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Nature, ISSN 0028-0836, 2014, Volume 505, Issue 7483, pp. 412 - 416
Journal Article
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 08/2012, Volume 47, Issue 2, pp. 178 - 185
Mucins are gel-forming proteins that are responsible for the characteristic viscoelastic properties of mucus. Mucin overproduction is a hallmark of asthma, but... 
Protein disulfide isomerase | Mucus | Airway epithelium | Asthma | Endoplasmic reticulum stress | AIRWAY MUCUS HYPERSECRETION | PROTEIN-DISULFIDE-ISOMERASE | protein disulfide isomerase | BIOCHEMISTRY & MOLECULAR BIOLOGY | airway epithelium | IL-4 RECEPTOR | asthma | ENDOPLASMIC-RETICULUM STRESS | endoplasmic reticulum stress | mucus | FAMILY | CELL BIOLOGY | MUC5AC MUCIN | EPITHELIAL-CELLS | RESPIRATORY SYSTEM | COLOCALIZATION | DISEASE | GENE-EXPRESSION | Asthma - metabolism | Mucoproteins - immunology | Bronchial Hyperreactivity - immunology | Mucin-5B - genetics | Humans | Endoplasmic Reticulum - metabolism | Th2 Cells - immunology | Bronchial Hyperreactivity - genetics | Allergens - administration & dosage | Inflammation - metabolism | Allergens - immunology | Mucin 5AC - immunology | Mucoproteins - biosynthesis | Respiratory Mucosa - immunology | Asthma - immunology | Hypersensitivity, Immediate - immunology | Bronchial Hyperreactivity - metabolism | Mucoproteins - genetics | Gene Expression | Mucin-5B - immunology | Hypersensitivity, Immediate - genetics | Mice, Transgenic | Inflammation - immunology | Th2 Cells - metabolism | Asthma - genetics | Proteins - immunology | Proteins - genetics | Mucin-5B - metabolism | Animals | Proteins - metabolism | Hypersensitivity, Immediate - metabolism | Endoplasmic Reticulum - immunology | Mucin 5AC - genetics | Cell Line, Tumor | Inflammation - genetics | Mucin 5AC - metabolism | Mice | Respiratory Mucosa - metabolism | Mucin 5AC - biosynthesis | Mucin-5B - biosynthesis | Mucoproteins - metabolism
Journal Article
NATURE IMMUNOLOGY, ISSN 1529-2908, 05/2019, Volume 20, Issue 5, pp. 571 - 571
Fine control of macrophage activation is needed to prevent inflammatory disease, particularly at barrier sites such as the lungs. However, the dominant... 
PATHWAYS | RESPONSES | CELLS | GLUCOSE-HOMEOSTASIS | PROTECTION | TH2 INDUCTION | TISSUE-RESIDENT MACROPHAGES | IMMUNOMETABOLISM | FETAL MONOCYTES | IMMUNOLOGY | PROMOTES | Macrophage Activation - genetics | Macrophages, Alveolar - parasitology | Mucin-5B - genetics | Strongylida Infections - genetics | Nippostrongylus - immunology | Pulmonary Surfactant-Associated Protein D - immunology | Inflammation - metabolism | Larva - immunology | Strongylida Infections - parasitology | Strongylida Infections - immunology | Lung - metabolism | Interleukin-4 - genetics | Macrophages, Alveolar - immunology | Lung - pathology | Mucin-5B - immunology | Macrophage Activation - immunology | Interleukin-4 - metabolism | Mice, Inbred C57BL | Mice, Transgenic | Inflammation - immunology | Pulmonary Surfactant-Associated Protein D - metabolism | Pulmonary Surfactant-Associated Protein D - genetics | Mice, Knockout | Interleukin-4 - immunology | Mucin-5B - metabolism | Animals | Nippostrongylus - physiology | Inflammation - genetics | Mice, Inbred BALB C | Larva - physiology | Macrophages, Alveolar - metabolism | Lung - immunology | Immune response | Lung diseases | Physiological aspects | Cell metabolism | Development and progression | Inflammation | Macrophages | Health aspects | Protein D | Activation | Allergic diseases | Metabolism | Disease control | Inflammatory diseases | Proteins | Microbiota | Interleukin 4 | Cell activation | Surfactant protein D | Mucin | Lungs | Parasitic diseases | Glycolysis | Extracellular matrix | Alveoli | Niches | Peritoneum
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2015, Volume 135, Issue 3, pp. 799 - 810.e7
Background Balanced activation and inhibition of the immune system ensures pathogen clearance while avoiding hyperinflammation. Siglecs, sialic acid–binding... 
Allergy and Immunology | Chronic rhinosinusitis | nuclear factor κB | inflammation | submucosal gland cells | siglecs | glycobiology | mucin 5B | F LIGANDS | ACTIVATION | PROTEIN | RECOGNITION | nuclear factor kappa B | GLYCANS | IMMUNOLOGY | ALLERGY | DEFICIENT | SECRETION | RECEPTORS | Antigens, CD - immunology | RNA, Small Interfering - genetics | Respiratory Mucosa - drug effects | Mucin-5B - genetics | Epithelial Cells - drug effects | Humans | Middle Aged | NF-kappa B - immunology | Male | Antigens, CD - genetics | Lipopolysaccharides | Respiratory Mucosa - pathology | Sialic Acid Binding Immunoglobulin-like Lectins - immunology | Sinusitis - genetics | Sinusitis - immunology | Respiratory Mucosa - immunology | Antigens, Differentiation, B-Lymphocyte - immunology | Sinusitis - pathology | Adult | Female | Mucin-5B - antagonists & inhibitors | Antigens, Differentiation, B-Lymphocyte - genetics | Cell Line | Rhinitis - immunology | Lectins - immunology | Mucin-5B - immunology | Signal Transduction | Gene Expression Regulation | Epithelial Cells - pathology | Tumor Necrosis Factor-alpha | Sialic Acid Binding Immunoglobulin-like Lectins - genetics | Rhinitis - genetics | NF-kappa B - genetics | Epithelial Cells - immunology | Lectins - genetics | Ligands | Primary Cell Culture | Rhinitis - pathology | Chronic Disease | RNA, Small Interfering - metabolism | Binding proteins | Organic acids | Protein binding | Proteins | Sinuses | Plasmids | Laboratories | Otolaryngology | Phosphatase | Immune system | NF-κB | chronic rhinosinusitis | MUC5B
Journal Article
The Journal of Immunology, ISSN 0022-1767, 11/2009, Volume 183, Issue 10, pp. 6698 - 6707
Journal Article
Journal of Immunology, ISSN 0022-1767, 08/2017, Volume 199, Issue 3, pp. 1170 - 1183
Secondhand smoke (SHS) exposure has been linked to the worsening of ongoing lung diseases. However, whether SHS exposure affects the manifestation and natural... 
CHRONIC-BRONCHITIS | ALLERGIC INFLAMMATION | SURFACE DEHYDRATION | INDUCED PULMONARY INFLAMMATION | IN-UTERO EXPOSURE | CHANNEL-OVEREXPRESSING MICE | TOBACCO-SMOKE | INNATE LYMPHOID-CELLS | IMMUNOLOGY | NEUTROPHIL RECRUITMENT | 2ND-HAND SMOKE | Epithelial Sodium Channels - deficiency | Lung Diseases, Obstructive - immunology | Cystic Fibrosis - physiopathology | Humans | Mucin-5B | Th2 Cells - immunology | Epithelial Sodium Channels - metabolism | Lung - cytology | Interleukin-33 - immunology | Cystic Fibrosis - immunology | Epithelial Sodium Channels - genetics | Bacterial Infections - immunology | Female | Mucus - metabolism | Th2 Cells - pathology | Disease Models, Animal | Neutrophils - pathology | Animals, Newborn | Lung - pathology | Signal Transduction | Cystic Fibrosis - microbiology | Down-Regulation | Neutrophils - immunology | Neutrophils - physiology | Epithelial Cells - pathology | Mice, Transgenic | Bacterial Load | Bacterial Infections - physiopathology | Lung Diseases, Obstructive - physiopathology | Interleukin-33 - genetics | Animals | Tobacco Smoke Pollution - adverse effects | Epithelial Cells - immunology | Immunoglobulin A - immunology | Interleukin-33 - metabolism | Mice | Goblet Cells - pathology | Lung - immunology | Cell Movement | Neonates | Epithelial cells | Hyperplasia | Immune clearance | Mucus | Lymphocytes T | Infections | Smoke | Immunoglobulin A | Recruitment | Consolidation | Rodents | Passive smoking | Bacteria | Respiratory tract diseases | Alveoli | Sodium channels | Lymphoid cells | Immune response | Bacterial infections | Secretion | Lung diseases | Neutrophils | Transgenic mice | Cystic fibrosis | Exposure | Secretions | Metaplasia | Self-propagating synthesis | Obstructive lung disease | Fibrosis | Smoking
Journal Article
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 06/2011, Volume 15, Issue 6, pp. 1355 - 1363
Airway inflammation and mucus hyperproduction play the central role in the development of asthma, although the mechanisms remain unclear. The aquaporin (AQP)‐5... 
aquaporins | chronic allergy | house dust mite | inflammation | mucin | House dust mite | Inflammation | Mucin | Aquaporins | Chronic allergy | MEDICINE, RESEARCH & EXPERIMENTAL | DENDRITIC CELLS | LACRIMAL GLANDS | AQP5 | WATER PERMEABILITY | HYPERRESPONSIVENESS | MUC5AC | CELL BIOLOGY | LUNG EPITHELIAL-CELLS | SECRETION | DECREASED EXPRESSION | MOUSE LUNG | Respiratory System - pathology | Asthma - metabolism | Bronchoalveolar Lavage Fluid - immunology | Hypersensitivity - immunology | Aquaporin 5 - deficiency | Th2 Cells - immunology | Aquaporin 5 - genetics | Inflammation - metabolism | Hypersensitivity - metabolism | Pyroglyphidae - metabolism | Gene Deletion | Asthma - immunology | Mucus - metabolism | Respiratory System - immunology | Cytokines - immunology | Cytokines - metabolism | Mice, Inbred C57BL | Mucus - secretion | Antigens - metabolism | Inflammation - immunology | Th2 Cells - metabolism | Mice, Knockout | Animals | Respiratory System - metabolism | Antigens - immunology | Mice | Mucus - immunology | Bronchoalveolar Lavage Fluid - chemistry | Mucin 5AC - biosynthesis | Mucin-5B - biosynthesis | Pyroglyphidae - immunology | Aquaporin 5 | Glands | Lung cancer | Interleukin | Mucus | Lymphocytes T | Kinases | Respiratory tract | Proteins | Bronchoalveolar lavage | Respiratory tract diseases | Alveoli | Antigens | Edema | Cytokines | Hypersensitivity | House dust | Bronchus | Gene expression | Epithelium | Asthma | Studies | Hospitals | Lungs | Interferon
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2014, Volume 9, Issue 11, p. e112469
The human intestinal parasite Schistosoma mansoni causes a chronic disease, schistosomiasis or bilharzia. According to the current literature, the parasite... 
PNEUMONIA VIRUS | HELMINTH INFECTION | INFLUENZA-A VIRUS | DENDRITIC CELLS | CYTOKINE RESPONSES | MULTIDISCIPLINARY SCIENCES | IN-VIVO | CD4(+) T-CELLS | NECROSIS-FACTOR-ALPHA | INNATE LYMPHOID-CELLS | TNF-ALPHA | Lung - pathology | Schistosomiasis mansoni - immunology | Coinfection - immunology | Orthomyxoviridae Infections - pathology | Bronchoalveolar Lavage | Schistosoma mansoni - immunology | Murine pneumonia virus - immunology | Mucin-5B - metabolism | Animals | Flow Cytometry | Etanercept | T-Lymphocytes, Helper-Inducer - immunology | Statistics, Nonparametric | Mucin 5AC - metabolism | Influenza A virus - immunology | Mice | Orthomyxoviridae Infections - immunology | Cytokines - blood | Antiviral agents | Health aspects | Analysis | Disease | Pathogenesis | Hyperplasia | Liver | Helper cells | Viruses | Mucus | Lymphocytes T | Infections | Immunity | Eggs | Tropical diseases | Defects | Mating | Respiratory tract | Schistosomiasis | Interleukin 5 | Interleukin 2 | Intestine | Tumor necrosis factor-TNF | Respiratory tract diseases | Chronic obstructive pulmonary disease | Immune system | Immune response | Cytokines | Granulocyte-macrophage colony-stimulating factor | Secretion | Dermatology | Inflammation | Interleukin 13 | Tumor necrosis factor-α | Interleukin 17 | Virology | Serum levels | Lungs | γ-Interferon | Influenza | Epigenetics | Interferon | Oviposition | Human behavior | Hygiene | Viral infections | Biodiversity and Ecology | Environmental Sciences
Journal Article
Journal Article
American Journal of Physiology - Lung Cellular and Molecular Physiology, ISSN 1040-0605, 11/2014, Volume 307, Issue 9, pp. L681 - L691
Molecular biomarkers are highly desired in idiopathic pulmonary fibrosis (IPF), where they hold the potential to elucidate underlying disease mechanisms,... 
Biomarker | Pulmonary fibrosis | Diagnosis | Prediction | SURROGATE END-POINTS | PHYSIOLOGY | MUC5B PROMOTER POLYMORPHISM | TGF-BETA | ALVEOLAR EPITHELIAL-CELLS | GENE-EXPRESSION PROFILES | diagnosis | ENDOPLASMIC-RETICULUM STRESS | biomarker | NONSPECIFIC INTERSTITIAL PNEUMONIA | SURFACTANT-PROTEIN-C | RESPIRATORY SYSTEM | pulmonary fibrosis | prediction | REGULATORY T-CELLS | LUNG FIBROBLASTS | Pulmonary Surfactant-Associated Proteins - genetics | Epithelial Cells - metabolism | Mucin-5B - genetics | Humans | Blood Proteins - metabolism | Idiopathic Pulmonary Fibrosis - metabolism | T-Lymphocytes, Regulatory - pathology | T-Lymphocytes, Regulatory - immunology | Telomerase - genetics | Blood Proteins - genetics | Respiratory Mucosa - immunology | Respiratory Mucosa - physiopathology | Telomerase - metabolism | Lung - metabolism | Toll-Like Receptor 3 - genetics | Biomarkers - metabolism | Pulmonary Surfactant-Associated Proteins - metabolism | Gene Expression | Epithelial Cells - pathology | Lung - physiopathology | Idiopathic Pulmonary Fibrosis - immunology | Immunity, Innate | Toll-Like Receptor 3 - metabolism | Mucin-5B - metabolism | Idiopathic Pulmonary Fibrosis - diagnosis | Epithelial Cells - immunology | Idiopathic Pulmonary Fibrosis - physiopathology | Mutation | Respiratory Mucosa - metabolism | Lung - immunology | Physiological aspects | Call for Papers
Journal Article
Journal of Reproductive Immunology, ISSN 0165-0378, 2011, Volume 92, Issue 1, pp. 8 - 20
Journal Article