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Clinical and Experimental Pharmacology and Physiology, ISSN 1440-1681, 09/2007, Volume 34, Issue 9, pp. 906 - 911
... (NO) synthesis and the development of vascular disease. • Arginase inhibits the production of NO via several potential mechanisms, including competition with NO synthase (NOS... 
smooth muscle cell proliferation | arginine | nitric oxide synthase | diabetes | endothelial dysfunction | hypertension | arginase | Hypertension | Arginine | Endothelial dysfunction | Smooth muscle cell proliferation | Arginase | Nitric oxide synthase | Diabetes | PHYSIOLOGY | MEDIATED DILATION | I EXPRESSION | TRANSLATIONAL CONTROL | AMINO-ACID TRANSPORTER | INTIMAL HYPERPLASIA | SYNTHASE ACTIVITY | MOUSE MODEL | L-ARGININE TRANSPORT | PHARMACOLOGY & PHARMACY | SMOOTH-MUSCLE-CELLS | Cardiovascular Diseases - physiopathology | Cell Proliferation | Reactive Oxygen Species - metabolism | Cardiovascular Diseases - metabolism | Muscle, Smooth, Vascular - metabolism | Humans | Arginase - metabolism | Endothelium, Vascular - physiopathology | Homeostasis | Cardiovascular Diseases - enzymology | Endothelium, Vascular - enzymology | Muscle, Smooth, Vascular - physiopathology | Arginine - analogs & derivatives | Collagen - metabolism | Animals | Endothelium, Vascular - metabolism | Urea - metabolism | Nitric Oxide Synthase Type III - metabolism | Nitric Oxide - metabolism | Muscle, Smooth, Vascular - enzymology | Arginine - metabolism | Muscle, Smooth, Vascular, metabolism | Collagen, metabolism | Endothelium, Vascular, physiopathology | Arginine, analogs and derivatives | Nitric Oxide, metabolism | Endothelium, Vascular, metabolism | Urea, metabolism | Nitric Oxide Synthase Type III, metabolism | Arginine, metabolism | Cardiovascular Diseases, enzymology | Endothelium, Vascular, enzymology | Muscle, Smooth, Vascular, physiopathology | Cardiovascular Diseases, physiopathology | Cardiovascular Diseases, metabolism | Arginase, metabolism | Muscle, Smooth, Vascular, enzymology | Reactive Oxygen Species, metabolism | Deicing chemicals | Physiological aspects | Endothelium-derived relaxing factors | Nitric oxide
Journal Article
Acta Neuropathologica, ISSN 0001-6322, 9/2011, Volume 122, Issue 3, pp. 293 - 311
...ORIGINAL PAPER Vascular b-amyloid and early astrocyte alterations impair cerebrovascular function and cerebral metabolism in transgenic arcAb mice Mario... 
Pathology | Neurosciences | Congophilic amyloid angiopathy | Medicine & Public Health | Alzheimer’s disease | Astrocytes | Cerebral glucose metabolism | Alzheimer's disease | MICROVASCULAR PATHOLOGY | ALZHEIMERS-DISEASE | NEUROVASCULAR MECHANISMS | PATHOLOGY | BLOOD-BRAIN-BARRIER | NEUROSCIENCES | CLINICAL NEUROLOGY | GLUCOSE | MOUSE MODEL | LACTATE | A-BETA | SMOOTH-MUSCLE-CELLS | ANGIOPATHY | Microdialysis - methods | Humans | Astrocytes - pathology | Dystroglycans - metabolism | Glucose Transporter Type 1 - metabolism | Muscle, Smooth - ultrastructure | Glial Fibrillary Acidic Protein - metabolism | Microscopy, Electron, Scanning - methods | Amyloid beta-Peptides - genetics | Amyloid beta-Peptides - metabolism | Cell Culture Techniques | Disease Models, Animal | Endothelium - pathology | Mice, Transgenic | Cerebral Arteries - ultrastructure | Basement Membrane - metabolism | Disease Progression | Symporters - metabolism | Astrocytes - ultrastructure | Blood-Brain Barrier - pathology | Cerebral Arteries - metabolism | Brain - pathology | Glucose - metabolism | Plaque, Amyloid - metabolism | Mice | Astrocytes - metabolism | Blood-Brain Barrier - physiopathology | Basement Membrane - pathology | Cerebral Amyloid Angiopathy - complications | Monocarboxylic Acid Transporters - metabolism | Platelet Endothelial Cell Adhesion Molecule-1 - metabolism | Hemorrhage - etiology | Amyloid beta-Protein Precursor - metabolism | Lactase - metabolism | Cerebral Amyloid Angiopathy - genetics | Astrocytes - drug effects | Plaque, Amyloid - pathology | Gene Expression Regulation - genetics | Hemorrhage - metabolism | Muscle, Smooth - metabolism | Cerebrovascular Disorders - etiology | Cerebral Amyloid Angiopathy - pathology | Animals | Endothelium - metabolism | Glucose Transporter Type 1 - genetics | Symporters - genetics | Cerebral Arteries - pathology | Laminin - metabolism | Monocarboxylic Acid Transporters - genetics | Cerebrovascular Disorders - pathology | Hemorrhage - pathology | Muscle, Smooth - pathology | Glucose transporter | Leakage | Brain | Neurodegenerative diseases | Cognitive ability | Transgenic mice | Blood vessels | Data processing | Smooth muscle | beta -Amyloid | Glucose transport | Metabolism | Amyloid precursor protein | Blood-brain barrier | Cerebral blood flow | Lactic acid | Mutation | Original Paper
Journal Article
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1524-4636, 2013, Volume 33, Issue 1, pp. 67 - 75
.... We investigated here whether Gal-3 is involved in Aldo-induced vascular fibrosis. Methods and Results-In rat vascular smooth muscle cells Gal-3 overexpression enhanced specifically collagen type I synthesis... 
collagen type I | aldosterone | HEART-FAILURE | LEFT-VENTRICULAR DYSFUNCTION | INFLAMMATION | ENDOTHELIAL-CELLS | fibrosis | SMOOTH-MUSCLE-CELLS | EXTRACELLULAR-MATRIX | MICE | galectin-3 | vascular smooth muscle cells | EXPRESSION | INSIGHTS | CARDIOTROPHIN-1 | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | Blood Pressure | Inflammation - chemically induced | Inflammation - pathology | Up-Regulation | Muscle, Smooth, Vascular - metabolism | Rats, Wistar | Humans | Galectin 3 - metabolism | Myocytes, Smooth Muscle - pathology | Male | Aldosterone | Muscle, Smooth, Vascular - physiopathology | Galectin 3 - deficiency | Inflammation - metabolism | Transfection | RNA Interference | Time Factors | Hypertension - chemically induced | Mineralocorticoid Receptor Antagonists - pharmacology | Hypertension - prevention & control | Hypertension - genetics | Myocytes, Smooth Muscle - drug effects | Vascular Stiffness | Myocytes, Smooth Muscle - metabolism | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | Mice, Inbred C57BL | Cells, Cultured | Rats | Galectin 3 - genetics | Hypertension - pathology | Hypertension - physiopathology | Hypertension - metabolism | Mice, Knockout | Collagen Type I - biosynthesis | Muscle, Smooth, Vascular - pathology | Animals | Galectin 3 - antagonists & inhibitors | Fibrosis | Inflammation - genetics | Inflammation - prevention & control | Mice | Inflammation - physiopathology | Cellular Biology | Life Sciences | Muscle, Smooth, Vascular | Mineralocorticoid Receptor Antagonists | Hypertension | Hematology | Biochemistry, Molecular Biology | Collagen Type I | Inflammation | Human health and pathology | Myocytes, Smooth Muscle | Galectin 3
Journal Article
Circulation (New York, N.Y.), ISSN 1524-4539, 2009, Volume 119, Issue 10, pp. 1424 - 1432
Background - Atherosclerosis is an inflammatory disease in which interferon (IFN)-gamma, the signature cytokine of Th1 cells, plays a central role. We... 
Smooth | Inflammation | Muscle | Interleukins | Lymphocytes | Coronary disease | CYTOKINES | CARDIAC & CARDIOVASCULAR SYSTEMS | lymphocytes | muscle, smooth | AUTOIMMUNE INFLAMMATION | HELPER-CELLS | ATHEROSCLEROSIS | IL-23 | interleukins | coronary disease | GROWTH-FACTOR-BETA | inflammation | DISEASE | IL-17 | PERIPHERAL VASCULAR DISEASE | DIFFERENTIATION | HEMATOLOGY | EXPRESSION | Vasculitis - physiopathology | Humans | Middle Aged | Receptors, Interleukin-17 - immunology | Vasculitis - etiology | Interleukin-17 - physiology | Myocytes, Smooth Muscle - pathology | Male | Interferon-gamma - metabolism | Chemokine CXCL10 - biosynthesis | Coronary Artery Disease - pathology | Inflammation Mediators - metabolism | Interferon-gamma - antagonists & inhibitors | Adult | Female | Interleukin-8 - metabolism | Interleukins - pharmacology | Receptors, Interferon - antagonists & inhibitors | Interleukin-6 - metabolism | Myocytes, Smooth Muscle - metabolism | Transforming Growth Factor beta1 - pharmacology | Cells, Cultured - drug effects | Coronary Vessels - drug effects | Interleukin-8 - biosynthesis | CD4-Positive T-Lymphocytes - metabolism | Interferon-gamma - physiology | Receptors, Interleukin-17 - antagonists & inhibitors | Interleukin-17 - biosynthesis | Gene Expression Regulation - drug effects | Interleukin-17 - metabolism | Muscle, Smooth, Vascular - pathology | Receptors, Interferon - immunology | Signal Transduction - drug effects | T-Lymphocyte Subsets - metabolism | Coronary Artery Disease - complications | Interleukin-6 - biosynthesis | Aged | Cells, Cultured - metabolism | Coronary Artery Disease - immunology | Chemokine CXCL10 - metabolism | Interferon-gamma - biosynthesis | muscle | smooth
Journal Article
Circulation Research, ISSN 0009-7330, 02/2010, Volume 106, Issue 2, pp. 363 - 372
Journal Article
Journal Article
Cell stem cell, ISSN 1934-5909, 2016, Volume 19, Issue 5, pp. 628 - 642
Journal Article