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Immunologic Research, ISSN 0257-277X, 10/2011, Volume 51, Issue 1, pp. 45 - 60
Complement system activation plays an important role in both innate and acquired immunity. Activation of the complement and the subsequent formation of C5b-9... 
Allergology | Signal transduction | Immunology | Medicine & Public Health | Cell cycle | Internal Medicine | Membrane attack complex | C5b-9 terminal complement complex | Transcriptional regulation | Medicine/Public Health, general | Apoptosis | ACTIVATED PROTEIN-KINASE | SYSTEMIC LUPUS-ERYTHEMATOSUS | GUILLAIN-BARRE-SYNDROME | ULCERATIVE-COLITIS | GLOMERULAR EPITHELIAL-CELLS | C5B-9 COMPLEX | IMMUNOLOGY | S-PROTEIN | MANNAN-BINDING LECTIN | ACUTE MYOCARDIAL-INFARCTION | SMOOTH-MUSCLE-CELLS | Forkhead Transcription Factors - immunology | CDC2 Protein Kinase | Complement Membrane Attack Complex - metabolism | Cyclin-Dependent Kinase 4 - genetics | Humans | Caspase 8 - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Complement Membrane Attack Complex - immunology | MAP Kinase Signaling System - immunology | Phosphatidylinositol 3-Kinases - immunology | Cell Cycle Proteins - immunology | Cyclin B - immunology | Forkhead Transcription Factors - metabolism | Muscle Proteins - metabolism | bcl-Associated Death Protein - metabolism | Cell Membrane - metabolism | Phosphorylation - immunology | Cyclin-Dependent Kinases | Proto-Oncogene Proteins c-akt - metabolism | BH3 Interacting Domain Death Agonist Protein - metabolism | Cyclin-Dependent Kinase 2 - metabolism | Mitogen-Activated Protein Kinase 3 - immunology | Muscle Proteins - immunology | G1 Phase - immunology | S Phase - immunology | Nerve Tissue Proteins - immunology | Cell Cycle Proteins - metabolism | bcl-Associated Death Protein - immunology | Cyclin-Dependent Kinase 4 - metabolism | Nerve Tissue Proteins - metabolism | Caspase 8 - immunology | Animals | Apoptosis - immunology | Proto-Oncogene Proteins c-akt - immunology | Mitogen-Activated Protein Kinase 3 - metabolism | BH3 Interacting Domain Death Agonist Protein - immunology | Cyclin B - metabolism | Cell Membrane - immunology | Forkhead Box Protein O1 | Cyclin-Dependent Kinase 2 - immunology | Medical colleges | Membranes | Immune system | apoptosis | membrane attack complex | cell cycle | signal transduction | transcriptional regulation
Journal Article
Trends in Immunology, ISSN 1471-4906, 2008, Volume 30, Issue 2, pp. 83 - 90
Complement is part of the innate immune defence and not only recognizes microbes but also unwanted host molecules to enhance phagocytosis and clearance. This... 
Allergy and Immunology | C-REACTIVE PROTEIN | APOPTOTIC CELL RECOGNITION | C4B-BINDING PROTEIN | AMYLOID-P COMPONENT | SYSTEMIC-LUPUS-ERYTHEMATOSUS | NECROTIC CELLS | MACULAR DEGENERATION | EXTRACELLULAR-MATRIX | IMMUNOLOGY | FACTOR-H BINDS | GLOBULAR HEAD | Alzheimer Disease - etiology | Macular Degeneration - immunology | Humans | Complement System Proteins - immunology | Intracellular Signaling Peptides and Proteins - immunology | Intracellular Signaling Peptides and Proteins - metabolism | Complement Activation - immunology | Complement C4b-Binding Protein | Complement System Proteins - metabolism | Prions - immunology | Muscle Proteins - metabolism | Alzheimer Disease - immunology | Histocompatibility Antigens - metabolism | Macular Degeneration - etiology | Histocompatibility Antigens - immunology | Muscle Proteins - immunology | Prions - metabolism | LIM Domain Proteins | Prion Diseases - immunology | Arthritis, Rheumatoid - etiology | Immunity, Innate | Animals | Prion Diseases - etiology | Protein Binding | Amyloid beta-Peptides - immunology | Ligands | Arthritis, Rheumatoid - immunology | Macular degeneration | Immune complexes | Immunoproteins | Protein binding | Proteins | Lupus | Aging | Alzheimers disease | Autoimmune diseases | Basic Medicine | Other Basic Medicine | Andra medicinska och farmaceutiska grundvetenskaper | Medical and Health Sciences | Medicin och hälsovetenskap | Medicinska och farmaceutiska grundvetenskaper
Journal Article
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 05/2017, Volume 127, Issue 5, pp. 1600 - 1612
The cardiac extracellular matrix (ECM) not only provides mechanical support, but also transduces essential molecular signals in health and disease. Following... 
ISCHEMIA-REPERFUSION INJURY | MEDICINE, RESEARCH & EXPERIMENTAL | VOLUME OVERLOAD | HEART-FAILURE | CHRONIC PRESSURE-OVERLOAD | LEFT-VENTRICULAR DILATION | MATRICELLULAR PROTEINS | SMOOTH-MUSCLE-CELLS | ANGIOTENSIN-II | CARDIAC FIBROSIS | MICE LACKING OSTEOPONTIN | Inflammation - pathology | Leukocytes - pathology | Myocardium - immunology | Myocytes, Cardiac - immunology | Humans | Extracellular Matrix - immunology | Extracellular Matrix - metabolism | Stress, Mechanical | Heart Failure, Diastolic - metabolism | Myocardial Infarction - immunology | Leukocytes - immunology | Intercellular Signaling Peptides and Proteins - metabolism | Signal Transduction - immunology | Inflammation - metabolism | Myocardial Infarction - pathology | Myocardium - metabolism | Extracellular Matrix Proteins - metabolism | Myocardium - pathology | Inflammation - immunology | Myocardial Infarction - metabolism | Heart Failure, Diastolic - pathology | Myocytes, Cardiac - pathology | Animals | Extracellular Matrix Proteins - immunology | Heart Failure, Diastolic - immunology | Myocytes, Cardiac - metabolism | Extracellular Matrix - pathology | Leukocytes - metabolism | Intercellular Signaling Peptides and Proteins - immunology | Extracellular matrix | Care and treatment | Research | Regeneration (Biology) | Heart attack | Heart failure | Hypertension | Myocardial infarction | Heart attacks | Pathogenesis | Spatial discrimination | Cardiomyocytes | Inflammation | Metabolism | Proteins | Ischemia | Collagen | Rodents | Fibroblasts | Plastics | Growth factors | Heart diseases | Binding sites
Journal Article
Annals of the New York Academy of Sciences, ISSN 0077-8923, 11/2015, Volume 1356, Issue 1, pp. 45 - 79
Ca 2+ release‐activated Ca 2+ (CRAC) channels mediate a specific form of Ca 2+ influx called store‐operated Ca 2+ entry (SOCE) that contributes to the function... 
calcium | disease | thrombocytopenia | channelopathy | enamel | CRAC channel | ameloblast | platelets | York platelet syndrome | mutation | STIM1 | muscular hypotonia | autoimmunity | Stormorken syndrome | SOCE | ORAI1 | skeletal muscle | tubular aggregate myopathy | Autoimmunity | Thrombocytopenia | Calcium | Disease | Channelopathy | Tubular aggregate myopathy | Enamel | Muscular hypotonia | Skeletal muscle | Mutation | Platelets | Ameloblast | OPERATED CA2+ ENTRY | SEVERE COMBINED IMMUNODEFICIENCY | IMMUNOLOGY | ACTIVATED CALCIUM-CHANNEL | CLASSIC KAPOSI-SARCOMA | DENTAL ENAMEL CELLS | INHERITED THROMBOCYTOPENIC DISORDER | T-CELL DEVELOPMENT | ELECTRON-DENSE CHAINS | Ca2 | STROMAL INTERACTION MOLECULE-1 | Stromal Interaction Molecule 1 | Muscle Hypotonia - genetics | Membrane Proteins - genetics | Humans | Autoimmune Diseases - immunology | Channelopathies - immunology | Neoplasm Proteins - immunology | Membrane Proteins - immunology | Severe Combined Immunodeficiency - immunology | Channelopathies - pathology | ORAI1 Protein | Autoimmune Diseases - genetics | Muscle Hypotonia - pathology | Severe Combined Immunodeficiency - genetics | Channelopathies - genetics | Calcium Channels - immunology | Severe Combined Immunodeficiency - pathology | Muscle Hypotonia - immunology | Neoplasm Proteins - genetics | Calcium Channels - genetics | Human | Proteins | Mutations | Teeth | Patients | Channels | Diseases | Defects
Journal Article
Nature Communications, ISSN 2041-1723, 04/2015, Volume 6, Issue 1, p. 6763
Asthma, a common disorder that affects 4250 million people worldwide, is defined by exaggerated bronchoconstriction to inflammatory mediators including... 
EXTRACELLULAR-MATRIX PROTEINS | ASPERGILLUS-FUMIGATUS | EPITHELIAL-CELLS | CONTRACTION | MULTIDISCIPLINARY SCIENCES | LUNG-DISEASE | SMOOTH-MUSCLE-CELLS | MECHANISMS | PEN C-13 | SERINE PROTEINASE | EXPRESSION | Histamine - biosynthesis | Calcium - metabolism | Humans | Immunoglobulin E - biosynthesis | Extracellular Matrix - immunology | Myocytes, Smooth Muscle - pathology | Calcium - immunology | Trachea - drug effects | Histamine - immunology | Bronchi - immunology | Extracellular Matrix - chemistry | Bronchi - drug effects | Allergens - administration & dosage | Allergens - immunology | Fungal Proteins - immunology | Bronchoconstriction - drug effects | Asthma - immunology | Bronchoconstriction - immunology | Female | Bacterial Proteins - immunology | rho GTP-Binding Proteins - immunology | Myocytes, Smooth Muscle - drug effects | Bronchi - pathology | Disease Models, Animal | Gene Expression | rho GTP-Binding Proteins - genetics | Extracellular Matrix - drug effects | Administration, Inhalation | Aspergillus fumigatus - chemistry | Bacterial Proteins - administration & dosage | Endopeptidases - administration & dosage | Endopeptidases - immunology | Trachea - immunology | Trachea - pathology | Animals | Aspergillus fumigatus - immunology | Myocytes, Smooth Muscle - immunology | Mice | Mice, Inbred BALB C | Primary Cell Culture | Asthma - pathology | Fungal Proteins - administration & dosage | Biodegradation | Allergens | Calcium | Serine | Medical treatment | Lung diseases | Muscles | Smooth muscle | RhoA protein | Inflammation | Asthma | Respiratory tract | Fungi | Histamine | Bronchoconstriction | Protease | Bradykinin | Extracellular matrix | Serine proteinase | Acetylcholine | Disruption | Calcium ions | Alkaline protease
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2016, Volume 138, Issue 3, pp. 700 - 710
Journal Article
Blood, ISSN 0006-4971, 11/2012, Volume 120, Issue 20, pp. 4182 - 4190
Journal Article