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International Journal of Cancer, ISSN 0020-7136, 12/2012, Volume 131, Issue 11, pp. 2553 - 2561
Estrogen receptor‐beta (ERβ) has been suggested to exert anti‐inflammatory and anti‐tumorigenic effects in the colon, providing a translational potential to... 
dextran sodium sulfate | estrogen receptor‐beta | colon cancer | azoxymethane | inflammatory bowel disease | estrogen receptor-beta | Estradiol - blood | Mucin-2 - genetics | Neoplasms - metabolism | Tumor Necrosis Factor-alpha - metabolism | Inflammation - pathology | Colitis - genetics | Colorectal Neoplasms - genetics | Tumor Necrosis Factor-alpha - genetics | Colitis - pathology | NF-kappa B - metabolism | Interferon-gamma - metabolism | Inflammatory Bowel Diseases - metabolism | Estrogen Receptor beta - metabolism | Cell Differentiation - genetics | Inflammation - metabolism | Estrogen Receptor beta - genetics | Proliferating Cell Nuclear Antigen - genetics | Neoplasms - genetics | Inflammatory Bowel Diseases - pathology | Inflammatory Bowel Diseases - genetics | Female | Interferon-gamma - genetics | Interleukin-6 - metabolism | Colorectal Neoplasms - metabolism | Mucin-2 - metabolism | Mucin-1 - metabolism | Interleukin-6 - genetics | Colon - pathology | Mice, Inbred C57BL | Caveolin 1 - genetics | Interleukin-17 - genetics | beta Catenin - metabolism | Colon - metabolism | beta Catenin - genetics | Mice, Knockout | Caveolin 1 - metabolism | Interleukin-17 - metabolism | Animals | NF-kappa B - genetics | Nitric Oxide Synthase Type II - genetics | Colitis - metabolism | Inflammation - genetics | Mice | Colorectal Neoplasms - pathology | Neoplasms - pathology | Proliferating Cell Nuclear Antigen - metabolism | Mucin-1 - genetics | Nitric Oxide Synthase Type II - metabolism | Estrogen receptor-beta
Journal Article
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 07/2011, Volume 286, Issue 28, pp. 25256 - 25264
Chronic infection of Helicobacter pylori in the stomach mucosa with translocation of the bacterial cytotoxin-associated gene A (CagA) effector protein via the... 
ANTIGEN-BINDING ADHESIN | OUTER-MEMBRANE PROTEINS | INTESTINAL METAPLASIA | BLOOD-GROUP ANTIGENS | BIOCHEMISTRY & MOLECULAR BIOLOGY | GASTRIC EPITHELIAL-CELLS | INFECTION | MONGOLIAN GERBILS | EXPRESSION | STOMACH | CAG PATHOGENICITY ISLAND | Mucin-2 - genetics | Inflammation - pathology | Interleukin-8 - genetics | Metaplasia - microbiology | Cricetulus | Metaplasia - metabolism | Adhesins, Bacterial - genetics | Humans | Helicobacter pylori - pathogenicity | Gastric Mucosa - pathology | Precancerous Conditions - metabolism | Gastric Mucosa - metabolism | Adhesins, Bacterial - metabolism | Inflammation - metabolism | Bacterial Adhesion - physiology | Gene Deletion | Helicobacter Infections - metabolism | Precancerous Conditions - pathology | Metaplasia - genetics | CHO Cells | Cricetinae | Inflammation - microbiology | Homeodomain Proteins - biosynthesis | Interleukin-8 - biosynthesis | Signal Transduction - genetics | Bacterial Secretion Systems - physiology | Helicobacter Infections - genetics | Precancerous Conditions - genetics | Homeodomain Proteins - genetics | Metaplasia - pathology | Gastric Mucosa - immunology | Animals | Chemokine CCL5 - biosynthesis | Chemokine CCL5 - genetics | Dogs | Inflammation - genetics | Lewis Blood-Group System - metabolism | Helicobacter pylori - physiology | Lewis Blood-Group System - genetics | Precancerous Conditions - microbiology | Mucin-2 - biosynthesis | Carbohydrate-binding Protein | BabA | Microbiology | Signal Transduction | Helicobacter pylori | Type IV Secretion System | Cancer Tumor Promoter | Bacteria | Carbohydrate | Glycoconjugate | Adhesion | Basic Medicine | Biological Sciences | Naturvetenskap | Medical and Health Sciences | Medicin och hälsovetenskap | Biokemi och molekylärbiologi | Biologiska vetenskaper | Medicinska och farmaceutiska grundvetenskaper | Cell and Molecular Biology | Biochemistry and Molecular Biology | Natural Sciences | Cell- och molekylärbiologi
Journal Article
BMC Gastroenterology, ISSN 1471-230X, 07/2013, Volume 13, Issue 1, pp. 113 - 113
Background: Celiac disease (CD) is an autoimmune disorder of the small intestine which is triggered by dietary gluten in genetically predisposed (HLA-DQ2/DQ8... 
Celiac disease | Microbiota | Duodenum | Gene expression | Host-microbe cross-talk | CROHNS-DISEASE | SERRATIA-MARCESCENS | MICROARRAY ANALYSIS | SMALL-INTESTINAL MICROBIOTA | HEALTHY-CHILDREN | INTERFERON-GAMMA | INFLAMMATION | CHEMOKINE CXCL16 | GASTROENTEROLOGY & HEPATOLOGY | T-CELLS | BACTERIAL-DNA | Mucin-2 - genetics | Intestinal Mucosa - metabolism | Humans | Middle Aged | Tumor Necrosis Factor-alpha - genetics | Child, Preschool | Homeostasis | Male | Receptors, Virus - genetics | Case-Control Studies | Celiac Disease - microbiology | Duodenum - microbiology | Duodenum - metabolism | Adult | Female | Interferon-gamma - genetics | Receptors, Chemokine - genetics | Child | Connexin 43 - genetics | Zonula Occludens-1 Protein - genetics | Gene Expression | Celiac Disease - genetics | Signal Transduction | Chemokine CXCL16 | Intestinal Mucosa - microbiology | Toll-Like Receptor 2 - metabolism | Chemokines, CXC - genetics | Pancreatitis-Associated Proteins | Proteins - genetics | Receptors, Scavenger - genetics | Interleukin-10 - genetics | Adolescent | Toll-Like Receptor 9 - metabolism | Metagenome | Celiac Disease - metabolism | Receptors, CXCR6 | Medical research | Microbiota (Symbiotic organisms) | Genes | Medicine, Experimental | Phylogeny | Pediatrics | Disease | Pathogenesis | Principal components analysis | Small intestine | Studies | Proteins | Nutrition research | Hospitals | Diet | Rodents | Ligands | Health risk assessment | Chemokines | Deoxyribonucleic acid--DNA | crohns-disease | microarray analysis | bacterial-dna | small-intestinal microbiota | inflammation | t-cells | serratia-marcescens | healthy-children | interferon-gamma | chemokine cxcl16
Journal Article
Journal of Proteome Research, ISSN 1535-3893, 07/2011, Volume 10, Issue 7, pp. 3040 - 3049
Proteomic analysis of samples isolated by laser capture microdissection from clinical specimens requires sample preparation and fractionation methods suitable... 
high resolution mass spectrometry | QUANTITATIVE PROTEOMICS | INCREASED EXPRESSION | SHOTGUN PROTEOMICS | N-GLYCOPROTEOME | colon cancer | SAMPLE PREPARATION | BIOCHEMICAL RESEARCH METHODS | CELL-GROWTH | laser capture microdissection | MASS-SPECTROMETRY | HUMAN COLORECTAL-CANCER | MITOTIC CHECKPOINT GENES | FFPE | FASP | MEMBRANE-PROTEINS | Mucin-2 - genetics | Colonic Neoplasms - genetics | Peptide Fragments | Proteome - genetics | Carcinoembryonic Antigen - chemistry | Transcription Factors - chemistry | Microdissection | Formaldehyde - chemistry | Cell Count | Humans | Gene Expression Regulation, Neoplastic | CD55 Antigens - genetics | Carcinoembryonic Antigen - genetics | Dextrans - chemistry | Polyethylene Glycols - chemistry | CD55 Antigens - chemistry | Proteome - chemistry | Mucin-2 - chemistry | Paraffin Embedding | Tandem Mass Spectrometry | Lasers | Mucin-1 - analysis | Mucin-1 - chemistry | Chromatography, Liquid | Colonic Neoplasms - chemistry | Proteomics - methods | CD55 Antigens - analysis | Biomarkers, Tumor - analysis | Mucin-2 - analysis | Fixatives - chemistry | Proteome - analysis | Transcription Factors - genetics | Carcinoembryonic Antigen - analysis | High-Throughput Screening Assays | Cell Line, Tumor | Biomarkers, Tumor - genetics | Transcription Factors - analysis | Biomarkers, Tumor - chemistry | Mucin-1 - genetics
Journal Article
Inflammatory Bowel Diseases, ISSN 1078-0998, 11/2011, Volume 17, Issue 11, pp. 2251 - 2260
Background: The transcription factor Atoh1/Hath1 plays crucial roles in the differentiation program of human intestinal epithelium cells (IECs). Although... 
Hath1 | Hes1 | ulcerative colitis | Notch signaling | CDX2 | GASTROINTESTINAL-TRACT | CANCER | AUTOREGULATION | MOUSE INTESTINE | INHIBITION | DISEASE | DIFFERENTIATION | GASTROENTEROLOGY & HEPATOLOGY | MATH1 EXPRESSION | REQUIREMENT | Mucin-2 - genetics | Colonic Neoplasms - genetics | Transcription Factor HES-1 | Luciferases - metabolism | Receptors, Notch - metabolism | Homeodomain Proteins - metabolism | Humans | Colitis, Ulcerative - genetics | Receptors, Notch - genetics | Intestines - metabolism | Promoter Regions, Genetic - genetics | Colonic Neoplasms - metabolism | Immunoenzyme Techniques | Basic Helix-Loop-Helix Transcription Factors - metabolism | Chromatin Immunoprecipitation | Kruppel-Like Transcription Factors - metabolism | Transcription, Genetic | Cell Differentiation | Tumor Cells, Cultured | Mucin-2 - metabolism | Basic Helix-Loop-Helix Transcription Factors - genetics | Signal Transduction | Colitis, Ulcerative - metabolism | RNA, Messenger - genetics | Gene Expression Regulation | Colitis, Ulcerative - pathology | Reverse Transcriptase Polymerase Chain Reaction | Goblet Cells - metabolism | Blotting, Western | Homeodomain Proteins - genetics | Colonic Neoplasms - pathology | CDX2 Transcription Factor | Kruppel-Like Transcription Factors - genetics | Goblet Cells - cytology | Intestines - cytology | Immunohistochemistry | Inflammatory bowel diseases | Transcription factors | Goblet cells | Intracellular signalling | CDX2 protein | Epithelium | Gene expression | Promoters | Homeobox | Polymerase chain reaction | Intestine | Notch protein | Differentiation | Ulcerative colitis
Journal Article
Mucosal Immunology, ISSN 1933-0219, 09/2012, Volume 5, Issue 5, pp. 501 - 512
Journal Article
Journal Article
Journal of Hepato-Biliary-Pancreatic Sciences, ISSN 1868-6974, 3/2010, Volume 17, Issue 2, pp. 108 - 124
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 08/2011, Volume 286, Issue 34, pp. 29964 - 29972
Journal Article
世界胃肠病学杂志:英文版(电子版), ISSN 1007-9327, 2014, Volume 20, Issue 42, pp. 15715 - 15726
AIM: To investigate esophageal Helicobacter pylori(H. pylori) colonization on esophageal injury caused by reflux and the related mechanisms. METHODS: An... 
Adenocarc | Metaplasia | pylori | Helicobacter | Esophagus | Adenocarcinoma | Animal model | Helicobacter pylori | INTESTINAL METAPLASIA | CDX2 | BARRETTS-ESOPHAGUS | ESOPHAGOGASTRODUODENAL ANASTOMOSIS | RAT MODEL | GASTROESOPHAGEAL-REFLUX | GASTRIC EPITHELIAL-CELLS | INFECTION | GASTROENTEROLOGY & HEPATOLOGY | EXPRESSION | Adenocarcinoma - pathology | Cell Proliferation | Homeodomain Proteins - metabolism | Ki-67 Antigen - metabolism | Male | RNA, Messenger - metabolism | Helicobacter Infections - pathology | Esophageal Neoplasms - pathology | Esophagus - microbiology | Adenocarcinoma - metabolism | Barrett Esophagus - pathology | Helicobacter Infections - metabolism | Disease Models, Animal | Barrett Esophagus - metabolism | bcl-2-Associated X Protein - metabolism | Esophagus - metabolism | Helicobacter Infections - genetics | Rats, Sprague-Dawley | Cyclin D1 - genetics | Proto-Oncogene Proteins c-myc - genetics | Helicobacter Infections - microbiology | Mucin-2 - genetics | Cyclin D1 - metabolism | Esophageal Neoplasms - microbiology | Helicobacter pylori - pathogenicity | Helicobacter Infections - complications | Proto-Oncogene Proteins c-bcl-2 - metabolism | Esophagitis, Peptic - pathology | Esophageal Neoplasms - metabolism | Adenocarcinoma - genetics | Barrett Esophagus - microbiology | Barrett Esophagus - genetics | bcl-2-Associated X Protein - genetics | Mucin-2 - metabolism | Severity of Illness Index | Adenocarcinoma - microbiology | Gene Expression Regulation | Esophagitis, Peptic - microbiology | Esophagitis, Peptic - metabolism | Transcription Factors - genetics | Proto-Oncogene Proteins c-myc - metabolism | Homeodomain Proteins - genetics | Transcription Factors - metabolism | Esophagus - pathology | Animals | Gastroesophageal Reflux - complications | Esophageal Neoplasms - genetics | Esophagitis, Peptic - genetics | CDX2 Transcription Factor | Proto-Oncogene Proteins c-bcl-2 - genetics | Apoptosis | Original
Journal Article
PLoS ONE, ISSN 1932-6203, 12/2011, Volume 6, Issue 12, p. e29094
MiRNAs play a relevant role in regulating gene expression in a variety of physiological and pathological conditions including autoimmune disorders. MiRNAs are... 
ACTIVATION | PATHWAY | MULTIDISCIPLINARY SCIENCES | DISEASE | BARRIER | GENE-EXPRESSION | TARGETS | PROLIFERATION | PATHOLOGY | EPITHELIAL DIFFERENTIATION | CHILDHOOD | Protein Binding - genetics | Transcription Factor HES-1 | Intestine, Small - pathology | Cell Count | Homeodomain Proteins - metabolism | Humans | 3' Untranslated Regions - genetics | Child, Preschool | Male | MicroRNAs - metabolism | Gene Expression Profiling | Case-Control Studies | Basic Helix-Loop-Helix Transcription Factors - metabolism | Kruppel-Like Transcription Factors - metabolism | HEK293 Cells | Female | Child | Diet, Gluten-Free | Mucin-2 - metabolism | Basic Helix-Loop-Helix Transcription Factors - genetics | Celiac Disease - genetics | Computational Biology | Gene Expression Regulation | Signal Transduction - genetics | Receptor, Notch1 - metabolism | Goblet Cells - metabolism | beta Catenin - metabolism | Homeodomain Proteins - genetics | Celiac Disease - pathology | Animals | Mice | MicroRNAs - genetics | Goblet Cells - pathology | Intestine, Small - metabolism | Receptor, Notch1 - genetics | Proteins | Immunohistochemistry | MicroRNA | Genes | Gluten-free diet | Physiological aspects | Gene expression | Cell proliferation | Biotechnology | Pediatrics | Phosphorylation | Wnt protein | Working groups | Laboratories | Gene regulation | Differentiation (biology) | Staining | Biochemistry | Small intestine | β-catenin | KLF4 protein | Rodents | Gastroenterology | Children | Localization | Bioinformatics | Active control | Gluten | Goblet cells | MiRNA | Cell lineage | Epithelium | Patients | Inflammatory bowel disease | Pathology | Celiac disease | Ribonucleic acids | Gastrointestinal diseases | Crypts | Notch protein | Autoimmune diseases | Health risk assessment
Journal Article