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1. Full Text A PGC1-α-dependent myokine that drives brown-fat-like development of white fat and thermogenesis
by Boström, Pontus and Wu, Jun and Jedrychowski, Mark P and Korde, Anisha and Ye, Li and Lo, James C and Rasbach, Kyle A and Boström, Elisabeth Almer and Choi, Jang Hyun and Long, Jonathan Z and Kajimura, Shingo and Zingaretti, Maria Cristina and Vind, Birgitte F and Tu, Hua and Cinti, Saverio and Højlund, Kurt and Gygi, Steven P and Spiegelman, Bruce M
Nature (London), ISSN 0028-0836, 01/2012, Volume 481, Issue 7382, pp. 463 - 468
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Fundamental and applied biological sciences. Psychology | Vertebrates: nervous system and sense organs | Obesity | Biological and medical sciences | Metabolic diseases | Medical sciences | Humans | Intracellular Signaling Peptides and Proteins - metabolism | Adipocytes - drug effects | Obesity - blood | Adipose Tissue, White - cytology | Mitochondrial Proteins - metabolism | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Models, Animal | Energy Metabolism - physiology | Intracellular Signaling Peptides and Proteins - genetics | Obesity - chemically induced | Mice, Transgenic | Trans-Activators - deficiency | Obesity - prevention & control | Plasma - chemistry | Adipose Tissue, Brown - drug effects | Exercise - physiology | Mice | Mice, Inbred BALB C | Transcription Factors | Energy Metabolism - drug effects | Thermogenesis - genetics | Adipose Tissue, White - metabolism | Adipocytes - cytology | Culture Media, Conditioned - pharmacology | Subcutaneous Fat - metabolism | Insulin Resistance - physiology | Cell Respiration - drug effects | Subcutaneous Fat - drug effects | Trans-Activators - genetics | Energy Metabolism - genetics | Gene Expression Regulation - genetics | Cells, Cultured | Hormones - secretion | Muscle Cells - metabolism | Physical Conditioning, Animal - physiology | Subcutaneous Fat - cytology | Gene Expression Regulation - drug effects | Adipose Tissue, Brown - cytology | Animals | Hormones - metabolism | Ion Channels - metabolism | Adipocytes - metabolism | Trans-Activators - secretion | Trans-Activators - metabolism | Adipose Tissue, Brown - metabolism | Thermogenesis - drug effects | Uncoupling Protein 1 | Adipose Tissue, White - drug effects | Index Medicus | Medicin och hälsovetenskap
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2. Full Text TWEAK, via its receptor Fn14, is a novel regulator of mesenchymal progenitor cells and skeletal muscle regeneration
by Weng, Shawn and Yagita, Hideo and Burkly, Linda C and Winkles, Jeffrey A and Browning, Beth and Zheng, Timothy S and Hsu, Yen-ming and Kostek, Christine A and Miller, Jeffrey Boone and Scott, Martin L and Brown, Sharron AN and Girgenrath, Mahasweta and Schneider, Pascal and Flavell, Richard A and Wang, Monica and Michaelson, Jennifer S and Allaire, Norm
The EMBO journal, ISSN 0261-4189, 12/2006, Volume 25, Issue 24, pp. 5826 - 5839
Fn14 | TWEAK | inflammation | regeneration | muscle | Regeneration | Inflammation | Muscle | Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Tumor Necrosis Factors - genetics | Humans | Cobra Cardiotoxin Proteins - pharmacology | TWEAK Receptor | Muscle, Skeletal - cytology | RNA, Messenger - metabolism | Myoblasts - drug effects | Mesenchymal Stromal Cells - cytology | Muscle, Skeletal - drug effects | Myoblasts - cytology | Receptors, Tumor Necrosis Factor - deficiency | Receptors, Tumor Necrosis Factor - genetics | Receptors, Tumor Necrosis Factor - metabolism | Mesenchymal Stromal Cells - drug effects | RNA, Messenger - genetics | Cells, Cultured | Tumor Necrosis Factors - metabolism | Muscle, Skeletal - physiology | Gene Expression Regulation - drug effects | Cytokine TWEAK | Regeneration - drug effects | Animals | Muscle Development - drug effects | Cell Differentiation - drug effects | Models, Biological | Cell Proliferation - drug effects | Mice | Cell Cycle - drug effects | Cellular biology | Molecular biology | Rodents | Inflammatory diseases | Stem cells | Index Medicus
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3. Full Text Myogenic Progenitor Cells Control Extracellular Matrix Production by Fibroblasts during Skeletal Muscle Hypertrophy
by Fry, Christopher S and Kirby, Tyler J and Kosmac, Kate and McCarthy, John J and Peterson, Charlotte A
Cell stem cell, ISSN 1934-5909, 01/2017, Volume 20, Issue 1, pp. 56 - 69
hypertrophy | Pax7 | exosomes | muscle | collagen | microRNA | fibrosis | muscle progenitor cells | extracellular matrix | satellite cells | Cell & Tissue Engineering | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Exosomes - drug effects | Exosomes - metabolism | NIH 3T3 Cells | Cell Survival - genetics | Extracellular Matrix - metabolism | MicroRNAs - metabolism | PAX7 Transcription Factor - metabolism | Muscle Fibers, Skeletal - drug effects | Muscle Fibers, Skeletal - metabolism | Stem Cells - metabolism | Gene Knockdown Techniques | Cell Differentiation - genetics | Extracellular Matrix - genetics | Cell Nucleus - metabolism | Gene Deletion | Collagen - genetics | Fibroblasts - metabolism | Cell Survival - drug effects | Ribonuclease III - metabolism | Extracellular Matrix - drug effects | Satellite Cells, Skeletal Muscle - metabolism | Down-Regulation - drug effects | Fibroblasts - pathology | Down-Regulation - genetics | Collagen - metabolism | Satellite Cells, Skeletal Muscle - drug effects | Animals | Carrier Proteins - metabolism | Muscle Development - drug effects | Cell Differentiation - drug effects | Models, Biological | Fibroblasts - drug effects | Muscle Fibers, Skeletal - pathology | Tamoxifen - pharmacology | Muscle Development - genetics | Stem Cells - drug effects | Mice | MicroRNAs - genetics | Muscle, Skeletal - pathology | Cell Nucleus - drug effects | Hypertrophy | Index Medicus | miRNA | Dicer | Muscle extracellular matrix
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4. Mouse models for drug discovery
: methods and protocols
by Proetzel, Gabriele and Wiles, Michael V
2010, Methods in molecular biology, ISBN 9781607610571, Volume 602., xii, 421
Drugs | Effect of drugs on | Animal models in research | Drug Design | Models, Animal | Mice | Drug development | Drug Evaluation, Preclinical | Physiological effect | Molecular aspects | Diseases
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5. Full Text Fasudil improves survival and promotes skeletal muscle development in a mouse model of spinal muscular atrophy
by Bowerman, Melissa and Murray, Lyndsay M and Boyer, Justin G and Anderson, Carrie L and Kothary, Rashmi
BMC medicine, ISSN 1741-7015, 03/2012, Volume 10, Issue 1, pp. 24 - 24
Muscle | NMJ | Survival motor neuron protein | Fasudil | Spinal muscular atrophy | Medicine, General & Internal | Life Sciences & Biomedicine | General & Internal Medicine | Science & Technology | Anterior Horn Cells - drug effects | Spinal Cord - drug effects | Longevity - drug effects | 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine - analogs & derivatives | Muscle Fibers, Skeletal - drug effects | Survival of Motor Neuron 2 Protein - metabolism | Motor Neurons - pathology | Dose-Response Relationship, Drug | Motor Endplate - drug effects | Spinal Cord - pathology | Motor Endplate - pathology | Muscle, Skeletal - drug effects | Weight Gain - drug effects | Survival of Motor Neuron 2 Protein - deficiency | Muscular Atrophy, Spinal - physiopathology | Motor Neurons - drug effects | Disease Models, Animal | Muscle, Skeletal - growth & development | Mice, Inbred C57BL | Anterior Horn Cells - pathology | 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine - therapeutic use | 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine - administration & dosage | Muscular Atrophy, Spinal - pathology | Gait - drug effects | Phenotype | Animals | Muscle Development - drug effects | 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine - pharmacology | Muscle, Skeletal - physiopathology | Muscle Fibers, Skeletal - pathology | Survival Analysis | Muscular Atrophy, Spinal - drug therapy | Mice | Spinal Cord - physiopathology | Muscle, Skeletal - pathology | Myogenin - metabolism | Motor Endplate - physiopathology | Prevention | Complications and side effects | Prognosis | Vasodilators | Patient outcomes | Dosage and administration | Infants | Research | Drug therapy | Atrophy, Muscular | Risk factors | Medical research | Musculoskeletal system | Pathogenesis | Laboratories | Colleges & universities | Mutation | Muscular system | FDA approval | Kinases | Drug dosages | Index Medicus | survival motor neuron protein | spinal muscular atrophy | fasudil | muscle
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6. Full Text Development and plasticity of meningeal lymphatic vessels
by Antila, Salli and Karaman, Sinem and Nurmi, Harri and Airavaara, Mikko and Voutilainen, Merja H and Mathivet, Thomas and Chilov, Dmitri and Li, Zhilin and Koppinen, Tapani and Park, Jun-Hee and Fang, Shentong and Aspelund, Aleksanteri and Saarma, Mart and Eichmann, Anne and Thomas, Jean-Léon and Alitalo, Kari
The Journal of experimental medicine, ISSN 0022-1007, 12/2017, Volume 214, Issue 12, pp. 3645 - 3667
Immunology | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Spinal Cord - drug effects | Humans | Injections, Intraventricular | Male | Lymphatic Vessels - drug effects | Microspheres | Sunitinib | Gene Deletion | Indoles - pharmacology | Lymph Nodes - drug effects | Biological Transport - drug effects | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - metabolism | Animals, Newborn | Dependovirus - metabolism | Lymphatic Vessels - physiology | Signal Transduction | Mice, Inbred C57BL | Lymph Nodes - metabolism | Meninges - drug effects | Vascular Endothelial Growth Factor Receptor-3 - metabolism | Lymphangiogenesis - drug effects | Pyrroles - pharmacology | Vascular Endothelial Growth Factor D - metabolism | Animals | Cerebrospinal Fluid - metabolism | Vascular Endothelial Growth Factor C - metabolism | Spinal Cord - physiology | Protein Kinase Inhibitors - pharmacology | Meninges - physiology | Tyrosine | Spinal nerves | Lymphatic system | Neuropathology | Developmental plasticity | Central nervous system | Muscles | Blood vessels | Smooth muscle | Cerebrospinal fluid | Wound drainage | Endothelial cells | Vascular endothelial growth factor receptors | Signal transduction | Inhibitors | Clonal deletion | Rodents | Nerves | Plasticity | Plastic properties | Mice | Meninges | Vascular endothelial growth factor | Protein-tyrosine kinase | Index Medicus | 301 | 320
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7. Full Text Vascular endothelial growth factor is upregulated by l-dopa in the parkinsonian brain: implications for the development of dyskinesia
by Ohlin, K. Elisabet and Francardo, Veronica and Lindgren, Hanna S and Sillivan, Stephanie E and O'Sullivan, Sean S and Luksik, Andrew S and Vassoler, Fair M and Lees, Andrew J and Konradi, Christine and Cenci, M. Angela
Brain (London, England : 1878), ISSN 0006-8950, 8/2011, Volume 134, Issue 8, pp. 2339 - 2357
blood-brain barrier | D1 receptors | Parkinson's disease | levodopa | angiogenesis | neuroplasticity | Neurosciences | Clinical Neurology | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Neurology | Biological and medical sciences | Toxicity: nervous system and muscle | Medical sciences | Drug toxicity and drugs side effects treatment | Pharmacology. Drug treatments | Embryo, Mammalian | Blood Vessels - metabolism | Cell Count | Humans | Blood-Brain Barrier - physiopathology | Male | Vascular Endothelial Growth Factor A - metabolism | RNA, Messenger - metabolism | Vascular Endothelial Growth Factor A - genetics | Antigens, CD - metabolism | Dose-Response Relationship, Drug | Time Factors | Dyskinesia, Drug-Induced - drug therapy | Parkinsonian Disorders - drug therapy | Aged, 80 and over | Antigens, Surface - metabolism | Behavior, Animal - drug effects | Female | Bromodeoxyuridine - metabolism | Dopamine - metabolism | Disease Models, Animal | Dyskinesia, Drug-Induced - etiology | Astrocytes - drug effects | Brain - cytology | Benserazide - adverse effects | Enzyme-Linked Immunosorbent Assay | Cells, Cultured | Enzyme Inhibitors - pharmacology | Rats | Neovascularization, Pathologic - chemically induced | Dyskinesia, Drug-Induced - pathology | Antiparkinson Agents - adverse effects | Blood-Brain Barrier - drug effects | Enzyme Inhibitors - therapeutic use | Rats, Sprague-Dawley | Brain - drug effects | Nerve Tissue Proteins - metabolism | Up-Regulation - drug effects | Animals | Analysis of Variance | Parkinsonian Disorders - pathology | Neovascularization, Pathologic - drug therapy | Levodopa - adverse effects | Blood Vessels - drug effects | Aged | Laminin - metabolism | Astrocytes - metabolism | Index Medicus | Abridged Index Medicus | blood–brain barrier | Original | Parkinson’s disease | Neurologi | Clinical Medicine | Medical and Health Sciences | Klinisk medicin | Medicin och hälsovetenskap
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