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1. Full Text Fibrosis — A Common Pathway to Organ Injury and Failure
by Rockey, Don C and Bell, P. Darwin and Hill, Joseph A
The New England journal of medicine, ISSN 0028-4793, 03/2015, Volume 372, Issue 12, pp. 1138 - 1149
Medicine, General & Internal | Life Sciences & Biomedicine | General & Internal Medicine | Science & Technology | Fibrosis - therapy | Fibrosis - physiopathology | Transforming Growth Factor beta - metabolism | Fibrosis - complications | Humans | Multiple Organ Failure - etiology | Cell proliferation | Care and treatment | Fibrosis | Fibroblasts | Inflammation | Research | Risk factors | Proteins | Medical research | Pathogenesis | Extracellular matrix | Tissues | Index Medicus | Abridged Index Medicus
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2. Full Text Kynurenine-3-monooxygenase inhibition prevents multiple organ failure in rodent models of acute pancreatitis
by Mole, Damian J and Webster, Scott P and Uings, Iain and Zheng, Xiaozhong and Binnie, Margaret and Wilson, Kris and Hutchinson, Jonathan P and Mirguet, Olivier and Walker, Ann and Beaufils, Benjamin and Ancellin, Nicolas and Trottet, Lionel and Bénéton, Véronique and Mowat, Christopher G and Wilkinson, Martin and Rowland, Paul and Haslam, Carl and McBride, Andrew and Homer, Natalie Z. M and Baily, James E and Sharp, Matthew G. F and Garden, O. James and Hughes, Jeremy and Howie, Sarah E. M and Holmes, Duncan S and Liddle, John and Iredale, John P
Nature medicine, ISSN 1078-8956, 02/2016, Volume 22, Issue 2, pp. 202 - 209
Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Cell Biology | Research & Experimental Medicine | Propionates - pharmacology | Benzoxazoles - pharmacology | Kynurenine 3-Monooxygenase - genetics | Kidney - pathology | Kynurenine 3-Monooxygenase - antagonists & inhibitors | Humans | Crystallography, X-Ray | Hepatocytes - metabolism | Pancreatitis - complications | RNA, Messenger - metabolism | Tryptophan - metabolism | Pancreatitis - genetics | Tandem Mass Spectrometry | Kidney - metabolism | HEK293 Cells | Chromatography, Liquid | Lung - metabolism | Disease Models, Animal | Multiple Organ Failure - pathology | Acute Disease | Lung - pathology | Pancreas - pathology | Rats | Multiple Organ Failure - genetics | Pancreas - metabolism | Mice, Knockout | Animals | Oxazolidinones - pharmacology | Multiple Organ Failure - etiology | Pancreatitis - pathology | Mice | In Vitro Techniques | Oxidases | Prevention | Enzymes | Pancreatitis | Multiple organ failure | Development and progression | Genetic aspects | Regulation | Health aspects | Pharmacology | Pancreas | Rodents | Inflammatory diseases | Index Medicus
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3. Full Text Lipotoxicity causes multisystem organ failure and exacerbates acute pancreatitis in obesity
by Navina, Sarah and Acharya, Chathur and DeLany, James P and Orlichenko, Lidiya S and Baty, Catherine J and Shiva, Sruti S and Durgampudi, Chandra and Karlsson, Jenny M and Lee, Kenneth and Bae, Kyongtae T and Furlan, Alessandro and Behari, Jaideep and Liu, Shiguang and McHale, Teresa and Nichols, Larry and Papachristou, Georgios Ioannis and Yadav, Dhiraj and Singh, Vijay P
Science translational medicine, ISSN 1946-6234, 11/2011, Volume 3, Issue 107, pp. 107ra110 - 107ra110
Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Cell Biology | Research & Experimental Medicine | Immunohistochemistry | Multiple Organ Failure - metabolism | Acinar Cells - metabolism | Humans | Pancreas - pathology | Pancreatitis, Acute Necrotizing - physiopathology | Necrosis - metabolism | Obesity - physiopathology | Fatty Acids, Unsaturated - metabolism | Pancreas - metabolism | Obesity - metabolism | Acinar Cells - pathology | Animals | Multiple Organ Failure - etiology | Necrosis - etiology | Pancreatitis, Acute Necrotizing - metabolism | Mice, Obese | Mice | Pancreatitis - metabolism | In Vitro Techniques | Lipolysis - physiology | Fatty Acids - metabolism | Pancreatitis - physiopathology | Pancreatitis, Acute Necrotizing - pathology | Index Medicus
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4. Full Text Mitochondrial function in sepsis: Acute phase versus multiple organ failure
by Singer, Mervyn
Critical care medicine, ISSN 0090-3493, 09/2007, Volume 35, Issue 9 Suppl, pp. S441 - S448
Mitochondria | Hibernation | Multiple organ failure | Oxygen consumption | Sepsis | Biogenesis | Metabolism | Life Sciences & Biomedicine | Critical Care Medicine | General & Internal Medicine | Science & Technology | Biological and medical sciences | Intensive care medicine | Emergency and intensive care: infection, septic shock | Medical sciences | Anesthesia. Intensive care medicine. Transfusions. Cell therapy and gene therapy | Emergency and intensive respiratory care | Multiple Organ Failure - metabolism | Acute Disease | Animals | Energy Metabolism | Sepsis - metabolism | Adenosine Triphosphate - metabolism | Humans | Oxygen Consumption | Mitochondria - metabolism | Mitochondria - ultrastructure | Index Medicus | Abridged Index Medicus
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5. Full Text TLR4 induced Wnt3a-Dvl3 restrains the intensity of inflammation and protects against endotoxin-driven organ failure through GSK3β/β-catenin signaling
by Yang, Dongqiang and Li, ShuJian and Duan, Xiaoxian and Ren, Junling and Liang, Shuang and Yakoumatos, Lan and Kang, Yi and Uriarte, Silvia M and Shang, Jia and Li, Wei and Wang, Huizhi
Molecular immunology, ISSN 0161-5890, 02/2020, Volume 118, pp. 153 - 164
β-catenin | Dvl3 | Inflammation regulation | TLR4 | Wnt3 | Biochemistry & Molecular Biology | Immunology | Life Sciences & Biomedicine | Science & Technology | Endotoxins - metabolism | Multiple Organ Failure - metabolism | Phosphorylation - physiology | Tumor Necrosis Factor-alpha - metabolism | Wnt3A Protein - metabolism | Cytokines - metabolism | Humans | Mice, Inbred C57BL | Dishevelled Proteins - metabolism | Monocytes - metabolism | Glycogen Synthase Kinase 3 beta - metabolism | Toll-Like Receptor 4 - metabolism | beta Catenin - metabolism | Inflammation - metabolism | Animals | Transcription Factor RelA - metabolism | Lipopolysaccharides - pharmacology | Signal Transduction - physiology | Mice | Index Medicus
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6. Full Text Inhibition of serum‐ and glucocorticoid‐inducible kinase 1 enhances TLR‐mediated inflammation and promotes endotoxin‐driven organ failure
by Zhou, Huaxin and Gao, Shegan and Duan, Xiaoxian and Liang, Shuang and Scott, David A and Lamont, Richard J and Wang, Huizhi
The FASEB journal, ISSN 0892-6638, 09/2015, Volume 29, Issue 9, pp. 3737 - 3749
inflammatory cytokines | TAK1 | SGK1 | LPS | Biochemistry & Molecular Biology | Biology | Life Sciences & Biomedicine - Other Topics | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Inflammation - chemically induced | RNA, Small Interfering - genetics | Multiple Organ Failure - chemically induced | Humans | Male | Transcription Factor RelA - genetics | Immediate-Early Proteins - metabolism | I-kappa B Kinase - metabolism | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Female | Toll-Like Receptors - metabolism | Cytokines - genetics | Protein-Serine-Threonine Kinases - metabolism | Lipopolysaccharides - toxicity | Cytokines - metabolism | MAP Kinase Kinase Kinases - genetics | Protein-Serine-Threonine Kinases - genetics | Mice, Transgenic | MAP Kinase Kinase Kinases - metabolism | Multiple Organ Failure - genetics | I-kappa B Kinase - genetics | Animals | Immediate-Early Proteins - genetics | Transcription Factor RelA - metabolism | Toll-Like Receptors - genetics | Multiple Organ Failure - enzymology | Inflammation - genetics | Mice | Inflammation - enzymology | Immediate-Early Proteins - antagonists & inhibitors | Index Medicus | Research Communication
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7. Full Text Angiotensin-converting enzyme 2 (ACE2): COVID 19 gate way to multiple organ failure syndromes
by loganathan, Sundareswaran and Kuppusamy, Maheshkumar and Wankhar, Wankupar and Gurugubelli, Krishna Rao and Mahadevappa, Vidyashree Hodagatta and Lepcha, Lhakit and Choudhary, Arbind kumar
Respiratory physiology & neurobiology, ISSN 1569-9048, 01/2021, Volume 283, pp. 103548 - 103548
COVID-19 | ACE2 | SARS-CoV-2 | Pneumonia and inflammation | ARDS | Respiratory System | Physiology | Life Sciences & Biomedicine | Science & Technology | Pandemics | Gastrointestinal Diseases - physiopathology | Humans | Heart Failure - physiopathology | Coronavirus Infections - physiopathology | Respiratory Distress Syndrome - physiopathology | Respiratory Distress Syndrome - immunology | Arrhythmias, Cardiac - physiopathology | Viral Load | Kidney - metabolism | Myocardium - metabolism | Arrhythmias, Cardiac - immunology | Heart Failure - immunology | Peptidyl-Dipeptidase A - metabolism | Lung - metabolism | Acute Kidney Injury - immunology | Gastrointestinal Diseases - immunology | Cytokines - immunology | Liver Diseases - immunology | Cytokine Release Syndrome - immunology | Cytokine Release Syndrome - physiopathology | Spike Glycoprotein, Coronavirus - metabolism | Liver - metabolism | Thromboembolism - immunology | Inflammation - immunology | Betacoronavirus - metabolism | Acute Kidney Injury - physiopathology | Coronavirus Infections - immunology | Pneumonia, Viral - immunology | Gastrointestinal Tract - metabolism | Pneumonia, Viral - physiopathology | Angiotensin-Converting Enzyme 2 | Endothelium, Vascular - metabolism | Erythrocytes - metabolism | Multiple Organ Failure - immunology | Thromboembolism - physiopathology | Liver Diseases - physiopathology | Multiple Organ Failure - physiopathology | Index Medicus
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