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Journal Article
Acta Pharmacologica Sinica, ISSN 1671-4083, 07/2013, Volume 34, Issue 7, pp. 901 - 911
Journal Article
Journal Article
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 12/2004, Volume 24, Issue 12, pp. 2320 - 2325
OBJECTIVE—Atherosclerosis is a chronic inflammatory response of the arterial wall to injury. High-mobility group box 1 (HMGB1) is a DNA-binding protein, which... 
High-mobility group box 1 | Inflammation | Cytokines | Macrophages | Atherosclerosis | atherosclerosis | RHEUMATOID-ARTHRITIS | PROTEIN-KINASE-C | PHOSPHATIDYLINOSITOL 3-KINASE | IFN-GAMMA | VASCULAR-LESIONS | NECROSIS-FACTOR-ALPHA | macrophages | SMOOTH-MUSCLE | high-mobility group box 1 | inflammation | SIGNALING PATHWAY | PERIPHERAL VASCULAR DISEASE | cytokines | TNF-ALPHA | END-PRODUCTS RAGE | HEMATOLOGY | Endothelium, Vascular - cytology | Monocytes - cytology | Humans | Myocytes, Smooth Muscle - pathology | Monocytes - metabolism | Protein Transport - physiology | HMGB1 Protein - immunology | Aorta, Thoracic - chemistry | Cytokines - physiology | Endothelium, Vascular - chemistry | HMGB1 Protein - genetics | HMGB1 Protein - secretion | RNA, Messenger - biosynthesis | HMGB1 Protein - metabolism | Muscle, Smooth, Vascular - chemistry | Aorta, Abdominal - pathology | Aorta, Abdominal - metabolism | Inflammation Mediators - physiology | Myocytes, Smooth Muscle - metabolism | Aorta, Thoracic - pathology | Muscle, Smooth, Vascular - physiology | Tunica Intima - chemistry | Macrophage Activation - physiology | Macrophages - physiology | Tunica Intima - physiology | Cells, Cultured | Myocytes, Smooth Muscle - physiology | Aorta, Thoracic - metabolism | Gene Expression Regulation - physiology | Myocytes, Smooth Muscle - chemistry | Immunohistochemistry - methods | Monocytes - chemistry | Muscle, Smooth, Vascular - pathology | Tunica Intima - pathology | Aorta, Abdominal - chemistry | Endothelium, Vascular - pathology | Arteriosclerosis - metabolism | Arteriosclerosis - pathology
Journal Article
Journal of Vascular Surgery, ISSN 0741-5214, 2016, Volume 66, Issue 5, pp. 1553 - 1564.e6
Abstract Objective Abdominal aortic aneurysm (AAA) is a frequent, potentially life-threatening, disease that can only be treated by surgical means such as open... 
Surgery | SURGERY | MYOCARDIN | RISK-FACTORS | ABDOMINAL AORTIC-ANEURYSM | PHENOTYPIC MODULATION | INFLAMMATION | UNIQUE | PERIPHERAL VASCULAR DISEASE | PATHOPHYSIOLOGY | Inflammation - pathology | Vascular Remodeling | Humans | Myocytes, Smooth Muscle - pathology | Cell Dedifferentiation | Aortic Aneurysm, Abdominal - diagnostic imaging | Popliteal Artery - chemistry | Extracellular Matrix - chemistry | Aortic Aneurysm, Abdominal - metabolism | Extracellular Matrix Proteins - analysis | Aortography - methods | Inflammation - metabolism | Transforming Growth Factor beta - analysis | Aorta, Abdominal - diagnostic imaging | Inflammation Mediators - analysis | Kruppel-Like Transcription Factors - analysis | Muscle, Smooth, Vascular - chemistry | Aneurysm - diagnostic imaging | Aorta, Abdominal - pathology | Inflammation - diagnostic imaging | Dilatation, Pathologic | Aneurysm - metabolism | Aortic Aneurysm, Abdominal - pathology | Angiogenic Proteins - analysis | Biomarkers - analysis | Myocytes, Smooth Muscle - chemistry | Aneurysm - pathology | Popliteal Artery - diagnostic imaging | Computed Tomography Angiography | Muscle, Smooth, Vascular - pathology | Phenotype | Aorta, Abdominal - chemistry | Popliteal Artery - pathology | Extracellular Matrix - pathology | Medicine, Experimental | Aneurysms | Medical research | Cells | Immunohistochemistry | Endothelial growth factors | Analysis | Smooth muscle | Bone morphogenetic proteins
Journal Article
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology: Journal of the American Heart Association, ISSN 1079-5642, 09/2003, Volume 23, Issue 9, pp. 1510 - 1520
Journal Article
Journal Article
Cytokine and Growth Factor Reviews, ISSN 1359-6101, 2015, Volume 26, Issue 6, pp. 673 - 685
Abstract Atherosclerosis, a chronic inflammatory disorder of the arteries, is responsible for most deaths in westernized societies with numbers increasing at a... 
Advanced Basic Science | Therapeutic avenues | Inflammation | Cytokines | Chemokines | Atherosclerosis | Atherosclerosis - immunology | Cytokines - metabolism | Atherosclerosis - physiopathology | Humans | Endothelium, Vascular - physiopathology | Extracellular Matrix - metabolism | Inflammation - immunology | Lipoproteins - blood | Molecular Targeted Therapy | Extracellular Matrix - chemistry | Macrophages - enzymology | Animals | Atherosclerosis - therapy | Muscle, Smooth, Vascular - chemistry | Myocardial Infarction - physiopathology | Chemokines - metabolism | Mice | Matrix Metalloproteinases - metabolism | Macrophages - immunology | Chemokines - immunology | Cytokines - immunology | Endothelium, Vascular - immunology | Inflammation - physiopathology | Development and progression | Blood lipids | Health aspects | GM-CSF, granulocyte macrophage colony-stimulating factor | ECM, extracellular matrix | RCT, reverse cholesterol transport | ICAM-1, intercellular adhesion molecule-1 | LDLr, low-density lipoprotein receptor | PECAM-1, platelet endothelial cell adhesion molecule-1 | ABC, ATP-binding cassette | IL-1RA, IL-1 receptor antagonist | ADRP, adipocyte differentiation related protein | SREBP, sterol response element binding protein | wt, wild type | G-CSF, granulocyte colony-stimulating factor | PAI, plasminogen activator inhibitor | CSF, colony-stimulating factor | PRR, pattern recognition receptor | TIMP, tissue inhibitor of metalloproteinases | NPC, Niemann-Pick disease, type C | TNF, tumor necrosis factor | TL1A, TNF-like protein 1A | NLR, NOD-like receptor | SMCs, smooth muscle cells | CR, chemokine receptor | Tregs, regulatory T cells | Survey | GDF-15, growth differentiation factor-15 | NK, natural killer | TNFSF12, TNF superfamily member 12 | IL, interleukin | NLRP3, NLR family, pyrin domain containing 3 | mmLDL, minimally modified LDL | LIGHT, homologous to lymphotoxin, exhibits inducible expression, and competes with HSV glycoprotein D for herpes virus entry mediator, a receptor