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Journal Article
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 10/2017, Volume 37, Issue 12, pp. 2311 - 2321
OBJECTIVE—Drug-eluting stent delivery of mTORC1 (mammalian target of rapamycin complex 1) inhibitors is highly effective in preventing intimal hyperplasia... 
Gene expression | Muscle, smooth | Hyperplasia | Drug-eluting stents | Therapeutics | MAMMALIAN TARGET | RAPAMYCIN | MIGRATION | MYOCARDIN | therapeutics | drug-eluting stents | MICE LACKING | INSULIN | muscle | IN-VIVO | hyperplasia | CELL DIFFERENTIATION | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | EXPRESSION | gene expression | smooth | REVEALS | Vascular System Injuries - genetics | Humans | Proto-Oncogene Proteins c-akt - deficiency | Myocytes, Smooth Muscle - pathology | Vascular System Injuries - enzymology | RNA, Messenger - metabolism | Proto-Oncogene Proteins c-akt - genetics | Vascular System Injuries - pathology | Neointima | Transfection | RNA Interference | Time Factors | Trans-Activators - genetics | Myocytes, Smooth Muscle - drug effects | Nuclear Proteins - genetics | Proto-Oncogene Proteins c-akt - metabolism | Binding Sites | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | Promoter Regions, Genetic | Genetic Predisposition to Disease | Myocytes, Smooth Muscle - enzymology | RNA, Messenger - genetics | Cells, Cultured | Gene Expression Regulation | Nuclear Proteins - metabolism | Transcription Factors - genetics | Vascular System Injuries - prevention & control | Sirolimus - pharmacology | Mice, Knockout | Transcription Factors - metabolism | Cell Movement - drug effects | Muscle, Smooth, Vascular - injuries | Muscle, Smooth, Vascular - pathology | Phenotype | Animals | Signal Transduction - drug effects | Cell Differentiation - drug effects | Trans-Activators - metabolism | Cell Proliferation - drug effects | Muscle, Smooth, Vascular - enzymology | differentiation | signal transduction | basic science research | vascular biology | Akt | FOXO4 | neointimal hyperplasia | cell signaling | smooth muscle proliferation and differentiation | myocardin | mTORC1 | mechanisms | rapamycin
Journal Article
Circulation Journal, ISSN 1346-9843, 2011, Volume 75, Issue 6, pp. 1287 - 1296
Journal Article
Journal Article
Circulation, ISSN 0009-7322, 04/2018, Volume 137, Issue 17, pp. 1824 - 1841
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 04/2009, Volume 29, Issue 4, pp. 480 - 487
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 03/2017, Volume 37, Issue 5, pp. 879 - 888
OBJECTIVE—Smooth muscle cells (SMCs) contribute to neointima formation after vascular injury. Although β-catenin expression is induced after injury, whether... 
beta-catenin | beta-catenin inhibitors | vascular injury | myocytes, smooth muscle | neointima | RECEPTOR EDG-1 | P53 GENE-TRANSFER | ACTIVATION | HUMAN SAPHENOUS-VEIN | PROLIFERATION | HYPERPLASIA | IN-VITRO | RAT CAROTID-ARTERY | CELL-MIGRATION | PERIPHERAL VASCULAR DISEASE | DIFFERENTIATION | HEMATOLOGY | Carotid Arteries - metabolism | Vascular Remodeling | Cell Proliferation | Muscle, Smooth, Vascular - metabolism | Humans | Myocytes, Smooth Muscle - pathology | Male | Dose-Response Relationship, Drug | Neointima | Time Factors | Pyrimidinones - pharmacology | Carotid Artery Injuries - genetics | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - metabolism | Carotid Artery Injuries - metabolism | Carotid Artery Injuries - pathology | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | Triazines - pharmacology | Signal Transduction | beta Catenin - deficiency | Cells, Cultured | Gene Expression Regulation | Genotype | beta Catenin - genetics | Mice, Knockout | Bridged Bicyclo Compounds, Heterocyclic - pharmacology | Muscle, Smooth, Vascular - injuries | Muscle, Smooth, Vascular - pathology | Phenotype | Animals | beta Catenin - antagonists & inhibitors | Carotid Arteries - pathology | Apoptosis | Cell Movement | β-catenin | smooth muscle cell | Vascular Biology | Vascular Disease | Pharmacology | β-catenin inhibitors | Smooth Muscle Proliferation and Differentiation | Basic Science Research
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 04/2011, Volume 31, Issue 4, pp. 851 - 860
OBJECTIVE—Proliferation of smooth muscle cells (SMC) in response to vascular injury is central to neointimal vascular remodeling. There is accumulating... 
vascular muscle | restenosis | growth factors | proliferation | HDAC inhibitors | vascular biology | gene expression | CANCER CELLS | CHROMATIN | CYCLIN D1 | TRANSCRIPTION | INJURY | ATHEROSCLEROSIS | KAPPA-B | IN-VIVO | GENE-EXPRESSION | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | TRICHOSTATIN | Cyclin D1 - metabolism | Transcription, Genetic - drug effects | Tunica Media - pathology | Phosphorylation | Tunica Media - drug effects | Hyperplasia | Myocytes, Smooth Muscle - pathology | Vascular System Injuries - enzymology | Tunica Media - injuries | Vascular System Injuries - drug therapy | Chromatin Assembly and Disassembly - drug effects | E2F Transcription Factors - metabolism | Quinolines - pharmacology | Vascular System Injuries - pathology | Cyclin-Dependent Kinase Inhibitor p27 - metabolism | RNA Interference | Time Factors | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Epigenesis, Genetic - drug effects | Acetylation | Hydroxylamines - pharmacology | Myocytes, Smooth Muscle - drug effects | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | Histone Deacetylases - genetics | Myocytes, Smooth Muscle - enzymology | Mice, Inbred C57BL | Retinoblastoma Protein - metabolism | Cell Cycle Proteins - metabolism | Cells, Cultured | Rats | Histone Deacetylases - metabolism | Muscle, Smooth, Vascular - injuries | Muscle, Smooth, Vascular - pathology | Animals | Histone Deacetylase Inhibitors - pharmacology | Cell Proliferation - drug effects | Mice | Histones - metabolism | Cell Cycle - drug effects | Muscle, Smooth, Vascular - enzymology | Tunica Media - enzymology | cyclin D1 | cell cycle | Vascular smooth muscle cells | HDAC
Journal Article