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by Fu, AKY and Hung, KW and Fu, WY and Shen, C and Chen, Y and Xia, J and Lai, KO and Ip, NY
NATURE NEUROSCIENCE, ISSN 1097-6256, 02/2011, Volume 14, Issue 2, pp. 181 - U263
Homeostatic plasticity is crucial for maintaining neuronal output by counteracting unrestrained changes in synaptic strength. Chronic elevation of synaptic... 
ANAPHASE-PROMOTING COMPLEX | DENDRITIC SPINE MORPHOGENESIS | GLUTAMATE RECEPTORS | UBIQUITIN-PROTEASOME SYSTEM | EPHB RECEPTORS | PROTEIN-DEGRADATION | EXCITATORY SYNAPSES | SURFACE EXPRESSION | SYNAPTIC PLASTICITY | NEUROSCIENCES | POSTSYNAPTIC DENSITY | Anaphase-Promoting Complex-Cyclosome | Electrophysiology | Neuronal Plasticity - drug effects | Receptor, EphA4 - metabolism | Neurons - cytology | Ubiquitin-Protein Ligase Complexes - genetics | Hippocampus - drug effects | Miniature Postsynaptic Potentials - drug effects | Excitatory Postsynaptic Potentials - drug effects | Excitatory Postsynaptic Potentials - physiology | Neuronal Plasticity - physiology | Ubiquitination - drug effects | Neurons - physiology | Statistics, Nonparametric | Ubiquitination - physiology | Neurons - drug effects | Receptors, AMPA - genetics | Receptors, AMPA - metabolism | GABA-A Receptor Antagonists - pharmacology | Miniature Postsynaptic Potentials - physiology | Synapses - drug effects | Synapses - physiology | Cells, Cultured | Bicuculline - pharmacology | Down-Regulation - drug effects | Hippocampus - cytology | Down-Regulation - physiology | Proteasome Endopeptidase Complex - genetics | Animals | Ubiquitin-Protein Ligase Complexes - metabolism | Analysis of Variance | RNA, Small Interfering | Proteasome Endopeptidase Complex - metabolism | Hippocampus - physiology
Journal Article
Biomaterials, ISSN 0142-9612, 2013, Volume 34, Issue 13, pp. 3290 - 3302
Abstract To design tissue-specific bioscaffolds with well-defined properties and 3-D architecture, methods were developed for preparing porous foams from... 
Advanced Basic Science | Dentistry | ECM (extracellular matrix) | Wound healing | Scaffold | Adipose tissue engineering | Collagen | Stem cell | MATERIALS SCIENCE, BIOMATERIALS | STEM-CELL FATE | VIVO | ENGINEERING, BIOMEDICAL | NICHE | INDUCTION | COLLAGEN SPONGES | MORPHOGENESIS | EXTRACELLULAR-MATRIX | SCAFFOLDS | DIFFERENTIATION | SAMPLES | Adipose Tissue - physiology | Rats, Wistar | Adipogenesis - drug effects | Humans | Adipose Tissue - cytology | Elastic Modulus - drug effects | Tissue Scaffolds - chemistry | Microspheres | Female | Lipid Metabolism - genetics | Models, Animal | Adipogenesis - genetics | Microscopy, Electron, Scanning | Intracellular Space - drug effects | Tissue Engineering - methods | Freeze Drying | Rats | Biocompatible Materials - pharmacology | Regeneration - physiology | Gene Expression Regulation - drug effects | Glycerolphosphate Dehydrogenase - metabolism | Regeneration - drug effects | Animals | Intracellular Space - metabolism | Lipid Metabolism - drug effects | Mechanical Phenomena | Porosity | Adipose Tissue - drug effects | Subcutaneous Tissue - drug effects | Adipose tissues | Phosphates | Glycerin | Enzymes | Crosslinked polymers | Analysis | Stem cells | Glycerol | Gene expression | Electric properties | Foams | Surgical implants | Regeneration | Biomedical materials | Differentiation | Beads | Scaffolds | Soft tissues
Journal Article
Plant Physiology, ISSN 0032-0889, 6/2015, Volume 168, Issue 2, pp. 735 - 751
Plants alter their development in response to changes in their environment. This responsiveness has proven to be a successful evolutionary trait. Here, we... 
Branching | Receptors | Plant growth | Genes | Branches | Plant roots | American culture | Biology | SIGNALING AND RESPONSE | Plants | Rice | PHOSPHORUS DEFICIENCY | LEAF SENESCENCE | AXILLARY BUD OUTGROWTH | PHOSPHATE DEFICIENCY | STRIGOLACTONE | ARABIDOPSIS | TRANSCRIPTION FACTOR | PROMOTES | F-BOX PROTEIN | APICAL-DOMINANCE | PLANT SCIENCES | Genes, Plant | DNA, Complementary - genetics | Molecular Sequence Data | Plant Roots - genetics | RNA, Messenger - metabolism | Plant Stems - genetics | Promoter Regions, Genetic - genetics | Plant Roots - drug effects | Biosynthetic Pathways - genetics | Petunia - radiation effects | Biosynthetic Pathways - radiation effects | Light | Plant Stems - radiation effects | Plant Proteins - metabolism | Signal Transduction - radiation effects | Principal Component Analysis | Gene Expression Regulation, Plant - radiation effects | Morphogenesis - radiation effects | RNA, Messenger - genetics | Petunia - drug effects | Petunia - growth & development | Transcription Factors - metabolism | Phosphorus - pharmacology | Plant Proteins - genetics | Gene Expression Regulation, Plant - drug effects | Biosynthetic Pathways - drug effects | Plant Stems - drug effects | Signal Transduction - drug effects | Petunia - genetics | Morphogenesis - drug effects | Environment | Plant Roots - radiation effects
Journal Article
New Phytologist, ISSN 0028-646X, 03/2016, Volume 209, Issue 4, pp. 1496 - 1512
Journal Article
Development (Cambridge), ISSN 0950-1991, 04/2015, Volume 142, Issue 7, pp. 1357 - 1367
Bone morphogenetic protein (BMP) signaling plays many roles in skull morphogenesis. We have previously reported that enhanced BMP signaling through the BMP... 
Craniofacial development | Neural crest cell | BMP | MDM2 | Nasal septum | SMAD | Apoptosis | P53 | SEPTAL CARTILAGE | FOREBRAIN DEVELOPMENT | DAMAGE-INDUCED PHOSPHORYLATION | DEVELOPMENTAL BIOLOGY | p53 | PROGRAMMED CELL-DEATH | TUMOR-SUPPRESSOR | NULL MICE | RABBIT SNOUT GROWTH | VOMERAL BONE DEFECT | TRANSGENIC MICE | Embryo, Mammalian - drug effects | Nose - embryology | Chondrogenesis - drug effects | Apoptosis - drug effects | Mesoderm - drug effects | Embryo, Mammalian - metabolism | Neural Crest - metabolism | Embryo Loss - metabolism | Toluene - analogs & derivatives | Fibroblast Growth Factors - metabolism | Proteolysis - drug effects | Bone Morphogenetic Proteins - metabolism | Nasal Cartilages - abnormalities | Nasal Cartilages - embryology | Cell Nucleus - metabolism | Embryo Loss - pathology | Protein Binding - drug effects | Integrases - metabolism | Toluene - pharmacology | Proto-Oncogene Proteins c-mdm2 - metabolism | Nasal Cartilages - pathology | Nose - metabolism | Animals, Newborn | Cell Survival - drug effects | Embryo, Mammalian - pathology | Mesoderm - embryology | Benzothiazoles - pharmacology | Tumor Suppressor Protein p53 - metabolism | Bone Morphogenetic Protein Receptors, Type I - metabolism | Mutation - genetics | Neural Crest - drug effects | Animals | Signal Transduction - drug effects | Neural Crest - embryology | Morphogenesis - drug effects | Mice | Cell Nucleus - drug effects | Nasal Cartilages - metabolism | Mesoderm - pathology | Smad Proteins - metabolism
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 7/2015, Volume 112, Issue 29, pp. E3855 - E3863
Nonsmall cell lung cancer (NSCLC) is the leading cause of cancer death worldwide. About 14% of NSCLCs harbor mutations in epidermal growth factor receptor... 
Innate drug resistance | Nonsmall cell lung cancer | ERK1/2 paradoxical activation | Tyrosine kinase inhibitors | EGFr inhibition | TRANSCRIPTIONAL ACTIVITY | PROTEIN | SRC | nonsmall cell lung cancer | TYROSINE KINASE | MULTIDISCIPLINARY SCIENCES | innate drug resistance | MEDIATED PHOSPHORYLATION | tyrosine kinase inhibitors | GROWTH-FACTOR RECEPTOR | TARGETED THERAPIES | EGFR inhibition | GENE | C-MET | EPITHELIAL MORPHOGENESIS | Proto-Oncogene Protein c-ets-1 - metabolism | Humans | Cyclins - genetics | ras Proteins - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Neoplasm Proteins - metabolism | RNA, Messenger - metabolism | Receptor, Epidermal Growth Factor - metabolism | Bcl-2-Like Protein 11 | Cyclins - metabolism | Protein Binding - drug effects | src-Family Kinases - metabolism | Membrane Proteins - metabolism | Gene Expression Regulation, Neoplastic - drug effects | Phosphorylation - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Lung Neoplasms - genetics | Proto-Oncogene Proteins - metabolism | Cell Survival - drug effects | Lung Neoplasms - enzymology | Carcinoma, Non-Small-Cell Lung - genetics | RNA, Messenger - genetics | Enzyme Activation - drug effects | Mutation - genetics | Apoptosis Regulatory Proteins - metabolism | MAP Kinase Signaling System - drug effects | Models, Biological | Cell Line, Tumor | Receptor, Epidermal Growth Factor - antagonists & inhibitors | Protein Kinase Inhibitors - pharmacology | Carcinoma, Non-Small-Cell Lung - enzymology | Quinazolines - pharmacology | Drug Resistance, Neoplasm - drug effects | Enzymes | Development and progression | Genetic aspects | Regulation | Genetic transcription | Lung cancer, Non-small cell | Health aspects | Biological Sciences | ERK1 | PNAS Plus | 2 paradoxical activation
Journal Article
Neuropharmacology, ISSN 0028-3908, 2005, Volume 48, Issue 6, pp. 903 - 917
Intrauterine inflammation is a major risk for offspring neurodevelopmental brain damage and may result in cognitive limitations and poor cognitive and... 
Learning | Morphogenesis | Neurotrophic factors | Memory | Hippocampus | Cytokine | cytokine | memory | hippocampus | WATER-MAZE | RAT | BRAIN-INJURY | learning | NEUROSCIENCES | NERVE GROWTH-FACTOR | morphogenesis | neurotrophic factors | NEURONS | PHARMACOLOGY & PHARMACY | LONG-TERM POTENTIATION | TNF-ALPHA | DENTATE GYRUS | TRANSGENIC MICE | Brain Chemistry - drug effects | Inflammation - chemically induced | Prenatal Exposure Delayed Effects | Reflex - physiology | Age Factors | Pregnancy Complications | Body Weight - drug effects | Reflex - drug effects | Male | Spleen - drug effects | Brain - growth & development | Psychomotor Performance - physiology | Lipopolysaccharides | Brain - metabolism | Exploratory Behavior - physiology | Inflammation - complications | Neurons - classification | Time Factors | Reaction Time - drug effects | Exploratory Behavior - drug effects | Behavior, Animal - drug effects | Female | Brain-Derived Neurotrophic Factor - metabolism | Neurons - drug effects | Spleen - growth & development | Gene Expression Regulation, Developmental - physiology | Brain - cytology | Nerve Growth Factor - metabolism | Brain - physiopathology | Mice, Inbred C57BL | Gene Expression Regulation, Developmental - drug effects | Psychomotor Performance - drug effects | Embryonic Development | Reaction Time - physiology | Brain Chemistry - physiology | Pregnancy | Organ Size - drug effects | Animals | Cell Count - methods | Spleen - metabolism | Mice | Body Weight - physiology | Embryonic development | Neurosciences | Pregnant women | Neurons | Brain damage | Inflammation | Universities and colleges | Mitogens
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 3, p. e17531
We characterized the differentiation of rat bone marrow-derived mesenchymal stem cells (BM-MSCs) into tenocyte-like cells in response to bone morphogenetic... 
ROOF PLATE | OSTEOBLASTS | GROWTH | BIOLOGY | PROLIFERATION | GENE-TRANSFER | DIFFERENTIATION | EXPRESSION | TENOMODULIN | TENOCYTES | FAMILY | Calcaneus - pathology | Tendons - drug effects | Cell Lineage - drug effects | Calcaneus - drug effects | Tissue Scaffolds | Mesenchymal Stromal Cells - cytology | Basic Helix-Loop-Helix Transcription Factors - metabolism | Female | Collagen - pharmacology | Membrane Proteins - metabolism | Bone Morphogenetic Proteins - pharmacology | Wound Healing - drug effects | Adult Stem Cells - drug effects | Tissue Engineering | Mesenchymal Stromal Cells - drug effects | Adult Stem Cells - cytology | Basic Helix-Loop-Helix Transcription Factors - genetics | Implants, Experimental | Bone Marrow Cells - cytology | Membrane Proteins - genetics | Cells, Cultured | Mesenchymal Stromal Cells - metabolism | Rats | Rats, Sprague-Dawley | Gene Expression Regulation - drug effects | Adult Stem Cells - metabolism | Animals | Cell Differentiation - drug effects | Tendons - pathology | Bone morphogenetic proteins | Tissue engineering | Collagen | Stem cells | Monomolecular films | Tenascin | Surgical implants | Collagen (type I) | Mesenchyme | Stem cell transplantation | Nervous system | Transplantation | Morphogenesis | Proteins | Engineering | Allografts | Bone marrow | Growth factors | Elongation | Biomedical engineering | Recombinant | Reconstruction | Tenascin C | Plastic surgery | Tendons | Medicine | Progeny | Fractures | Cell number | Skin & tissue grafts | Biomarkers | Bone | Differentiation | Scaffolds | Colonies | scaffolds
Journal Article
American Journal of Pathology, ISSN 0002-9440, 12/2012, Volume 181, Issue 6, pp. 2126 - 2137
Journal Article
Angiogenesis, ISSN 0969-6970, 3/2011, Volume 14, Issue 1, pp. 47 - 59
Using a fibrin-based angiogenesis model, we have established that there is no canonical mechanism used by endothelial cells (ECs) to degrade the surrounding... 
Urokinase plasminogen activator | Matrix metalloproteinases | Oncology | Adipose-derived stem cells | Biomedicine general | Plasmin | Cell Biology | Angiogenesis | Biomedicine | Cancer Research | Ophthalmology | Cardiology | Mesenchymal stem cells | CAPILLARY MORPHOGENESIS | PLASMINOGEN-ACTIVATOR UPA | TISSUE | PROTEOLYSIS | TUBE FORMATION | HUMAN ENDOTHELIAL-CELLS | UROKINASE RECEPTOR | PERIPHERAL VASCULAR DISEASE | EXTRACELLULAR-MATRIX | GROWTH-FACTOR | Neovascularization, Physiologic - drug effects | Blood Vessels - growth & development | Coculture Techniques | Humans | Adipose Tissue - cytology | Extracellular Matrix - metabolism | Gene Expression Profiling | Mesenchymal Stromal Cells - cytology | Bone Marrow Cells - drug effects | Cytokines - genetics | Fibrinolysin - metabolism | Fibroblasts - metabolism | Mesenchymal Stromal Cells - drug effects | Cytokines - metabolism | Morphogenesis - genetics | Bone Marrow Cells - cytology | Dipeptides - pharmacology | Extracellular Matrix - drug effects | Fibrinolysin - antagonists & inhibitors | Cells, Cultured | Mesenchymal Stromal Cells - metabolism | Gene Expression Regulation - drug effects | Phenotype | Cell Differentiation - drug effects | Fibroblasts - drug effects | Blood Vessels - drug effects | Matrix Metalloproteinase Inhibitors | Morphogenesis - drug effects | Fibroblasts - cytology | Matrix Metalloproteinases - metabolism | Bone Marrow Cells - metabolism | Usage | Tissue plasminogen activator | Stem cells | Physiological aspects | Extracellular matrix | Genetic aspects | Research | Neovascularization | Health aspects
Journal Article
Journal Article