | Recombinant Proteins - administration & dosage | Absorbable Implants | Hyperalgesia - physiopathology | Animals | Constriction, Pathologic - drug therapy | Constriction, Pathologic - surgery | Pain - physiopathology | Collagen | Transforming Growth Factor beta - administration & dosage | Hyperalgesia - etiology | Sciatic Neuropathy - physiopathology | Pain Measurement | Chronic Disease
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 11/2002, Volume 22, Issue 22, pp. 9980 - 9989
Hypersensitivity resulting from nerve injury or morphine tolerance/ hyperalgesia is predicted to involve similar cellular and molecular mechanisms. One... 
Morphine tolerance | Interleukin-1β | Neuropathy | Tumor necrosis factor-α | Glia | Hyperalgesia | Interleukin-6 | GLIAL ACTIVATION | morphine tolerance | PERSISTENT PAIN | TOLERANCE | INVOLVEMENT | MECHANISMS | MODEL | glia | NEUROSCIENCES | interleukin-6 | OPIOID-INDUCED HYPERALGESIA | tumor necrosis factor-alpha | interleukin-1 beta | NERVE-INJURED RATS | MICROGLIA | neuropathy | hyperalgesia | IMMUNE FUNCTION | Hyperalgesia - chemically induced | Tumor Necrosis Factor-alpha - metabolism | Interleukin-1 - genetics | Interleukin-6 - antagonists & inhibitors | Sialoglycoproteins - administration & dosage | Spinal Cord - drug effects | Neuroglia - pathology | Tumor Necrosis Factor-alpha - genetics | Mononeuropathies - physiopathology | Male | RNA, Messenger - metabolism | Behavior, Animal | Lumbosacral Region | Spinal Cord - pathology | Neuralgia - etiology | Drug Tolerance - immunology | Interleukin-6 - metabolism | Spinal Nerves - injuries | Disease Models, Animal | Injections, Spinal | Spinal Nerves - physiopathology | Interleukin-1 - metabolism | Drug Administration Schedule | Interleukin-6 - genetics | Morphine - pharmacology | Mononeuropathies - complications | Rats | Mononeuropathies - drug therapy | Pain Measurement - drug effects | Receptors, Tumor Necrosis Factor - administration & dosage | Rats, Sprague-Dawley | Neuralgia - drug therapy | Hyperalgesia - physiopathology | Antibodies - administration & dosage | Animals | Interleukin 1 Receptor Antagonist Protein | Neuroglia - metabolism | Spinal Cord - physiopathology | Substance Withdrawal Syndrome - physiopathology | Morphine - adverse effects | ARTICLE | tumor necrosis factor-α | interleukin-1β
Journal Article
International Journal of Rheumatic Diseases, ISSN 1756-1841, 09/2017, Volume 20, Issue 9, pp. 1297 - 1302
Journal Article
European Journal of Pain, ISSN 1090-3801, 2006, Volume 11, Issue 6, pp. 677 - 684
Abstract Complex regional pain syndromes (CRPS) are disabling pain syndromes that can develop after trauma or minor tissue injury affecting a limb.... 
Anesthesia & Perioperative Care | Pain Medicine | Post-ischemia pain | Ischemia-reperfusion-injury | Inflammation | Rat | CRPS | Post‐ischemia pain | Ischemia‐reperfusion‐injury | THERMAL HYPERALGESIA | rat | REGIONAL PAIN SYNDROME | NEUROSCIENCES | CLINICAL NEUROLOGY | C-FIBERS | NEUROPATHIC PAIN | inflammation | SYNDROME-TYPE-I | REFLEX SYMPATHETIC DYSTROPHY | ANESTHESIOLOGY | TISSUE ACIDOSIS | ANIMAL-MODEL | SYNDROME TYPE-1 | ischemia-reperfusion-injury | post-ischemia pain | PERIPHERAL MONONEUROPATHY | Sensation Disorders - physiopathology | Rats, Wistar | Sensation Disorders - etiology | Male | Complex Regional Pain Syndromes - physiopathology | Peripheral Nervous System Diseases - physiopathology | Complex Regional Pain Syndromes - diagnosis | Behavior, Animal | Femoral Artery - injuries | Hindlimb - physiopathology | Peripheral Nerves - metabolism | Reperfusion Injury - complications | Hyperalgesia - diagnosis | Disease Models, Animal | Peripheral Nervous System Diseases - etiology | Complex Regional Pain Syndromes - etiology | Femoral Artery - physiopathology | Rats | Hindlimb - innervation | Regional Blood Flow | Hyperalgesia - physiopathology | Peripheral Nerves - physiopathology | Animals | Reperfusion Injury - physiopathology | Hyperalgesia - etiology | Pain Threshold | Ligation - adverse effects | Sensation Disorders - diagnosis | Pain Measurement
Journal Article
Journal of Veterinary Internal Medicine, ISSN 0891-6640, 07/2018, Volume 32, Issue 4, pp. 1397 - 1409
Journal Article