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Oncogene, ISSN 0950-9232, 07/2007, Volume 26, Issue 32, pp. 4617 - 4626
Infection with Helicobacter pylori cagA-positive strains is associated with gastric adenocarcinoma. Intestinal metaplasia is a precancerous lesion of the... 
β-catenin | Gastric adenocarcinoma | Helicobacter pylori CagA | E-cadherin | Intestinal metaplasia | beta-catenin | TARGET | ACTIVATION | PROTEIN | CDX1 | BIOCHEMISTRY & MOLECULAR BIOLOGY | CANCER | CELL BIOLOGY | gastric adenocarcinoma | GENE | ONCOLOGY | intestinal metaplasia | WNT PATHWAY | ATROPHIC GASTRITIS | GENETICS & HEREDITY | INFECTION | EXPRESSION | Intestinal Mucosa - metabolism | Phosphorylation | Adenocarcinoma - pathology | Cadherins - metabolism | beta Catenin - analysis | Adenocarcinoma - etiology | Homeodomain Proteins - metabolism | Humans | Transcriptional Activation | Gene Expression Regulation, Neoplastic | Stomach Neoplasms - metabolism | Cytoplasm - metabolism | Gastric Mucosa - pathology | Precancerous Conditions - metabolism | Stomach Neoplasms - pathology | Bacterial Proteins - analysis | Gastric Mucosa - metabolism | Adenocarcinoma - metabolism | Cell Nucleus - metabolism | Cyclin-Dependent Kinase Inhibitor p21 - genetics | Cell Transformation, Neoplastic - genetics | Mucins - metabolism | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Precancerous Conditions - pathology | Stomach Neoplasms - etiology | Cytoplasm - chemistry | Cell Line | Cadherins - analysis | Cell Transformation, Neoplastic - metabolism | Precancerous Conditions - genetics | beta Catenin - metabolism | Homeodomain Proteins - genetics | Mucin-2 | Tyrosine - metabolism | Antigens, Bacterial - analysis | Bacterial Proteins - metabolism | Cell Nucleus - chemistry | Cell Transformation, Neoplastic - pathology | Antigens, Bacterial - metabolism | Intestinal Mucosa - pathology
Journal Article
International Journal of Cancer, ISSN 0020-7136, 12/2012, Volume 131, Issue 11, pp. 2553 - 2561
Estrogen receptor‐beta (ERβ) has been suggested to exert anti‐inflammatory and anti‐tumorigenic effects in the colon, providing a translational potential to... 
dextran sodium sulfate | estrogen receptor‐beta | colon cancer | azoxymethane | inflammatory bowel disease | estrogen receptor-beta | Estradiol - blood | Mucin-2 - genetics | Neoplasms - metabolism | Tumor Necrosis Factor-alpha - metabolism | Inflammation - pathology | Colitis - genetics | Colorectal Neoplasms - genetics | Tumor Necrosis Factor-alpha - genetics | Colitis - pathology | NF-kappa B - metabolism | Interferon-gamma - metabolism | Inflammatory Bowel Diseases - metabolism | Estrogen Receptor beta - metabolism | Cell Differentiation - genetics | Inflammation - metabolism | Estrogen Receptor beta - genetics | Proliferating Cell Nuclear Antigen - genetics | Neoplasms - genetics | Inflammatory Bowel Diseases - pathology | Inflammatory Bowel Diseases - genetics | Female | Interferon-gamma - genetics | Interleukin-6 - metabolism | Colorectal Neoplasms - metabolism | Mucin-2 - metabolism | Mucin-1 - metabolism | Interleukin-6 - genetics | Colon - pathology | Mice, Inbred C57BL | Caveolin 1 - genetics | Interleukin-17 - genetics | beta Catenin - metabolism | Colon - metabolism | beta Catenin - genetics | Mice, Knockout | Caveolin 1 - metabolism | Interleukin-17 - metabolism | Animals | NF-kappa B - genetics | Nitric Oxide Synthase Type II - genetics | Colitis - metabolism | Inflammation - genetics | Mice | Colorectal Neoplasms - pathology | Neoplasms - pathology | Proliferating Cell Nuclear Antigen - metabolism | Mucin-1 - genetics | Nitric Oxide Synthase Type II - metabolism | Estrogen receptor-beta
Journal Article
Gastroenterology, ISSN 0016-5085, 2009, Volume 137, Issue 6, pp. 2052 - 2062
Background & Aims The winged helix transcription factors Foxa1 and Foxa2 are expressed in all epithelia of the gastrointestinal tract from its embryonic origin... 
Gastroenterology and Hepatology | RESPONSE ELEMENT | GLUCOSE-HOMEOSTASIS | GLUCAGON-LIKE PEPTIDE-1 | PROGLUCAGON GENE-TRANSCRIPTION | IN-VIVO | INSULIN-SECRETION | PANCREATIC BETA-CELLS | GASTROENTEROLOGY & HEPATOLOGY | ISLET CELLS | EXPRESSION | ENDOCRINE-CELLS | Immunohistochemistry | Hepatocyte Nuclear Factor 3-beta - genetics | Intestine, Small - pathology | Peptide YY - metabolism | Homeodomain Proteins - metabolism | Male | RNA, Messenger - metabolism | Cell Differentiation | Enteroendocrine Cells - metabolism | Proglucagon - metabolism | Somatostatin - metabolism | Repressor Proteins - metabolism | Hepatocyte Nuclear Factor 3-beta - metabolism | Mucin-2 - metabolism | Enteroendocrine Cells - pathology | Somatostatin-Secreting Cells - pathology | Glucagon-Like Peptide 1 - metabolism | Glucagon-Like Peptide 2 - metabolism | Hepatocyte Nuclear Factor 3-alpha - deficiency | Hepatocyte Nuclear Factor 3-alpha - genetics | PAX6 Transcription Factor | Goblet Cells - metabolism | Hepatocyte Nuclear Factor 3-alpha - metabolism | Mice, Knockout | Mucin-5B - metabolism | Animals | Eye Proteins - metabolism | Mucin 5AC - metabolism | LIM-Homeodomain Proteins | Mice | Transcription Factors | Goblet Cells - pathology | Intestine, Small - metabolism | Somatostatin-Secreting Cells - metabolism | Hepatocyte Nuclear Factor 3-beta - deficiency | Paired Box Transcription Factors - metabolism | Chromatin | Messenger RNA | DNA binding proteins | Cholecystokinin | Peptides | Mucins
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 4/2009, Volume 106, Issue 17, pp. 6950 - 6955
Journal Article
Cell Host and Microbe, ISSN 1931-3128, 07/2017, Volume 22, Issue 1, pp. 25 - 37.e6
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2013, Volume 8, Issue 9, p. e74963
Excessive mucin degradation by intestinal bacteria may contribute to inflammatory bowel diseases because access of luminal antigens to the intestinal immune... 
VIRULENCE FACTORS | INTERFERON-GAMMA | MACROPHAGES | MULTIDISCIPLINARY SCIENCES | ULCERATIVE-COLITIS | GASTROINTESTINAL-TRACT | MOUSE MODEL | MICROBIOTA | PERSISTENT INFECTION | INTESTINAL EPITHELIAL-CELLS | BOWEL-DISEASE | Mucin-2 - metabolism | Symbiosis | Tumor Necrosis Factor-alpha - metabolism | Interleukin-1 - metabolism | Inflammation - microbiology | Salmonella Infections - complications | Salmonella typhimurium - pathogenicity | Lymph Nodes - metabolism | Interleukin-12 - metabolism | Interferon-gamma - metabolism | RNA, Messenger - metabolism | Mice, Inbred C3H | Verrucomicrobia - physiology | Macrophages - metabolism | Animals | Intestines - microbiology | Germ-Free Life | Salmonella Infections - microbiology | Mice | Chemokine CXCL10 - metabolism | Interleukin-6 - metabolism | Salmonella | Microbiota (Symbiotic organisms) | RNA | Gastrointestinal diseases | Bacteria | Inflammation | Histochemistry | Inflammatory bowel diseases | Homeostasis | Mucus | mRNA | Infections | Lymph nodes | Degradation | Interleukin 6 | Microbiota | Histopathology | E coli | Intestine | IP-10 protein | Digestive tract | Immune system | Biodegradation | Antigens | Goblet cells | Nutrition | Cytokines | Digestive system | Cecum | Interleukin 12 | Tumor necrosis factor-α | Interleukin 17 | Sulfation | Studies | Inflammatory bowel disease | Mucin | γ-Interferon | Interferon
Journal Article
American Journal of Physiology - Gastrointestinal and Liver Physiology, ISSN 0193-1857, 09/2013, Volume 305, Issue 5, pp. G357 - G363
Journal Article
Journal Article
The Journal of Cell Biology, ISSN 0021-9525, 7/2004, Volume 166, Issue 1, pp. 37 - 47
TCF and SOX proteins belong to the high mobility group box transcription factor family. Whereas TCFs, the transcriptional effectors of the Wnt pathway, have... 
Intestines | Transcription factors | Epithelial cells | Genes | Crypts | Cell lines | Stem cells | Epithelium | Cells | Intestinal mucosa | SOX9 | CDX2 | Differentiation | Intestinal epithelium | Wnt | differentiation | AUTOSOMAL SEX REVERSAL | CAMPOMELIC DYSPLASIA | II COLLAGEN GENE | EMBRYONIC INTESTINE | BETA-CATENIN | CELL BIOLOGY | SRY-RELATED GENE | NEURAL CREST DEVELOPMENT | CHONDROCYTE-SPECIFIC ENHANCER | COLORECTAL-CANCER | intestinal epithelium | HOMEOBOX GENE | Immunohistochemistry | Neoplasm Transplantation | Homeodomain Proteins - metabolism | Humans | Gene Expression Regulation, Neoplastic | Intestines - metabolism | Stem Cells - metabolism | Colonic Neoplasms - metabolism | Cell Nucleus - metabolism | Transfection | Mucins - metabolism | Cytoskeletal Proteins - metabolism | Transcription, Genetic | Cell Differentiation | RNA - metabolism | Epithelium - metabolism | High Mobility Group Proteins - metabolism | Signal Transduction | beta Catenin | DNA - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Mucin-2 | Transcription Factors - metabolism | Blotting, Northern | Phenotype | Animals | Image Processing, Computer-Assisted | Cell Line, Tumor | Trans-Activators - metabolism | Mice | Carcinoma - metabolism | CDX2 Transcription Factor | SOX9 Transcription Factor | Microscopy, Fluorescence | Genetic transcription | Research | SOX9; Wnt; CDX2; intestinal epithelium; differentiation
Journal Article
American Journal of Physiology - Gastrointestinal and Liver Physiology, ISSN 0193-1857, 11/2009, Volume 297, Issue 5, pp. G940 - G949
Khailova L, Dvorak K, Arganbright KM, Halpern MD, Kinouchi T, Yajima M, Dvorak B. Bifidobacterium bifidum improves intestinal integrity in a rat model of... 
Adherens junctions | Tight junctions | Intestinal barrier function | Mucins | PHYSIOLOGY | intestinal barrier function | TREFOIL FACTORS | PROBIOTICS | PRETERM INFANTS | mucins | PARACELLULAR PERMEABILITY | adherens junctions | IN-VITRO | INFLAMMATORY-BOWEL-DISEASE | tight junctions | TNF-ALPHA | BACTERIAL-INFECTION | GASTROENTEROLOGY & HEPATOLOGY | COLONIC EPITHELIAL-CELLS | Mucin-2 - genetics | Ileum - pathology | Cadherins - metabolism | Enterocolitis, Necrotizing - therapy | Enterocytes - metabolism | Gene Expression - genetics | Enterocolitis, Necrotizing - metabolism | Tumor Necrosis Factor-alpha - genetics | Stress, Physiological | Ileum - metabolism | Enterocolitis, Necrotizing - microbiology | Intestines - metabolism | Adherens Junctions - metabolism | Incidence | Occludin | Catenins - metabolism | Interleukins - genetics | Trefoil Factor-3 | Asphyxia - complications | Membrane Proteins - metabolism | Neuropeptides - genetics | Enterocolitis, Necrotizing - pathology | Mucin-3 - genetics | Disease Models, Animal | Animals, Newborn | Mucin-2 - metabolism | Tight Junctions - metabolism | Cold Temperature | Bifidobacterium | Intestines - pathology | Rats | Neuropeptides - metabolism | Rats, Sprague-Dawley | Claudin-3 | Animals | Intestines - microbiology | Probiotics - therapeutic use | Junctional complexes (Epithelium) | Enterocolitis, Pseudomembranous | Enterocolitis, Neonatal necrotizing | Physiological aspects | Cell junctions | Development and progression | Models | Properties | Mediators | Inflammation | Immunity
Journal Article
世界胃肠病学杂志:英文版(电子版), ISSN 1007-9327, 2014, Volume 20, Issue 42, pp. 15715 - 15726
AIM: To investigate esophageal Helicobacter pylori(H. pylori) colonization on esophageal injury caused by reflux and the related mechanisms. METHODS: An... 
Adenocarc | Metaplasia | pylori | Helicobacter | Esophagus | Adenocarcinoma | Animal model | Helicobacter pylori | INTESTINAL METAPLASIA | CDX2 | BARRETTS-ESOPHAGUS | ESOPHAGOGASTRODUODENAL ANASTOMOSIS | RAT MODEL | GASTROESOPHAGEAL-REFLUX | GASTRIC EPITHELIAL-CELLS | INFECTION | GASTROENTEROLOGY & HEPATOLOGY | EXPRESSION | Adenocarcinoma - pathology | Cell Proliferation | Homeodomain Proteins - metabolism | Ki-67 Antigen - metabolism | Male | RNA, Messenger - metabolism | Helicobacter Infections - pathology | Esophageal Neoplasms - pathology | Esophagus - microbiology | Adenocarcinoma - metabolism | Barrett Esophagus - pathology | Helicobacter Infections - metabolism | Disease Models, Animal | Barrett Esophagus - metabolism | bcl-2-Associated X Protein - metabolism | Esophagus - metabolism | Helicobacter Infections - genetics | Rats, Sprague-Dawley | Cyclin D1 - genetics | Proto-Oncogene Proteins c-myc - genetics | Helicobacter Infections - microbiology | Mucin-2 - genetics | Cyclin D1 - metabolism | Esophageal Neoplasms - microbiology | Helicobacter pylori - pathogenicity | Helicobacter Infections - complications | Proto-Oncogene Proteins c-bcl-2 - metabolism | Esophagitis, Peptic - pathology | Esophageal Neoplasms - metabolism | Adenocarcinoma - genetics | Barrett Esophagus - microbiology | Barrett Esophagus - genetics | bcl-2-Associated X Protein - genetics | Mucin-2 - metabolism | Severity of Illness Index | Adenocarcinoma - microbiology | Gene Expression Regulation | Esophagitis, Peptic - microbiology | Esophagitis, Peptic - metabolism | Transcription Factors - genetics | Proto-Oncogene Proteins c-myc - metabolism | Homeodomain Proteins - genetics | Transcription Factors - metabolism | Esophagus - pathology | Animals | Gastroesophageal Reflux - complications | Esophageal Neoplasms - genetics | Esophagitis, Peptic - genetics | CDX2 Transcription Factor | Proto-Oncogene Proteins c-bcl-2 - genetics | Apoptosis | Original
Journal Article
Journal Article
World Neurosurgery, ISSN 1878-8750, 2016, Volume 96, pp. 85 - 90
Abstract Background Neurenteric cysts are rare central nervous system lesions derived from an endodermal origin. There is no consensus concerning pathogenesis... 
Neurosurgery | Neuroendodermal cysts | Immunohistochemical | Endodermal cysts | Neurenteric cysts | Pathogenesis | OVARY | SURGERY | MANAGEMENT | CDX2 | CEREBELLOPONTINE ANGLE | PATHOLOGY | CLINICAL NEUROLOGY | ENTITY | MUC2 | CK20 | RESPIRATORY EPITHELIAL CYST | EXPRESSION | Immunohistochemistry | Central Nervous System Diseases - pathology | Recurrence | Thyroid Nuclear Factor 1 | Neural Tube Defects - diagnostic imaging | Cysts - surgery | Humans | Middle Aged | Alkaline Phosphatase - metabolism | Neural Tube Defects - pathology | Infant | Male | Neural Tube Defects - metabolism | Central Nervous System Diseases - metabolism | Young Adult | Cysts - metabolism | Isoenzymes - metabolism | Platelet Endothelial Cell Adhesion Molecule-1 - metabolism | Adult | Female | Retrospective Studies | Child | Endoderm | Mucin-2 - metabolism | Diagnosis, Differential | Central Nervous System Diseases - diagnostic imaging | Nuclear Proteins - metabolism | Neural Tube Defects - surgery | Cysts - pathology | Chorionic Gonadotropin - metabolism | GPI-Linked Proteins - metabolism | Transcription Factors - metabolism | CDX2 Transcription Factor - metabolism | Cysts - diagnostic imaging | Keratin-7 - metabolism | Central Nervous System Diseases - surgery | Mucin 5AC - metabolism | Aged | Keratin-20 - metabolism | Keratin | Medical research | Phosphatases | Chorionic gonadotropin | Mucins | Medicine, Experimental | Cell differentiation | Index Medicus
Journal Article