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Journal of Neuroscience, ISSN 0270-6474, 06/2012, Volume 32, Issue 25, pp. 8703 - 8715
Journal Article
The Journal of cell biology, ISSN 1540-8140, 2009, Volume 187, Issue 6, pp. 761 - 772
Selective degeneration and death of one or more classes of neurons is the defining feature of human neurodegenerative disease... 
Motor neurons | Nervous system diseases | Reviews | Neurodegenerative diseases | Neurons | Astrocytes | Motor neuron disease | Huntington disease | Parkinson disease | Amyotrophic lateral sclerosis | Prion diseases | MOTOR-NEURONS | NEURON-SPECIFIC EXPRESSION | CU/ZN-SUPEROXIDE-DISMUTASE | SLOW DISEASE PROGRESSION | MONOAMINE-OXIDASE-B | LINKED SOD1 MUTANTS | AMYOTROPHIC-LATERAL-SCLEROSIS | FRONTOTEMPORAL LOBAR DEGENERATION | TRANSGENIC MOUSE MODEL | GENOME-WIDE ASSOCIATION | CELL BIOLOGY | Neurons - pathology | Axonal Transport | Superoxide Dismutase - genetics | Capillaries - pathology | T-Lymphocytes - enzymology | Humans | Astrocytes - pathology | Stress, Physiological | Nerve Degeneration - genetics | Astrocytes - enzymology | Endoplasmic Reticulum - pathology | Cell Death | Superoxides - metabolism | Microglia - pathology | Amyotrophic Lateral Sclerosis - enzymology | T-Lymphocytes - pathology | Superoxide Dismutase - metabolism | Endoplasmic Reticulum - enzymology | Nerve Degeneration - enzymology | Amyotrophic Lateral Sclerosis - genetics | Microglia - enzymology | Capillaries - enzymology | Nerve Degeneration - pathology | Amyotrophic Lateral Sclerosis - pathology | Animals | Neurons - enzymology | Glutamic Acid - metabolism | Mutation | Superoxide Dismutase-1 | Care and treatment | Physiological aspects | Superoxide dismutase | Genetic aspects | Disease susceptibility | Research | Health aspects | Index Medicus
Journal Article
Scientific Reports, ISSN 2045-2322, 03/2017, Volume 7, Issue 1, p. 44989
Journal Article
The Journal of clinical investigation, ISSN 1558-8238, 2019, Volume 129, Issue 9, pp. 3738 - 3753
Journal Article
Lancet, The, ISSN 0140-6736, 2016, Volume 388, Issue 10047, pp. 919 - 931
Summary Early reports of cognitive and behavioural deficits in motor neuron disease might have been overlooked initially, but the concept of a frontotemporal dementia-motor neuron disease continuum... 
Internal Medicine | HEXANUCLEOTIDE REPEAT EXPANSION | MEDICINE, GENERAL & INTERNAL | PTDP-43 PATHOLOGY | LOBAR DEGENERATION | EXPANDED C9ORF72 | ANTISENSE TRANSCRIPTS | BRAIN NETWORK CONNECTIVITY | C9ORF72 EXPANSION | COGNITIVE IMPAIRMENT | AMYOTROPHIC-LATERAL-SCLEROSIS | RNA FOCI | Neuroimaging | Neuroprotective Agents - therapeutic use | Prognosis | Humans | Patient Care Team | Cognition | Frontotemporal Dementia - psychology | Activities of Daily Living | Brain - metabolism | DNA-Binding Proteins - metabolism | DNA Repeat Expansion | DNA Methylation | Motor Neuron Disease - therapy | Motor Neuron Disease - genetics | Riluzole - therapeutic use | C9orf72 Protein | Executive Function | Frontotemporal Dementia - pathology | Frontotemporal Dementia - genetics | DNA-Binding Proteins - genetics | Frontotemporal Dementia - therapy | Motor Neuron Disease - pathology | Neuropsychological Tests | Proteins - genetics | Proteins - metabolism | Image Processing, Computer-Assisted | Brain - pathology | Motor Neuron Disease - psychology | Mutation | Medical colleges | Neurosciences | Neurons | Epidemiology | Dementia | Protein binding | Brain | Gene expression | Patients | Studies | Pathology | Stem cells | DNA methylation | Epigenetics | Genetics | Chromosomes | Deoxyribonucleic acid--DNA
Journal Article
Journal Article
Acta neuropathologica, ISSN 1432-0533, 2015, Volume 130, Issue 1, pp. 63 - 75
... determined. Blinded examination of CNS biosamples from ALS patients with a repeat expansion of C9ORF72 showed that antisense foci are present at a significantly higher frequency in cerebellar Purkinje neurons... 
Immunohistochemistry | Pathology | Neurosciences | Dipeptide repeat protein | Medicine & Public Health | Amyotrophic lateral sclerosis | C9ORF72 | RNA foci | EXPANSIONS | ALS | NEURODEGENERATION | AMYOTROPHIC-LATERAL-SCLEROSIS | PATHOLOGY | FRONTOTEMPORAL LOBAR DEGENERATION | TRANSLATION | NEUROSCIENCES | CLINICAL NEUROLOGY | SEQUESTRATION | MESSENGER-RNA | DIPEPTIDE-REPEAT PROTEINS | HEXANUCLEOTIDE REPEAT | Frontotemporal Lobar Degeneration - pathology | Humans | Middle Aged | Male | Motor Neurons - pathology | DNA-Binding Proteins - metabolism | DNA Repeat Expansion | Frontotemporal Lobar Degeneration - metabolism | Female | C9orf72 Protein | Inclusion Bodies - metabolism | RNA, Antisense | Purkinje Cells - metabolism | Amyotrophic Lateral Sclerosis - genetics | Cerebellum - metabolism | Hippocampus - pathology | Cerebellum - pathology | Motor Neurons - metabolism | Proteins - genetics | Hippocampus - metabolism | Amyotrophic Lateral Sclerosis - pathology | Inclusion Bodies - genetics | Proteins - metabolism | Amyotrophic Lateral Sclerosis - metabolism | Inclusion Bodies - pathology | Frontotemporal Lobar Degeneration - genetics | Purkinje Cells - pathology | Proteins | Crosslinked polymers | Neurons | Antisense RNA | Protein binding | Index Medicus | Original Paper
Journal Article