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Gastroenterology (New York, N.Y. 1943), ISSN 0016-5085, 2008, Volume 134, Issue 2, pp. 511 - 522
Background & Aims: Loss of gastric parietal cells is a critical precursor to gastric metaplasia and neoplasia. However, the origin of metaplasia remains... 
Gastroenterology and Hepatology | OXYNTIC ATROPHY | LOCALIZATION | COMPLEX | ZYMOGENIC CELLS | ADENOCARCINOMA | RISK | INFECTION | DEFICIENT MICE | TISSUES | GASTROENTEROLOGY & HEPATOLOGY | CANCER | Metaplasia - metabolism | Humans | Parietal Cells, Gastric - metabolism | Stomach Neoplasms - metabolism | Gastric Mucosa - pathology | Precancerous Conditions - metabolism | Stomach Neoplasms - pathology | beta-Defensins | Fetal Proteins - metabolism | Gastric Mucosa - metabolism | Stomach Neoplasms - physiopathology | Minichromosome Maintenance Complex Component 3 | Atrophy | DNA-Binding Proteins - metabolism | Peptides - metabolism | Basic Helix-Loop-Helix Transcription Factors - metabolism | Mucins - metabolism | Muscle Proteins - metabolism | Precancerous Conditions - pathology | Trefoil Factor-2 | Gastric Fundus - metabolism | Chief Cells, Gastric - metabolism | Mice, Inbred C57BL | Cell Cycle Proteins - metabolism | Microtubule-Associated Proteins | Nuclear Proteins - metabolism | Gastrins - metabolism | Cell Transformation, Neoplastic - metabolism | Metaplasia - pathology | Mice, Knockout | Animals | Carrier Proteins - metabolism | Parietal Cells, Gastric - pathology | Epididymal Secretory Proteins - metabolism | Chief Cells, Gastric - pathology | Mice | Cell Transformation, Neoplastic - pathology | Gastric Fundus - pathology | Intrinsic Factor - metabolism | Vasoactive intestinal peptides | Analysis | Polypeptides
Journal Article
Gastroenterology (New York, N.Y. 1943), ISSN 0016-5085, 2009, Volume 137, Issue 6, pp. 2052 - 2062
Background & Aims The winged helix transcription factors Foxa1 and Foxa2 are expressed in all epithelia of the gastrointestinal tract from its embryonic origin... 
Gastroenterology and Hepatology | RESPONSE ELEMENT | GLUCOSE-HOMEOSTASIS | GLUCAGON-LIKE PEPTIDE-1 | PROGLUCAGON GENE-TRANSCRIPTION | IN-VIVO | INSULIN-SECRETION | PANCREATIC BETA-CELLS | GASTROENTEROLOGY & HEPATOLOGY | ISLET CELLS | EXPRESSION | ENDOCRINE-CELLS | Immunohistochemistry | Hepatocyte Nuclear Factor 3-beta - genetics | Intestine, Small - pathology | Peptide YY - metabolism | Homeodomain Proteins - metabolism | Male | RNA, Messenger - metabolism | Cell Differentiation | Enteroendocrine Cells - metabolism | Proglucagon - metabolism | Somatostatin - metabolism | Repressor Proteins - metabolism | Hepatocyte Nuclear Factor 3-beta - metabolism | Mucin-2 - metabolism | Enteroendocrine Cells - pathology | Somatostatin-Secreting Cells - pathology | Glucagon-Like Peptide 1 - metabolism | Glucagon-Like Peptide 2 - metabolism | Hepatocyte Nuclear Factor 3-alpha - deficiency | Hepatocyte Nuclear Factor 3-alpha - genetics | PAX6 Transcription Factor | Goblet Cells - metabolism | Hepatocyte Nuclear Factor 3-alpha - metabolism | Mice, Knockout | Mucin-5B - metabolism | Animals | Eye Proteins - metabolism | Mucin 5AC - metabolism | LIM-Homeodomain Proteins | Mice | Transcription Factors | Goblet Cells - pathology | Intestine, Small - metabolism | Somatostatin-Secreting Cells - metabolism | Hepatocyte Nuclear Factor 3-beta - deficiency | Paired Box Transcription Factors - metabolism | Chromatin | Messenger RNA | DNA binding proteins | Cholecystokinin | Peptides | Mucins
Journal Article
Journal Article
Journal of allergy and clinical immunology, ISSN 0091-6749, 2013, Volume 131, Issue 2, pp. 501 - 511.e1
Background Sphingosine-1-phosphate (S1P), which is produced by 2 sphingosine kinase (SphK) isoenzymes, SphK1 and SphK2, has been implicated in IgE-mediated... 
Allergy and Immunology | airway hyperresponsiveness | asthma | nuclear factor κB | mast cells | sphingosine kinase | Sphingosine-1-phosphate | IMMUNITY | FC-EPSILON-RI | nuclear factor kappa B | FACTOR-KAPPA-B | IMMUNOLOGY | MULTIPLE FEATURES | REGULATES MUCIN PRODUCTION | RESPONSES | CYTOKINE PRODUCTION | ALLERGY | GENE-EXPRESSION | MICE | Tumor Necrosis Factor-alpha - metabolism | Lysophospholipids - metabolism | Asthma - metabolism | Bronchial Hyperreactivity - drug therapy | Humans | Goblet Cells - drug effects | NF-kappa B - metabolism | Amino Alcohols - pharmacology | Interferon-gamma - metabolism | Interleukins - metabolism | Inflammation - metabolism | Inflammation - drug therapy | Methacholine Chloride - pharmacology | Mast Cells - metabolism | Bronchial Hyperreactivity - metabolism | Female | Chemokine CCL2 - metabolism | Hyperplasia - drug therapy | Lung - metabolism | Sphingosine - metabolism | Hyperplasia - metabolism | Asthma - enzymology | Immunoglobulin E - metabolism | Mice, Inbred C57BL | Cells, Cultured | Ovalbumin - pharmacology | Asthma - drug therapy | Goblet Cells - metabolism | Mast Cells - drug effects | Asthma - chemically induced | Phosphotransferases (Alcohol Group Acceptor) - metabolism | Bronchial Hyperreactivity - enzymology | Phosphotransferases (Alcohol Group Acceptor) - antagonists & inhibitors | Bronchial Hyperreactivity - pathology | Sphingosine - analogs & derivatives | Animals | Lung - drug effects | Mice | Bronchoalveolar Lavage Fluid - chemistry | Phosphates | Medical colleges | Isoenzymes | Analysis | Sphingosine | Inflammation | Asthma | Studies | Cytokines | Lungs | Rodents | Kinases | Laboratory animals | Allergies
Journal Article
Nature medicine, ISSN 1546-170X, 2002, Volume 8, Issue 10, pp. 1089 - 1097
The intracellular signaling mechanisms that specify tissue-specific responses to the interleukin-6 (IL-6) family of cytokines are not well understood. Here, we... 
MEDICINE, RESEARCH & EXPERIMENTAL | HEPATIC CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | CELL BIOLOGY | INTESTINAL INFLAMMATION | RESPONSES | INTERLEUKIN-6 FAMILY | COLITIS | IN-VIVO | DISEASE | CYTOKINE RECEPTORS | TRANSGENIC MICE | SIGNAL TRANSDUCER | Protein Tyrosine Phosphatase, Non-Receptor Type 11 | Membrane Glycoproteins - metabolism | Transcriptional Activation | Peptides - genetics | Neuropeptides | Protein Tyrosine Phosphatases - metabolism | Wound Healing | Antigens, CD - genetics | Antigens, CD - metabolism | Growth Substances - genetics | Peptides - metabolism | Digestive System Physiological Phenomena | Protein Phosphatase 2 | Interleukin-6 - metabolism | Cytokine Receptor gp130 | Colon - pathology | Interleukin-11 - metabolism | Mice, Transgenic | Spleen - cytology | Colon - metabolism | STAT1 Transcription Factor | Mice | Intestinal Mucosa - pathology | Intestinal Mucosa - metabolism | Homeostasis | Stomach Neoplasms - metabolism | Stomach Neoplasms - pathology | Stomach - metabolism | Adenoma - metabolism | DNA-Binding Proteins - metabolism | Muscle Proteins | Trefoil Factor-3 | Trefoil Factor-2 | Cell Line | Stomach - pathology | Gene Expression Regulation | Intracellular Signaling Peptides and Proteins | Membrane Glycoproteins - genetics | Proteins - genetics | Animals | Proteins - metabolism | Spleen - metabolism | Mucins | Stomach - cytology | Adenoma - pathology | Signal Transduction - physiology | Trans-Activators - metabolism | Growth Substances - metabolism | Mitogen-Activated Protein Kinases - metabolism
Journal Article
Cancer cell, ISSN 1535-6108, 2006, Volume 9, Issue 4, pp. 261 - 272
Journal Article
Placenta (Eastbourne), ISSN 0143-4004, 2008, Volume 29, Issue 3, pp. 274 - 281
Abstract Obesity and pregnancy are associated with a combination of insulin resistance and inflammatory changes which exacerbate in combination. Based on the... 
Internal Medicine | Obstetrics and Gynecology | Pregnancy | Obesity | Placenta | Cytokines | Fetus | Inflammation | Macrophages | MATERNAL OBESITY | HOFBAUER CELLS | pregnancy | LONGITUDINAL CHANGES | GESTATIONAL DIABETES-MELLITUS | NECROSIS-FACTOR-ALPHA | DEVELOPMENTAL BIOLOGY | BODY-COMPOSITION | OBSTETRICS & GYNECOLOGY | macrophages | REPRODUCTIVE BIOLOGY | inflammation | INSULIN-RESISTANCE | placenta | cytokines | TNF-ALPHA | obesity | fetus | GLUCOSE-TOLERANCE | ADIPOSE-TISSUE | Tumor Necrosis Factor-alpha - metabolism | Inflammation - pathology | Leukocytes, Mononuclear - metabolism | Pregnancy Complications - blood | Membrane Glycoproteins - metabolism | Receptors, G-Protein-Coupled - metabolism | Obesity - immunology | Cell Count | Humans | Tumor Necrosis Factor-alpha - genetics | Antigens, CD - genetics | Obesity - blood | Antigens, CD - metabolism | Lipopolysaccharide Receptors - metabolism | Inflammation - complications | Placenta - cytology | Mucins - metabolism | Adult | Female | Pregnancy Complications - metabolism | Interleukin-6 - metabolism | Interleukin-6 - genetics | Placenta - immunology | Obesity - complications | Cells, Cultured | Macrophages - cytology | Fetal Blood - metabolism | Antigens, Differentiation, Myelomonocytic - genetics | Membrane Glycoproteins - genetics | Obesity - metabolism | Macrophages - metabolism | Antigens, Differentiation, Myelomonocytic - metabolism | Placenta - pathology | Lipopolysaccharide Receptors - genetics | Receptors, G-Protein-Coupled - genetics | Mucins - genetics | Pregnancy Complications - immunology | Pregnant women | programming
Journal Article
Cell host & microbe, ISSN 1931-3128, 2017, Volume 22, Issue 1, pp. 25 - 37.e6
Journal Article
Archives of pathology & laboratory medicine (1976), ISSN 0003-9985, 2014, Volume 138, Issue 8, pp. 1015 - 1026
Journal Article
Digestive and Liver Disease, ISSN 1590-8658, 2015, Volume 48, Issue 3, pp. 223 - 230
Journal Article