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Gastroenterology, ISSN 0016-5085, 2008, Volume 134, Issue 2, pp. 511 - 522
Background & Aims: Loss of gastric parietal cells is a critical precursor to gastric metaplasia and neoplasia. However, the origin of metaplasia remains... 
Gastroenterology and Hepatology | OXYNTIC ATROPHY | LOCALIZATION | COMPLEX | ZYMOGENIC CELLS | ADENOCARCINOMA | RISK | INFECTION | DEFICIENT MICE | TISSUES | GASTROENTEROLOGY & HEPATOLOGY | CANCER | Metaplasia - metabolism | Humans | Parietal Cells, Gastric - metabolism | Stomach Neoplasms - metabolism | Gastric Mucosa - pathology | Precancerous Conditions - metabolism | Stomach Neoplasms - pathology | beta-Defensins | Fetal Proteins - metabolism | Gastric Mucosa - metabolism | Stomach Neoplasms - physiopathology | Minichromosome Maintenance Complex Component 3 | Atrophy | DNA-Binding Proteins - metabolism | Peptides - metabolism | Basic Helix-Loop-Helix Transcription Factors - metabolism | Mucins - metabolism | Muscle Proteins - metabolism | Precancerous Conditions - pathology | Trefoil Factor-2 | Gastric Fundus - metabolism | Chief Cells, Gastric - metabolism | Mice, Inbred C57BL | Cell Cycle Proteins - metabolism | Microtubule-Associated Proteins | Nuclear Proteins - metabolism | Gastrins - metabolism | Cell Transformation, Neoplastic - metabolism | Metaplasia - pathology | Mice, Knockout | Animals | Carrier Proteins - metabolism | Parietal Cells, Gastric - pathology | Epididymal Secretory Proteins - metabolism | Chief Cells, Gastric - pathology | Mice | Cell Transformation, Neoplastic - pathology | Gastric Fundus - pathology | Intrinsic Factor - metabolism | Vasoactive intestinal peptides | Analysis | Polypeptides | Index Medicus | Abridged Index Medicus
Journal Article
Gastroenterology, ISSN 0016-5085, 2009, Volume 137, Issue 6, pp. 2052 - 2062
Background & Aims The winged helix transcription factors Foxa1 and Foxa2 are expressed in all epithelia of the gastrointestinal tract from its embryonic origin... 
Gastroenterology and Hepatology | RESPONSE ELEMENT | GLUCOSE-HOMEOSTASIS | GLUCAGON-LIKE PEPTIDE-1 | PROGLUCAGON GENE-TRANSCRIPTION | IN-VIVO | INSULIN-SECRETION | PANCREATIC BETA-CELLS | GASTROENTEROLOGY & HEPATOLOGY | ISLET CELLS | EXPRESSION | ENDOCRINE-CELLS | Immunohistochemistry | Hepatocyte Nuclear Factor 3-beta - genetics | Intestine, Small - pathology | Peptide YY - metabolism | Homeodomain Proteins - metabolism | Male | RNA, Messenger - metabolism | Cell Differentiation | Enteroendocrine Cells - metabolism | Proglucagon - metabolism | Somatostatin - metabolism | Repressor Proteins - metabolism | Hepatocyte Nuclear Factor 3-beta - metabolism | Mucin-2 - metabolism | Enteroendocrine Cells - pathology | Somatostatin-Secreting Cells - pathology | Glucagon-Like Peptide 1 - metabolism | Glucagon-Like Peptide 2 - metabolism | Hepatocyte Nuclear Factor 3-alpha - deficiency | Hepatocyte Nuclear Factor 3-alpha - genetics | PAX6 Transcription Factor | Goblet Cells - metabolism | Hepatocyte Nuclear Factor 3-alpha - metabolism | Mice, Knockout | Mucin-5B - metabolism | Animals | Eye Proteins - metabolism | Mucin 5AC - metabolism | LIM-Homeodomain Proteins | Mice | Transcription Factors | Goblet Cells - pathology | Intestine, Small - metabolism | Somatostatin-Secreting Cells - metabolism | Hepatocyte Nuclear Factor 3-beta - deficiency | Paired Box Transcription Factors - metabolism | Chromatin | Messenger RNA | DNA binding proteins | Cholecystokinin | Peptides | Mucins | Index Medicus | Abridged Index Medicus
Journal Article
Oncogene, ISSN 0950-9232, 07/2007, Volume 26, Issue 32, pp. 4617 - 4626
Infection with Helicobacter pylori cagA-positive strains is associated with gastric adenocarcinoma. Intestinal metaplasia is a precancerous lesion of the... 
β-catenin | Gastric adenocarcinoma | Helicobacter pylori CagA | E-cadherin | Intestinal metaplasia | beta-catenin | TARGET | ACTIVATION | PROTEIN | CDX1 | BIOCHEMISTRY & MOLECULAR BIOLOGY | CANCER | CELL BIOLOGY | gastric adenocarcinoma | GENE | ONCOLOGY | intestinal metaplasia | WNT PATHWAY | ATROPHIC GASTRITIS | GENETICS & HEREDITY | INFECTION | EXPRESSION | Intestinal Mucosa - metabolism | Phosphorylation | Adenocarcinoma - pathology | Cadherins - metabolism | beta Catenin - analysis | Adenocarcinoma - etiology | Homeodomain Proteins - metabolism | Humans | Transcriptional Activation | Gene Expression Regulation, Neoplastic | Stomach Neoplasms - metabolism | Cytoplasm - metabolism | Gastric Mucosa - pathology | Precancerous Conditions - metabolism | Stomach Neoplasms - pathology | Bacterial Proteins - analysis | Gastric Mucosa - metabolism | Adenocarcinoma - metabolism | Cell Nucleus - metabolism | Cyclin-Dependent Kinase Inhibitor p21 - genetics | Cell Transformation, Neoplastic - genetics | Mucins - metabolism | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Precancerous Conditions - pathology | Stomach Neoplasms - etiology | Cytoplasm - chemistry | Cell Line | Cadherins - analysis | Cell Transformation, Neoplastic - metabolism | Precancerous Conditions - genetics | beta Catenin - metabolism | Homeodomain Proteins - genetics | Mucin-2 | Tyrosine - metabolism | Antigens, Bacterial - analysis | Bacterial Proteins - metabolism | Cell Nucleus - chemistry | Cell Transformation, Neoplastic - pathology | Antigens, Bacterial - metabolism | Intestinal Mucosa - pathology | Index Medicus
Journal Article
Nature Medicine, ISSN 1078-8956, 10/2002, Volume 8, Issue 10, pp. 1089 - 1097
The intracellular signaling mechanisms that specify tissue-specific responses to the interleukin-6 (IL-6) family of cytokines are not well understood. Here, we... 
MEDICINE, RESEARCH & EXPERIMENTAL | HEPATIC CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | CELL BIOLOGY | INTESTINAL INFLAMMATION | RESPONSES | INTERLEUKIN-6 FAMILY | COLITIS | IN-VIVO | DISEASE | CYTOKINE RECEPTORS | TRANSGENIC MICE | SIGNAL TRANSDUCER | Protein Tyrosine Phosphatase, Non-Receptor Type 11 | Membrane Glycoproteins - metabolism | Transcriptional Activation | Peptides - genetics | Neuropeptides | Protein Tyrosine Phosphatases - metabolism | Wound Healing | Antigens, CD - genetics | Antigens, CD - metabolism | Growth Substances - genetics | Peptides - metabolism | Digestive System Physiological Phenomena | Protein Phosphatase 2 | Interleukin-6 - metabolism | Cytokine Receptor gp130 | Colon - pathology | Interleukin-11 - metabolism | Mice, Transgenic | Spleen - cytology | Colon - metabolism | STAT1 Transcription Factor | Mice | Intestinal Mucosa - pathology | Intestinal Mucosa - metabolism | Homeostasis | Stomach Neoplasms - metabolism | Stomach Neoplasms - pathology | Stomach - metabolism | Adenoma - metabolism | DNA-Binding Proteins - metabolism | Muscle Proteins | Trefoil Factor-3 | Trefoil Factor-2 | Cell Line | Stomach - pathology | Gene Expression Regulation | Intracellular Signaling Peptides and Proteins | Membrane Glycoproteins - genetics | Proteins - genetics | Animals | Proteins - metabolism | Spleen - metabolism | Mucins | Stomach - cytology | Adenoma - pathology | Signal Transduction - physiology | Trans-Activators - metabolism | Growth Substances - metabolism | Mitogen-Activated Protein Kinases - metabolism | Index Medicus
Journal Article
Journal of Thoracic Oncology, ISSN 1556-0864, 04/2010, Volume 5, Issue 4, pp. 442 - 447
The emergence of treatments for non-small cell lung carcinoma (NSCLC) with differential efficacy and toxicity between subtypes has highlighted the importance... 
Immunohistochemistry | NSCLC subtype | Non-small cell lung carcinoma | NSCLC-NOS | Bronchial biopsy diagnosis | Predictive value | Undifferentiated carcinoma | TTF1 | p63 | P63 | DIFFERENTIAL-DIAGNOSIS | PRIMARY LUNG-CANCER | TTF-1 | OBSERVER VARIABILITY | HISTOLOGICAL CLASSIFICATION | PULMONARY ADENOCARCINOMAS | ONCOLOGY | RESPIRATORY SYSTEM | TISSUE MICROARRAY | TRANSCRIPTION FACTOR-I | EXPRESSION | Thyroid Nuclear Factor 1 | Adenocarcinoma - pathology | Prognosis | Prospective Studies | Adenocarcinoma - classification | Carcinoma, Large Cell - pathology | Carcinoma, Squamous Cell - metabolism | Carcinoma, Squamous Cell - pathology | Humans | Lung Neoplasms - metabolism | Lung Neoplasms - pathology | Immunoenzyme Techniques | Adenocarcinoma - metabolism | Carcinoma, Non-Small-Cell Lung - classification | Sensitivity and Specificity | Mucins - metabolism | Biomarkers, Tumor - metabolism | Bronchi - metabolism | Cell Differentiation | Bronchi - pathology | Carcinoma, Non-Small-Cell Lung - pathology | S100 Calcium Binding Protein A7 | Tumor Suppressor Proteins - metabolism | Carcinoma, Large Cell - metabolism | Carcinoma, Non-Small-Cell Lung - metabolism | Nuclear Proteins - metabolism | S100 Proteins - metabolism | Survival Rate | Transcription Factors - metabolism | Biopsy, Fine-Needle | Lung Neoplasms - classification | Carcinoma, Squamous Cell - classification | Keratin-5 - metabolism | Carcinoma, Large Cell - classification | Trans-Activators - metabolism | Keratin-6 - metabolism | Neoplasm Staging | Index Medicus
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2012, Volume 131, Issue 2, pp. 501 - 511.e1
Background Sphingosine-1-phosphate (S1P), which is produced by 2 sphingosine kinase (SphK) isoenzymes, SphK1 and SphK2, has been implicated in IgE-mediated... 
Allergy and Immunology | airway hyperresponsiveness | asthma | nuclear factor κB | mast cells | sphingosine kinase | Sphingosine-1-phosphate | IMMUNITY | FC-EPSILON-RI | nuclear factor kappa B | FACTOR-KAPPA-B | IMMUNOLOGY | MULTIPLE FEATURES | REGULATES MUCIN PRODUCTION | RESPONSES | CYTOKINE PRODUCTION | ALLERGY | GENE-EXPRESSION | MICE | Tumor Necrosis Factor-alpha - metabolism | Lysophospholipids - metabolism | Asthma - metabolism | Bronchial Hyperreactivity - drug therapy | Humans | Goblet Cells - drug effects | NF-kappa B - metabolism | Amino Alcohols - pharmacology | Interferon-gamma - metabolism | Interleukins - metabolism | Inflammation - metabolism | Inflammation - drug therapy | Methacholine Chloride - pharmacology | Mast Cells - metabolism | Bronchial Hyperreactivity - metabolism | Female | Chemokine CCL2 - metabolism | Hyperplasia - drug therapy | Lung - metabolism | Sphingosine - metabolism | Hyperplasia - metabolism | Asthma - enzymology | Immunoglobulin E - metabolism | Mice, Inbred C57BL | Cells, Cultured | Ovalbumin - pharmacology | Asthma - drug therapy | Goblet Cells - metabolism | Mast Cells - drug effects | Asthma - chemically induced | Phosphotransferases (Alcohol Group Acceptor) - metabolism | Bronchial Hyperreactivity - enzymology | Phosphotransferases (Alcohol Group Acceptor) - antagonists & inhibitors | Bronchial Hyperreactivity - pathology | Sphingosine - analogs & derivatives | Animals | Lung - drug effects | Mice | Bronchoalveolar Lavage Fluid - chemistry | Phosphates | Medical colleges | Isoenzymes | Analysis | Sphingosine | Inflammation | Asthma | Studies | Cytokines | Lungs | Rodents | Kinases | Laboratory animals | Allergies | Index Medicus | Abridged Index Medicus
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 12/2012, Volume 122, Issue 12, pp. 4667 - 4674
Journal Article
Cancer Cell, ISSN 1535-6108, 2006, Volume 9, Issue 4, pp. 261 - 272
Journal Article
Placenta, ISSN 0143-4004, 2008, Volume 29, Issue 3, pp. 274 - 281
Abstract Obesity and pregnancy are associated with a combination of insulin resistance and inflammatory changes which exacerbate in combination. Based on the... 
Internal Medicine | Obstetrics and Gynecology | Pregnancy | Obesity | Placenta | Cytokines | Fetus | Inflammation | Macrophages | MATERNAL OBESITY | HOFBAUER CELLS | pregnancy | LONGITUDINAL CHANGES | GESTATIONAL DIABETES-MELLITUS | NECROSIS-FACTOR-ALPHA | DEVELOPMENTAL BIOLOGY | BODY-COMPOSITION | OBSTETRICS & GYNECOLOGY | macrophages | REPRODUCTIVE BIOLOGY | inflammation | INSULIN-RESISTANCE | placenta | cytokines | TNF-ALPHA | obesity | fetus | GLUCOSE-TOLERANCE | ADIPOSE-TISSUE | Tumor Necrosis Factor-alpha - metabolism | Inflammation - pathology | Leukocytes, Mononuclear - metabolism | Pregnancy Complications - blood | Membrane Glycoproteins - metabolism | Receptors, G-Protein-Coupled - metabolism | Obesity - immunology | Cell Count | Humans | Tumor Necrosis Factor-alpha - genetics | Antigens, CD - genetics | Obesity - blood | Antigens, CD - metabolism | Lipopolysaccharide Receptors - metabolism | Inflammation - complications | Placenta - cytology | Mucins - metabolism | Adult | Female | Pregnancy Complications - metabolism | Interleukin-6 - metabolism | Interleukin-6 - genetics | Placenta - immunology | Obesity - complications | Cells, Cultured | Macrophages - cytology | Fetal Blood - metabolism | Antigens, Differentiation, Myelomonocytic - genetics | Membrane Glycoproteins - genetics | Obesity - metabolism | Macrophages - metabolism | Antigens, Differentiation, Myelomonocytic - metabolism | Placenta - pathology | Lipopolysaccharide Receptors - genetics | Receptors, G-Protein-Coupled - genetics | Mucins - genetics | Pregnancy Complications - immunology | Pregnant women | Index Medicus | programming
Journal Article
The Journal of Nutritional Biochemistry, ISSN 0955-2863, 01/2015, Volume 26, Issue 1, pp. 91 - 98
We have previously shown that high-protein (HP) diet ingestion causes marked changes in the luminal environment of the colonic epithelium. This study aimed to... 
Mucus | Goblet cells | Basal gut inflammatory status | Intestine | High-protein diet | IMMUNE HOMEOSTASIS | RECOGNITION | BIOCHEMISTRY & MOLECULAR BIOLOGY | GUT | MICROBIOTA | NUTRITION & DIETETICS | EPITHELIAL-CELLS | METABOLISM | CONSEQUENCES | FERMENTATION | NITROGEN | THREONINE | Immunohistochemistry | Dietary Proteins - administration & dosage | Up-Regulation | Intestinal Mucosa - metabolism | Epithelial Cells - metabolism | Rats, Wistar | Enterocytes - metabolism | Epithelial Cells - drug effects | Transforming Growth Factor beta1 - metabolism | Colon - drug effects | Immunoglobulin A - genetics | Ileum - metabolism | Fatty Acids, Volatile - genetics | Goblet Cells - drug effects | Male | Fatty Acids, Volatile - metabolism | Intestinal Mucosa - drug effects | Ileum - drug effects | Interleukin-10 - metabolism | Interleukin-6 - metabolism | Mucin-3 - genetics | Tight Junctions - metabolism | Immunoglobulin A - metabolism | Mucin-3 - metabolism | Interleukin-6 - genetics | Acetates - metabolism | Down-Regulation | CD4-Positive T-Lymphocytes - metabolism | Rats | Toll-Like Receptor 4 - genetics | Interleukin-13 - metabolism | Toll-Like Receptor 4 - metabolism | Colon - metabolism | Animals | Diet | Enterocytes - drug effects | Physiological aspects | Cytokines | Gene expression | Mucins | Index Medicus | Life Sciences | Food and Nutrition
Journal Article
Archives of Pathology and Laboratory Medicine, ISSN 0003-9985, 2014, Volume 138, Issue 8, pp. 1015 - 1026
Context.-Distinction of medullary carcinoma of the large intestine from other cytokeratin (CK) 7(-)/CK20(-) carcinomas can be challenging when working on a... 
MEDICINE, RESEARCH & EXPERIMENTAL | MARKER | MICROSATELLITE INSTABILITY | COLON | PATHOLOGY | CYTOKERATIN-20 | GASTROINTESTINAL ADENOCARCINOMAS | COLORECTAL CARCINOMAS | IMMUNOHISTOCHEMICAL SURVEY | GENE-EXPRESSION | FACTOR CDX2 | DIFFERENTIATION | MEDICAL LABORATORY TECHNOLOGY | Immunohistochemistry | MutL Protein Homolog 1 | Cadherins - metabolism | Tissue Array Analysis | Cecal Neoplasms - pathology | Humans | Middle Aged | Carcinoma, Medullary - diagnosis | Colonic Neoplasms - diagnosis | Carcinoma, Medullary - metabolism | Male | Neoplasm Proteins - metabolism | Colonic Neoplasms - metabolism | Cecal Neoplasms - metabolism | DNA-Binding Proteins - metabolism | Cecum - metabolism | Sensitivity and Specificity | DNA Repair Enzymes - metabolism | Aged, 80 and over | Female | Mismatch Repair Endonuclease PMS2 | Biomarkers - metabolism | Colon - pathology | Matrix Attachment Region Binding Proteins - metabolism | Down-Regulation | Adenosine Triphosphatases - metabolism | Cecal Neoplasms - diagnosis | Carcinoma, Medullary - pathology | Nuclear Proteins - metabolism | Tumor Burden | Colon - metabolism | Cecum - pathology | Transcription Factors - metabolism | Colonic Neoplasms - pathology | Aged | Adaptor Proteins, Signal Transducing - metabolism | Keratin | Carcinoma | Diagnosis | Colorectal cancer | Mucins | Cancer | Index Medicus | Abridged Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2014, Volume 9, Issue 1, pp. e87368 - e87368
There are several animal models illustrating dry eye pathophysiology. Current study would like to establish an ex vivo tissue culture model for characterizing... 
OCULAR SURFACE EPITHELIUM | MATRIX METALLOPROTEINASES | MUCIN GENE-EXPRESSION | KERATOCONJUNCTIVITIS SICCA | MULTIDISCIPLINARY SCIENCES | CORNEAL EPITHELIAL-CELLS | SQUAMOUS METAPLASIA | LACRIMAL GLAND | FLOW-CYTOMETRIC ANALYSIS | BENZALKONIUM CHLORIDE | INFLAMMATORY MARKERS | Immunohistochemistry | Tumor Necrosis Factor-alpha - metabolism | Epithelial Cells - metabolism | Homeodomain Proteins - metabolism | Humans | Keratin-10 - genetics | Keratin-16 - genetics | Conjunctiva - metabolism | Conjunctiva - pathology | Matrix Metalloproteinase 9 - metabolism | Time Factors | Interleukin-1beta - metabolism | Mucins - metabolism | Repressor Proteins - metabolism | Keratin-10 - metabolism | Gene Expression | Enzyme-Linked Immunosorbent Assay | Mucin-4 - metabolism | Keratin-16 - metabolism | Dry Eye Syndromes - genetics | PAX6 Transcription Factor | Air | Reverse Transcriptase Polymerase Chain Reaction | Mucin-4 - genetics | Keratin-19 - genetics | Eye Proteins - metabolism | Dry Eye Syndromes - metabolism | Mucin 5AC - genetics | Mucin 5AC - metabolism | Keratin-19 - metabolism | Tissue Culture Techniques - methods | Mucins - genetics | Paired Box Transcription Factors - metabolism | Apoptosis | Keratin | Analysis | Mucins | Cell culture | Cornea | Animal models | Media (culture) | Wnt protein | Disease | Laboratories | Science | Tissue culture | Eye | Signal transduction | Epidermal growth factor | Stratification | Air conditioners | MUC16 gene | Explants | Immunoglobulins | Air exposure | Cultures | Inflammation