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The Journal of physiology, ISSN 0022-3751, 10/2014, Volume 592, Issue 20, pp. 4575 - 4589
... pathways involved in their degradation. Here we show that administration of an antioxidant prevents redox imbalance, but does not prevent activation of catabolic pathways and muscle atrophy... 
Neurosciences | Physiology | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Up-Regulation | Dynamins - metabolism | Superoxide Dismutase - genetics | Oxidative Stress | SKP Cullin F-Box Protein Ligases - genetics | Mitochondria, Muscle - metabolism | Male | Muscle Fibers, Skeletal - drug effects | Muscle Fibers, Skeletal - metabolism | RNA, Messenger - metabolism | Autophagy | Tripartite Motif Proteins | Chromans - pharmacology | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Muscle Proteins - metabolism | Apoptosis Regulatory Proteins - genetics | NF-E2-Related Factor 2 - genetics | Muscular Atrophy - prevention & control | Muscle Fibers, Skeletal - physiology | Superoxide Dismutase - metabolism | Beclin-1 | Muscular Atrophy - metabolism | Catalase - genetics | Mice, Inbred C57BL | RNA, Messenger - genetics | Ubiquitin-Protein Ligases - metabolism | Dynamins - genetics | SKP Cullin F-Box Protein Ligases - metabolism | Antioxidants - pharmacology | Transcription Factors - genetics | Apoptosis Regulatory Proteins - metabolism | Catalase - metabolism | Muscle Proteins - genetics | Transcription Factors - metabolism | Antioxidants - therapeutic use | Animals | Chromans - therapeutic use | Hindlimb Suspension - adverse effects | NF-E2-Related Factor 2 - metabolism | Mice | Mitochondria, Muscle - drug effects | Superoxide Dismutase-1 | Ubiquitin-Protein Ligases - genetics | Index Medicus | Muscle
Journal Article
Journal Article
The FEBS journal, ISSN 1742-464X, 09/2013, Volume 280, Issue 17, pp. 4294 - 4314
Journal Article
The New England journal of medicine, ISSN 1533-4406, 11/2017, Volume 377, Issue 18, pp. 1723 - 1732
In this phase 3 trial, among infants with spinal muscular atrophy, those who received nusinersen were more likely to achieve major motor milestones and less likely to need permanent assisted... 
Medicine, General & Internal | Life Sciences & Biomedicine | General & Internal Medicine | Science & Technology | Injections, Spinal | Spinal Muscular Atrophies of Childhood - genetics | Double-Blind Method | Oligonucleotides - therapeutic use | Humans | Spinal Muscular Atrophies of Childhood - drug therapy | Infant | Male | Oligonucleotides - adverse effects | Oligonucleotides, Antisense - adverse effects | RNA, Messenger - metabolism | Survival of Motor Neuron 2 Protein - metabolism | Respiration, Artificial | Disease-Free Survival | Oligonucleotides, Antisense - therapeutic use | Survival of Motor Neuron 2 Protein - genetics | Age of Onset | Survival Analysis | Female | Spinal Muscular Atrophies of Childhood - mortality | Spinal Muscular Atrophies of Childhood - physiopathology | Motor Skills | RNA, Messenger - drug effects | Care and treatment | Usage | Surgery | Patient outcomes | Motor ability | Analysis | Spinal muscular atrophy | Pediatrics | Neuromuscular diseases | Cell survival | Splicing | Neurons | SMN protein | Antisense oligonucleotides | mRNA | Infants | Motor task performance | Hereditary diseases | Children & youth | Babies | Proteins | Neurology | Ventilation | Death | Mutation | Drug therapy | Drug dosages | Age | Index Medicus | Abridged Index Medicus | trials | Pediatrik
Journal Article
The Journal of clinical investigation, ISSN 0021-9738, 01/2013, Volume 123, Issue 2, pp. 611 - 622
Journal Article