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The Journal of Physiology, ISSN 0022-3751, 10/2014, Volume 592, Issue 20, pp. 4575 - 4589
Key points Oxidative stress is widely considered a major cause of muscle loss not only in disuse but also in most chronic diseases, triggering carbonylation of... 
TIBIALIS ANTERIOR | SKELETAL-MUSCLE | GAMMA COACTIVATOR 1-ALPHA | OXIDATIVE STRESS | PHYSIOLOGY | MITOCHONDRIAL BIOGENESIS | GENE-EXPRESSION | TRANSCRIPTIONAL COACTIVATOR | UBIQUITIN LIGASES | PROTEIN-SYNTHESIS | NEUROSCIENCES | ANTIOXIDANT CAPACITY | Up-Regulation | Dynamins - metabolism | Superoxide Dismutase - genetics | Oxidative Stress | SKP Cullin F-Box Protein Ligases - genetics | Mitochondria, Muscle - metabolism | Male | Muscle Fibers, Skeletal - drug effects | Muscle Fibers, Skeletal - metabolism | RNA, Messenger - metabolism | Autophagy | Tripartite Motif Proteins | Chromans - pharmacology | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Muscle Proteins - metabolism | Apoptosis Regulatory Proteins - genetics | NF-E2-Related Factor 2 - genetics | Muscular Atrophy - prevention & control | Muscle Fibers, Skeletal - physiology | Superoxide Dismutase - metabolism | Beclin-1 | Muscular Atrophy - metabolism | Catalase - genetics | Mice, Inbred C57BL | RNA, Messenger - genetics | Ubiquitin-Protein Ligases - metabolism | Dynamins - genetics | SKP Cullin F-Box Protein Ligases - metabolism | Antioxidants - pharmacology | Transcription Factors - genetics | Apoptosis Regulatory Proteins - metabolism | Catalase - metabolism | Muscle Proteins - genetics | Transcription Factors - metabolism | Antioxidants - therapeutic use | Animals | Chromans - therapeutic use | Hindlimb Suspension - adverse effects | NF-E2-Related Factor 2 - metabolism | Mice | Mitochondria, Muscle - drug effects | Superoxide Dismutase-1 | Ubiquitin-Protein Ligases - genetics | Muscle
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 02/2013, Volume 123, Issue 2, pp. 611 - 622
Journal Article
Nature Cell Biology, ISSN 1465-7392, 08/2018, Volume 20, Issue 8, pp. 917 - 927
Fibro-adipogenic progenitors (FAPs) are typically activated in response to muscle injury, and establish functional interactions with inflammatory and muscle... 
SKELETAL-MUSCLE | SATELLITE CELL NICHE | DYSTROPHIC MUSCLES | REGENERATION | ANTI-INTERLEUKIN-6 MONOCLONAL-ANTIBODY | INJURY | MICE | INTERLEUKIN-6 | CANCER | FIBRO/ADIPOGENIC PROGENITORS | CELL BIOLOGY | Interleukin-6 - antagonists & inhibitors | Adipogenesis - drug effects | Coculture Techniques | Humans | Amyotrophic Lateral Sclerosis - prevention & control | Male | Spinal Cord Injuries - genetics | Cardiotoxins | Muscular Atrophy, Spinal - prevention & control | Spinal Cord Injuries - pathology | Muscular Atrophy, Spinal - genetics | Myoblasts, Skeletal - pathology | Neuromuscular Agents - pharmacology | Quadriceps Muscle - drug effects | Muscular Atrophy - prevention & control | Quadriceps Muscle - innervation | Quadriceps Muscle - pathology | Interleukin-6 - metabolism | STAT3 Transcription Factor - genetics | STAT3 Transcription Factor - metabolism | Disease Models, Animal | Myoblasts, Skeletal - metabolism | Cell Line | Muscular Atrophy, Spinal - metabolism | Denervation - methods | Interleukin-6 - genetics | Muscular Atrophy - metabolism | Signal Transduction | Spinal Cord Injuries - metabolism | Amyotrophic Lateral Sclerosis - genetics | Mice, Inbred C57BL | Muscular Atrophy - pathology | Mice, Transgenic | Muscular Atrophy - genetics | Muscular Atrophy, Spinal - pathology | Spinal Cord Injuries - prevention & control | Amyotrophic Lateral Sclerosis - pathology | Animals | Sciatic Nerve - surgery | Amyotrophic Lateral Sclerosis - metabolism | Fibrosis | Superoxide Dismutase-1 - genetics | Mutation | Myoblasts, Skeletal - drug effects | Quadriceps Muscle - metabolism | STAT3 Transcription Factor - antagonists & inhibitors | Animal models | Spinal cord | Mental disorders | Pathogenesis | Stem cell transplantation | Activation | Spinal cord injury | Macrophages | Inactivation | Spinal muscular atrophy | Interleukin 6 | Atrophy | Cell activation | Rodents | Neuromuscular diseases | Deactivation | Stat3 protein | Muscles | Amyotrophic lateral sclerosis | Inflammation | Regeneration | Signaling | Stem cells | Neuromuscular junctions | Infiltration | Denervation
Journal Article
Circulation, ISSN 0009-7322, 01/2010, Volume 121, Issue 3, pp. 419 - 425
Journal Article
The Journal of Physiology, ISSN 0022-3751, 11/2007, Volume 585, Issue 1, pp. 203 - 215
Oxidative stress promotes controlled mechanical ventilation (MV)-induced diaphragmatic atrophy. Nonetheless, the signalling pathways responsible for oxidative... 
CHAIN MESSENGER-RNA | DISUSE ATROPHY | SKELETAL-MUSCLE | OXIDATIVE STRESS | VITAMIN-E | IN-VITRO | PHYSIOLOGY | SOLEUS MUSCLE | FOXO TRANSCRIPTION FACTORS | UBIQUITIN LIGASES | EXPRESSION | NEUROSCIENCES | Diaphragm - drug effects | Insulin - physiology | SKP Cullin F-Box Protein Ligases - genetics | Oxidative Stress - physiology | Insulin-Like Growth Factor I - genetics | Proto-Oncogene Proteins c-akt - genetics | Tripartite Motif Proteins | Chromans - pharmacology | Respiration, Artificial - adverse effects | Forkhead Transcription Factors - metabolism | Muscle Proteins - metabolism | Female | Muscular Atrophy - prevention & control | Diaphragm - metabolism | Muscular Atrophy - etiology | Diaphragm - physiopathology | Ubiquitin-Protein Ligases - metabolism | Rats | Proto-Oncogene Proteins c-akt - physiology | SKP Cullin F-Box Protein Ligases - metabolism | Antioxidants - pharmacology | Forkhead Transcription Factors - genetics | Nerve Tissue Proteins - genetics | Rats, Sprague-Dawley | Muscle Proteins - genetics | Nerve Tissue Proteins - metabolism | Phosphatidylinositol 3-Kinases - genetics | Antioxidants - therapeutic use | Animals | Chromans - therapeutic use | Insulin-Like Growth Factor I - physiology | Phosphatidylinositol 3-Kinases - physiology | Signal Transduction - physiology | Oxidative Stress - drug effects | Forkhead Box Protein O3 | Ubiquitin-Protein Ligases - genetics | Antioxidants | Physiological aspects | Protein kinases | Respiratory
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2012, Volume 7, Issue 9, p. e45048
Journal Article