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2010, ISBN 9781405179546, xi, 297
"Serving the needs of pigment cell biologists, cellular physiologists, developmental geneticists, researchers interested in melanoma and more, this new book... 
genetics | Color | physiology | Models, Animal | Mice | Models, Genetic | Gene Regulatory Networks | Pigmentation | Mice as laboratory animals | Pigments, Biological | Genetics
Book
Acta Physiologica, ISSN 1748-1708, 02/2019, Volume 225, Issue 2, pp. e13237 - n/a
Journal Article
2008, 1. Aufl., Current topics in microbiology and immunology, ISBN 3540756477, Volume 324., xiii, 199
Book
2006, Practical approach series., ISBN 9780198550013, xxv, 334
The groundbreaking isolation of embryonic stem cells (or 'ES cells') of the mouse in the early 1980s triggered a sustained expansion of global research into... 
Embryonic stem cells | Medicine: General Issues
Book
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 1/2011, Volume 108, Issue 2, pp. 751 - 756
Sphingosine 1-phosphate (S1P), a lysophospholipid, has gained relevance to multiple sclerosis through the discovery of FTY720 (fingolimod), recently approved... 
Receptors | Multiple sclerosis | Spinal cord | Astrocytes | Neurons | Lymphocytes | Central nervous system | Cell lines | Null condition | Mice | G protein-coupled receptor | Lysophospholipid | Neuroprotection | Knockout | knockout | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | ACTIVATION | ORAL FINGOLIMOD | WHITE-MATTER | MULTIDISCIPLINARY SCIENCES | LYMPHOCYTE EGRESS | AGONISTS | neuroprotection | FIBRILLARY ACIDIC PROTEIN | SPHINGOSINE-1-PHOSPHATE | IMMUNOSUPPRESSANT FTY720 | CENTRAL-NERVOUS-SYSTEM | lysophospholipid | Astrocytes - cytology | Lymphocytes - metabolism | Propylene Glycols - pharmacology | Encephalomyelitis, Autoimmune, Experimental - metabolism | Receptors, G-Protein-Coupled - metabolism | Signal Transduction | Encephalomyelitis, Autoimmune, Experimental - drug therapy | Mice, Inbred C57BL | Gene Expression Regulation | Fingolimod Hydrochloride | Mice, Knockout | Sphingosine - pharmacology | Sphingosine - analogs & derivatives | Animals | Female | Receptors, Lysosphingolipid - metabolism | Immunosuppressive Agents - pharmacology | Disease Models, Animal | Multiple Sclerosis - drug therapy | Physiological aspects | Care and treatment | Studies | Prescription drugs | Phosphorylation | Immunology | Metabolites | Rodents | Lipids | Models | Gene expression | Index Medicus | Animal models | Cell lineage | Data processing | Neuromodulation | Experimental allergic encephalomyelitis | sphingosine 1-phosphate receptors | Sphingosine 1-phosphate | Signal transduction | Gliosis | Demyelination | FTY720 | Glia | Protein transport | Biological Sciences
Journal Article
Epilepsia, ISSN 0013-9580, 07/2018, Volume 59, Issue 7, pp. 1455 - 1468
ObjectiveNa(+)/K+-ATPase dysfunction, primary (mutation) or secondary (energy crisis, neurodegenerative disease) increases neuronal excitability in the brain.... 
epilepsy | D801N | 1A3 | alternating hemiplegia of childhood | ATP | parvalbumin | ATP1A3 | ALTERNATING HEMIPLEGIA | DYSTONIA-PARKINSONISM | STRATUM-ORIENS | SPREADING DEPRESSION | CLINICAL NEUROLOGY | DE-NOVO MUTATIONS | SODIUM-PUMP | CA1 REGION | RAT HIPPOCAMPUS | RAPID-ONSET | FOCAL EPILEPSY | Interneurons - physiology | Sodium-Potassium-Exchanging ATPase - genetics | Sodium-Potassium-Exchanging ATPase - physiology | Humans | Evoked Potentials | Electroencephalography | Epilepsy - physiopathology | Mice, Neurologic Mutants | gamma-Aminobutyric Acid - physiology | Patch-Clamp Techniques | Animals | DNA Mutational Analysis | Hemiplegia - genetics | Pyramidal Cells - physiology | Epilepsy - genetics | Mice | Hemiplegia - physiopathology | In Vitro Techniques | Child | Hippocampus - physiopathology | Genetic Carrier Screening | Disease Models, Animal | Immunohistochemistry | Nervous system diseases | Neurons | Analysis | Epilepsy | GABA | Paralysis | Adenosine triphosphatase | Phenotypes | Interneurons | EEG | Brain slice preparation | Excitability | Action potential | Na+/K+-exchanging ATPase | Parvalbumin | Electrical stimuli | Depolarization | γ-Aminobutyric acid | Excitatory postsynaptic potentials | Pyramidal cells | Rodents | Hemiplegia | Glutamatergic transmission | Children | Mutation | Hippocampus | Bicuculline | Inhibitory postsynaptic potentials | Index Medicus
Journal Article
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